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1 -beam CT (EBCT), and size of Achilles tendon xanthomas.
2 apoE knockout mice developed small choroidal xanthomas.
3 es, leading to the clinical finding eruptive xanthomas.
4 ight be responsible for creation of eruptive xanthomas.
5 e cutaneous paraneoplastic syndromes such as xanthomas, acanthosis nigricans, carcinoid syndrome, unu
6 s and frequently develop tendon and tuberous xanthomas, accelerated atherosclerosis, and premature co
7 n the plasma and develop tendon and tuberous xanthomas, accelerated atherosclerosis, and premature co
8  the plasma, and develop tendon and tuberous xanthomas, accelerated atherosclerosis, and premature co
9 terol levels and develop tendon and tuberous xanthomas, accelerated atherosclerosis, and premature co
10 glycerides and LDL cholesterol, and can have xanthomas and premature CHD.
11 nome of an 11-month-old breast-fed girl with xanthomas and very high plasma cholesterol levels (1023
12  cholesterol, reduced LDL receptor function, xanthomas, and cardiovascular disease before age 20 year
13 abdominal pain, acute pancreatitis, eruptive xanthomas, and lipemia retinalis.
14                                              Xanthomas appeared in all mice fed the high-cholesterol
15                        In Cyp27a1(-/-) mice, xanthomas are not formed, despite a significant cholesta
16 ate the fat-water content of Achilles tendon xanthomas at baseline and after treatment and to compare
17  causes intractable pruritus and disfiguring xanthomas because of retained bile acids and cholesterol
18 ion of VX lesions that demonstrated positive xanthoma cell reactivity with antibodies RM3/1, 25F9, an
19              VX lesions are characterized by xanthoma cells (lipid-laden macrophages or foam cells) f
20 l effects of probucol on atherosclerosis and xanthomas despite its HDL-lowering effects and suggest t
21                                More advanced xanthomas disrupted adjoining neural tissue in the forni
22 venile xanthogranuloma, histiocytic sarcoma, xanthoma disseminatum, interdigitating dendritic cell sa
23   The data indicate that L-arginine prevents xanthoma formation and reduces atherosclerosis in LDL re
24 < 0.01), whereas development of gross dorsal xanthoma (&gt;5 mm diameter) was effectively reduced to zer
25 ht Achilles tendons with clinically apparent xanthomas in 24 patients with familial hypercholesterole
26 EBD) is effective for relief of pruritus and xanthomas in AGS patients who fail conventional medical
27 reduces atherosclerosis in animal models and xanthomas in humans.
28 n macrophages was sufficient to cause dermal xanthomas in hyperlipidemic LDLR-deficient mice.
29 acerbates development of atherosclerosis and xanthomas in the 2/2 mice.
30 ith sitosterolemia presenting with prominent xanthomas in the subcutaneous tissue of both elbows who
31 n Most of the enlargement of Achilles tendon xanthomas is due to an increase in water content rather
32 hologic intimal thickening (PIT), or intimal xanthoma (IXA).
33                More remarkably, the tuberous xanthomas on her elbows had completely regressed.
34       Clinical manifestations include tendon xanthomas, premature coronary artery disease, hemolytic
35                Three patients with extensive xanthomas prior to PEBD had complete resolution within 1
36 velop spontaneous atherosclerotic plaque and xanthoma resulted in expression and secretion of apoE.
37 d MR imaging-based parameters were higher in xanthoma tendons compared with those in control tendons
38 nit volume was 71% higher (42.0% +/- 6.7) in xanthoma tendons than in control tendons (24.5% 6 5.8; P
39                                  Verruciform xanthoma (VX) is a benign lesion that primarily affects
40                                      Smaller xanthomas were confined mainly to the choroid plexus and
41                                           No xanthomas were observed in aged wild-type controls fed t