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1  with respect to the presence/absence of the xenobiotic.
2 nce caused by temperature, pH, oxidants, and xenobiotics.
3 mation pathway to assess overall exposure to xenobiotics.
4 uential, multi-organ metabolism of drugs and xenobiotics.
5 he N-methylation of endogenous and exogenous xenobiotics.
6 ffspring by enhancing metabolic tolerance to xenobiotics.
7 on of reduced glutathione (GSH) to endo- and xenobiotics.
8 ilization of nutrients and the processing of xenobiotics.
9 nsformations that may activate or deactivate xenobiotics.
10 CCSs for a set of endogenous metabolites and xenobiotics.
11 tructurally diverse endogenous compounds and xenobiotics.
12  displayed increased susceptibility to these xenobiotics.
13 is involved in the metabolism of over 50% of xenobiotics.
14 olism, especially in metabolism of drugs and xenobiotics.
15  to peptides, proteins, polysaccharides, and xenobiotics.
16 t UGT8 is involved in metabolism of endo- or xenobiotics.
17 conjugation of both endogenous compounds and xenobiotics.
18 , catabolism, gut microbiome activities, and xenobiotics.
19 conditions on the transformation pathways of xenobiotics.
20 t role in the metabolisms of drugs and other xenobiotics.
21 d initiating transport of multiple drugs and xenobiotics.
22 ardiovascular disease risk and metabolism of xenobiotics.
23 s a crucial role in the cellular response to xenobiotics.
24 abolism of drugs, steroids, fatty acids, and xenobiotics.
25 nism of activation of human CAR by drugs and xenobiotics.
26  in vitro and in vivo metabolism pathways of xenobiotics.
27 spective transporter mutants to the specific xenobiotics.
28 expressed by immune cells and binds numerous xenobiotics.
29 ne responses and the biological responses to xenobiotics.
30 en metabolize many pharmaceuticals and other xenobiotics, a feature that is valuable in a biotechnolo
31 the efflux of structurally diverse drugs and xenobiotics across cell membranes, affecting drug pharma
32 in the extrusion of endogenous compounds and xenobiotics across the plasma membrane.
33 ntagonist and promising therapeutic tool.The xenobiotic-activated human pregnane X receptor (hPXR) re
34                                              Xenobiotic activation of the aryl hydrocarbon receptor (
35                      These data suggest that xenobiotic adaptation in this specialist herbivore is th
36 nd lipocalins, also reflect the evolution of xenobiotic adaptation.
37 bsequently developed specific and remarkable xenobiotic adaptations.
38 venger by binding and/or hydrolyzing various xenobiotic agents and toxicants, many of which target th
39 f P450 expression in response to exposure to xenobiotic agonists of the nuclear receptors constitutiv
40 ontrol metabolism of glucose, nutrients, and xenobiotics; alterations in its activity may contribute
41 he pregnane X receptor (PXR), a modulator of xenobiotic and detoxification responses, which can sense
42 rostane receptor (CAR) is a key regulator of xenobiotic and endobiotic metabolism.
43  across tissue sections to build an image of xenobiotic and endogenous compound distribution to asses
44  androstane receptor (CAR) regulates hepatic xenobiotic and energy metabolism, as well as promotes ce
45  toxicity pathways being affected, including xenobiotic and lipid metabolism and the thyroid hormone
46 he role of the microbiome metabolism of this xenobiotic and the impact on host responses is unclear.
47                Associations between selected xenobiotics and antinuclear antibodies in the National H
48          Genes involved in the metabolism of xenobiotics and antioxidant mechanisms were primarily do
49 ns play important roles in detoxification of xenobiotics and during physiological and developmental p
50 nhance the detoxification and elimination of xenobiotics and endobiotics by modulating the expression
51 ular export of a chemically diverse array of xenobiotics and endogenous compounds.
52 orters to essentially detoxify and eliminate xenobiotics and endotoxins from the body.
53 naling (IIS) pathway increases resistance to xenobiotics and extends lifespan in Drosophila, and can
54 akes key contributions to the elimination of xenobiotics and greatly mediates the toxicity.
55 ntribution of food to the bioaccumulation of xenobiotics and hence toxicity is still an ambiguous iss
56 which has a central role in the clearance of xenobiotics and in cancer resistance to chemotherapy.
57  regulates inducible metabolism of drugs and xenobiotics and is activated or inhibited by various PCB
58 al mechanisms involved in insect immunity to xenobiotics and pathogens as well as the mechanisms by w
59 interfere with metabolism of drugs and other xenobiotics and provides a molecular mechanism for this
60 t functions as a sensor to a wide variety of xenobiotics and regulates expression of several drug met
61 or that mediates the biochemical response to xenobiotics and the toxic effects of a number of environ
62     Concurrent exposure to a wide variety of xenobiotics and their combined toxic effects can play a
63 ion (MATE) transporters mediate excretion of xenobiotics and toxic metabolites, thereby conferring mu
64 are critically involved in the metabolism of xenobiotics and toxins.
65 ield of bioorthogonal catalysis by producing xenobiotics and uncaging biomolecules in living systems.
66 cid-related, 4 non-esterified fatty acids, 6 xenobiotics, and 5 unknown compounds).
67 and endogenous stresses, including oxidants, xenobiotics, and excessive nutrient/metabolite supply.
68 ital enzyme for the metabolic elimination of xenobiotics, and it is prone to induction by xenobiotics
69 acterized by resistance to several unrelated xenobiotics, and poses significant challenges to managin
70 e variant in CYP1B1, encoding the homonymous xenobiotic- and hormone-processing enzyme.
71 otic metabolism, together with resistance to xenobiotics, are frequent correlates of lifespan extensi
72 vation, and sporadic differentiation through xenobiotic aryl hydrocarbon receptor signaling.
73 et cell differentiation, indole acts via the xenobiotic aryl hydrocarbon receptor to increase express
74 is gives a coherent picture of pathogens and xenobiotics as important drivers of adaptive evolution o
75 e aryl hydrocarbon receptor (AHR) recognizes xenobiotics as well as natural compounds such as tryptop
76 obtained with the Activated Sludge Model for Xenobiotics (ASM-X)) with representative measured data f
77  (WATS) model and Activated Sludge Model for Xenobiotics (ASM-X).
78 igh weight rabbits, and pathways related to "xenobiotics biodegradation" and "various types of N-glyc
79 we determined the thermal sensitivity of the xenobiotic biotransformation enzymes.
80 wn detoxification process to eliminate small xenobiotics, but its impacts on nanoparticle retention,
81 n the overrepresented pathways Metabolism of Xenobiotics by Cytochrome P450 and Butanoate and Tryptop
82 ins, as well as in the metabolism of various xenobiotics by cytochrome P450.
83  is the sensitive detection of intracellular xenobiotics by transcriptional activators, activating ex
84 rganism with innate resistance toward select xenobiotics by triggering their efflux.
85 ults show that biotransformation of phenolic xenobiotics can be significantly altered by coexposure,
86                              Many classes of xenobiotics can significantly suppress or enhance immune
87  the reduction and oxidation of biogenic and xenobiotic carbonyls.
88  a ubiquitous DNA lesion, formed not only by xenobiotic carcinogens but also by the endogenous methyl
89                         Despite the strongly xenobiotic character of some of these substituents, biod
90 thogen P. aeruginosa and to treatment with a xenobiotic chelator, phenanthroline, in C. elegans.
91 oic acid and a variety of endocrine factors, xenobiotic chemicals, or metabolites that can directly b
92 f by-products from each production and avoid xenobiotic chemicals, we have achieved beneficial improv
93 d Escherichia coli to assimilate melamine, a xenobiotic compound containing nitrogen.
94 ribute to defense against microorganisms and xenobiotic compounds acquired while foraging.
95 o known as P-glycoprotein, actively extrudes xenobiotic compounds across the plasma membrane of diver
96 e transporters are involved in the efflux of xenobiotic compounds and are responsible for decreasing
97 lay a key role in both the detoxification of xenobiotic compounds and the metabolism of drugs and pro
98  Mammalian systems inactivate endobiotic and xenobiotic compounds by linking them to a glucuronic aci
99 holesterol homeostasis, and the breakdown of xenobiotic compounds, including many current drugs.
100 ta have been implicated in the metabolism of xenobiotic compounds, motivating studies of microbe-driv
101 ptor (NR), involved in the detoxification of xenobiotic compounds.
102                     Benzotriazoles (BTs) are xenobiotic contaminants widely distributed in aquatic en
103 e major mitochondrial autoantigen of PBC and xenobiotic cross reactive chemicals.
104          TLR2 acts as a central regulator of xenobiotic defense via the multidrug transporter ABCB1/M
105 xidative transformations in drug metabolism, xenobiotic degradation, and natural product biosynthesis
106 fs based on their variance to assess whether xenobiotic-derived substructures are among the most-vari
107 22 out of the 30 most-variant Mass2Motifs to xenobiotic-derived substructures including paracetamol/a
108                                              Xenobiotic detoxification and immune genes are similarly
109 ntestine, which results in activation of the xenobiotic detoxification enzyme flavin-containing monoo
110 ese findings underline the importance of the xenobiotic detoxification in the mitohormetic, longevity
111 tegorized in the Gene Ontology (GO) term of "Xenobiotic Detoxification Program" (XDP).
112 w that KLF-1 activates genes involved in the xenobiotic detoxification programme and identified cytoc
113 liver plays a pivotal role in metabolism and xenobiotic detoxification, processes that must be partic
114 of transcripts encoding proteins involved in xenobiotic detoxification.
115 nes involved in metabolism, proteolysis, and xenobiotic detoxification.
116 ly increased resistance of Drosophila to the xenobiotic dichlordiphenyltrichlorethan (DDT), by artifi
117 d-activated transcription factor that senses xenobiotics, diet, and gut microbial-derived metabolites
118                                Exposure to a xenobiotic during early development induced persistent f
119 ing cellular resistance to oxidative stress, xenobiotic efflux, proliferation, and metabolic reprogra
120 est that larvae may be less able to detoxify xenobiotics encountered in diet than adults, and these f
121 he cell to carry out conjugation without the xenobiotic entering into the interior of the cell.
122 ential role in for example bile formation or xenobiotic export.
123 ome that should be considered when assessing xenobiotic exposure and as potential countermeasures to
124                  To test the hypothesis that xenobiotic exposure at environmentally relevant concentr
125  effects of nanomaterials, and the impact of xenobiotic exposure on the developing immune system.
126 sions of both HO-1 and P450s are affected by xenobiotic exposure, changes in HO-1 expression can pote
127 cotinic receptor antagonist, suggesting that xenobiotic exposure, rather than nicotinic receptor sign
128 lso show that Pdr1 is rapidly degraded after xenobiotic exposure, which leads to a desensitization of
129  data suggest that background levels of most xenobiotic exposures typical in the U.S. population are
130  detecting biological responses to low-level xenobiotic exposures, these data can aid the classificat
131 hways regulating cell metabolism (lipids and xenobiotics), extracellular matrix, and chemokine signal
132 hesis as well as clearing waste products and xenobiotics from the fetal circulation.
133   Although the detoxification mechanisms for xenobiotics have been extensively studied in mammalian c
134 l, we quantified the amount of a fluorinated xenobiotic (heptafluorobutyric acid, HFBA) in three diff
135  the pharmacokinetics (PK) and toxicities of xenobiotics in a patient-specific manner so that persona
136 the relative contribution of food-associated xenobiotics in bioaccumulation depends on species, subst
137 m-specific contribution to the metabolism of xenobiotics in complex biological samples has not been p
138 y and contamination of various compounds and xenobiotics in environmental biology and microbial ecolo
139  genotype variation might affect the fate of xenobiotics in food-producing species such as cattle.
140 protein plays a central role in clearance of xenobiotics in humans and is implicated in cancer resist
141 uced a broad protective response to multiple xenobiotics in male offspring.
142 of all clinically prescribed drugs and other xenobiotics in the human body.
143 ing contamination of reclaimed wastewater by xenobiotics including pharmaceuticals.
144 active steroids and fatty acids), as well as xenobiotics including therapeutic/chemotherapeutic drugs
145  into PXR-mediated cellular responses toward xenobiotics including therapeutics.
146 s that are responsible for the metabolism of xenobiotics, including drugs such as irinotecan and osel
147 xenobiotics, and it is prone to induction by xenobiotics, including phenobarbital via constitutive an
148  endobiotic estradiol-17-glucuronide and the xenobiotic indomethacin-acyl-glucuronide are found to ex
149 itivity C-reactive protein (CRP) paired with xenobiotic-induced immune signalling.
150 was the most appropriate model for detecting xenobiotic-induced metabolomic perturbations.
151 major role for SXR/PXR in protection against xenobiotic-induced oxidative stress by maintaining prope
152  of particular interest in the prevention of xenobiotic-induced toxicities.
153  factor that has been intensively studied in xenobiotic-induced toxicity.
154 ricting the entry of endogenous compounds or xenobiotics into tissues and it plays important roles in
155 r, SKN-1, regulates cellular defenses during xenobiotic intoxication and bacterial infection.
156 aminar and radial concentration gradients of xenobiotics introduced in multilayer gel arrangements an
157  The fundamental role of AOX in metabolizing xenobiotics is also due to the attempt of medicinal chem
158                        Thyroid disruption by xenobiotics is associated with a broad spectrum of sever
159 eased knowledge of Ahr-mediated responses to xenobiotics is imperative.
160 on of the aryl hydrocarbon receptor (AHR) by xenobiotics is known to affect epidermal differentiation
161 anisms involved in in situ biodegradation of xenobiotics, like pesticides, in natural and engineered
162 ing amino acids, nucleotides, carbohydrates, xenobiotics, lipids, and other classes.
163 BCRP in limiting fetal exposure to drugs and xenobiotics, long-term use of these medications may affe
164                         Many drugs and other xenobiotics may reach systemic concentrations where they
165 cant changes in expression of genes encoding xenobiotic metabolism (cyp4) and moulting (cut).
166 n sites were involved in pathways including, xenobiotic metabolism (e.g., Cyp1a4), lipid/bile acid ho
167 eceptor-controlled cholesterol/bile acid and xenobiotic metabolism among the top deregulated pathways
168 eeks post-smoking-cessation were involved in xenobiotic metabolism and anti-apoptotic functions respe
169 gut microbes, genes, and enzymes involved in xenobiotic metabolism are poorly understood.
170        Differences in amino acid, lipid, and xenobiotic metabolism distinguished ASD and typically de
171 rs, the function of PXR in the regulation of xenobiotic metabolism has been extensively studied, and
172 t in recent years, since its contribution in xenobiotic metabolism has not always been identified bef
173 4 is a major contributor to hepatic drug and xenobiotic metabolism in human adults.
174  the distinct evolution of genes involved in xenobiotic metabolism in mites.
175 l be important to determine whether enhanced xenobiotic metabolism is also a correlated, rather than
176                                              Xenobiotic metabolism is complex, and accounting for bio
177               The chemistry of gut microbial xenobiotic metabolism is often distinct from that of hos
178                          We investigated the xenobiotic metabolism of the red-blooded Gobionotothen g
179 ng on the association of starch, sucrose and xenobiotic metabolism pathway with longevity is consiste
180 analyses found a significant enrichment for "xenobiotic metabolism signaling" and "PXR/RXR activation
181 , a molecular understanding of gut microbial xenobiotic metabolism will guide personalized medicine a
182 (body weight, protein, chitobiase, catalase, xenobiotic metabolism, and acetylcholinesterase) were me
183 etabolism, peroxisome, bile acid metabolism, xenobiotic metabolism, and adipogenesis.
184 tant toxicity pathways, such as induction of xenobiotic metabolism, and some integrative indicators d
185 ne receptor (CAR) plays an important role in xenobiotic metabolism, energy homeostasis, and cell prol
186  and gene coexpression networks enriched for xenobiotic metabolism, hormone metabolism, and immune re
187 tration, and significant gene alterations in xenobiotic metabolism, including a decrease in ABCB1/mul
188 ession of mRNA genes associated with hepatic xenobiotic metabolism, oxidative DNA damage, or alterati
189 ith daily alterations in lipid, glucose, and xenobiotic metabolism, protein turnover, and redox balan
190 validation of previously reported effects in xenobiotic metabolism, the innate immune system, and glu
191    Increased expression of genes involved in xenobiotic metabolism, together with resistance to xenob
192 erimental animal models poorly predict human xenobiotic metabolism.
193 tor of the cellular antioxidant response and xenobiotic metabolism.
194 tor (PXR) plays a central role in regulating xenobiotic metabolism.
195 tion enzymes, including some associated with xenobiotic metabolism.
196 as an important cellular role in addition to xenobiotic metabolism.
197 bpopulations in the gut responsible for this xenobiotic metabolism.
198 indicates four pathways (starch, sucrose and xenobiotic metabolism; immune response and inflammation;
199 zymes, suggesting broad influence of ESR1 on xenobiotics metabolism in human liver.
200 ent were enriched with genes associated with xenobiotics metabolism.
201 e sequestration of TCBZ and additional toxic xenobiotic metabolites.
202 pport the hypothesis that NAT1 is not just a xenobiotic metabolizing enzyme and may have a role in en
203 sues, is classically described as a phase-II xenobiotic metabolizing enzyme but can also catalyze the
204 , the AhR has been studied as a regulator of xenobiotic metabolizing enzyme genes, notably cytochrome
205                                     Drug and xenobiotic metabolizing enzymes (DXME) play important ro
206 nooxygenases (FMOs) are primarily studied as xenobiotic metabolizing enzymes with a prominent role in
207 m stress, DNA damage responses, induction of xenobiotic metabolizing enzymes, and changes in sphingol
208 m and defending themselves via an arsenal of xenobiotic metabolizing enzymes.
209 ed A287P or WT POR, as were those of several xenobiotic-metabolizing cytochromes P450, indicating tha
210 e N-acetyltransferase 1 (NAT1) is a phase II xenobiotic-metabolizing enzyme that also has a role in c
211  Human cytochrome P450 3A4 (CYP3A4) is a key xenobiotic-metabolizing enzyme that oxidizes and clears
212 and vitamin D receptor, which regulate major xenobiotic-metabolizing enzymes and efflux transporters.
213 n-regulation of genes encoding digestive and xenobiotic-metabolizing enzymes, and transcription facto
214  into how peptide transporters interact with xenobiotic molecules and provide a template for further
215              The BBB protects the brain from xenobiotic neurotoxicants and harmful endogenous metabol
216                                              Xenobiotic nucleic acids (XNA) are nucleic acid analogue
217 that uniquely adds to the emerging family of xenobiotic nucleic acids (XNAs).
218                                Resistance to xenobiotic nucleosides used to treat acute myeloid leuke
219  clearing potentially harmful endobiotic and xenobiotic OAs from the eye.
220 t" study demonstrates that human exposure to xenobiotics occurs through ingestion of reclaimed wastew
221 g., peak height) to decide whether a certain xenobiotic of interest (XOI) is present/absent, yielding
222 Supplying essential growth nutrients through xenobiotic or ecologically rare chemicals provides micro
223 ime extremely potent activators of the other xenobiotic or hormonal receptors, namely PXR, AHR, and v
224 ed sugar water feeders amended with either a xenobiotic or solvent only (control).
225 es boosting overall metabolic elimination of xenobiotic or toxic compounds.
226 ath caused by serum deprivation, toxicity of xenobiotics or high concentrations of extracellular aden
227 rance, and transformational toxicity of most xenobiotics owing to its abundance in cytochrome P450 (C
228 s are encompassed in the broad definition of xenobiotic particles.
229             The lysolipid and food and plant xenobiotic pathways were most strongly associated with d
230 mised by diverse genetic, environmental, and xenobiotic perturbations that generate orphan r-proteins
231                Targeted analysis revealed 13 xenobiotics, phytoestrogens, and endogenous hormones in
232 ct the conceptus from exposure to toxins and xenobiotics present in the maternal circulation.
233 aine, accompanied by hepatic upregulation of xenobiotic processing genes, and enhanced drug clearance
234                              The steroid and xenobiotic receptor [SXR; also known as the pregnane X r
235 vation of either beta-catenin or the nuclear xenobiotic receptor CAR.
236                      CINPA1 1) is a specific xenobiotic receptor inhibitor and has no cytotoxic effec
237 s constituting the first class of potent pan-xenobiotic receptor ligands that can serve as potential
238 a nuclear receptor considered to be a master xenobiotic receptor that coordinately regulates the expr
239               Pregnane X receptor (PXR) is a xenobiotic receptor that regulates the detoxification an
240  receptor, was originally characterized as a xenobiotic receptor that senses xenotoxicants.
241 arbon (AHR) receptors, it is referred to as "xenobiotic receptor".
242 ounterproductive in tissues where both these xenobiotic receptors are present and active.
243 ved xenobiotics, several carnivores lost the xenobiotic receptors NR1I3 and NR1I2 Finally, the carniv
244                                          The xenobiotic receptors pregnane X receptor (PXR) and const
245 ry rate </=0.10, only 4-ethylphenylsufate, a xenobiotic related to benzoate metabolism, was significa
246 ity during adaptive evolution in response to xenobiotics released into the environment.
247 any up-regulation of genes involved in multi-xenobiotic resistance (MXR) or detoxification (phases I
248   Thus, in Drosophila IIS mutants, increased xenobiotic resistance and enhanced longevity are not cau
249                                              Xenobiotic resistance is commonly found in species with
250 6, as an essential mediator of the increased xenobiotic resistance of IIS mutant flies.
251 d long-lived in the absence of DHR96 and the xenobiotic resistance that it conferred.
252 iction increased lifespan without increasing xenobiotic resistance, confirming that the two traits ca
253 recognition sequence called the nonconsensus xenobiotic response element (NC-XRE).
254   AhR activity depends on its binding to the xenobiotic response element (XRE) in partnership with th
255         Exposure to 2,5-DCB induced multiple xenobiotic response genes, such as cytochrome P-450 and
256 nd stimulated expression of predicted target xenobiotic response-related genes AHRR (P = 1.13 x 10-62
257 sm of toxicity associated with a plethora of xenobiotics, responsible for preventing the effective tr
258                        How honey bees assess xenobiotic risk to nestmates as they forage is poorly un
259 ath of an individual, targeted or untargeted xenobiotic screening of post-mortem samples is normally
260 XR plays many other roles in addition to its xenobiotic-sensing function.
261    The pregnane X receptor (PXR, NR1I2) is a xenobiotic-sensing nuclear receptor that modulates the m
262        Pregnane X receptor (PXR) is a master xenobiotic-sensing transcription factor and a validated
263  constitutive androstane receptor (CAR) is a xenobiotic sensor expressed in hepatocytes that activate
264 xtensively studied, and the role of PXR as a xenobiotic sensor has been well established.
265 r (PXR, NR1I2), initially characterized as a xenobiotic sensor, has been functionally linked to the r
266 ng pathways beyond its established role as a xenobiotic sensor.
267 ptor (CAR) and pregnane X receptor (PXR) are xenobiotic sensors that enhance the detoxification and e
268 the reduced need to metabolize plant-derived xenobiotics, several carnivores lost the xenobiotic rece
269 rbon receptor (AHR), a conserved detector of xenobiotic small molecules.
270 the mammalian metabolism of amine-containing xenobiotics, some N-acylation reactions were not previou
271 transport and sterol metabolism for BMI, and xenobiotic stimuli response for smoking, showed >1.5 tim
272 A receptor DOP-1 activates the expression of xenobiotic stress response genes involved in pathogenic
273 ignal to frontline epithelia to activate the xenobiotic stress response so as to maintain proteostasi
274 h Mates interact with both physiological and xenobiotic substances but also substantially differ with
275  of a healthy, smoking male subject, several xenobiotic substances such as benzene, toluene, styrene,
276 he toxicokinetics and tissue distribution of xenobiotic substances, such as perfluorooctanoic acid (P
277  body, protecting against the penetration of xenobiotic substances.
278  with C. albicans nomenclature) could efflux xenobiotics such as berberine, rhodamine 123, and paraqu
279                                              Xenobiotics such as the neonicotinoid pesticide, imidacl
280  hypothesized that glucuronide conjugates of xenobiotics, such as the anticancer drug sorafenib, can
281 e I IFN and TLR trafficking and signaling in xenobiotic systemic mercury-induced autoimmunity (HgIA).
282 llen, or propolis, as well as five synthetic xenobiotics that frequently contaminate hives-two herbic
283    Endocrine disrupting chemicals (EDCs) are xenobiotics that mimic the interaction of natural hormon
284 LI) is an adverse reaction to drugs or other xenobiotics that occurs either as a predictable event wh
285 etoxify numerous and potentially deleterious xenobiotics; this activity extends to many drugs, giving
286 mplex mixture of organic contaminants (i.e., xenobiotics) through diet, environment, and behavior.
287 rders of magnitude for analytes ranging from xenobiotics to endogenous lipids, and facilitated the re
288 ology by taking into account the spectrum of xenobiotics to which bees are exposed.
289 Embryonic development is highly sensitive to xenobiotic toxicity and in utero exposure to environment
290 l hydrocarbon receptor (AhR), a regulator of xenobiotic toxicity, is a member of the eukaryotic Per-A
291 , a transcription factor known for mediating xenobiotic toxicity, is expressed in B cells, which are
292 stem, enabling quantitative determination of xenobiotic-transcription factor (TF) interactions in rea
293 ons to the metabolism of ingested compounds (xenobiotics), transforming hundreds of dietary component
294 eractions resulting from drug, metabolite or xenobiotic transport between organs are key components o
295 4(+) T effector (Teff) cells upregulated the xenobiotic transporter, Mdr1, in the ileum to maintain h
296 acycline through increased expression of the xenobiotic transporters mexEF-oprN and MexXY.
297 single exposure/coexposure to other phenolic xenobiotics (triclosan, tetrabromobisphenol A, and bisph
298 s confer cellular protection against diverse xenobiotics via not only binding, but also catalysis.
299 xposure, location of exposure, and causative xenobiotic were evaluated.
300 ut is a first point of contact with ingested xenobiotics, where chemicals are metabolized directly by

 
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