1 that is consistent with their higher risk of xerophthalmia and mortality.

2 cently because of the adverse events such as xerophthalmia and trigeminal neuralgia.

3  Sjogren's syndrome (SS) is characterized by xerophthalmia and xerostomia resulting from loss of secr

4 glands leading to xerostomia (dry mouth) and xerophthalmia (dry eyes).

5  lacrimal dysfunction (Schirmer's tear test, xerophthalmia), Lee cGVHD Symptom Scores, and NIH organ

6  dysfunction (P = 0.010) and xerostomia with xerophthalmia (r = 0.32, P = 0.001); and limited mouth-o

7 7.4% (P < 0.04) of children with and without xerophthalmia, respectively.

8 nked to age-related macular degeneration and xerophthalmia, respectively.

9  and 13.7% (NS) of children with and without xerophthalmia, respectively.

10 ndemic, dietary counseling for children with xerophthalmia should be extended to their younger siblin

11 e anomalies in the JHS/EDS-HT group included xerophthalmia, steeper corneas, pathologic myopia, and v

12            Self-reported facial swelling and xerophthalmia that was not explained by the effects of m

13 y contracted to its importance in preventing xerophthalmia, until this ophthalmologist stumbled, quit

14                             The frequency of xerophthalmia was not significantly different between th

15 of age, with (n = 118) and without (n = 118) xerophthalmia were assigned to receive oral vitamin A (6

16 lds with a 1-6-y-old child with a history of xerophthalmia were compared with dietary patterns of 81

17                       Historically, signs of xerophthalmia were used to determine vitamin A deficienc