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1 nt cells as well as in WT cells treated with xyloside.
2 or by treatment of WT cells with heparin or xyloside.
3 th 20 or 50 mM sodium chlorate, or 1 mM beta-xyloside.
4 case of the beta-mannoside than of the beta-xyloside.
5 epitopes at each antenna and containing core xyloside.
8 erein we report the discovery of the novel O-xyloside 7c that inhibits SGLT2 in vitro and urinary glu
9 on of the main diastereoisomer of 3-methyl-1-xyloside-allene was determined for the first time by sin
10 uman gene C3ORF21 encodes a UDP-xylose:alpha-xyloside alpha1,3-xylosyltransferase, acting on xylose-a
11 the core proteins of proteoglycans by beta-d-xylosides also reduced incorporation of LTBP1 into the E
12 vity of the glycoside, we synthesized beta-D-xyloside analogs in which the hydroxyls were substituted
13 signing inhibitors of GAG synthesis based on xyloside analogs with reactive groups in key positions.
14 on fibrinogen were inhibited by both beta-d xyloside and after cleavage of chondroitin sulfate from
15 hat these differ in composition depending on xyloside and cell type, detailed knowledge regarding a s
16 ation, two more ACNs (cyanidin-3-galactoside-xyloside and cyanidin-3-galactoside-xyloside-glucoside-s
17 d as N-(4-methoxyphenyl)formamide 2-O-beta-D-xyloside and was revealed to have the potential to enhan
18 hydrolase specificity hint that, to enzymes, xylosides and arabinofuranosides are closely resemblant.
20 ns can be modified by bisecting GlcNAc, core xylosides and fucosides, and extended N-acetyl lactosami
21 ein glycoform, the addition of benzyl-beta-d-xyloside, and a UDP-xylose: core protein beta-d-xylosylt
22 sulfate side chain formation inhibitor beta-xyloside, and soluble heparin on the overexpression of p
24 ta-D-glucosides, beta-D-galactosides, beta-L-xylosides, beta-D-arabinosides), similar to the native e
25 by assaying the transfer of galactose to the xylosides by galactosyltransferase I (UDP-D-galactose:xy
30 cterized by its unique capacity to take over xyloside derivatives linked to a hydrophobic aglycone as
31 -h incubation in 4-methylumbelliferyl-beta-D-xyloside did not attenuate the ability of LPL to reduce
32 inidin 3-O-glucoside, quercetin 3-O-glucosyl-xyloside, dihydroquercetin, and quercetin 3-O-glucuronid
35 nds from black carrot pomace with cyanidin-3-xyloside-galactoside-glucoside-ferrulic acid (C3XGGF, 60
36 ajor ACN, followed by cyanidin-3-galactoside-xyloside-glucoside-coumaric acid, and cyanidin-3-galacto
37 arified BCJ contained cyanidin-3-galactoside-xyloside-glucoside-ferulic acid as the major ACN, follow
38 actoside-xyloside and cyanidin-3-galactoside-xyloside-glucoside-sinapic acid) were also identified.
40 the PDGF effect, but not in the presence of xyloside, indicating that GAG synthesis that results fro
43 examined as well as protecting groups on the xyloside moiety and the influence of the substituents on
47 paran sulfate side chain formation with beta-xyloside or the addition of soluble heparin prevented ER
48 lycan synthesis (4-methylumbelliferyl-beta-D-xyloside) or sulfation (sodium chlorate) enhanced the re
50 dications that this could be mediated by the xyloside-primed glycosaminoglycans (GAGs) and that these
51 of CS synthesis, 4-methylumbelliferyl-beta-D-xyloside, reduced the amount of the xyloside-CS chains b
52 treated with chlorate or substituted beta-D-xylosides, resulting in undersulfation or secretion of G
56 OPCs and by treating demyelinated mice with xyloside to reduce the CSPG deposition that occurred fol
57 bronectin immunostaining was altered in beta-xyloside-treated eyes, whereas in cell culture, chlorate
58 Ac were only slightly larger than those with xyloside treatment alone (K(av) of 0.55 compared with th
60 toside, peonidin-3-O-glucoside, cyanidin-3-O-xyloside were separated and identified by LC/DAD/MS and
61 hlorate, an inhibitor of sulfation, and beta-xyloside, which provides a competitive nucleation point
62 onation of differently substituted propargyl xylosides with s-BuLi/TMEDA followed by protonation with
63 -keto derivative) and benzyl-4-methyl-beta-D-xyloside, with a methyl group in place of an axial hydro
64 p-nitrophenyl beta-d xylopyranoside (beta-d xyloside), wound microvascular endothelial cells were ca
65 mbination of the food components vitexin-2-O-xyloside (X), raphasatin (4-methylsulphanyl-3-butenyl is
68 cubation of these cells with naphthol-beta-D-xyloside (Xylbeta-O-Np) resulted in accumulation of link