戻る
「早戻しボタン」を押すと検索画面に戻ります。 [閉じる]

コーパス検索結果 (1語後でソート)

通し番号をクリックするとPubMedの該当ページを表示します
1 enotypes, inactivates mismatch repair in the yeast cell.
2 a level, is then extended to the rest of the yeast cell.
3 , there are 57,000 to 60,000 nucleosomes per yeast cell.
4 n animal feed additive, from a low number of yeast cells.
5  beta (Abeta) peptides accumulate around the yeast cells.
6 nked to cellular growth rate in RP-deficient yeast cells.
7 of the plasma membrane during endocytosis in yeast cells.
8 t sulfate, when heterogeneously expressed in yeast cells.
9  endogenous Sod1 (Sod1-KD), and in sod1Delta yeast cells.
10 ed by living and isolated single bacteria or yeast cells.
11 an overcome resistance of turgor pressure in yeast cells.
12 es that can form viable endosymbionts within yeast cells.
13 f Escherichia coli endosymbionts within host yeast cells.
14  cytoplasmic iron-sulfur cluster assembly in yeast cells.
15 eviate the nystatin-sensitivity of lam2Delta yeast cells.
16 As are localized to distinct granules within yeast cells.
17 escently labeled circular chromosome in live yeast cells.
18 ies of formaldehyde-cross-linking in budding yeast cells.
19 rion fusions to encode synthetic memories in yeast cells.
20 anslational modification found in animal and yeast cells.
21 redominant form of mtDNA replication in rho+ yeast cells.
22  that mitophagy is perturbed in CL-deficient yeast cells.
23 s been demonstrated using both bacterial and yeast cells.
24 accumulation, unlike the OsPCS2b transformed yeast cells.
25 in vitro, and blocked autophagy induction in yeast cells.
26 ggregate activity observed in living budding yeast cells.
27 f m(6)A-modified FAA1 transcripts in haploid yeast cells.
28 to monitor glutathione import into the ER of yeast cells.
29 ry of stress adaptation is encoded in single yeast cells.
30 e joint distributions of mRNA populations in yeast cells.
31 bacteria use lactic acid to communicate with yeast cells.
32 izations as well as phenotypes of expressing yeast cells.
33 artments and cell membrane when expressed in yeast cells.
34 ion is closely guided by experiments in live yeast cells.
35  nodes and contractile rings in live fission yeast cells.
36 isappearance of mitochondria from the mutant yeast cells.
37 f NAD into peroxisomes against AMP in intact yeast cells.
38 reams and lysis of Rhodosporidium toruloides yeast cells.
39 ges in senescing and post-senescent survivor yeast cells.
40 s cytoplasmic hydrogen ion concentrations in yeast cells.
41 near invaginations at the plasma membrane of yeast cells.
42 l2-L-13 induces mitophagy in Atg32-deficient yeast cells.
43 critical role in metabolic energy control in yeast cells.
44 he toxicity of human alpha-synuclein in live yeast cells.
45 m can be visualized on the surface of living yeast cells.
46  our well-understood models of mammalian and yeast cells.
47 hnology can be modified to allow analysis of yeast cells.
48 ions for imaging mEos3.2-tagged molecules in yeast cells.
49 ities, namely, Escherichia coli bacteria and yeast cells.
50 rrested, growing, and synchronously dividing yeast cells.
51 inds to antigenic epitopes on the surface of yeast cells.
52 odulates SOS1 activity by activating SOS2 in yeast cells.
53  ultrastructural changes in the cytoplasm of yeast cells.
54 ntly decreases cellular longevity in diploid yeast cells.
55 in translocation are not directly coupled in yeast cells.
56 ,000 ORFs in exponentially growing wild-type yeast cells.
57 ges at 2 to 24 h after infection with viable yeast cells.
58 e Gal4 target genes GAL3 and GAL10 in living yeast cells.
59 icochemical parameters of replicatively aged yeast cells.
60 rity, endocytosis, adhesion, and invasion in yeast cells.
61                                   In fission yeast cells, a microtubule-dependent network has been id
62            Here, we show that the cytosol of yeast cells acidifies modestly in early aging and sharpl
63                   In response to pheromones, yeast cells activate a MAPK pathway to direct processes
64 val in response to sudden glucose depletion, yeast cells activate lipid-droplet (LD) consumption thro
65                                              Yeast cells activate RNR in response to genotoxic stress
66                        These multifunctional yeast cells adhere to the gold sensor surface while simu
67 isolated UPEC was subsequently determined by yeast cell agglutination and immunofluorescence microsco
68 ow that, in wolf lichens, Tremella occurs as yeast cells also in thalli that lack basidiomata and inf
69 , we measure gene network activity in single yeast cells and find that the activity of the compensate
70             C. albicans grows both as single yeast cells and hyphal filaments in the planktonic mode
71 s of Rcf1/Rcf2 single and double null-mutant yeast cells and mitochondria, we further explored how Rc
72 ther media used for culture of mammalian and yeast cells and phosphate-buffered saline.
73                                       Intact yeast cells and plasmolyzed yeast, i.e. yeast cell wall
74 s rapid screening of ADAR variants in single yeast cells and provides quantitative evaluation for enz
75                           Uptake assays with yeast cells and radiolabeled (32)P revealed that PvPht1;
76 evels of tension at kinetochores in dividing yeast cells and relating these measurements to kinetocho
77 ies to track the replicative aging of single yeast cells and reveal that the temporal patterns of het
78 correlations across hundreds of thousands of yeast cells and reveals ample evidence of both vertical
79 d these antibody libraries on the surface of yeast cells and selected antibodies that strongly recogn
80 ycosylation sites, is poorly glycosylated in yeast cells and STT3A-deficient human cells.
81  sensitive marker of increased ROS levels in yeast cells and suggest that changes in ribosomes may be
82 significant decrease in both phagocytosis of yeast cells and the frequency of nonlytic exocytosis.
83                               Screening with yeast cells and various strains of both Gram-positive an
84 sed mitochondria and cytoplasm isolated from yeast cells, and [(35)S]cysteine to detect cytoplasmic F
85 d, stabilise the position and orientation of yeast cells, and demonstrate independent control over mu
86 vents of cell division in both mammalian and yeast cells, and in fission yeast a single mitotic cycli
87  events cause genetic instability in diploid yeast cells, and propose that similar, heterozygous muta
88 mplate for DNA double-strand break repair in yeast cells, and Rad52, a member of the homologous recom
89                                Nevertheless, yeast cells appear to avoid being misled by responding t
90                                         When yeast cells are challenged by a fluctuating environment,
91                                 Fast growing yeast cells are predicted to perform significant amount
92 The trends of measured lateral migrations of yeast cells are similar to the corresponding Clausius-Mo
93                            We show that most yeast cells arrest in G1 before death with low nuclear l
94       Furthermore, the lateral migrations of yeast cells as a function of the ac frequency were measu
95 , the continuous separation of live and dead yeast cells as well as the yeast cells with targeted dia
96 be opens new avenues for studies focusing on yeast cells, as well as other cells with a degradable ce
97 itinated ribosomes from oxidatively stressed yeast cells at 3.5-3.2 angstrom resolution.
98 LM), we probed this question in live fission yeast cells at unprecedented resolution.
99 As confer a competitive fitness advantage to yeast cells because expression of these non-coding molec
100 f molecular noise that is inevitable in tiny yeast cells, because mistakes in sequencing cell cycle e
101 scriptionally formed G4 DNA in vivo and that yeast cells become highly sensitivity to G4-stabilizing
102 gnificant regulatory variation in individual yeast cells, both before and after stress.
103 rgo) and demonstrate its utility not only in yeast cells, but also in cultured mammalian cells, Droso
104 on a genomic scale have been investigated in yeast cells, but comparable experiments have not been do
105 nd is toxic to wheat, tobacco, bacterial and yeast cells, but not to Z. tritici itself.
106 ublocations within mitochondria of respiring yeast cells by fusing a pH-sensitive GFP to proteins res
107 MG inhibits the growth of glucose-fermenting yeast cells by inducing endocytosis and degradation of t
108 ed for oxidative stress responses in fission yeast cells by promoting transcription initiation.
109 platform, we measure noise dynamics in aging yeast cells by tracking the generation-specific activity
110 ully folded bioactive cyclotides inside live yeast cells by using intracellular protein trans-splicin
111 ned Nanobody on the surface of an individual yeast cell can be monitored through a covalent fluoropho
112          However, after several generations, yeast cells can adapt to the loss of mtDNA.
113                                Metabolism in yeast cells can be manipulated by supplying different ca
114                     Our results suggest that yeast cells can use differential protein interactions wi
115 stributes homogeneously in wild-type fission yeast cells, can be made to concentrate at cell ends by
116                   Here, we show that fission yeast cells carrying a mutation in the DNA-binding prote
117                          Exposure of haploid yeast cells, carrying mating type "a," to "alpha pheromo
118        The major chitin synthase activity in yeast cells, Chs3, has become a paradigm in the study of
119  Au level was increased in COPT2, expressing yeast cells compared to vector transformed control.
120 rnal, Stahl et al (2019) reveal that budding yeast cells confer a growth advantage to their daughters
121 duce rejuvenated daughters, dividing budding yeast cells confine aging factors, including protein agg
122 e presence of a proteasome inhibitor or when yeast cells contained mutations in the CDC48 or SSA1 gen
123 nal microscopic diagnosis, as characteristic yeast cells could be observed only in 14 pus samples.
124                                        Using yeast, cell culture, and mouse models of LGMDD1, we foun
125                                Using fission yeast cell cycle as an example, we uncovered that the no
126 ability, speed and robustness of the fission yeast cell cycle oscillations.
127 we provide a stochastic model of the budding yeast cell cycle that accurately accounts for the variab
128 rom peripheral leukocytes, brain tissue, and yeast cell cycle, revealed novel marker genes that were
129 ture are linked to ER inheritance during the yeast cell cycle.
130 t of H3K56Ac on transcription throughout the yeast cell cycle.
131                                   In budding yeast, cell cycle progression and ribosome biogenesis ar
132 lity, as observed in the budding and fission yeast cell- cycle.
133 er investigations of the budding and fission yeast cell-cycle, we identify two generic dynamical rule
134                                   In fission yeast cells, Cyclin B(Cdc13) scales with size, and we pr
135 rus, F1L does not prevent caspase-9-mediated yeast cell death.
136                         We show that fission yeast cells deficient in ER-PM contacts exhibit aberrant
137                       In the absence of LDs, yeast cells display alterations in their phospholipid co
138              We show that RNase H2-deficient yeast cells displayed elevated frequency of Rad52 foci,
139 s antigenic preference is also observed with yeast cells displaying Ab fragments.
140                           Rod-shaped fission yeast cells divide at a threshold size partly due to Cdr
141 r9 positions mitotic spindles during budding yeast cell division.
142 or single-cell RNA sequencing (scRNA-seq) of yeast cells do not match the throughput and relative sim
143 RNA quantities is apparent in single fission yeast cells during a normal cell cycle.
144                         Our images of single yeast cells during aging, show that the abundance of sev
145 ined the range of proteins that aggregate in yeast cells during normal growth and after exposure to s
146                                       Normal yeast cells eliminate the large majority of prion varian
147 w that both vegetative and pheromone-treated yeast cells exhibit discrete and asynchronous Ca(2+) bur
148                                      Budding yeast cells exist in two mating types, a and alpha, whic
149      We study cell polarization when fission yeast cells exit starvation.
150  the transcriptomes of >2,000 single fission yeast cells exposed to various environmental conditions
151 o the time course of fluorescence signal per yeast cell expressing mEos3.2.
152                      Transport analyses with yeast cells expressing a truncated, vacuole-targeted ver
153                                Compared with yeast cells expressing Arabidopsis thaliana Pht1;5, cell
154 probe the effects of polyethylene glycol and yeast cell extract as crowding agents.
155 alogs to thiophosphorylate its substrates in yeast cell extracts as well as when produced as recombin
156                                              Yeast cell factories encounter physical and chemical str
157 molecules, have been proposed to explain how yeast cells filter fluctuations and detect shallow gradi
158 terized by emergence of a germ tube from the yeast cell followed by mold-like growth of branching hyp
159                  Purification of Ty1-IN from yeast cells followed by mass spectrometry (MS) analysis
160 eplication of hundreds of individual fission yeast cells for over seventy-five generations.
161 nction of pH and protein phase separation in yeast cells for pH values close to the isoelectric point
162 ngation-can be recapitulated in vitro with a yeast cell-free system.
163            The HPF1 repeat expansion shifted yeast cells from a sedentary to a buoyant state, thereby
164                                              Yeast cells from colonies or liquid cultures are lysed b
165 for the SWI/SNF complex in the transition of yeast cells from fermentative to respiratory modes of me
166                Here we find DnaJB6-protected yeast cells from polyglutamine toxicity and cured yeast
167                           Here, we find that yeast cell fusion is negatively regulated by components
168                           Rod-shaped fission yeast cells grow in a highly polarized manner, and genet
169                                          For yeast cells grown in synthetic defined (SD) medium, the
170 rformed a genome-wide expression analysis in yeast cells grown in the presence or absence of the drug
171 we identified 377 IVC-associated proteins in yeast cells grown under steady-state low-glucose conditi
172 responding eIF2gamma-I318M mutation impaired yeast cell growth and derepressed GCN4 expression, an in
173 nment and that either K113E or E195K induces yeast cell growth defects rescued by E/K.
174 lly generated metabolic flux data to predict yeast cell growth.
175  selected differently in haploid and diploid yeast cells: haploid cells bud in an axial manner, while
176                                              Yeast cells harboring [SMAUG(+)] downregulate a coherent
177                                      Budding yeast cells have a finite replicative life span; that is
178 n early after telomerase inactivation (ETI), yeast cells have accelerated mother cell aging and mildl
179 y responded to sphingolipid, suggesting that yeast cells have, in addition to Orm phosphorylation, an
180  oilseed rape in the microsomal fractions of yeast cells heterologously expressing these enzymes.
181 the budding pattern and pre-mRNA splicing in yeast cells; however, no Bud13p homologs have been ident
182 d to improve the enrichment of low-abundance yeast cells in an iDEP channel.
183 HIF-1alpha creates a hostile environment for yeast cells in human macrophages by interrupting the abi
184 s, namely (i) the chromosome interactions of yeast cells in quiescence and in exponential growth, and
185 e measurement of ac-DEP lateral migration of yeast cells in solutions with different electrical condu
186                         The DEP behaviors of yeast cells in suspending media with different ionic con
187 of Mediator subunits in wild-type and mutant yeast cells in which RNA polymerase II promoter escape i
188 function is required for sterol secretion in yeast cells, indicating that members of this superfamily
189                         The growth rate of a yeast cell is controlled by the target of rapamycin kina
190 ions, the time-integrated mEos3.2 signal per yeast cell is similar in live cells and fixed cells imag
191                            Mating of budding yeast cells is a model system for studying cell-cell int
192                                              Yeast cells lacking either Put6 or Put7 exhibit a pronou
193  yeast expression system and discovered that yeast cells lacking endogenous potassium channels could
194 function of Dna2 in end resection in budding yeast cells lacking exonuclease 1.
195                                              Yeast cells lacking LDs are severely defective in PSM gr
196                                              Yeast cells lacking Msn2 and Msn4 exhibit prevalent repr
197 to cellular toxicity and cell cycle delay in yeast cells lacking PSH1, but not in cells lacking UBR1,
198 rnover as Cse4 degradation is compromised in yeast cells lacking RCY1 Excessive Cse4 accumulation in
199                                 We show that yeast cells lacking seipin displayed altered sensitivity
200 d cell death caused by DNA damage in budding yeast cells lacking the Rad53 checkpoint protein kinase
201             Inhibition of Hsp104 function in yeast cells leads to a failure to generate new propagons
202 nt in tension over multiple isogenic budding yeast cell lines by genetically altering the magnitude o
203                                              Yeast cell lines were genetically engineered to display
204  and formation of a shmoo-like morphology in yeast cells, lower pheromone doses elicit elongated cell
205  purified a high quantity of mRNA from crude yeast cell lysate compared to a phenol/chloroform extrac
206 closporine A with cyclophilin A protein in a yeast cell lysate is successfully detected and quantifie
207 ffer conditions likely by permeabilizing the yeast cell membrane.
208 ancement was also shown on a separation of a yeast cell metabolite extract, where the enhanced TIC fo
209         Our results support a model in which yeast cells mobilize, and perhaps compartmentalize, mult
210 mimetic FUS reduces aggregation in human and yeast cell models, and can ameliorate FUS-associated cyt
211           We conclude that aneuploid budding yeast cells mount the ESR, rather than the CAGE signatur
212                                              Yeast cells must grow to a critical size before committi
213 by both plasmolysed (PYC) and nonplasmolysed yeast cell (NPYC) and stability of thymoquinone and bioa
214  homeostasis of cation concentrations in the yeast cells of S. cerevisiae.
215 mely, the geometrical effect of the dividing yeast cell on the diffusion of protein aggregates, and t
216 at constitutive membrane anchoring of GIV in yeast cells or rapid membrane translocation in mammalian
217                                           In yeast cells, overexpression of the top2-F1025Y,R1128G al
218 l tubes had increased adhesion compared with yeast cells ( P < 0.05).
219                                              Yeast cell populations harboring the same defined aneupl
220 ryotype-specific dosage effects in aneuploid yeast-cell populations with random and diverse chromosom
221                                  In dividing yeast cells, protein aggregates that form under stress o
222 1R) ) in the FRB domain of Tor2 that renders yeast cells rapamycin resistant and temperature sensitiv
223 show that upon growth at higher temperature, yeast cells relax the retention of DNA circles, which ac
224 r fit, our model quantitatively predicts the yeast cell response to pheromone gradient providing an i
225   Nitrogen replenishment of nitrogen-starved yeast cells resulted in substantial transcriptome change
226         Compromising the activity of Ddi1 in yeast cells results in the accumulation of polyubiquitin
227  (B. cereus, E. coli, and S. enterica) and a yeast cell (S. cerevisiae), ranging in size from 1 to 6.
228 , cancer cells (MCF-7, MDA-435 and CD34(+)), yeast cells (saccharomyces cerevisiae, listeria innocua
229                                      Haploid yeast cells secrete mating pheromones that are sensed by
230 report comprehensive ribosome profiling of a yeast cell size series from the time of cell birth, to i
231                                   In budding yeast, cell size is thought to be controlled almost enti
232                                   In budding yeast, cell size primarily modulates the duration of the
233 nked to the end of DNA in RNase H2-deficient yeast cells, supporting this model.
234                                       We use yeast cell surface display to engineer E6AP to exclusive
235 designs and assessment of their stability on yeast cell surface, detailed biophysical characterizatio
236 g1 in removing exposed beta-glucans from the yeast cell surface.
237                                          The yeast cell-surface glucose sensors Rgt2 and Snf3 functio
238                    Similarly to CL-deficient yeast cells, TAZ-KO cells exhibited elevated sensitivity
239                                           In yeast cells that do not readily take up pyruvate, the ad
240 (GET) pathway was described in mammalian and yeast cells that serve as a blueprint of TA protein inse
241                               As observed in yeast cells, the TATA-binding protein (TBP) typically di
242 , promotes the symmetric division of fission yeast cells through spatial control of cytokinesis.
243                        We also elucidate how yeast cells thwart Ty3/Gypsy proliferation by blocking t
244                             The ability of a yeast cell to propagate [PSI(+) ], the prion form of the
245                  Diverse bacteria can elicit yeast cells to acquire [GAR(+)], although the molecular
246 he predicted 2nd WD40 propeller was shown in yeast cells to bind Vernalization 5 (VRN5), which contai
247 on dramatically alters the susceptibility of yeast cells to ergosterol biosynthesis inhibitors.
248 pression assays in Nicotiana benthamiana and yeast cells to examine its functionality.
249     Transient exposure to lactic acid caused yeast cells to heritably circumvent glucose repression.
250 tion localization microscopy of live fission yeast cells to improve the spatial resolution to approxi
251 scence microscopy techniques in live budding yeast cells to investigate how Mex67 facilitates mRNA ex
252                                 As seen from yeast cells to mammalian cells, size homeostasis is main
253 novative single-molecule imaging approach in yeast cells to measure chromatin association of individu
254  CSLAs from different species, we programmed yeast cells to produce an HM backbone composed exclusive
255 possibilities by using clonal populations of yeast cells to quantify the inherent relationships betwe
256 llular traffic of the Chs3 protein, allowing yeast cells to regulate morphogenesis, depending on envi
257 a stress-induced survival strategy, allowing yeast cells to save energy, protect proteins from degrad
258                 The gene that allows budding yeast cells to switch their mating type evolved from a n
259  and mathematical modeling in single fission yeast cells to uncover the precise molecular mechanisms
260  highly conserved from simple systems, e.g., yeast cells, to the much more complex human system.
261 d chromosomal loci during interphase in live yeast cells together with polymer models of chromatin ch
262 t MAb 4D1 binds to and recognizes conidia to yeast cells' transition inside of a human monocyte-like
263 de enzymes produced by SSF were utilised for yeast cell treatment leading to simultaneous release of
264                                              Yeast cells unable to synthesize inositol pyrophosphates
265 popular green-to-red PCFP mEos3.2 in fission yeast cells under a wide range of imaging conditions.
266 t, we systematically monitored the growth of yeast cells under various frequencies of oscillating osm
267 shown that in response to pheromone, budding yeast cells undergo a rise of cytosolic Ca(2+) that is m
268               Upon acute glucose starvation, yeast cells undergo drastic physiological and metabolic
269 osed to a high dose of mating pheromone, the yeast cell undergoes growth arrest and forms a shmoo-lik
270                                              Yeast cells undergoing the diauxic response show a strik
271                  To describe the dynamics of yeast cells upon osmotic upshift, we extended the model
272 im32 proteins are essential for viability of yeast cells upon treatment with the redox mediators gall
273 his work, we measured LacI binding in living yeast cells using a fluorescent repressor operator syste
274 mmobilizing fungal laccase on the surface of yeast cells using synthetic biology techniques.
275 was found in glycolytic oscillations in real yeast cells, verifying that chronotaxicity could be used
276 n1, a DNA/RNA helicase that is essential for yeast cell viability and homologous to human senataxin,
277                    In the present study, the yeast cell wall fractionation process involving enzymati
278                                          The yeast cell wall integrity MAPK Slt2 mediates the transcr
279 sently, commercial MOS is being derived from yeast cell wall mannan and is widely used as prebiotic i
280                                          The yeast cell wall of Saccharomyces cerevisiae is an import
281                             Native yeast and yeast cell wall particles (YCWPs) were used as model cel
282 tact yeast cells and plasmolyzed yeast, i.e. yeast cell wall particles (YCWPs), of Saccharomyces cere
283 ion of plant HM: up to 30% of glycans in the yeast cell wall.
284 f purified mannoproteins (MP), isolated from yeast cell walls upon the enzymatic treatment, revealed
285  the ability of adsorbing color molecules by yeasts' cell walls was assessed.
286 bility of the CBD to bind to the surfaces of yeast cells was found to be unperturbed by this modifica
287 the maximum accumulation of both ions in the yeast cells was observed.
288 er to monitor Fus3 and Kss1 activity in live yeast cells, we demonstrate that overall mating MAPK act
289  wild-type levels of mcm10-m2,3,4 in budding yeast cells, we observed a severe growth defect and a su
290  from both wild-type and hydroxyurea-treated yeast cells, we show that our model is more accurate tha
291 longating transcript sequencing (NET-seq) to yeast cells, we show that Xrn1 functions mainly as a tra
292                                              Yeast cells were encapsulated in nanoliter volumes by dr
293 ntial to aid the enrichment of low-abundance yeast cells when filler volume fractions approximately 1
294                                              Yeast cells, when exposed to stress, can enter a protect
295                         Unlike in animal and yeast cells, which have single SEIPIN genes, plants have
296                                              Yeast cells with arginine-to-alanine mutations in the H4
297                Third, treatment of wild-type yeast cells with E9591 or LMT generated cellular defects
298  of live and dead yeast cells as well as the yeast cells with targeted diameter and dielectric proper
299 rowth and DNA replication defects in budding yeast cells, with diminished DDK phosphorylation of Mcm2
300 improved the ability to metabolize xylose of yeast cells without adaptive evolution, suggesting that

 
Page Top