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1 GFR2 signaling or using a vascular deficient zebrafish mutant.
2 epileptic activities observed in a mind bomb zebrafish mutant.
3 nt mutant ARL13B rescued the Arl13b scorpion zebrafish mutant.
4 opment necessary for the characterization of zebrafish mutants.
5 examine cmlc2 and vmhc expression in several zebrafish mutants.
6 ge of defects similar to those of dorsalized zebrafish mutants.
7 drocyte number and myelinated axons in gpr56 zebrafish mutants.
8 tive sarcomere assembly in smyd1a and smyd1b zebrafish mutants.
9                     Here, we discuss work on zebrafish mutants affecting gastrulation and patterning
10 ated heat shock factor 1 (hsf(-/-)) knockout zebrafish mutants and analyzed hsp gene expression and m
11                     Here we analyse a set of zebrafish mutants and arrive at a different interpretati
12                                        Using zebrafish mutants and in vivo imaging, we identified the
13 the exocyst complex, by analyzing both exoc5 zebrafish mutants, and photoreceptor-specific Exoc5 knoc
14 , we report on the novel diabetic pdx1 (-/-) zebrafish mutant as a model for diabetic retinopathy tha
15 ervations support the alms1 loss-of-function zebrafish mutant as a monogenic model for mechanistic in
16 zygous kcnh2 mutations and expand the use of zebrafish mutants as a model system to study human arrhy
17 on in somite measurements across a number of zebrafish mutants; (b) the delayed formation of somites
18 ral patterns of gene expression in different zebrafish mutant backgrounds allow further quantitative
19 netic analyses of a recessive, larval lethal zebrafish mutant, bal(a69), characterized by severe eye
20 cterized the ocular defects in the recessive zebrafish mutant blowout that presents with a variably p
21 , the study was focused on a reduced melanin zebrafish mutant, brass.
22 roadly rescue morphology and motility in the zebrafish mutant, but alter motor axon morphology, demon
23  those of phenotypic rescue of headless/tcf3 zebrafish mutants by mouse Six3, demonstrate that region
24                   Here, we describe a unique zebrafish mutant, caliban (clbn), with arrested developm
25                          A homozygous lethal zebrafish mutant, cassiopeia (csp), was identified by a
26 sible for the cell cycle defects seen in the zebrafish mutant, cease&desist (cds).
27                   We previously identified a zebrafish mutant ceylon (cey) that has a severe reductio
28 ellular, and electrophysiological basis of a zebrafish mutant characterized by ventricular asystole.
29  We performed a deep characterization of the zebrafish mutant Chihuahua, that carries a G574D (p.G736
30                                       In the zebrafish mutant cloche (clo), SCL expression is nearly
31 t hematopoietic and vascular deficits in the zebrafish mutant cloche.
32                                          The zebrafish mutant colgate (col)/histone deacetylase1 (hda
33                                  We utilized zebrafish mutant combinations that disrupt Chordin and m
34                           Here we describe a zebrafish mutant crimsonless (crs) with a developmental
35                                       In the zebrafish mutant cyclops (Cyc(b16)), most embryos have t
36 alling pathway, such as those mutated in the zebrafish mutants cyclops, squint and one-eyed pinhead (
37                            We characterize a zebrafish mutant defective in definitive haematopoiesis
38       From a genetic screen, we identified a zebrafish mutant defective in the leo1 gene.
39 ovel genes for congenital muscle diseases, a zebrafish mutant, designated patchytail, was identified
40                               The corneas of zebrafish mutants display severe abnormalities of the ep
41 2) knockdown in these cells or its loss in a zebrafish mutant disrupted cell extrusion.
42      Analysis of heart development in lamb1a zebrafish mutant embryos reveals mild morphogenetic defe
43                             Adult rspo3(-/-) zebrafish mutants exhibit a dysmorphic cranial skeleton
44 Moreover, when expressed in mfsd2aa-morphant zebrafish, mutants failed to rescue microcephaly, BBB br
45 ectron microscopy to evaluate the corneas of zebrafish mutant for a crumbs locus oko meduzy (ome) and
46                        Here, we found that a zebrafish mutant for a gene which in humans causes one t
47 enetic screen yielded malbec (mlb(bw306)), a zebrafish mutant for cdh5, with normal embryonic and def
48        Moreover, Wnt activity is enhanced in zebrafish mutant for cohesin subunits stag2b and rad21.
49                                              Zebrafish mutant for foxp3a displayed excess T lymphocyt
50 ected the mechanisms of scoliosis onset in a zebrafish mutant for the rpgrip1l gene encoding a ciliar
51                                        Using zebrafish mutants for cav1, cav3, and cavin1b, we show t
52        To address this question, we utilized zebrafish mutants for ewsa and tp53.
53 sidedness to organ chirality, we examined 12 zebrafish mutants for initial heart tube position and la
54                                 We generated zebrafish mutants for pkd1 and noted cystic kidney and m
55                                              Zebrafish mutants for slc12a2b also display swellings of
56        Here, we report the identification of zebrafish mutants for the gene encoding Structure specif
57                           Here we describe a zebrafish mutant, frascati (frs), that shows profound hy
58 polarity in vertebrates, we have studied the zebrafish mutant fused somites (fss), because its paraxi
59                                  Analyses of zebrafish mutants have confirmed the action of chordin (
60                                         Many zebrafish mutants have specific defects in axon guidance
61 ies using in vitro chick models and systemic zebrafish mutants have suggested that FGF signaling is r
62                                              Zebrafish mutants have traditionally been obtained by us
63      Here, we report the identification of a zebrafish mutant, heart and mind, which exhibits multipl
64 , we find that the lama5 mutant is the first zebrafish mutant identified in which the pectoral fins f
65                We identified an adult-viable zebrafish mutant in which the negative feedback on the s
66 LZK cell-type-specific functions, we created zebrafish mutants in each gene.
67 zer coordinates morphogenetic movements, and zebrafish mutants in T-box mesoderm-specific genes help
68                                 We show that zebrafish mutants in uhrf1 and dnmt1 have defects in len
69 ucted to characterize ocular defects in five zebrafish mutants in which core components of the v-ATPa
70                                        Using zebrafish mutants in which OPCs migrate out of the spina
71  mechanosensory neurons of touch-insensitive zebrafish mutants indicates that in three mutant lines t
72 that the hypochromic anaemia in shiraz (sir) zebrafish mutants is caused by deficiency of glutaredoxi
73             Using whole-genome sequencing of zebrafish mutants isolated in an unbiased genetic screen
74  (vlt(m651)) is one of only five "bloodless" zebrafish mutants isolated through large-scale chemical
75                          In contrast, gpr156 zebrafish mutants lack the smaller mechanically evoked s
76  is capable of rescuing hypomyelination in a zebrafish mutant lacking BACE1.
77                  In this study, we generated zebrafish mutants lacking all four zebrafish Mesp genes
78                        Here, we show that in zebrafish mutants lacking ErbB3 function, neural crest c
79                                  Analysis of zebrafish mutants lacking functional Pten revealed that
80                      Here, we report that in zebrafish mutants lacking Schwann cells in peripheral ne
81                             We have cloned a zebrafish mutant, lauscher (lau), identified by its swol
82               Here, we present analysis of a zebrafish mutant line carrying a truncation mutation, W8
83 activated HIF transcriptional responses in a zebrafish mutant line harboring a point mutation in the
84                         We have identified a zebrafish mutant line in which deficient emi1 gene expre
85             We generated a stable homozygous zebrafish mutant line using the CRISPR/Cas9 system.
86 d genetic screen, we previously identified a zebrafish mutant line, tgct, which develops spontaneous
87                                          The zebrafish mutant line, twister, models SCS in terms of a
88 aB-crystallin, we generated loss-of-function zebrafish mutant lines by utilizing the CRISPR/Cas9 syst
89                            However, managing zebrafish mutant lines derived from mutagenesis screens
90 ion, we screened a collection of insertional zebrafish mutant lines for expression of the HSC marker,
91 suitable for large-scale deep-phenotyping of zebrafish mutant lines, which uses optical projection to
92 cal characterization of two loss-of-function zebrafish mutant lines: wfs1aC825X and wfs1bW493X.
93                                  The classic zebrafish mutant longfint2 develops and regenerates dram
94 quencing and mRNA rescue that the dorsalized zebrafish mutant lost-a-fin (laf) is defective in the ge
95               Furthermore, we found that the zebrafish mutants mariner (myo7aa) and sputnik (cad23) t
96 ificant risk factor for glaucoma, the bugeye zebrafish mutant may be a model organism for the disease
97                                          The zebrafish mutants merlot (mot) and chablis (cha) exhibit
98                              In summary, our zebrafish mutant model of eif2b3 provides novel insights
99 decreased cilia) in a CRISPR/Cas9-engineered zebrafish mutant model provide additional support for AR
100 aling in both human embryonic stem cells and zebrafish mutant models, as well as abnormal expression
101                                    Moreover, zebrafish mutants (mut) with loss of function for the on
102                                           In zebrafish, mutant MYL4 leads to disruption of sarcomeric
103 e as a screening assay, we have identified a zebrafish mutant named fantasma (fan), which displays re
104 imary and secondary hindbrain neurons in the zebrafish mutant neckless (nls), which disrupts retinald
105  we identified a homolog of the Stil gene in zebrafish mutant (night blindness b, nbb), which showed
106                           Here we describe a zebrafish mutant, no optokinetic response f(w21) (nof),
107                             Studies with the zebrafish mutant nrc have revealed that loss of SynJ1 al
108                                          The zebrafish mutant nrc is a possible model for human retin
109  Here we present crystal, an optically clear zebrafish mutant obtained by combining different viable
110                          Here, we describe a zebrafish mutant of upf1, encoding the central component
111                                          The zebrafish mutants of CCTbeta are known to exhibit the ey
112 their functions in axon injury, we generated zebrafish mutants of each gene, labeled motor neurons (M
113                                          The zebrafish mutant, partial optokinetic response b (pob),
114                                  A red-blind zebrafish mutant, partial optokinetic response b (pob),
115 irst identified as the locus affected in the zebrafish mutant pescadillo, which exhibits severe defec
116                   The striking similarity of zebrafish mutant phenotypes and human diseases emphasize
117 approach is based on the rescue of embryonic zebrafish mutant phenotypes by "humanized" zebrafish ort
118              In comparing mouse knockout and zebrafish mutant phenotypes, we propose a pathway for er
119          Consistent with this notion, tfap2a zebrafish mutants present with abnormal alx3 expression
120                     A recent analysis of the zebrafish mutant prometheus points to a previously unkno
121                                        A new zebrafish mutant provides further insight into how the a
122                                          The zebrafish mutant rapunzel has heterozygous defects in bo
123   Together, our study showed that the rlbp1a zebrafish mutant recapitulated many features of human bl
124                          Here, we identify a zebrafish mutant, redhead (rhd(mi149)), that exhibits em
125                                          The zebrafish mutant represents the first genetically 'accur
126                       Here we characterize a zebrafish mutant, retsina (ret), that exhibits an erythr
127   Retinal examination of the novel ush2au507 zebrafish mutant revealed a slowly progressive degenerat
128                                            A zebrafish mutant (s434 mutation) was identified that dis
129                                          The zebrafish mutant sauternes (sau) has a microcytic, hypoc
130                                          Our zebrafish mutant serves as a model for developing therap
131  CRISPR/Cas9-mediated REEP5 loss-of-function zebrafish mutants show sensitized cardiac dysfunction up
132 ion of chordin in D-V patterning, dorsalized zebrafish mutants showed expanded domains of chordin exp
133              Here, we report that a dominant zebrafish mutant, smoothback (smb), fails to develop a d
134                                Analysis of a zebrafish mutant, spondo, whose spine is dysmorphic, pro
135                 Genetic screening identifies zebrafish mutants, such as fat free, that show normal di
136 mutant phenotype of the strongest dorsalized zebrafish mutant swirl/bmp2b, revealing equivalent genet
137 ns of positional cloning, we reveal that the zebrafish mutant tennismatch is a hypomorphic allele of
138                           Here, we present a zebrafish mutant that ceases mitosis at the beginning of
139 ified split top, a recessive maternal-effect zebrafish mutant that disrupts embryonic patterning upst
140 e identified brambleberry, a maternal-effect zebrafish mutant that disrupts karyomere fusion, resulti
141             Using a novel hypomorphic hnf1ba zebrafish mutant that exhibits pancreas hypoplasia, as o
142 ycat from the deletion interval of cloche, a zebrafish mutant that has dramatically reduced hematopoi
143                  A recent report describes a zebrafish mutant that provides us with some enticing clu
144 ered expression patterns of lft1 and lft2 in zebrafish mutants that affect midline development sugges
145 ssues, we analyzed otic placode induction in zebrafish mutants that are deficient in prospective otic
146                                      Work on zebrafish mutants that develop supernumerary neuromasts
147 crowding with in vivo studies of aquaporin 0 zebrafish mutants that disrupt its regulation.
148                                 By analyzing zebrafish mutants that disrupt sonic hedgehog (shh) expr
149                                          Two zebrafish mutants that do not develop brain ventricles a
150                            Here, we identify zebrafish mutants that form supernumerary long bones in
151 o rescues retinal axon pathfinding errors in zebrafish mutants that have a partial functional loss of
152                                  Analysis of zebrafish mutants that have defects in motor behavior ca
153  primary motoneuron specification in several zebrafish mutants that have distinct effects on paraxial
154  in one-eyed pinhead (oep) and cyclops (cyc) zebrafish mutants, the pattern is altered.
155    Here, we use a temperature-sensitive mitf zebrafish mutant to conditionally control endogenous MIT
156                                  To identify zebrafish mutants to serve as models for hepatic patholo
157                      Here we characterized a zebrafish mutant--too few (tof)--that develops hindbrain
158 its C terminus, cause the touch-unresponsive zebrafish mutant touchdown.
159                                       In the zebrafish mutant unplugged, two of the three segmental m
160  Here we quantifiy four eye movements in the zebrafish mutant valentino and hox3 knockdowns, and find
161                                          The zebrafish mutant violet beauregarde (vbg) can be identif
162 e describe the positional cloning of a blind zebrafish mutant, wait until dark (wud), which encodes a
163                                  Because the zebrafish mutant was a global knockout, we also observed
164                                  The pdx1-/- zebrafish mutant was recently established as a novel ani
165 pproach we mapped each of the five different zebrafish mutants we sequenced and identified likely cau
166                                        Using zebrafish mutants we show here that in the zebrafish gbx
167 ession studies and the analysis of different zebrafish mutants, we have assembled a molecular pathway
168                        Using pax7a and pax7b zebrafish mutants, we identified a previously unknown re
169 sponsible for the hypochromic anaemia of the zebrafish mutant weissherbst.
170            We firstly generated ghrelin( / ) zebrafish mutant, which displayed hyperactive, attention
171                       yquem (yqe(tp61)) is a zebrafish mutant with a photosensitive porphyria syndrom
172 dx4 as the locus mutated in kugelig (kgg), a zebrafish mutant with an early defect in haematopoiesis
173 esis screen to identify no blastema (nbl), a zebrafish mutant with an early fin regeneration defect.
174                            Here we isolate a zebrafish mutant with heart malformations, called 34c.
175 terized the phenotype of chardonnay (cdy), a zebrafish mutant with hypochromic, microcytic anemia, an
176 l ATPase inhibitory factor 1 (atpif1) from a zebrafish mutant with profound anaemia, pinotage (pnt (t
177                   We have identified a blind zebrafish mutant with rapid degeneration of cone photore
178 e cellular bases of heart development and on zebrafish mutants with cardiac abnormalities whose study
179 left-right asymmetry defects, reminiscent of zebrafish mutants with defective cilia.
180                            Today, studies of zebrafish mutants with defective heart function are prov
181  reagents, future studies of Hb switching in zebrafish mutants with defective hematopoiesis will be p
182                          We identified three zebrafish mutants with defects in biliary development.
183 s screens for developmental mutants, several zebrafish mutants with defects in blood development have
184 ide a useful molecular probe for identifying zebrafish mutants with defects in granulopoiesis.
185  and will ultimately be useful in evaluating zebrafish mutants with defects in hemoglobin production
186 n important framework for future analyses of zebrafish mutants with defects in this process.
187 s epilepsy and autism, we determined whether zebrafish mutants with grossly abnormal brain structure
188 ipid mass, we performed a genetic screen for zebrafish mutants with hepatic steatosis, a pathological
189 unc45b, hsp90aa1.1 and smyd1b is specific to zebrafish mutants with myosin folding defects, and is no
190 RISPR-Cas9-mediated genome editing generated zebrafish mutants with novel missense and frameshift all
191                              We generated 27 zebrafish mutants with presumptive protein-truncating mu
192 nt and myelination, we undertook a screen of zebrafish mutants with previously characterized neural d
193  Behavioral screens have uncovered dozens of zebrafish mutants with striking visual defects.

 
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