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1 BB6F1-Kit(W/Wv) mice) was able to affect AYP-induced itch.
2 and to identify the molecular basis of IL-31-induced itch.
3 nel acting together with TRPA1 to mediate CQ-induced itch.
4 ced by histamine but did not prevent cowhage-induced itch.
5 ch but did not significantly alter histamine-induced itch.
6 distinct from CMi fibers mediating histamine-induced itch.
7 ce that TRPV4 is a key mediator of serotonin-induced itch.
8 portant role of myelinated fibers in cowhage-induced itch: (1) a selective conduction block in myelin
9                                Spinal opioid-induced itch, a prevalent side effect of pain management
10 eater dlPFC and caudate activation to nocebo-induced itch also demonstrated greater dlPFC and caudate
11            Subjects reporting greater nocebo-induced itch also demonstrated greater placebo reduction
12 rphine and explain several aspects of opioid-induced itch and analgesia.
13                  Long persistence of cowhage-induced itch and diminished histamine-induced flare in n
14 unrecognized spinal circuit for mechanically-induced itch and elucidated a mechanism that keeps it in
15                                          BDL-induced itch and hypersensitivity involved a minor contr
16 kin with topical capsaicin abolished cowhage-induced itch but did not significantly alter histamine-i
17        Brain activity likely supports nocebo-induced itch, but is currently unknown.
18 mechanisms underlying cowhage- and histamine-induced itch differ.
19                   The persistence of cowhage-induced itch for at least 30 min and a histamine-induced
20 ht to the brain mechanisms supporting nocebo-induced itch in AD, thus aiding our understanding of the
21  scratching on skin blood flow and histamine-induced itch in healthy volunteers.
22     Together, these data suggest that opioid-induced itch is an active process concomitant with but i
23             These results indicate that 5-HT-induced itch is linked to TRPV4.
24      Providing potential insight into opioid-induced itch prevention, we demonstrate that molecular a
25  (SDH) is required selectively for histamine-induced itch sensation.
26 There was no significant difference in IL-31-induced itch start time, duration and intensity between
27                      The late onset of IL-31-induced itch supports the notion that IL-31 exerts its p
28                                     Allergen-induced itch was evaluated in 20 patients with AD after
29 itive neurons (pruriceptive fibers), and AYP-induced itch was reduced by the selective GRP receptor a
30                             Importantly, AYP-induced itch was reduced by treatment with either the se
31                                        IL-31-induced itch was significantly reduced in TRPV1-deficien

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