ライフサイエンスコーパス: -infected

1 udy of matrix turnover in HIV type 1 (HIV-1)-infected and -uninfected TB patients and controls, and a

2 Although most herpes simplex virus 1 (HSV-1)-infected individuals shed the virus in their body fluids

3 (PIB) +/- ribavirin (RBV) in HCV genotype 1-infected patients with prior virologic failure to HCV DA

4 In 44.6% of HCV genotype 1-infected patients, only the variant rs12785878 in the DH

5 in chronic hepatitis C virus genotype (GT)1-infected treatment-naive and prior-relapse patients.

6 alating study of BMS-936559, including HIV-1-infected adults aged >18 to <70 years on suppressive ant

7 ebo-controlled, non-inferiority trial, HIV-1-infected adults were screened and enrolled at 119 hospit

8 MMP activity differed between HIV-1-infected and -uninfected TB patients and corresponded wi

9 lones segregated based on responses to HIV-1-infected and peptide-loaded target cells.

10 dant capacity in exosomes derived from HIV-1-infected and uninfected macrophages.

11 Thus, the proliferation of HIV-1-infected cells may play a role in viral persistence, but

12 y ET at 5-days post-infection, whereas HIV-1-infected cells surrounded by pools of free virions were

13 hown to increase the susceptibility of HIV-1-infected cells to ADCC despite the activity of Vpu.

14 elated with enhanced susceptibility of HIV-1-infected cells to ADCC.

15 nctional antibodies that can eliminate HIV-1-infected cells.

16 ations in longitudinal samples from 10 HIV-1-infected children who initiated ART when viral diversity

17 s collected from 70 virally suppressed HIV-1-infected individuals from Rakai District, Uganda, who ha

18 y in which 15 virologically suppressed HIV-1-infected individuals on antiretroviral therapy received

19 Studies treating HIV-1-infected individuals with latency reactivation agents to

20 the extent of proviral integration in HIV-1-infected MDDCs was unaffected by the absence of Vpr, the

21 ls accumulate in the blood of aviremic HIV-1-infected patients on long-term antiretroviral therapy, a

22 ively eliminate the reactivated latent HIV-1-infected T cells.

23 ols, and a prospective cohort study of HIV-1-infected TB patients at risk of TB immune reconstitution

24 he importance of multiscale imaging of HIV-1-infected tissues and are adaptable to other animal model

25 we demonstrate that mTORC1 activity in HSV-1-infected cells is largely insensitive to stress induced

26 reduced virus reactivation in latently HSV-1-infected mice and HSV-2-infected guinea pigs.

27 is of trigeminal ganglia from latently HSV-1-infected, glutamine-treated WT mice showed upregulation

28 Based on the brain cytokine levels, MAV-1-infected Unc93b1(-/-) mice had a very different inflamma

29 on in latently HSV-1-infected mice and HSV-2-infected guinea pigs.

30 Enrichment of GT in Aspergillus-infected neutropenic lung correlated with fungal burden

31 se, and outcome between individual S. aureus-infected ICU patients remains enigmatic, suggesting a ne

32 oiesis and effective resolution of S. aureus-infected wounds, revealing a potential antibiotic-free s

33 In part B, subtype B-infected subjects received 40 mg or 80 mg GSK3532795 onc

34 Plasmodium chabaudi- and Plasmodium berghei-infected mice and the 48-hour in vitro cycle of P falcip

35 egulated in the livers of Plasmodium berghei-infected mice; hepatic activin B was also upregulated at

36 of AnxA1 was investigated in L. braziliensis-infected BALB/c mice.

37 I. scapularis nymphal ticks, B. burgdorferi-infected nymphal ticks and B. mayonii-infected nymphal t

38 monstrate that CLQ treatment of Burkholderia-infected Madagascar hissing cockroaches (HCs) increases

39 sponses in a cohort of untreated HIV clade C-infected persons.

40 type analyses in a cohort of HIV-1 subtype C-infected patients (n = 168), together with site-directed

41 are higher than the virus burdens in HAdV-C5-infected ones because more of the permissive hepatocytes

42 The virus burdens in the livers of HAdV-C6-infected hamsters are higher than the virus burdens in H

43 tantly, exogenous superoxide addition to CB3-infected NOD.Ncf1(m1J) bone marrow-derived macrophages r

44 tected in supernatants from ICOVIR-15K-cBiTE-infected cells, and the secreted BiTEs bound specificall

45 id wasting to extend survival of V. cholerae-infected flies.

46 28S rRNA and microscopically counted chytrid-infected diatoms.

47 c varicosities in the cortical neurons of CM-infected brain.

48 es in the blood and the brains of murine CMV-infected mice.

49 HIV/CMV co-infected persons despite prolonged viral suppression oft

50 le and SVR12 rates of 96.7% among HIV/HCV co-infected patients participating in an Italian compassion

51 Patients co-infected with hepatitis C virus (HCV) and human immunode

52 ctive than a cathepsin inhibitor in SARS-CoV-infected mice.

53 superfamily homolog in Drosophila, Coxiella-infected flies exhibit reduced mortality from infection.

54 and potency of NAb responses in 98 CRF07_BC-infected individuals using a large, multi-subtype panel

55 al-3 with N-acetyl-d-lactosamine in T. cruzi-infected mice led to a significant reduction of cardiac

56 In CVB3-infected RAW macrophages, both alarmins increased MIP-2

57 d content from chronic wasting disease (CWD)-infected white-tailed deer brain homogenates and found t

58 gic and rheologic alterations in P cynomolgi-infected human reticulocytes that are strikingly similar

59 xodes ticks, mice fed upon by the DeltacheY2-infected ticks did not develop a persistent infection in

60 (TSC2) inhibits lipophagy induction in DENV-infected cells and decreases viral replication.

61 lipophagy, but not basal autophagy, in DENV-infected cells.

62 Together, the results for the L. donovani-infected livers of chemokine-deficient mice (CXCR6(-/-)

63 3 (IRF3) and NF-kappaB was observed in EBOV-infected, but not in RESTV-infected, MDMs.

64 ls have been reported to respond against EBV-infected B cells in the lytic cycle and to control the v

65 , LMP1 blocks IRF5-mediated apoptosis in EBV-infected cells.

66 promotes efficient lytic reactivation in EBV-infected epithelial cells by enhancing expression of the

67 Thus, in EBV-infected epithelial cells, LMP1 expression is promoted b

68 In P. falciparum-infected patients, Vgamma9Vdelta2 T cells presented incr

69 ctor mechanism against Plasmodium falciparum-infected erythrocytes (IE); however, current phagocytosi

70 tage determination for Plasmodium falciparum-infected red blood cells (Pf-iRBCs).

71 ulation of spleen cells from R. typhi(GFPuv)-infected BALB/c mice elicits gamma interferon (IFN-gamma

72 In contrast, GoGV-infected boa constrictors remained free of clinical sign

73 ed substitutions identified in NS3 from GT1a-infected patients who failed therapy with grazoprevir (i

74 826-like sequences can be identified in H7N9-infected patients, healthy adults, and newborn babies.

75 al swab (NPS) samples from all acutely HBoV1-infected children and from controls with nonacute infect

76 Mice with high levels of viremia had HBV-infected liver progenitor cells.

77 Significantly, BI-2536 administration to HBV-infected humanized liver FRG mice strongly inhibited HBV

78 Instead, we found that in HCMV-infected cells glucose carbon can be used for lipid synt

79 that expanded when a hepatitis C virus (HCV)-infected HLA-A2(-) individual received an HLA-A2(+) live

80 HCV-infected patients receiving cancer treatment at our inst

81 antly among IgM(+) memory B cells of all HCV-infected patients analyzed.

82 ir expression in cell culture models and HCV-infected human livers.

83 as an intervention to identify HIV- and HCV-infected PWID and MSM who are unaware of their status an

84 for hepatic stellate-cell activation by HCV-infected hepatocytes are underexplored.

85 iR-19a carried through the exosomes from HCV-infected hepatocytes activates HSC by modulating the SOC

86 ding protein 1 (ID1) were upregulated in HCV-infected cells or viral core gene-transfected cells.

87 ly reversible epigenetic field defect in HCV-infected liver.

88 pigenetic and transcriptional changes in HCV-infected livers in comparison with control, uninfected l

89 o analyse the effect of this strategy in HCV-infected people who inject drugs in a US city.

90 dication rates are pushing 100% for many HCV-infected populations, including patients with HIV/HCV co

91 ripheral blood B cells of 30 MC-negative HCV-infected patients and 15 healthy controls revealed that

92 ESRD than those who were nonchronically HCV-infected (HR, 2.11, 95% CI 1.16-3.86, and HR, 3.06, 95%

93 1.83-7.07) compared with nonchronically HCV-infected subjects.

94 Short exposure of HCV-infected cells to daclatasvir reduced viral assembly and

95 ducing enhanced cytolytic elimination of HCV-infected Huh7.5A2 cells.

96 dictors for viral decline in a cohort of HCV-infected postpartum women.

97 protective effect of PD-L1 expression on HCV-infected hepatoma cells against HCV-specific CD8 T cells

98 fects was blunted by PD-L1 expression on HCV-infected Huh7.5A2 cells, resulting in the improved viabi

99 We find the HCV-infected liver to have a pattern of acquisition of DNA m

100 We further observed that these HCV-infected hepatocytes express transforming growth factor

101 additional cost of identifying 1 unaware HCV-infected PWID was US$13 (site range: US$7-US$140).

102 Here we used HCV-infected cells and liver biopsies to study how HCV modul

103 events in human immunodeficiency virus (HIV)-infected HCV-seropositive and incidence density-matched

104 Human immunodeficiency virus (HIV)-infected individuals are at increased risk of chronic ki

105 l tracked human immunodeficiency virus (HIV)-infected individuals for 10 years following ART initiati

106 rapid in human immunodeficiency virus (HIV)-infected infants.

107 ancers in human immunodeficiency virus (HIV)-infected patients in Zambia.

108 E), among human immunodeficiency virus (HIV)-infected patients with and without hepatitis B virus (HB

109 Selected human immunodeficiency virus (HIV)-infected patients with end organ failure can safely rece

110 ortion of human immunodeficiency virus (HIV)-infected patients with specific comorbidities receiving

111 rt of 961 human immunodeficiency virus (HIV)-infected people who inject drugs in Vancouver, Canada, b

112 ly active human immunodeficiency virus (HIV)-infected persons be tested at least annually for syphili

113 TDR among human immunodeficiency virus (HIV)-infected PWUD, and assessed its impacts on first-line AR

114 HIV-infected frail men (n = 155) were matched to nonfrail, H

115 HIV-infected individuals had a higher proportion of total at

116 HIV-infected individuals have excess congestive heart failur

117 HIV-infected older adults and a demographically-matched grou

118 HIV-infected participants enrolled in a study of cerebrospin

119 HIV-infected patients were significantly less accurate on th

120 HIV-infected persons compared with HIV-uninfected persons we

121 HIV-infected persons who achieve undetectable viral loads on

122 HIV-infected subjects had higher immune activation and micro

123 RDS identified 4,051 HIV-infected persons, of whom 2,325 (57.4%) were unaware of

124 ng condomless anal intercourse with >/=1 HIV-infected or unknown-serostatus man within 90 days.

125 riological status at diagnosis among 127 HIV-infected patients starting TB treatment.

126 ober 1, 2012) to identify and study 1431 HIV-infected KT candidates from the first point of active st

127 was determined in 370 participants (218 HIV-infected and 152 HEU participants) by means of chart rev

128 asthma was identified in 75 (34%) of 218 HIV-infected participants and 38 (25%) of 152 HEU participan

129 ctive, observational cohort study of 221 HIV-infected men undergoing male circumcision (MC) was condu

130 P) at 6 weeks and 6 months of age in 272 HIV-infected infants who either died (cases) or survived (co

131 POCT detected all 30 HIV-infected neonates (sensitivity 100%; 95% CI 88.4-100) wi

132 eriority trial, we randomly assigned 440 HIV-infected adults who had talaromycosis, confirmed by eith

133 This cohort study analyzed 455 HIV-infected and 1945 HIV-uninfected patients, all of them m

134 Among 500 HIV-infected adults receiving ART (median ART duration, 4.5

135 In this prospective study of 558 HIV-infected men enrolled in the Multicenter AIDS Cohort Stu

136 We recruited 64 HIV-infected and 107 HIV-uninfected patients.

137 We recruited 77 HIV-infected participants (37 PPI+ and 40 PPI-) and 20 HIV-u

138 warranted to screen all sexually active HIV-infected adults for syphilis, chlamydia, and gonorrhea.

139 arch databases were used to select adult HIV-infected patients from each payer.

140 of universal AC screening offered to all HIV-infected women.

141 Frailty among HIV-infected men was associated with increased inflammation

142 covariates, frailty was associated among HIV-infected men with higher interleukin 6 and high-sensitiv

143 ross-sectional study was conducted among HIV-infected patients on antiretroviral therapy at 20 AIDS c

144 HCV treatment was highly effective among HIV-infected patients who received care within an integrated

145 Among HIV-infected patients with advanced immunosuppression, enhan

146 egravir (RAL) on hepatic steatosis among HIV-infected patients with nonalcoholic fatty liver disease

147 equently, particularly postpartum, among HIV-infected women after initial VS in this setting.

148 Births among HIV-infected women who started 3-drug ART regimens before th

149 infected women and 20.5% and 43.6% among HIV-infected women.

150 ion allows organ transplantation from an HIV-infected donor under exceptional medical circumstances a

151 reased chemotaxis of both uninfected and HIV-infected human monocytes, suggesting a role for sPrP(c)

152 ution with antiretroviral therapy (ART), HIV-infected individuals remain highly susceptible to tuberc

153 infants to be incorrectly identified as HIV-infected.

154 safety of organ transplantation between HIV-infected persons prompted a change in US American law to

155 For decades, HIV-infected patients were excluded from consideration for k

156 ipants from a TB-endemic setting, either HIV-infected or uninfected and with latent or active TB (aTB

157 Zimbabwe, Malawi, and Kenya, we enrolled HIV-infected adults and children 5 years of age or older who

158 ing virologically suppressed NVP-exposed HIV-infected children >/=3 years of age from LPV/r to EFV ar

159 onfirms the utility of the UAD assay for HIV-infected adults comparing favorably with other diagnosti

160 y single-tablet regimen is available for HIV-infected children under 12 years.

161 Evidence suggests that for HIV-infected ESRD patients, KT is associated with a signific

162 firmatory testing, LE was 26.2 years for HIV-infected infants and 61.4 years for all HIV-exposed infa

163 Lifetime CVD risk was 64.8% for HIV-infected males compared to 54.8% for males in the US gen

164 ized trial of behavioral weight loss for HIV-infected patients (n = 40).

165 optimal methods to predict MI risks for HIV-infected persons remain unclear.

166 nonuclear cells in six samples from four HIV-infected donors (one with viremia and not on ART and thr

167 peripheral blood mononuclear cells from HIV-infected ART-treated individuals in response to M. tuber

168 d to increase utilization of organs from HIV-infected donors for transplantation in Canada.

169 al blood mononuclear cells isolated from HIV-infected individuals with suppressed infection.

170 ately 1 in 10(6) cells and in cells from HIV-infected patients on suppressive antiretroviral therapy

171 Peripheral blood mononuclear cells from HIV-infected women on antiretroviral therapy who were HSV-2

172 t Treatment Panel III recommendations in HIV-infected adults and evaluated associations with carotid

173 M. tuberculosis-specific CD4 T cells in HIV-infected and HIV-uninfected adults with LTBI.

174 We tested this approach in HIV-infected cells grown in the lab and in animal infections

175 roach performed by our novel compound in HIV-infected cells provides a means to bridge the gap betwee

176 ions, but its role on CD4 T cells and in HIV-infected children is poorly understood.

177 py (ART) suppresses viral replication in HIV-infected individuals but does not eliminate the reservoi

178 tes to loss of immune control of LTBI in HIV-infected individuals, although the precise mechanisms wh

179 fic CD4 T cells was markedly impaired in HIV-infected individuals, compared with HIV-uninfected indiv

180 M. tuberculosis-specific CD4 T cells in HIV-infected individuals.

181 high rates of cardiovascular disease in HIV-infected individuals.

182 -restricted cellular immune responses in HIV-infected individuals.

183 a use is a risk factor for CV disease in HIV-infected men ages 40-60, independent of tobacco smoking

184 In HIV-infected patients receiving ART, chronic co-infection wi

185 ecommendation of a single dose of BPG in HIV-infected patients with early syphilis.

186 iated with increased incidence of NHL in HIV-infected patients.

187 xacerbated neurocognitive dysfunction in HIV-infected patients.

188 eumonia cases were due to pneumococci in HIV-infected South African adults.

189 As a result, most HIV-infected adults in the United States are >50 years of ag

190 In a cohort of 53 antiretroviral-naive HIV-infected subjects, the measure of thymic volume, as a re

191 men (n = 155) were matched to nonfrail, HIV-infected (n = 141) and HIV-uninfected (n = 150) men by a

192 massive apoptosis, a small population of HIV-infected cells survive infection, silence viral replicat

193 time in part by expansion of the pool of HIV-infected cells.

194 estored CD4 expression to the surface of HIV-infected cells.

195 Expansion of HIV-infected CMV/EBV-specific CD4 + T cells may contribute t

196 (Tim-3)-expressing pDCs in the blood of HIV-infected donors.

197 at could compromise the health status of HIV-infected individuals might not be ethically warranted.

198 Serial specimens from a trial of HIV-infected individuals receiving antiretroviral treatment

199 A subset of HIV-infected individuals termed elite controllers (ECs) main

200 at Cx43 is dysregulated in the hearts of HIV-infected individuals.

201 an increased risk to female partners of HIV-infected men resuming sex early after male circumcision.

202 ecognized as an important comorbidity of HIV-infected patients, however, the exact molecular mechanis

203 Millions of HIV-infected people worldwide receive antiretroviral therapy

204 rsons per day, and the detection rate of HIV-infected persons with viremia (regardless of their aware

205 g organ allocation practices, optimizing HIV-infected donor and recipient selection, managing donor-d

206 Among perinatally HIV-infected children born in 2002-2013, 836 (63.0%) of the

207 against native antigens on persistently HIV-infected cells and recombinant antigens on Env-transfect

208 Compared with HIV-uninfected persons, HIV-infected individuals were slightly younger (mean age, 52

209 wn to be safe and effective for selected HIV-infected patients with hematological malignancies.

210 l, we enrolled virologically suppressed, HIV-infected children from five hospital clinics in Uganda,

211 tor functions that specifically targeted HIV-infected cells.

212 in a longitudinal cohort of ART-treated HIV-infected South Africans.

213 Lymph node cells from untreated HIV-infected individuals revealed that TFR cells harbored th

214 ar year, and antiretroviral therapy use (HIV-infected men only).

215 Using HIV-infected humanized mice, we demonstrated that in vivo bl

216 ly upregulated in human papillomavirus (HPV)-infected cell lines and tissues.

217 xceptionally large collection of 5,570 HPV16-infected case-control samples to determine whether viral

218 was up-regulated in the brains of influenza-infected mice and was elevated in cerebrospinal fluid of

219 144 with severe influenza in three influenza-infected cohorts characterized by different levels of in

220 DAT and rescued antibiotic-treated influenza-infected mice.

221 JHMV-infected CD19(-/-) mice were thus used to determine how

222 Thus the increase of miRNP stability in Ld-infected cells curtails production of proinflammatory cy

223 thering, also shows reduced expression in Ld-infected cells.

224 ted levels of Ago2-bound cytokine mRNA in Ld-infected macrophages.

225 ory lesions formed in the ears of Leishmania-infected C57BL/6 and Tlr3/7/9(-/-) mice, indicating that

226 AVV and treated with NP siRNA-LNP, with MARV-infected animals beginning treatment four or five days a

227 orferi-infected nymphal ticks and B. mayonii-infected nymphal ticks by measuring metabolism every 24

228 Importantly, in mosquito-infected animals ZIKV tissue distribution was limited to

229 ESAT-6, IL-6 and phosphorylated-STAT3 in Mtb-infected mouse lungs.

230 Finally, selective killing of Mtb-infected macrophages and subsequent bacterial release en

231 ion, resulting in increased apoptosis of Mtb-infected macrophages.

232 delayed clearance, rechallenged C. muridarum-infected mice were highly immune.

233 (CRISPR)-enzymatically inactive Cas9 in MVM-infected cells increased both cyclin B1 protein and RNA

234 not previously been applied in mycobacteria-infected animals.

235 urvival of Nippostrongylus brasiliensis (Nb)-infected hRETNTg(+) mice after a fatal LPS dose compared

236 atal LPS dose compared with naive mice or Nb-infected hRETNTg(-) mice.

237 rentially expressed between infected and non-infected birds.

238 ng inflammation only in the neighboring, non-infected epithelium.

239 sed on the gene expression levels in the non-infected cells, and demonstrated reasonable performance

240 Pathogen-infected &CMFDA-labeled MoDCs long-lasting survival was

241 dium falciparum The percentage of Plasmodium-infected (iRBCs) with bound platelets during the ascendi

242 espiratory status of mitochondria from prion-infected animals is unknown.

243 how that a prophylactic inoculation of prion-infected animals with an anti-prion delays the onset of

244 ypical features of immune responses in PRRSV-infected pigs are delayed onset and low levels of virus

245 ng this tool for Yersinia pseudotuberculosis-infected lymphatic tissues, we revealed numerous alterat

246 family cytokines was performed in H. pylori-infected and uninfected gastric biopsy specimens.

247 egulated approximately 4.5-fold in H. pylori-infected gastric biopsy specimens.

248 and loss of cell junctions seen in H. pylori-infected host cells.

249 Treatment of CrPV(R146A)-infected cells with actinomycin D, which represses trans

250 t four or five days after infection and RAVV-infected animals starting treatment three or six days af

251 acterize the immune profiles of rDEN2Delta30-infected subjects and to compare the profiles with those

252 that the lack of immune activation in RESTV-infected MDMs contributes to lower pathogenicity by prev

253 observed in EBOV-infected, but not in RESTV-infected, MDMs.

254 Rickettsia spp. in 19 ticks (8%); Rickettsia-infected ticks contained R. rhipicephali (16 of 250, 6.4

255 Within the gut, Salmonella-infected enterocytes are expelled into the lumen, limiti

256 om heterozygous (ARQ/VRQ or ARH/ARQ) scrapie-infected Rasa Aragonesa sheep was analyzed using this MR

257 on to PrP(res) when transfected into scrapie-infected N2a neuroblastoma cells, likely due to segregat

258 und in the spleen and liver samples of SFTSV-infected hamsters.

259 umulate in a long-lasting manner in Shigella-infected cells, causing subsequent formation of covalent

260 Finally, ARVs administered to mice and SIV-infected macaques resulted in neuronal damage and BACE1

261 IV neutralizing antibodies, or sera from SIV-infected macaques.

262 oss-species recognition of human and MCM SIV-infected CD4(+) T cells.

263 Here, using ART-treated, SIV-infected rhesus macaques, we show that CTLA-4(+)PD-1(-)

264 ls were depleted from blood in the SIVmac239-infected animals.

265 nriched leukocyte populations from SIVmac251-infected macaques with or without CD8(+) lymphocyte depl

266 iler's murine encephalomyelitis virus (TMEV)-infected transgenic FVB mice that express the D(b) class

267 maintaining the viability of C. trachomatis-infected primary human cells.

268 M. tuberculosis-infected IL-21R KO mice had enhanced bacterial burden an

269 cells in lungs compared with M. tuberculosis-infected WT mice.

270 Mycobacterium tuberculosis-infected macrophages and dendritic cells are limited in

271 cell populations were decreased in vDeltaK1L-infected ears.

272 Virus-infected bees had greater expression of genes involved i

273 To counter premature death of a virus-infected cell, poxviruses use a range of different molec

274 (representative of Italian hepatitis C virus-infected patients in care).

275 e considered in genotype 1 hepatitis C virus-infected patients who are treatment-naive, do not have c

276 s in a real-life cohort of hepatitis C virus-infected policy 1, "universal," treat all patients, rega

277 ne levels in supernatant obtained from virus-infected fetal brain cells were measured simultaneously

278 High titers of ADCC-Abs against H7N9 virus-infected cells were detected in sera from adults and chi

279 Human immunodeficiency virus-infected individuals on cART (predominantly containing t

280 onducted in 302 human immunodeficiency virus-infected patients who had a CD4 T-cell count <100 cells/

281 that creates a cellular environment in virus-infected cells that permits productive virus infection.

282 at the difficulty of detecting RNAi in virus-infected mammalian cells reflects the expression of high

283 zed and challenged mice into influenza virus-infected mice resulted in reduced morbidity and viral bu

284 ty is essential for the elimination of virus-infected and cancerous cells by NK cells.

285 fined by nonspecific innate killing of virus-infected and tumor cells.

286 ral therapy, a persistent reservoir of virus-infected cells remains.

287 protein was detectable only in Sendai virus-infected PHHs from individuals with the dG allele, where

288 cell survival mechanisms is unique to virus-infected cells and relies on regulation of MCL-1 mitocho

289 People's Democratic Republic in O. viverrini-infected adults.

290 vation of CNS macrophages (microglia) in WNV-infected SCSC while inhibiting the expression of genes a

291 ilitates WNV neuroinvasion by recruiting WNV-infected PMNs into the brain.

292 nfection is significantly lower in Wolbachia-infected females.

293 rearing infrastructure specific to Wolbachia-infected mosquitoes.

294 In WT-infected mice, infiltrates in gastric tissues were predo

295 hether repellents are effective against ZIKV-infected mosquitoes, in part because of the ethical conc

296 to inflammatory mediators derived from ZIKV-infected macrophages led to the degradation of the ZO-1

297 d or uninfected cells could be found in ZIKV-infected brain tissues.

298 lowing clearance of peripheral virus in ZIKV-infected individuals.

299 evaluated the CD8(+) T cell response in ZIKV-infected LysMCre(+)IFNAR(fl/fl) C57BL/6 (H-2(b)) mice la

300 hloroquine (CQ) extends the lifespan of ZIKV-infected interferon signalling-deficient AG129 mice.