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1 was Prox1-positive, CD31-positive and LYVE-1-negative, bearing a different molecular signature from b
2  May and December, 2015, compared with mcr-1-negative isolates from rectal swabs of inpatients (colon
3 hese data, ACA11 overexpression in a t(4;14)-negative MM cell line, MM1.S, demonstrated enhanced reac
4 -1R, externally induced DNA damage in IGF-1R-negative cells caused G1 cell cycle arrest and S phase f
5 isk human epidermal growth factor receptor 2-negative breast cancer compared with the TC6 regimen.
6 145 human epidermal growth factor receptor 2-negative patients younger than 35 years in TEXT, 5-year
7 OG-ab NMOSD and 87.5% in the heterogenous ab-negative NMOSD cohort.
8  (Cohen d; Abeta-positive impaired vs. Abeta-negative normal) were evaluated in another phase 2 study
9 he adjacent matrix enriched with the ALDH1A1-negative axons.
10  risk TFs associated with ESR1-positive and -negative breast cancer can physically interact.
11 here mRNA from PGE2-G response-positive and -negative cell lines was subjected to transcriptome-wide
12 criminating between the cancer-positive and -negative cells, without any amplification step, in less
13 ilar 5-year EFS and OS for MRD-positive and -negative patients using an MRD threshold of 0.01%.
14 e growth of multiple AR-positive, but not AR-negative, prostate cancer cells.
15 hal in argininosuccinate synthetase 1 (ASS1)-negative cancers, including mesothelioma.
16 ist in finding a diagnosis for their autopsy-negative child SUD cases.
17 Five of 8 C4d-positive recipients turned C4d-negative in 5-week follow-up biopsies, while another 2 r
18  compared to cells infected with either cagA-negative or cag pathogenicity island (PAI) mutant.
19       Thus, calbindin-positive and calbindin-negative SNc neurons differ substantially in their calci
20                 During inhibition, calbindin-negative neurons exhibit increased sensitivity to excita
21  increase excitation efficacy onto calbindin-negative cells during dopamine inhibition, suggesting th
22  divided into two populations: the calbindin-negative ventral tier, which is vulnerable to neurodegen
23 roduced by 243 isolates and 51 carbapenemase-negative isolates included porin mutants and producers o
24  Due to the highly challenging carbapenemase-negative isolates, specificities were lower than typical
25 ocytic leukaemia (CLL)-phenotype MBL and CD5-negative MBL, as well as differences in absolute monoclo
26 mal abnormalities in Philadelphia chromosome-negative (CCA/Ph(-)) metaphases emerge as patients with
27 icant differences were found between B2A-CIC-negative and control group patients.
28                          If they are B2A-CIC-negative patients, they have the same risk as the contro
29 er serology, Bartonella serology) in culture-negative cases.
30   Defining the microbial etiology of culture-negative prosthetic joint infection (PJI) can be challen
31 lar-weight isoform of cyclin E (cytoplasmic)-negative) is a reliable prognostic biomarker in ER posit
32 ngs do not support the classification of DIF-negative patients, meeting the clinical criteria for ocu
33 all intent-to-treat minimal residual disease-negative (MRD(-)) remission rate for this phase 1 study
34                                   A dominant-negative (dn) atypical PKM selectively reversed associat
35 in the gene KCNA2, causing either a dominant-negative loss-of-function or a gain-of-function of the v
36 d that the mutated kinase acts in a dominant-negative manner to reduce CaMKIIalpha-WT autophosphoryla
37        Transgenic mice expressing a dominant-negative Orai1 mutant (E108Q) increases albuminuria, and
38 monolayers led to the proposal of a dominant-negative trafficking mechanism to explain AE1-associated
39 e, we show that overexpression of a dominant-negative version of DA1 enhances leaf size in a broad ra
40  such effects (gain of function and dominant-negative activity) in lung adenocarcinoma are unclear.
41 e of both pathways by Atg5(-/-) and dominant-negative rab5, ER cholesterol fails to increase when mTO
42                       Wild-Type and dominant-negative-DISC1 (DN-DISC1) mice were injected with THC (1
43                   Rather than being dominant-negative regulators, Tcf1 short isoforms are adequate in
44  OMPs that assemble slowly can form dominant-negative interactions with the Bam complex.
45  palmitoylated by GODZ using a GODZ dominant-negative plasmid.
46     We found that overexpression of dominant-negative (DN) forms of NSF or knockdown of the expressio
47 esized that selective expression of dominant-negative C-terminus-truncated human DISC1 (mutant DISC1)
48 tivity, whereas the introduction of dominant-negative NRF-1 repressed such activity.
49 ion and, contrary to their proposed dominant-negative role, did not interfere with the expression of
50                  Here, we show that dominant-negative mutants of ATL1 in PC-12 cells inhibit nerve gr
51 ignaling by RNAi, expression of the dominant-negative Rac1 (Rac1 DN), or the specific Rac1 inhibitor
52 us transcription factors, including a double-negative feedback loop between the microRNA-200 (miR-200
53 h CAL-101 [idelalisib]), interrupts a double-negative feedback loop, enhancing GC-regulated transcrip
54 status (e.g., EGFR-positive [EGFR+] vs. EGFR-negative) was assessed using the Wilcoxon rank-sum test.
55                                      In ePET-negative patients, 5-year PFS rates in the F group were
56                                      In ePET-negative patients, noninferiority of ABVD only could not
57          We conducted a GWAS using 21,468 ER-negative cases and 100,594 controls combined with 18,908
58  10 of 11 variants previously reported in ER-negative disease or BRCA1 mutation carrier GWAS and obse
59 and observed consistent associations with ER-negative disease for 105 susceptibility variants identif
60 anti-hormone therapeutic efficacy in ERalpha-negative breast cancer.
61 tly inhibited breast tumor growth in ERalpha-negative mouse xenografts, especially when combined with
62 p are highly effective in inhibiting ERalpha-negative breast cancer due at least in part to epigeneti
63                                        False-negative results were found in 2 patients and occurred a
64  (PPV3), 30.4% (95% CI: 29.9%, 30.9%); false-negative rate, 4.8 per 1000 (95% CI: 4.6, 5.0); sensitiv
65 ive, true-negative, false-positive and false-negative patients as classified against any reference st
66 true- and false-positive and true- and false-negative results were extracted to fit a cross-tabulatio
67 ems associated with false-positive and false-negative results, inconsistencies and low reactivity of
68 rasitemia, and PfHRP-II tests can give false-negative results when P. falciparum strains do not expre
69  Seven of the 1982 patients (0.4%) had false-negative results with the staged algorithm.
70  higher analytic sensitivity and lower false-negative rate of HTS improves upon FC for MRD detection
71 test; Cepheid, Sunnyvale, CA) found no false-negative and 4 false-positive cobas Cdiff test results.
72 opriate antimicrobial therapy, with no false-negative cases.
73                          There were no false-negative ratings.
74 s significantly lower expressed in PET false-negative cases (5.3-fold change, P < .001) which provide
75 levant number of patients with FDG-PET false-negative MM and a strong association between hexokinase-
76               However, the risk of PET false-negative results in the presence of carbidopa is a conce
77 cigarette use), indicating substantial false-negative rates.
78  test reliability, as indicated by the false-negative (FN), false-positive (FP), and fixation loss (F
79 ith human and porcine fVIII leading to false-negative HSM results.
80 mber of antimalarial treatments due to false-negative RDT results.
81 erformance and factors contributing to false-negative results in longitudinal studies, we examined re
82 ) version 2 score to better understand false-negative lesion characteristics.
83 owever, pfhrp2 gene deletions yielding false-negative RDTs, first reported in South America in 2010,
84  third of metastases < 1.0 cm were (18)F-FET-negative, most likely because of scanner resolution and
85 g cranial computed tomography-positive from -negative cases (AUC = 0.921, 0.923, and 0.646, respectiv
86 mice represent a novel mouse model of fusion-negative RMS.
87                                Among the GCA-negative group, herpes zoster antigen was not detected i
88       METHODS AND A total of 160 AC genotype-negative probands for 5 AC desmosomal genes by conventio
89 cadherin 2 (CDH2) gene in unrelated genotype-negative patients with ARVC.
90                                         Gram-negative bacteria remodel their surfaces to interact wit
91                                         Gram-negative bacteria such as Escherichia coli are protected
92             Pseudomonas aeruginosa is a Gram-negative bacterial pathogen associated with acute and ch
93 regatibacter actinomycetemcomitans is a Gram-negative commensal bacterium of the oral cavity which ha
94 al activity, including activity against Gram-negative pathogens.
95  aureus and Streptococcus pyogenes) and gram-negative bacteria (Pseudomonas aeruginosa and Escherichi
96 ty against a panel of Gram-positive and Gram-negative bacteria revealed structure-activity relationsh
97 lin is active against Gram-positive and Gram-negative bacteria using a microdilution assay.
98 DR pathogens, such as malaria, HIV, and Gram-negative bacteria.
99 mensal and pathogenic Gram-positive and Gram-negative bacteria.
100  into the backbone of Gram-positive and Gram-negative bacterial PG utilizing metabolic cell wall recy
101 gram-positive Staphylococcus aureus and gram-negative Pseudomonas aeruginosa (99.3 +/- 1.9% and 88.5
102  Burkholderia cepacia complex (Bcc) are Gram-negative opportunisitic bacteria that are capable of cau
103 dal protein that limits contact between Gram-negative bacteria and the colonic epithelial surface.
104  four bacterial strains, including both Gram-negative and Gram-positive bacteria, showing great poten
105 ell-cell interactions between competing Gram-negative bacteria.
106 also relevant for other T3SS-containing Gram-negative bacteria.
107 tal of 210 Bactec bottles demonstrating Gram-negative bacilli were prospectively enrolled for this st
108  understand morphology in the dimorphic Gram-negative bacterium Caulobacter crescentus.
109 e zauPzapA operon is present in diverse Gram-negative bacteria, indicating a common mechanism for Z-r
110  in the extracellular matrix of enteric Gram-negative bacteria.
111 ultidrug resistant bacteria, especially Gram-negative bacteria for which the situation is particularl
112 and showed that HFM increases rat fecal Gram-negative bacteria, elevates lipopolysaccharides (LPS), a
113 exes constitute a primary mechanism for Gram-negative bacteria to expel toxic molecules for survival.
114 e, we chose the pilus protein FimG from Gram-negative bacteria and a disulfide-bonded variant of the
115        Mechanisms of drug resistance in gram-negative bacteria (GNB) are numerous; beta-lactamase gen
116 otein H-NS is a key global regulator in Gram-negative bacteria and is believed to be a crucial player
117                                      In Gram-negative bacteria, efflux pumps are able to prevent effe
118 velopment of resistance particularly in Gram-negative bacteria, illustrates the urgent need for new m
119                                      In Gram-negative bacteria, some of these pumps form multi-protei
120 arrel outer membrane proteins (OMPs) in Gram-negative bacteria.
121 e of Type Vd secreted phospholipases in Gram-negative bacteria.
122 timicrobial resistance, particularly in Gram-negative hospital pathogens, which has led to renewed ef
123 ella spp. are facultative intracellular Gram-negative bacteria that cause the zoonotic disease brucel
124 e and human RELMbeta selectively killed Gram-negative bacteria by forming size-selective pores that p
125 fective permeability barrier that makes Gram-negative bacteria inherently resistant to many antibioti
126  of novel therapeutic options to manage Gram-negative infections.
127 sociated with secretion systems in many Gram-negative and Gram-positive bacteria.
128 tilization of enterobactin permits many Gram-negative bacteria to thrive in environments where low so
129                                    Many Gram-negative bacteria use type 2 secretion systems (T2SSs) t
130                                 In many Gram-negative bacteria, including Rhodobacter capsulatus, cyt
131 nalool-IC-NFs inhibited growth of model Gram-negative (E. coli) and Gram-positive (S. aureus) bacteri
132 posed of 15 conserved proteins in model gram-negative species.
133 scriminate between viable and nonviable Gram-negative bacteria to tune the immune response, thereby l
134 rthfield, IL) for the identification of Gram-negative bacilli.
135  rapidly traverse the outer membrane of Gram-negative bacteria and accumulate inside these cells, mak
136                    Although a number of Gram-negative bacteria are known to catabolize quinate and sh
137                    The cell envelope of gram-negative bacteria, a structure comprising an outer (OM)
138 e gene exchange between five species of Gram-negative bacteria, and that the identity of the genetic
139 ad antibiotic resistance, especially of Gram-negative bacteria, has become a severe concern for human
140 r detecting lipopolysaccharide (LPS) of Gram-negative bacteria, was immobilized on both a large area
141 xcellent sensitivity to trace levels of Gram-negative bacteria, while remaining insensitive to both G
142  the space between the two membranes of Gram-negative bacteria.
143 roteins into the outer membrane (OM) of Gram-negative bacteria.
144              The outer membrane (OM) of Gram-negative is a unique lipid bilayer containing LPS in its
145 aratus functions in the injectisomes of gram-negative pathogens to export virulence factors into host
146 olysaccharide, a cell wall component of Gram-negative Proteobacteria and known inducer of lupus in mi
147 he difference in mechanism of action on gram-negative and gram-positive bacteria may be less pronounc
148  Antimicrobial resistance in pathogenic gram-negative bacteria is one of the most pressing challenges
149                         Many pathogenic Gram-negative bacteria use the type III secretion system (T3S
150  Pseudomonas aeruginosa is a pathogenic gram-negative organism that has the ability to cause blinding
151 ibility profiles of clinically relevant Gram-negative bacteria within two hours of antibiotic introdu
152 r the prevention of multidrug-resistant gram-negative bacteria (MDR-GNB) in adult intensive care unit
153 Bcc) are a group of multidrug-resistant gram-negative bacteria rarely reported in patients without cy
154               Colonization by resistant gram-negative bacteria was significantly associated with an i
155 associated with antimicrobial-resistant Gram-negative pathogens.
156 and export of amyloid protein sequences.Gram-negative bacteria assemble biofilms from amyloid fibres,
157 evelopment for the treatment of serious Gram-negative infections.
158                        Twenty-one small Gram-negative motile coccobacilli were isolated from 15 syste
159 ns are widespread in toxins that target Gram-negative bacteria.
160                      The mission of the Gram-Negative Committee is to advance our knowledge of these
161  we have addressed this question in the Gram-negative model bacterium Burkholderia thailandensis E264
162 Bioinformatic screens reveal that these gram-negative bacteria carry genes coding for thiol-disulfide
163 3 ligase (NEL) domain that is unique to Gram-negative pathogens and whose activity is repressed by a
164 t Ags, and they are more susceptible to Gram-negative sepsis.
165 a low light dose (0.6 J cm(-2) ) toward Gram-negative bacteria E. coli, making it a remarkably effici
166 am-positive rods and other uncultivable Gram-negative rods, and, rarely, opportunistic microorganisms
167 I secretion (T2S) is one means by which Gram-negative pathogens secrete proteins into the extracellul
168 sing candidate to treat infections with Gram-negative bacteria.
169 nical needs in treating infections with Gram-negative bacteria.
170 n monocytes isolated from patients with Gram-negative sepsis compared with healthy control subjects.
171     We identified 85863 HCV-positive and HCV-negative men in the PSM population.
172                     Among 1391 initially HCV-negative participants followed prospectively (1644.5 per
173 an epidermal growth factor receptor 2 (HER2)-negative tumors, 82.8% of luminal B-like HER2-positive t
174 esistant, hormone receptor-positive and HER2-negative advanced breast cancer.
175 ogenic contributions of EVI1 in ER- and HER2-negative subsets of breast cancer.
176 ependent data set, 2 of 9 (22.2%) ERBB2/HER2-negative BrCa switched to ERBB2/HER2-positive with 1 BrM
177 ing AGTR1 overexpression in a subset of HER2-negative breast cancers, and they provide a mechanistic
178 HER2 gene amplification and HER2-low or HER2-negative breast cancers following radiotherapy or endocr
179 d metastatic hormone receptor-positive, HER2-negative breast cancer.
180 ly confirmed hormone-receptor-positive, HER2-negative, locally advanced or metastatic breast cancer,
181  patient has hormone receptor-positive, HER2-negative, node-positive breast cancer, the MammaPrint as
182            In hormone receptor-positive/HER2-negative (n = 318) disease, BCSS was 100% in patients wi
183  (HGF-positive, MET-positive) and MKN45 (HGF-negative, MET-positive) and 4 patient-derived xenografts
184 ebo-controlled double-blind study of 162 HIV-negative RV144 vaccine recipients, we evaluated 2 additi
185          We studied these factors in 168 HIV-negative South African adolescent females aged 16 to 22
186 or specimens from 23 HIV-positive and 17 HIV-negative patients (29 men and 11 women; mean [SD] age, 5
187                               Among 6751 HIV-negative HHCs with baseline blood samples, 192 had secon
188 0.008, -0.01 to -0.004; p=0.001) than in HIV-negative people, but the magnitudes of difference were s
189 were detected in both partners in 60% of HIV-negative couples and 96% of HIV-positive couples over 2
190 ral therapy (ART) is approaching that of HIV-negative people.
191  separate clinical studies of high-risk, HIV-negative persons conducted in Botswana and Thailand.
192 numbers of circulating CD4+ cells in the HIV-negative (HIV-) brain-dead donor (BDD) is not known.
193 overall life expectancies than did their HIV-negative counterparts in the general population [29.1 ye
194 ncerns for nephrotoxicity also extend to HIV-negative individuals using tenofovir disoproxil fumarate
195 ctions by comparing newly diagnosed with HIV-negative participants.
196                   Human papillomavirus (HPV)-negative oropharyngeal squamous cell carcinoma (OPSCC) h
197 ermined based on 8 (6 HPV-positive and 2 HPV-negative) control samples.
198 ing cT1-2 N1-2b pathologically confirmed HPV-negative OPSCC in 2010 to 2012 were identified using the
199 ted redundancy for NSD family members in HPV-negative HNSCCs and suggest a potential role for impaire
200 ing chromatin regulators is warranted in HPV-negative HNSCCs driven by aberrant H3K36 methylation.
201 me deregulation in the genesis of 13% of HPV-negative HNSCCs.
202 all survival rates than do patients with HPV-negative HNSCC, but the mechanisms underlying this pheno
203 lts with infection-positive versus infection-negative systemic inflammation.
204 racing confirmed that LDB1-depleted, insulin-negative beta cells express NEUROG3 but do not adopt alt
205 dies were performed in PD-L1-positive, PD-L1-negative, and mixed tumor-bearing severe combined immuno
206  with Hirshfeld charges shows that the least-negative Cl discriminates active from nonactive substrat
207     Furthermore, lotus tetragonolobus lectin-negative/p-Creb-positive cyst segments (re)-expressed Nc
208 2(-/-) or Rag2(-/-)gammac(-/-) These lineage-negative CD45(+)/Thy1.2(+) cells are found within the my
209 liferated normally in vitro, whereas lineage-negative progenitors displayed impaired NK-cell differen
210 herapy for AS (58% vs 22%, P < 0.01), margin-negative resection (100% vs 73%, P < 0.01), reconstructi
211 in a subset analysis of patients with margin-negative resection (HR, 0.97; 95% CI, 0.66-1.45; P = .88
212 nalysis of peripheral blood B cells of 30 MC-negative HCV-infected patients and 15 healthy controls r
213                               FDA microscopy-negative bacteria in these pretreatment samples may be a
214  and endocervical cells transfected with miR-negative control, miR-143 or miR-145 were used in cell p
215 ant patients have worse outcomes than FC MRD-negative patients.
216  Conclusion Our results demonstrate that MRD-negative status surpasses the prognostic value of CR ach
217      Twelve pre-surgical TLE patients (7 MRI-negative) and age-matched healthy volunteers were scanne
218                                     Mutation-negative patients therefore either have distinct disease
219 CLP (NME1 R18Q in an IRF6 and GRHL3 mutation-negative patient with van der Woude syndrome and NME2 G7
220          In the cohort of patients with node-negative disease, the 3-year rate of invasive-disease-fr
221                                          Non-negative Matrix Factorization (NMF) algorithms associate
222                            We proposed a non-negative matrix factorization based method to rank, so a
223 ariate resolution curve fitting known as non-negative matrix factorization with alternating least-squ
224 were classified as HER2-positive (HER2+) or -negative (HER2-) based on fluorescence in situ hybridiza
225                           We estimated panel-negative binomial models on a subset of beneficiaries to
226  1) or 3 (cohort 2) cycles of weekly BV; PET-negative patients (Deauville score </=2) proceeded to au
227 d significantly higher CT densities than PET-negative LNs, irrespective of the type of cancer.
228 g PfHRP2-only RDTs in the presence of PfHRP2-negative parasites caused an increase in prevalence, red
229 cribed in adult Philadelphia chromosome (Ph)-negative B-cell precursor (BCP) acute lymphoblastic leuk
230  Philadelphia chromosome (Ph)-positive or Ph-negative B-cell acute lymphoblastic leukaemia who were d
231 HCM after family screening, 74 FG+/phenotype-negative relatives, and 9 with noncompaction or dilated
232 ed as phenotype-positive (n=72) or phenotype-negative (n=102).
233 ce harboring PSMA-positive CWR22Rv1 and PSMA-negative PC-3 tumor xenografts.
234 aP and PC-3 (prostate cancer cell line; PSMA-negative) tumors.
235 reated with antimalarials (n = 104), (2) RDT-negative children whose untreated P. falciparum infectio
236 % of RDT-positive (n = 185) and 31.2% of RDT-negative samples (n = 340) were available and selected f
237                         In estrogen receptor-negative (ER(-)) and triple-negative breast cancer (TNBC
238 eterogeneity = .10) versus estrogen receptor-negative disease.
239 ositive cancer and 63% for estrogen receptor-negative progesterone- receptor-negative cancer.
240 re similar among women with hormone receptor-negative disease, although the HRs were substantially sm
241 ormone receptor-positive vs hormone receptor-negative), nodal status (0 vs 1-3 vs or >/=4 positive no
242 ositive disease and 36% had hormone-receptor-negative disease.
243 gen receptor-negative progesterone- receptor-negative cancer.
244 ontribute to the pathogenesis of retinopathy-negative CM.
245 ribution of malaria infection to retinopathy-negative CM.
246 evere respiratory syncytial virus/rhinovirus-negative wheezing episode (adjusted OR, 8.0; P = .001),
247 candidate gene sequencing in additional SBDS-negative SDS cases or molecularly undiagnosed IBMFS case
248 trio whole-exome sequencing (WES) in an sbds-negative SDS family and candidate gene sequencing in add
249 n D deficiency is potentially causal of sero-negative RA and provide new insights into the pathogenes
250 ciation study (vGWAS) of six cohorts of sero-negative RA recruited in Europe and the US that were gen
251      The remaining three DSM265-treated, TBS-negative participants of cohort 2 developed transient su
252 rter was stringently repressed in telomerase-negative human cells in a histone deacetylase (HDAC)-dep
253 pdm09 and 305 B(Victoria) cases and 926 test-negative controls.
254 ion strategies to identify pathogens in test-negative children with CAP and asymptomatic controls.
255                                       Triple-negative (TN) breast cancer (BC) shares histological cha
256                                       Triple-negative breast cancer (TNBC) comprises approximately 20
257                                       Triple-negative breast cancer (TNBC) is a breast cancer subtype
258                                       Triple-negative breast cancer (TNBC) is an aggressive disease l
259                                       Triple-negative breast cancer (TNBC) patients commonly exhibit
260                                       Triple-negative breast cancer is viewed clinically as an aggres
261                                       Triple-negative breast cancer, compared with non-TNBC, likely a
262                                       Triple-negative breast cancers (TNBC) are highly aggressive, la
263                                       Triple-negative breast cancers (TNBCs) are more common among Af
264 strogen receptor-negative (ER(-)) and triple-negative breast cancer (TNBC), nitric oxide synthase-2 (
265 like tumors (24% v 8%; P < .001), and triple-negative tumors (21% v 6%; P = .08), but not for human e
266 owed a significant difference between triple-negative breast cancer and non-triple-negative breast ca
267                    New treatments for triple-negative breast cancer (TNBC) are urgently needed.
268 s supporting AKT-targeted therapy for triple-negative breast cancer.
269 luding regression in PDX samples from triple-negative breast cancer.
270 he effects of elevated LDL-C in human triple-negative (MDA-MB-231) and mouse Her2/Neu-overexpressing
271  in a mouse orthotopic model of human triple-negative breast cancer.
272 een reported to be a driving force in triple-negative breast cancer (TNBC), contributing to the maint
273 tially lower level than Akt3/+S472 in triple-negative breast cancer cells, specific ablation of Akt3/
274  of tumor-infiltrating lymphocytes in triple-negative breast cancers and while MEK inhibition can pro
275  yields potent synthetic lethality in triple-negative breast tumors and other aggressive tumors chara
276                      We show that, in triple-negative-breast cancer (TNBC) cells enriched with TICs,
277 by many aggressive cancers, including triple-negative breast cancers (TNBC), to utilize glutamine for
278 le stratification of mesenchymal-like triple-negative breast cancer (TNBC) cells that differ from one
279 JNK is highly activated in basal-like triple-negative breast cancer (TNBC).
280     Purpose Trop-2, expressed in most triple-negative breast cancers (TNBCs), may be a potential targ
281                         In fact, most triple-negative breast cancers are poor-prognosis tumors with a
282 triple-negative breast cancer and non-triple-negative breast cancer (P = .009, .003, and .001, respec
283  involvement of ECs in the process of triple-negative breast cancer (TNBC) metastasis has not been ad
284 wledge related to the pathogenesis of triple-negative breast cancer.
285 iew, we describe the heterogeneity of triple-negative disease, focusing on the histologic and molecul
286  of HER2-enriched tumors, and 7.8% of triple-negative tumors.
287 in for patients with HER2-positive or triple-negative subtypes and extracranial metastases.
288 er patients, those diagnosed with the triple-negative breast cancer (TNBC) subtype have the worst pro
289                           Among them, triple-negative breast cancer MDA-MB-231, which has the highest
290    Results Eighteen tumors (22%) were triple-negative breast cancers.
291  to be obese and to be diagnosed with triple-negative breast cancer (TNBC).
292 A signaling is highly correlated with triple-negative breast cancer (TNBC).
293 d/allowed calculation of true-positive, true-negative, false-positive and false-negative patients as
294 nd TBR in positive lobes compared with tumor-negative lobes.
295 wn tissue was composed of normal-sized, UCP1-negative unilocular adipocytes, with mitochondrial netwo
296 ting mutations in SMIM1 defines the rare Vel-negative phenotype.
297 immunodeficiency virus and hepatitis D virus-negative patients with pretransplant HBV DNA undetectabl
298  are not driven by MCPyV and that such virus-negative MCCs, which can be quite reliably identified by
299 2 (19%) MCC tumors in this cohort were virus-negative using this multimodal system.
300 state (differentiation) happened via an XIST-negative XaXa intermediate.

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