ライフサイエンスコーパス: -precipitated withdrawal

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1 splay both abstinence-induced and antagonist-precipitated withdrawal signs, indicative of the develop

2 avior during both spontaneous and antagonist-precipitated withdrawal was measured by potentiation of

3 regions with chronic morphine and antagonist-precipitated withdrawal.

4 of amino acids before and during antagonist-precipitated withdrawal in unanesthetized rats was exami

5 utonomic manifestations of opioid antagonist-precipitated withdrawal in morphine-tolerant rats.

6 ce, as indicated by a less severe antagonist-precipitated withdrawal syndrome.

7 us (LC) during nor-binaltorphimine (nor-BNI)-precipitated withdrawal from butorphanol, (5alpha,7alpha

8 ar rapid blocking of naloxone (10 microg/kg)-precipitated withdrawal hyperalgesia and unmasking of pr

9 noted after nor-BNI (48 nmol/50 microl, LC)-precipitated withdrawal in rats that had been intracereb

10 during acute morphine exposure and naloxone-precipitated withdrawal in two regions associated with t

11 Chronic morphine exposure and naloxone-precipitated withdrawal increase activity of spinal MMP-

12 ly promotes analgesic tolerance and naloxone-precipitated withdrawal, whereas downregulation of G9a i

13 o induce dependence, as revealed by naloxone-precipitated withdrawal in saline or mismatch-pretreated

14 morphine dependence as assessed by naloxone-precipitated withdrawal jumping.

15 ependence liability as indicated by naloxone-precipitated withdrawal studies.

16 orphine dependence (i.e., decreased naloxone-precipitated withdrawal jumping).

17 Low-dose naloxone-precipitated withdrawal hyperalgesia is a reliable indic

18 s.c. naloxone (10 mg/kg) to induce naloxone-precipitated withdrawal syndrome on the final day of the

19 Here, we show that somatic signs of naloxone-precipitated withdrawal were associated with increased P

20 markedly attenuates development of naloxone-precipitated withdrawal-jumping in physical dependence a

21 an L-type Ca2+ channel blocker, on naloxone-precipitated withdrawal and elevations of glutamate leve

22 ns of the PKA inhibitor Rp-cAMPS on naloxone-precipitated withdrawal from morphine was examined.

23 as evaluated by quantifying/scoring naloxone-precipitated withdrawal signs.

24 the maintenance of dependence until naloxone-precipitated withdrawal.

25 hen dependence was maintained until naloxone-precipitated withdrawal.

26 hrough pairings of one chamber with naloxone-precipitated withdrawal and the other chamber with salin

27 tly, stimuli repeatedly paired with naloxone-precipitated withdrawal provoked heroin consumption and

28 dase (beta-gal) expression during naltrexone-precipitated withdrawal.

29 attenuating development of tolerance and NLX-precipitated withdrawal-jumping during chronic cotreatme

30 940 also produced hypothermia and rimonabant-precipitated withdrawal in WT, but not CB1KO, mice.

31 40 did not produce hypothermia or rimonabant-precipitated withdrawal in CB1KO mice.

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