ライフサイエンスコーパス: 1-octanol

1 for the detection of the analyte molecules (1-octanol).

2 se to high osmolarity or volatile chemicals (1-octanol).

3 bolished the effects of subsequently applied 1-octanol.

4 f their modulation by an anesthetic alcohol, 1-octanol.

5 r modulation of T-channels in nRT neurons by 1-octanol.

6 N-acetyl arginine methyl ester from water to 1-octanol.

7 in the presence or absence of chloroform or 1-octanol.

8 anone (0.08-7.39%), 1-hexanol (0.56-16.39%), 1-octanol (0.49-10.86%) and methylbutanoic acid (0.53-21

9 ls 5 and 6 were converted via oxabicyclo[3.2.1]octanols 10 and 14 to target oxolanes 8 and 7 where C-

10 2-ethylhexyl) phosphate as the extractant in 1-octanol 2% v/v, extraction time 10min, stirring rate 7

11 n aqueous alkali), only minor proportions of 1-octanol (2.4%) are formed accompanied by very signific

12 ime methodology as follows: organic solvent: 1-octanol+2.5% (V/V) di-(2-ethylhexyl) phosphate, applie

13 alcohols were obtained from TBDMS ethers of 1-octanol, 2-phenoxyethanol, and racemic alpha-phenyl et

14 L), 6-mercapto-1-hexanol (6-MHL), 8-mercapto-1-octanol (8-MOL), 9-mercapto-1-nonanol (9-MNL), 11-merc

15 1-Octanol also antagonized ethanol inhibition of BMP-7 m

16 nd corresponding iron(III) complexes between 1-octanol and MOPS buffer (pH 7.4) are also influenced.

17 1-Octanol and related compounds may prove useful in diss

18 improves the peaks for 2,6-dimethyl aniline, 1-octanol, and 1-decanol implying a decrease in the colu

19 ed cell-cell adhesion (L1 adhesion), whereas 1-octanol antagonizes this action.

20 mised extraction conditions were as follows: 1-octanol as an organic solvent, pHdonor=2, pHacceptor=9

21 The optimum extraction conditions were 1-octanol as immobilized solvent; toluene and acetone at

22 s for the oxidative methyl esterification of 1-octanol at 60 degrees C in methanol.

23 ipid solubility of AZT, as determined by the 1-octanol/buffer partition coefficient (P), was 16-fold

24 erved for Cl-CyMe4-BTBP and Br-CyMe4-BTBP in 1-octanol but in the absence of a phase-transfer agent.

25 erase inhibited by 5 mM chloroform or 0.7 mM 1-octanol by 19.7% and 18.8%, respectively.

26 Gap junction uncouplers (1-octanol, carbenoxolone, 18beta-glycyrrhetinic acid and

27 (1.0 mL of sample solution, using 1.0 muL of 1-octanol containing of 0.5 muL of air bubble, at 40 deg

28 ed, five of which (pentanal, (E)-2-heptenal, 1-octanol, eucalyptol and 2-pentylfuran) are reported fo

29 Recently, 1-octanol has been shown to have efficacy in treating pa

30 ized by 1-pentanol (IC(50) = 715 microM) and 1-octanol (IC(50) = 3.6 microM).

31 We found that 1-octanol inhibited native T-currents at subanesthetic c

32 arity across the strongly associating, water-1-octanol interface is dominated by a nonpolar, alkane-l

33 The primary metabolite of 1-octanol is octanoic acid (OA), which is now thought to

34 A hanging microliter drop of 1-octanol is shown to be an excellent preconcentration m

35 upported liquid membranes composed of one of 1-octanol (octan-1-ol), 1,9-decadiene (deca-1,9-diene),

36 n order of magnitude for 24-DNP dissolved in 1-octanol or embedded in secondary organic material (SOM

37 To explore the mechanisms underlying higher 1-octanol potency in inhibiting native nRT T-currents, w

38 absence of phorbol ester or diacylglycerol, 1-octanol potently activated PKCalpha in a concentration

39 The very low vapor pressure of 1-octanol results in minimal evaporation of the microdro

40 n steady-state inactivation, indicating that 1-octanol stabilized inactive states of the channel.

41 d reactivity of those compounds in the water/1-octanol system that can lead to inconsistent and inacc

42 also been observed for the aliphatic octanal/1-octanol system.

43 als that may be slowly reactive in the water/1-octanol system.

44 he photoactivatable analogs of 1-butanol and 1-octanol, to photolabel the purified Ig1-4 domain of hu

45 Antagonism by 1-octanol was complete, reversible, and noncompetitive.

46 s of DC18C6 in chloroform, nitrobenzene, and 1-octanol was undetectable.

47 In contrast, 1-octanol was up to 30-fold less potent in inhibiting re

48 greement with hydrophobicities assessed from 1-octanol-water partitioning of N-acetyl amino acid amid

49 Reliable air/water (K(AW)), 1-octanol/water (K(OW)), and octanol/air partition coeff

50 Sensitivity analysis highlighted that the 1-octanol/water partition coefficient, the metabolism ra

51 Reversed-phase chromatography and 1-octanol/water partition measurements demonstrate that

52 The effects of 1-octanol were also abolished by chelation of intracellu

53 queous phase (DI water) to an organic phase (1-octanol) were performed to determine the relationship

54 hol is only marginally less hydrophobic than 1-octanol, whereas 2-nonanol, for which n = 6, was able

55 ch as nonadecafluoro-1-decanol and perfluoro-1-octanol, while multiply charged oligonucleotide anions

56 The developed method requires 11 muL of 1-octanol without the need for dispersive solvents.