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1 ective states in humans (FG7142, rimonabant, 13-cis retinoic acid).
2 arrow transplantation (ABMT) with or without 13 cis-retinoic acid.
3 f the type 2 17beta-HSD was not inhibited by 13-cis retinoic acid.
4 and ASCT was performed on day 0, followed by 13-cis-retinoic acid.
5 od stem-cell rescue, local radiotherapy, and 13-cis-retinoic acid.
7 e of the risk factors for MGD is exposure to 13-cis retinoic acid (13-cis RA), a metabolite of vitami
8 o determine whether combination therapy with 13-cis-retinoic acid (13-CRA) plus interferon alfa-2a (I
10 istered as a single agent and in combination 13-cis retinoic acid (13cRA) in children with refractory
11 To conduct a phase III trial of adjuvant 13-cis-retinoic acid (13cRA) plus interferon alfa (IFN-a
12 tudy was to test interferon alfa (IFNalpha), 13-cis-retinoic acid (13cRA), and cisplatin biochemother
13 ative fluorescence technique, that Accutane (13-cis-retinoic acid), a drug used in the treatment of a
15 ng the following search terms: isotretinoin, 13-cis-retinoic acid, Accutane, retinoids, acitretin, su
16 ta-apo-12'-carotenal, beta-apo-8'-carotenal, 13-cis-retinoic acid, all-trans-retinoic acid, beta-caro
17 At high concentrations, the combination of 13-cis-retinoic acid and each of the five other drugs re
18 site, achieving mixed clinical results (with 13-cis-retinoic acid and etretinate) in superficial blad
19 at C-4 of all-trans-retinoic acid (ATRA) and 13-cis-retinoic acid, and modification of terminal carbo
21 f other hydroxylase in response to 9-cis and 13-cis retinoic acid application to adult human skin in
22 ame preparations were unable to use 9-cis or 13-cis retinoic acid as substrate for the hydroxylation
24 d the combination of phenylbutyrate (PB) and 13-cis retinoic acid (CRA) as a differentiation and anti
29 3-0.150 microM); sulindac sulfide, NDGA, and 13-cis-retinoic acid had intermediate potency (IC50 = 4-
32 bination of low-dose MK886, ciglitazone, and 13-cis-retinoic acid interacted at least in a superaddit
33 produced by isomerization of both 9-cis and 13-cis retinoic acids is the likely inducer of the 4-hyd
35 of the follicle, or (ii) in the presence of 13-cis-retinoic acid, is due to the reduced expression o
37 alone, and whether subsequent treatment with 13-cis-retinoic acid (isotretinoin) further improves eve
41 e have evaluated whether IFN-alpha-2a and/or 13-cis-retinoic acid (RA) enhance radiation cytotoxicity
42 ctive component of the acne drug Accutane is 13-cis-retinoic acid (RA), and it is highly teratogenic
43 nd, and NDGA with paclitaxel, cisplatin, and 13-cis-retinoic acid, regardless of drug-resistance phen
44 everal of these agents (including tamoxifen, 13-cis-retinoic acid, retinyl palmitate, and an acyclic
46 of other inhibitors such as fenretinide and 13-cis-retinoic acid showed multiple advantages of Ret-N
47 lower levels of all-trans retinoic acid and 13-cis retinoic acid than control subjects but similar 9
48 he 130 patients who were assigned to receive 13-cis-retinoic acid than among the 128 patients assigne
49 tion of 0.1% all-trans, 0.1% 9-cis, and 0.1% 13-cis retinoic acid to human skin for 2 d resulted in i
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