ライフサイエンスコーパス: 17beta-estradiol

1 ge from 0 (no activity) to 1 (bioactivity of 17beta-estradiol).

2 ESR1) in the presence of its cognate ligand, 17beta-estradiol.

3 onal regulation of the SNAT2 transporter via 17beta-estradiol.

4 ment for NO/cGMP/PKG signaling downstream of 17beta-estradiol.

5 enotype by antagonizing the growth effect of 17beta-estradiol.

6 y prevented when cells were pre-treated with 17beta-estradiol.

7 apeutic effect of post-SCI administration of 17beta-estradiol.

8 (ER) alpha and beta differ from that used by 17beta-estradiol.

9 eported 35-mer aptamer for a small molecule, 17beta-estradiol.

10 aptamer, which highly specifically binds to 17beta-estradiol.

11 ed with equol compared to those treated with 17beta-estradiol.

12 able changes in ER levels and sensitivity to 17beta-estradiol.

13 ith a silastic capsule containing vehicle or 17beta-estradiol.

14 trauma-hemorrhage, which were normalized by 17beta-estradiol.

15 re compared with each other and with that of 17beta-estradiol.

16 t of ERalpha-AF2 in the metabolic actions of 17beta-estradiol.

17 ignaling mechanisms from ERalpha, ERbeta, or 17beta-estradiol.

18 M site for steroid hormone estrogens such as 17beta-estradiol.

19 indicate that this transporter is induced by 17beta-estradiol.

20 wine samples, as well as cocaine (1 nM) and 17beta-estradiol (0.2 nM) in spiked synthetic urine and

21 were 0.045 ng/L for estrone, 0.086 ng/L for 17beta-estradiol, 0.030 ng/L for estriol, 0.049 ng/L for

22 ere randomly assigned to receive either oral 17beta-estradiol (1 mg per day, plus progesterone [45 mg

23 resuscitation, rats received either vehicle, 17beta-estradiol (1 mg/kg), or 17beta-estradiol plus the

24 ectomized and then injected over 2 days with 17beta-estradiol (10 mug, s.c.), which was repeated ever

25 al, as shown by vesicular transport of [(3)H]17beta-estradiol-17-beta-(D-glucuronide) and doxorubicin

26 sformation of two natural steroid estrogens [17beta-estradiol (17beta-E2) and estrone (E1)] and two s

27 ne hormones, 17alpha-estradiol (17alpha-E2), 17beta-estradiol (17beta-E2), and estrone (E1), are rout

28 Addition of 17beta-estradiol (17betaE2) protected cultured neurons a

29 PET with the ER ligand 16alpha-(18)F-fluoro-17beta-estradiol ((18)F-FES) and to evaluate whether tra

30 16alpha-(18)F-fluoro-17beta-estradiol ((18)F-FES) is a PET tracer for ER with

31 16alpha-(18)F-fluoro-17beta-estradiol ((18)F-FES) is an estrogen receptor (ER

32 ression with the tracer 16alpha-(18)F-fluoro-17beta-estradiol ((18)F-FES) may be valuable to select o

33 16alpha-(18)F-fluoro-17beta-estradiol ((18)F-FES) with PET is a noninvasive t

34 reated for 5 weeks with cyclic injections of 17beta-estradiol-3-benzoate (EB, 10 mug) or oil vehicle.

35 ed with a (14)C labeled prototype conjugate, 17beta-estradiol-3-glucuronide (E2-3G).

36 emical transformation from parent compounds (17beta-estradiol, 4-nonylphenolpolyethoxylates, and 4-no

37 Accordingly, 17beta-estradiol administration regulated key metabolic

38 ently found that the protection conferred by 17beta-estradiol against obesity and insulin resistance

39 recursor prodrug of the main human estrogen, 17beta-estradiol, alleviates hot flushes in rat models o

40 sensitive and highly selective detection of 17beta-estradiol, an EDC that is frequently detected in

41 n, some methods reported false positives for 17beta-estradiol and 17alpha-ethynylestradiol in unspike

42 In these studies, we assessed the ability of 17beta-estradiol and equol to regulate markers of hippoc

43 Similarly, 17beta-estradiol and equol treatment had no effect on mi

44 endocrine-disrupting chemicals, particularly 17beta-estradiol and estrone, and fish exposed to the po

45 These data indicate that 17beta-estradiol and GPER independently regulate hippoca

46 17beta-estradiol and IL-6 may act synergistically to pro

47 Females received 17beta-estradiol and males received testosterone.

48 hondrial damage using synergistic effects of 17beta-estradiol and methylene blue, previously shown by

49 st cancer cells, cultured in the presence of 17beta-estradiol and tamoxifen, were used as a model sys

50 sed in size and number at 24 hours following 17beta-estradiol and, in particular diarylpropionitrile,

51 Here we show that physiologic estrogen (17beta-estradiol) and progesterone reciprocally regulate

52 nts were respectively theophylline, digoxin, 17beta-estradiol, and aldrin.

53 In WTP influents, estrogens (estrone, 17beta-estradiol, and estriol), androgens (androstenedio

54 ted the fate and transport of 4-nonylphenol, 17beta-estradiol, and estrone in a 10-km reach of the Re

55 ural estrogenic hormones (17alpha-estradiol, 17beta-estradiol, and estrone) in aqueous solutions blen

56 of the initial amounts of 17alpha-estradiol, 17beta-estradiol, and estrone, respectively.

57 tain florfenicol, pyrimethamine, estrone and 17beta-estradiol at levels from 0.095 to 2.7 mug/kg.

58 anure excreted by implanted (40 mg TBA, 8 mg 17beta-estradiol) beef cattle.

59 n terms of research and monitoring have been 17beta-estradiol (beta-E2) and 17alpha-ethinylestradiol,

60 f Fe(3+)-saturated montmorillonite catalyzed 17beta-estradiol (betaE2) transformation was investigate

61 he alpha4beta2-selective positive modulators 17beta-estradiol (betaEST) and desformylflustrabromine (

62 BPA and 17beta-estradiol bind to ERs in a similar fashion, where

63 This effect was associated with increased 17beta-estradiol but not with insulin-like growth factor

64 Across women, lower 17beta-estradiol concentrations were related to more pro

65 17beta-estradiol-D5 was used as internal standard.

66 We found that post-SCI administration of 17beta-estradiol decreased neuronal loss in the ventral

67 Additionally, 17beta-estradiol decreased synaptophysin levels in the C

68 Neutrophils treated with 17beta-estradiol demonstrated an enhanced oxidative burs

69 yo implantation is coordinately regulated by 17beta-estradiol (E(2)) and progesterone (P(4)).

70 his study was to elucidate the mechanisms of 17beta-estradiol (E(2)) antioxidant and neuroprotective

71 17beta-estradiol (E(2)) can enhance reproductive, cognit

72 OH-estrone (4-OHE(1)) with barely detectable 17beta-estradiol (E(2)) conversion following expression

73 We showed that 17beta-estradiol (E(2)) favors pancreatic beta-cell surv

74 The affinity of 17beta-estradiol (E(2)) for the estrogen receptor is wea

75 17beta-Estradiol (E(2)) hinders cytotoxic drug-induced c

76 l studies indicates that the gonadal hormone 17beta-estradiol (E(2)) impacts the structure and functi

77 To learn whether 17beta-estradiol (E(2)) is essential for vitamin D(3)-me

78 hin 1 y, and tumor growth occurs with either 17beta-estradiol (E(2)) or tamoxifen.

79 ted kinase (ERK) activation is necessary for 17beta-estradiol (E(2)) to enhance novel object recognit

80 ylation are critical for the potent estrogen 17beta-estradiol (E(2)) to enhance object recognition me

81 (PKC) signaling can be activated rapidly by 17beta-estradiol (E(2)) via nontraditional signaling in

82 The G protein-coupled receptor GPR30 binds 17beta-estradiol (E(2)) yet differs from classic estroge

83 ablation blocked the reduction of T(SKIN) by 17beta-estradiol (E(2)), which occurred in the environme

84 Herein, we demonstrate that 17beta-estradiol (E(2))-induced uterine epithelial proli

85 The mechanisms responsible for 17beta-estradiol (E(2))-stimulated breast cancer growth

86 The estrogen 17beta-estradiol (E) increases the axospinous synaptic d

87 ormone replacement paradigm involving either 17beta-estradiol (E), or E plus progesterone (E+P).

88 pha-dihydrotestosterone; DHT) or estrogenic (17beta-estradiol; E(2)) metabolites.

89 -enhancing effects of the modulatory hormone 17beta-estradiol E2.

90 17beta-estradiol (E2 or estrogen) is an endogenous stero

91 mug L(-1)), while that of the native hormone 17beta-estradiol (E2) (1 muM, i.e., 272 mug L(-1)) was 1

92 alpha-containing cancer cells, the estrogen, 17beta-estradiol (E2) activates the UPR through a phosph

93 We found recently that 17beta-estradiol (E2) acutely suppresses GABAergic inhib

94 17beta-estradiol (E2) also influences hypothalamic progr

95 fluctuations and systemic administration of 17beta-estradiol (E2) alter spine density in the dorsal

96 s often found in treated and natural waters, 17beta-estradiol (E2) and 17alpha-ethynylestradiol (EE).

97 We investigated the potential of 17beta-estradiol (E2) and estrogen receptor (ER) signali

98 ch were transformed into the free estrogens, 17beta-estradiol (E2) and estrone (E1), respectively.

99 owever, few studies have assessed both serum 17beta-estradiol (E2) and exogenous hormone therapy (HT)

100 Both 17beta-estradiol (E2) and lithium influence NMDAR expres

101 to analogues similar to progesterone such as 17beta-estradiol (E2) and norethisterone (NET).

102 e sought to Determine the mechanism by which 17beta-estradiol (E2) and progesterone (P4) increase IL-

103 We demonstrated that 17beta-estradiol (E2) and the GPER-selective ligand G-1

104 tudy, we demonstrate for the first time that 17beta-estradiol (E2) and the selective GPER ligand G-1

105 The neuroprotective effects of 17beta-estradiol (E2) and three synthetic nonfeminizing

106 ve shown that the neuroprotective effects of 17beta-estradiol (E2) are dependent upon mitochondrial f

107 using thyroid stimulating hormone (TSH) and 17beta-estradiol (E2) as model analytes, respectively.

108 -ethylbenzthiazoline-6-sulfonate) (ABTS) and 17beta-estradiol (E2) as the probing substrates.

109 We previously observed that 17beta-estradiol (E2) augments ischemic borderzone vascu

110 study examined whether the steroid hormone, 17beta-estradiol (E2) can exert long-lasting beneficial

111 this study was to investigate if exposure to 17beta-estradiol (E2) causes abnormal development in lar

112 overexpressed in the ERalpha-negative lines, 17beta-estradiol (E2) decreased slug and increased E-cad

113 Central administration of 17beta-estradiol (E2) decreases food intake in controls

114 to affect TMJ-responsive neurons, we applied 17beta-estradiol (E2) directly at the spinomedullary (Vc

115 While 17beta-estradiol (E2) enhanced ERalpha binding to p53 an

116 Our laboratory has demonstrated that 17beta-estradiol (E2) enhances hippocampal memory consol

117 monstrated by an immunospecific detection of 17beta-Estradiol (E2) following the competitive inhibiti

118 tives were synthesized and evaluated against 17beta-estradiol (E2) for their estrogenic activity in M

119 ensor for the rapid, label-free detection of 17beta-estradiol (E2) from femtomolar to micromolar leve

120 Although chronic 17beta-estradiol (E2) has been shown to be a cognition-p

121 17beta-Estradiol (E2) has been shown to regulate GM-CSF-

122 e molecular and cellular mechanisms by which 17beta-estradiol (E2) impacts the microenvironment and m

123 Suwannee River NOM-sensitized photolysis of 17beta-estradiol (E2) in freshwater and saline media.

124 ptor alpha (ERalpha) mediates the effects of 17beta-estradiol (E2) in normal mammary gland, and it is

125 We previously found that the hormone 17beta-estradiol (E2) in part mediates this benefit.

126 It is well known that many of the actions of 17beta-estradiol (E2) in the central nervous system are

127 ing ultrasensitive colorimetric detection of 17beta-estradiol (E2) in water and urine samples using D

128 Although systemically injected 17beta-estradiol (E2) increases CA1 dendritic spine dens

129 Previously, we found that 17beta-estradiol (E2) increases T-type channel expressio

130 Previously we demonstrated that 17beta-Estradiol (E2) induced rapid Ca(2+) influx via L-

131 Intriguingly, recent reports have shown that 17beta-estradiol (E2) induces Noxa expression, although

132 Here, we report that 17beta-estradiol (E2) influences production of runx1+ HS

133 We found 17beta-estradiol (E2) inhibited hepatic gluconeogenic ge

134 The uterotropic response of the uterus to 17beta-estradiol (E2) is genetically controlled, with ma

135 pressed during estrous cycle stages in which 17beta-estradiol (E2) is naturally high (e.g., proestrus

136 Neuromodulation of synaptic plasticity by 17beta-estradiol (E2) is thought to influence informatio

137 The steroid 17beta-estradiol (E2) is well known to influence hippoca

138 of BSEP was inversely correlated with serum 17beta-estradiol (E2) levels before, during, and after g

139 In cystic fibrosis, 17beta-estradiol (E2) may inhibit store-operated Ca(2+)

140 The estrogen 17beta-estradiol (E2) modulates dendritic spine plastici

141 This study investigated the effects of 17beta-estradiol (E2) on gene regulation in human cardia

142 t stimulation of ERalpha-positive cells with 17beta-estradiol (E2) promotes global citrullination of

143 17beta-estradiol (E2) regulates the activity of the gona

144 ive and positive feedback effects of ovarian 17beta-estradiol (E2) regulating release of gonadotropin

145 d subsequently explored the distribution and 17beta-estradiol (E2) regulation of kisspeptin mRNA (Kis

146 Given that chronic 17beta-estradiol (E2) replacement of ovariectomized rats

147 We previously found that 17beta-estradiol (E2) stimulates apolipoprotein A-IV (ap

148 Recent work suggests that timing of 17beta-estradiol (E2) therapy may be critical for observ

149 The ability of 17beta-estradiol (E2) to enhance hippocampal object reco

150 We have previously reported that 17beta-estradiol (E2) treatment activates Notch signalin

151 17beta-Estradiol (E2) treatment limits the pathology ass

152 Whereas 17beta-estradiol (E2) treatment up-regulates pancreatic

153 Here we show that 17beta-estradiol (E2) up-regulates total CaM level in en

154 (s) of D3 action and compare it with that of 17beta-estradiol (E2) using both in vitro and in vivo ut

155 An aptamer capable of binding to our target 17beta-estradiol (E2) was isolated by SELEX with dissoci

156 The classic estrogen 17beta-estradiol (E2) was recently identified as a novel

157 gned and synthesized nine new derivatives of 17beta-estradiol (E2) with a bulky side chain attached t

158 17beta-estradiol (E2), acting through ERalpha, and pro-i

159 case for the transformation of estrone (E1), 17beta-estradiol (E2), and 17alpha-ethinylestradiol (EE2

160 maceuticals, 17alpha-ethinylestradiol (EE2), 17beta-estradiol (E2), and diclofenac throughout Europea

161 t ovarian sex-steroid hormones, particularly 17beta-estradiol (E2), are important trophic factors tha

162 Estrogens, particularly 17beta-estradiol (E2), are powerful neuroprotective agen

163 AROM), the enzyme converting testosterone to 17beta-estradiol (E2), contributes to the regulation of

164 G protein-coupled receptor reported to bind 17beta-estradiol (E2), couple to the G proteins Gs and G

165 eration in either sexes by acute exposure to 17beta-estradiol (E2), dihydrotestosterone (DHT), a cock

166 termination of four estrogens [estrone (E1), 17beta-estradiol (E2), estriol (E3), and 17alpha-ethinyl

167 Estrogen, 17beta-estradiol (E2), is a powerful therapeutic agent t

168 2-bis(4-hydroxyphenyl)-proprionitrile (DPN), 17beta-estradiol (E2), or vehicle using Alzet minipumps

169 n dihydrotestosterone (DHT) and the estrogen 17beta-estradiol (E2), testosterone can mediate its effe

170 spensable for the atheroprotective action of 17beta-estradiol (E2), the main ligand of ERs.

171 suggests that the hippocampus is a source of 17beta-estradiol (E2), the physiological role of this ne

172 roles in reproductive tissues--specifically, 17beta-estradiol (E2), the primary estrogen, which is se

173 decreases the conversion of testosterone to 17beta-estradiol (E2), thereby reducing E2-dependent vit

174 zed into 5alpha-dihydrotestosterone (DHT) or 17beta-estradiol (E2), which activate different hormonal

175 bound SP1 and C/EBPbeta is essential for the 17beta-estradiol (E2)-induced activation of human prolac

176 least 17 CoRs from nuclear extracts bound to 17beta-estradiol (E2)-liganded estrogen receptor-alpha o

177 m), in both intact males and ovariectomized, 17beta-estradiol (E2)-treated females.

178 was decreased in bone in SPI-fed, but not in 17beta-estradiol (E2)-treated rats.

179 l aromatase, which produces the neurosteroid 17beta-estradiol (E2).

180 ironmental fate of steroid hormones, such as 17beta-estradiol (E2).

181 estrogen were observed by co-treatment with 17beta-estradiol (E2).

182 AR-dependent synaptic plasticity and memory, 17beta-estradiol (E2).

183 mouse responsive to long-term treatment with 17beta-estradiol (E2).

184 ontributes to the neuroprotective effects of 17beta-estradiol (E2); however, the mechanisms associate

185 eak proestrus levels of the primary estrogen 17beta-estradiol (E2; 10 mug/kg, i.p., 1-h pretreatment)

186 tics), genistein (GEN; found in soybean), or 17beta-estradiol (E2; an endogenous estrogen).

187 distinct cellular signaling pathways (i.e., 17beta-estradiol [E2] and TNF-alpha).

188 cused primarily on males, although estrogen (17beta-estradiol, E2) affects calcium-dependent activiti

189 We have shown that estrogen (17beta-estradiol, E2) inhibits expression of these genes

190 pes) after exposure to two concentrations of 17beta-estradiol (E2beta; 2 ng/L and 50 ng/L) during fou

191 ively at an average concentration of 10 ng/l 17beta-estradiol equivalent (EEQ), 26 ng/l testosterone

192 Results showed that 17beta-estradiol equivalent levels were higher than thos

193 Previous findings suggest that 17beta-estradiol (estradiol) has a suppressive effect on

194 e estrogens included for study were estrone, 17beta-estradiol, estriol, 17alpha-ethinylestradiol, and

195 When cells were pre-treated with 100 nM 17beta-estradiol (estrogen) for 1 h and then co-treated

196 Four steroidal estrogens (17beta-estradiol, estrone, estriol, and 17alpha-estradio

197 Because administration of 17beta-estradiol following trauma-hemorrhage improves ca

198 Administration of 17beta-estradiol following trauma-hemorrhage restored ca

199 Although 17beta-estradiol has long been known to regulate memory

200 Additionally, post-SCI administration of 17beta-estradiol improved skilled forelimb function and

201 me inexpensive and easy-to-use monitoring of 17beta-estradiol in environmental samples such as efflue

202 were induced by both diethylstilbestrol and 17beta-estradiol in estrogen receptor alpha (ERalpha)-po

203 58 genes previously shown to be regulated by 17beta-estradiol in MCF-7 cells, the meta-analysis combi

204 , showing the ability of detecting traces of 17beta-estradiol in serum at concentrations lower than t

205 We here report on the immunodetection of 17beta-estradiol in serum by antibody-immobilized microc

206 n castrated male mice, and IL-6 induction by 17beta-estradiol in splenocytes from naive female mice (

207 construct a voltammperometric biosensor for 17beta-estradiol in the 0.9-11 pM range.

208 This study revealed that 17beta-estradiol in the brain mediated the physiological

209 et of seizures by promoting the synthesis of 17beta-estradiol in the brain.

210 ot only the expression of P450arom, but also 17beta-estradiol in the cerebral cortex.

211 In summary, findings indicate that while 17beta-estradiol induced a significant increase in sever

212 17beta-Estradiol is a multi-active steroid that imparts

213 4+CD25+FoxP3+ regulatory T-cells (Tregs) and 17beta-estradiol is crucial in the pathogenesis of sex b

214 lower threshold for synaptic plasticity when 17beta-estradiol is elevated.

215 The steroid 17beta-estradiol is known to acutely potentiate glutamat

216 rete sequence and suggest that the effect of 17beta-estradiol is mediated by actions on single subuni

217 Attenuation of 17beta-estradiol (k(stream) = -3.2 +/- 1.0 day(-1)) was

218 ensor surface, and a higher concentration of 17beta-estradiol leads to less fluorescence-labeled DNA

219 17beta-estradiol led to a significant upregulation in pe

220 utcomes were percent change from baseline in 17beta-estradiol levels (E2) and tricuspid annular plane

221 A P450arom inhibitor, letrozole, reduced 17beta-estradiol levels and completely suppressed the el

222 eceptor-dependent inhibition in females when 17beta-estradiol levels are elevated.

223 tolerance, and suggest approaches to restore 17beta-estradiol levels as a novel treatment option for

224 onbreeding conditions by manipulating plasma 17beta-estradiol levels in wild-caught female Gambel's w

225 n Cyp19a1 suppression, decreased circulating 17beta-estradiol levels, abnormal fat accumulation, and

226 (Cyp19a1) expression and reduced circulating 17beta-estradiol levels.

227 elopment, and photoproducts alter whole-body 17beta-estradiol levels.

228 ial to influence the human daily exposure to 17beta-estradiol like activity in various risk groups wi

229 that treatment of postmenopausal women with 17beta-estradiol markedly enhances TLR-7- and TLR-9-depe

230 Thus, the anti-oxidative effects of 17beta-estradiol may be involved in the prevention of se

231 om naive female mice (p<0.05) suggested that 17beta-estradiol may enhance sex bias through IL-6 induc

232 Osteocalcin and 17beta-estradiol mediate their effects through G protein

233 f synthetic ERbeta-specific ligands, but not 17beta-estradiol, mediate recruitment of CtBP corepresso

234 These results suggest that the 17beta-estradiol-meditated improvement in cardiac functi

235 vitro culture of isolated uterine ILC2s with 17beta-estradiol modified expression of a number of gene

236 about the molecular mechanisms through which 17beta-estradiol modulates hippocampal memory.

237 port and extend previous findings indicating 17beta-estradiol modulation of hippocampal opioid peptid

238 ha-androstanediol-glucuronide (N = 4767) and 17beta-estradiol (N = 2014) in Caucasian men.

239 The acute vasodilatory effects of 17beta-estradiol (non-specific estrogen receptor (ER) ag

240 itment to the cell cycle following strain or 17beta-estradiol occurs within 30 min, as determined by

241 this, we have investigated the influence of 17beta-estradiol on acute wound repair in castrated male

242 investigated whether the salutary effects of 17beta-estradiol on cardiac function are mediated via Ak

243 characterize the effects of DHEA, prolactin, 17beta-estradiol on insulin-growth factor-1 and -2 (IGF-

244 published datasets addressing the effect of 17beta-estradiol on MCF-7 cells at early (3-4 hours) and

245 to determine the molecular level effects of 17beta-estradiol on single MCF-7 cells using Fourier tra

246 DC lineage abrogated the enhancing effect of 17beta-estradiol on their TLR-mediated production of IFN

247 Delivery of 17beta-estradiol or an estrogen receptor (ER)-alpha (but

248 ranscriptional activity include the agonists 17beta-estradiol or conjugated estrogens with the antago

249 There were no significant effects of either 17beta-estradiol or equol treatment on glycolytic protei

250 on proestrous morning, when serum levels of 17beta-estradiol peak, the nonspecific opioid receptor a

251 women with unopposed estrogen, we implanted 17beta-estradiol pellets in adult female Pten heterozygo

252 ther vehicle, 17beta-estradiol (1 mg/kg), or 17beta-estradiol plus the phosphoinositide 3-kinase inhi

253 17beta-Estradiol potentiates activation of neuronal nico

254 We found that 17beta-estradiol prevented the trauma-hemorrhage-induced

255 mice synchronized into estrus by delivery of 17beta-estradiol prior to intravaginal challenge with wi

256 specificity, and treatment of male mice with 17beta-estradiol prolonged their survival during the cou

257 data suggest that post-SCI administration of 17beta-estradiol protected both the gray and white matte

258 In conclusion, 17beta-estradiol protects osteocytes against apoptosis b

259 study, we elucidated the mechanism by which 17beta-estradiol regulates the transcription of SNAT2.

260 17beta-estradiol replacement in SERT (-/-) mice reversed

261 that the anti-apoptotic effects of cGMP and 17beta-estradiol required BAD phosphorylation on Ser(136

262 Akt and ERK1/2 activation by 17beta-estradiol required PKG II, and cGMP mimicked the

263 dihydrotestosterone and 16alpha-(18)F-fluoro-17beta-estradiol, respectively.

264 Coinjection with 17beta-estradiol resulted in a decrease in (18)F-FES upt

265 that maximal binding occurred at the highest 17beta-estradiol serum concentration.

266 In contrast, 17beta-estradiol significantly increased the abundance o

267 DHEA-sulfate (DHEA-S), DHEA, and 17beta-estradiol stimulated keratinocyte and fibroblast

268 g of each of these proteins nearly abolished 17beta-estradiol-stimulated SNAT2 promoter activity.

269 17beta-Estradiol stimulation of MCF-7 cells is a widely

270 ith estrogen receptor alpha (ER-alpha) after 17beta-estradiol stimulation.

271 Ovariectomized females supplemented with 17beta-estradiol succumbed to P. aeruginosa challenge ea

272 sses seizures, focusing on the regulation of 17beta-estradiol synthesis in the brain.

273 estrogens (o-CEE), 0.45 mg/d, or transdermal 17beta-estradiol (t-E2), 50 mcg/d, each with 200 mg of o

274 Here we demonstrate that 17beta-estradiol, tamoxifen, and fulvestrant induce nucl

275 t cancer cell line, restoring sensitivity to 17beta-estradiol, tamoxifen, and retinoids.

276 tes with PMCA with or without treatment with 17beta-estradiol, thapsigargin, or G-1.

277 In animals, 17beta-estradiol (the major estrogen in most mammals, re

278 vels increased in MCF-7 cells in response to 17beta-estradiol, the ERbeta-specific agonist diarylprop

279 with potential carcinogenic effects such as 17beta-estradiol, the most powerful substance with estro

280 Like 17beta-estradiol, the non-steroid Br-PBTC only requires

281 HED for the development of a brain-selective 17beta-estradiol therapy to relieve hot flushes without

282 little effect on this gene in the absence of 17beta-estradiol, they can potentiate ER activity in an

283 and that transcript levels are modulated by 17beta-estradiol through the estrogen receptor (ER)alpha

284 how that a single intracerebral injection of 17beta-estradiol to ovariectomized female rats immediate

285 hoeae following treatment with water-soluble 17beta-estradiol to promote long-term gonococcal infecti

286 tion of VMH PI3K activity blocked effects of 17beta-estradiol to stimulate energy expenditure, but di

287 scriptional coregulator that is recruited by 17beta-estradiol to the promoters of estrogen target gen

288 17beta-Estradiol-treated ovariectomized female mice demo

289 17beta-estradiol treatment of B cells and WT276 cells in

290 Results indicated that 17beta-estradiol treatment significantly increased expre

291 vertebra (C5) followed by administration of 17beta-estradiol via a slow release pellet (0.5 or 5.0 m

292 ation reaction between 17alpha-estradiol and 17beta-estradiol via estrone was observed in aqueous sol

293 17beta-estradiol was detected below its quantitation lim

294 trone was detected in all of the samples and 17beta-estradiol was detected in one.

295 The dose-response curve of 17beta-estradiol was established and a detection limit w

296 Coinjection of (18)F-FES with 17beta-estradiol was performed to determine whether trac

297 For comparison, estrone and 17beta-estradiol were modeled and are likely capable of

298 mples containing different concentrations of 17beta-estradiol were premixed with a given concentratio

299 d as vitellogenesis-related and regulated by 17beta-estradiol were significantly enriched among those

300 17beta-estradiol, with or without progesterone, altered