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5 iry cell leukemia (HCL) after treatment with 2-chlorodeoxyadenosine (2-CdA) using immunohistochemical
7 steroids, radiation, vinblastine/prednisone, 2-Chlorodeoxyadenosine (2-CdA), or cytosine arabinoside
12 d with interferon alfa (IFN-alpha; P = .27), 2-chlorodeoxyadenosine (2CDA; P = .37), or deoxycoformyc
13 , a clinical protocol was designed combining 2-chlorodeoxyadenosine (CdA), an inhibitor of ribonucleo
14 s fludarabine, 2'-deoxycoformycin (DCF), and 2-chlorodeoxyadenosine (CdA), commonly used in the treat
15 te the absolute bioavailability (F value) of 2-chlorodeoxyadenosine (cladribine; 2-CdA) after multipl
17 box-binding protein (TBP) were disrupted on 2-chlorodeoxyadenosine monophosphate (CldAMP)-substitute
19 ndefined cases have been shown to respond to 2-chlorodeoxyadenosine therapy after failing treatment w
20 nal processes would therefore be affected by 2-chlorodeoxyadenosine triphosphate (CldATP) incorporati
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