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1 ation of polyPs in the acidocalcisomes using 4',6-diamidino-2-phenylindole.
2 hese embryos were dispersed and treated with 4',6-diamidino-2-phenylindole.
3 rmed by visualization of nuclei stained with 4,6'-diamidino-2-phenylindole.
4 ization of polyP in the dense granules using 4',6-diamidino-2-phenylindole and by its release togethe
5 taining with anti-alpha-tubulin antibody and 4',6-diamidino-2-phenylindole and flow cytometric analys
7 pected for intracapsid steric restriction of 4',6-diamidino-2-phenylindole (DAPI) binding to packaged
9 , we show that the ICD of minor-groove-bound 4',6-diamidino-2-phenylindole (DAPI) originates from an
11 tending toxin (Cdt), connective tissue (CT), 4',6-diamidino-2-phenylindole (DAPI), human gingival epi
12 tron microscope after labeling in vitro with 4',6-diamidino-2-phenylindole (DAPI), intracellular inje
14 nuclei frequently do not stain equally with 4',6-diamidino-2-phenylindole (DAPI), suggesting that by
15 ex affinity exhibited by 5PTB, netropsin and 4',6-diamidino-2-phenylindole (DAPI), two AT-specific mi
16 surement of changes in the fluorescence of a 4',6-diamidino-2-phenylindole (DAPI)-dsDNA complex upon
17 ssay based on changes in the fluorescence of 4',6-diamidino-2-phenylindole (DAPI)-dsDNA complexes.
22 t methods: staining intact chloroplasts with 4',6-diamidino-2-phenylindole (DAPI); staining at the si
24 le chromosome probes, single-copy genes, and 4'-6-diamidino-2-phenylindole (DAPI-) and G-banded chrom
25 ick-end labeling (TUNEL) in conjunction with 4'6'-diamidino-2-phenylindole (DAPI) staining and by flu
27 nted in this investigation clearly show that 4,6-diamidino-2 phenylindole (DAPI) is superior to both
29 inor groove binding compounds, netropsin and 4,6-diamidino-2-phenylindole (DAPI), and a DNA hairpin h
30 of cell extracts, and staining of cells with 4,6-diamidino-2-phenylindole (DAPI), and the polyP-bindi
32 Cytochemical staining of cellular DNA with 4,6-diamidino-2-phenylindole demonstrated TNF-alpha-indu
33 ensitive fluorescent reagents SYPRO Ruby and 4',6-diamidino-2-phenylindole dihydrochloride (DAPI), to
35 /PMS kit and of dead (apoptotic) cells using 4',6-diamidino-2-phenylindole dihydrochloride nuclear st
36 uorescent dye molecules per CPMV using DAPI (4',6-diamidino-2-phenylindole dihydrochloride), propidiu
38 NA was stained with propidium iodide (PI) or 4',6-diamidino-2-phenylindole, dihydrochloride (DAPI).
39 Furthermore, microtubule immunostaining and 4,6-diamidino-2-phenylindole DNA staining demonstrated t
40 in condensation, as observed with the use of 4,6-diamidino-2-phenylindole-fluorescent staining, and b
42 cell level using double immunostaining plus 4,6-diamidino-2-phenylindole (for nuclei) counterstainin
45 unctate nuclear localization correlated with 4,6-diamidino-2-phenylindole light regions and is exclud
46 niformly; locally high levels accumulated in 4',6-diamidino-2-phenylindole-negative zones containing
49 of fixed permeabilized red blood cells with 4',6'-diamidino-2-phenylindole or YOYO-1, a sensitive nu
50 ite-specific probes, in addition to inverted 4,6-diamidino-2-phenylindole or conventional G-banding a
51 fluorescent foci that colocalize with DAPI (4',6'-diamidino-2-phenylindole)-positive material and fo
52 ence contrast and fluorescence microscopy of 4',6-diamidino-2-phenylindole-stained log phase cells.
53 undergoing apoptosis verifies that the FD of 4',6-diamidino-2-phenylindole-stained nuclear structures
55 distribution of the actin cytoskeleton, DNA (4', 6-diamidino-2-phenylindole staining), and calcofluor
56 e-contrast microscopy, and in selected cases 4',6'-diamidino-2-phenylindole staining and DNA fragment
58 poptotic, as assessed by Annexin V staining, 4',6'-diamidino-2-phenylindole staining, and DNA fragmen
60 enome was developed based on the patterns of 4',6-diamidino-2-phenylindole staining of the Nipponbare
61 ase-mediated dUTP nick-end labeling (TUNEL), 4',6-diamidino-2-phenylindole staining, and immunohistoc
62 ristic apoptotic changes, as demonstrated by 4',6-diamidino-2-phenylindole staining, lack of either D
63 also localized to the contractile vacuole by 4',6-diamidino-2-phenylindole staining, suggesting, with
66 romatin fragmentation by acridine orange and 4'6-diamidino-2-phenylindole staining, and (3) agarose g
67 nd changes in nuclear morphology detected by 4,6-diamidino-2-phenylindole staining, DNA fragmentation
70 mal fragmentation of cellular DNA, and DAPI (4',6-diamidino-2-phenylindole) staining revealed condens
71 deoxyuridine (BrdU) pulse-labeling and DAPI (4',6-diamidino-2-phenylindole) staining, which precisely
72 sitive for cytokeratins 8, 18, and/or 19 and 4',6-diamidino-2-phenylindole were considered to be CTCs
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