ライフサイエンスコーパス: 4AP

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1 4AP applied to the double mutant blocked the final trans

2 4AP promoted a pattern of firing which was observed, alb

3 d in gating currents, is almost normal for a 4AP-occupied channel; only the final opening transition

4 zygotes was tested directly by administering 4AP while recording force produced by failing motor unit

5 Although 4AP increased peak forces during unit tetanic activation

6 The drug 4-aminopyridine (4AP) blocks voltage-dependent potassium conductances and

7 sium channel blocking agent 4-aminopyridine (4AP) can sometimes cause ectopic activity in demyelinati

8 The convulsant 4-aminopyridine (4AP) facilitates the synchronous firing of interneurons

9 t a model for the action of 4-aminopyridine (4AP) on K channels.

10 culline methiodide (BMI) or 4-aminopyridine (4AP).

11 e potassium channel blocker 4-aminopyridine (4AP).

12 l types were also found for 4-aminopyridine (4AP).

13 zed TTX-sensitive sodium current (I(Na)) and 4AP-sensitive and TEA-resistant potassium current (I(K))

14 synchronized IPSPs in CA3 cells elicited by 4AP in the presence of ionotropic glutamate receptor blo

15 or units could be significantly increased by 4AP, whereas no effect was observed in a nonfailing moto

16 its in HCSMA homozygotes can be increased by 4AP.

17 conclude that the ectopic spiking induced by 4AP is generated by membrane potential oscillations asso

18 Once in the open channel, 4AP's major action is to bias the activation gate toward

19 dy, we determined whether and to what extent 4AP could enhance muscle force production in HCSMA.

20 SqKv1A's affinity for 4AP was poor at rest and increased after activation, whe

21 Unexpectedly, seizure-like events in 4AP were desynchronous events, both in comparison with i

22 nchronized interneuron oscillatory firing in 4AP.

23 ns, both in the presence and absence of 5 mM 4AP.

24 The ability of 4AP to increase force in failing units may be related to

25 The effect of 4AP is well modeled as a selective block of the final ga

26 n both the initiation and the maintenance of 4AP-facilitated inhibitory circuit oscillations.

27 Possible sites of 4AP action are considered.

28 cations, arterial perfusion of either BMI or 4AP induced focal limbic SLEs.

29 Bursts in 4-amino-pyridine (4AP) were highly synchronous events.

30 Systemic 4AP (1-2 mg/kg) increased nerve-evoked whole muscle twit

31 We propose that 4AP, like tetraethylammonium ion and other quaternary am

32 rest and increased after activation, whereas 4AP block occurred much more readily at rest with native

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