1 ntrast to the classical role ascribed to the 5-HT1 receptors.

2 tryptamine (5-CT), a nonselective agonist of 5-HT1 receptors.

3 lay neurons to 5-carboxytryptamine (5-CT), a 5-HT1 receptor agonist that preferentially activates the

4 ylserotonin (100 microg kg-1, i.a.), and the 5-HT1 receptor agonist, 5-carboxamidotryptamine (100 mic

5 re elevated but then returned to normal by a 5-HT1 receptor agonist, sumatriptan.

6 e investigated the inhibitory effects of six 5-HT1 receptor agonists in this model: 3-(1,2,5,6-tetrah

7 ylation in response to serotonin, SSRIs, and 5-HT1 receptor agonists.

8 ced melatonin suppression was blocked by the 5-HT(1) receptor antagonist, methiothepin, but not by th

9                                              5-HT1 receptor antagonists have been discovered with goo

10 ght involve inhibition of MAP kinases, since 5-HT1 receptors can increase mitogen-activated protein (

11                       Instead, activation of 5-HT1 receptors caused a slow and remarkably prolonged i

12 tter in bulbospinal descending pathways, and 5-HT(1) receptors have been shown to inhibit synaptic tr

13 emonstrate that activation of the endogenous 5-HT1 receptor is coupled to calcium signaling pathways

14 c interneurons and direct inhibition via the 5-HT1 receptors on MCs.

15 stablish the existence of triptan-sensitive (5-HT1) receptors on postsynaptic central trigeminal neur

16   This may largely be due to activation of a 5-HT1 receptor subtype and not to desensitization.

17 SSOFRAs with improved selectivity over other 5-HT(1) receptor subtypes.

18 s for further understanding the roles of the 5-HT1 receptor subtypes and providing new approaches for

19 brane environment these four closely related 5-HT1 receptor subtypes exhibit different G protein coup