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1 and 5-HT4 receptor antagonists, but not by a 5-HT3 receptor antagonist.
2 was less affected by VMN infusions with the 5-HT3 receptor antagonist.
3 nged efficacy, and acts synergistically with 5-HT3 receptor antagonists.
4 ative efficacy of the 5-hydroxytryptamine-3 (5-HT(3)) receptor antagonists.
8 sponses to 2-methyl-5-HT were blocked by the 5-HT(3) receptor antagonist alosetron (2 x 10(-7) M), wh
9 kg-1, i.v.), or treatment with the selective 5-HT3 receptor antagonist alosetron (30 microg kg-1, i.v
11 1) receptor antagonist in conjunction with a 5-HT3 receptor antagonist and corticosteroid in patients
14 isetron; 500 ng), or were coinfused with the 5-HT3 receptor antagonist and the 5-HT2A/2C receptor ago
15 rolapitant, in combination with a serotonin (5-HT3) receptor antagonist and dexamethasone, for the pr
16 d Fos-LI in the DVC of ondansetron (1 mg/kg; 5-HT3 receptor antagonist) and vehicle-treated rats foll
19 f ondansetron, a selective serotonin type-3 (5-HT3) receptor antagonist, attenuates cholecystokinin (
20 etic risk radiation therapy should receive a 5-HT(3) receptor antagonist before each fraction and for
21 by DPPIV inhibition or by cholecystokinin or 5-HT3 receptor antagonists, but was inhibited by atropin
23 or any highly emetic agents should receive a 5-HT(3) receptor antagonist, dexamethasone, and a neurok
24 etron (300 microg kg(-1), I.V.), a selective 5-HT(3) receptor antagonist, eliminated the afferent's r
25 ) and palonosetron (a 5-hydroxytryptamine-3 [5-HT3] receptor antagonist) for the prevention of acute
28 -HT response was completely abolished by the 5-HT3 receptor antagonist, granisetron (0.5 mg kg-1).
30 wide dynamic range neurons are inhibited by 5-HT3 receptor antagonists in rats following spinal nerv
33 TS-TH-EGFP neurons, an effect blocked by the 5-HT(3) receptor antagonist ondansetron and mimicked by
34 coefficient = 1.8) that were blocked by the 5-HT3 receptor antagonist ondansetron (IC50 = 103 pM) an
35 led to do so in Tph1(-/-) colon; and (9) the 5-HT3 receptor antagonist ondansetron, which reduced CMM
43 response to acetylcholine was blocked by the 5-HT(3) receptor antagonist renzapride with a similar IC
44 njection of tropisetron (200 microg kg-1), a 5-HT3 receptor antagonist, significantly attenuated the
45 whisking frequencies, and selective 5-HT2 or 5-HT3 receptor antagonists suppress this rhythmic firing
46 Unilateral infusion of selective 5-HT2 or 5-HT3 receptor antagonists suppresses ipsilateral whiski
48 otetralin (DPAT; 0.1 mg/kg and 0.3 mg/kg) or 5-HT3 receptor antagonist tropisetron (0.3 mg/kg) treatm
51 urthermore, this mutation also converted the 5-HT3 receptor antagonist/very weak partial agonist, apo
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