ライフサイエンスコーパス: 5-HT1A agonist

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1 s previously been reported for ipsapirone, a 5-HT1A agonist.

2 , but blunted by 10 microM buspirone, a weak 5-HT1A agonist.

3 t serotonergic neurotransmission such as the 5-HT1A agonist 8-hydroxy-2-(di-n-propyl-amino)tetralin (

4 cal microdialysis perfusion of the selective 5-HT1A agonist 8-hydroxy-2-(di-n-propylamino) tetralin (

5 ctions of AVP in combination with either the 5-HT1A agonist 8-hydroxy-2-(di-n-propylamino) tetraline

6 [WAY100635] 0.5 mg/kg, intravenous) and the 5-HT1A agonist 8-hydroxy-2-(di-n-propylamino)tetralin) [

7 Treatment with the 5-HT1A agonist 8-hydroxy-2-(di-n-propylmino)tetralin (8-

8 However, the 5-HT1A agonist 8-hydroxy-di-propylaminotetralin (8-OH-DP

9 onist NAN-190 (1 microM) and mimicked by the 5-HT1A agonist 8-OH-DPAT (1 microM).

10 Presession treatment with the 5-HT1A agonist 8-OH-DPAT (subcutaneous injections at a d

11 pharmacological approach, we found that the 5-HT1A agonist 8-OH-DPAT also potentiated the antidepres

12 Administration of low doses of the selective 5-HT1A agonists 8-OH-DPAT (5-20 micrograms/kg) and ipsap

13 ts (CP-93,129 and anpirtoline), but not by a 5-HT1A agonist (8-OH-DPAT), indicating that 5-HT1B recep

14 before and after unilaterally dialyzing the 5-HT1A agonist (+/-)-8-hydroxy-2-(dipropylamino)-tetrali

15 +/- 13 % of control (P < 0.001), as did the 5-HT1A agonist, 8-OH-DPAT (52.5 +/- 17 %, P < 0.001) and

16 complex (MS/DB) following injections of the 5-HT1A agonist, 8-OH-DPAT, into the median raphe nucleus

17 Next, a 5-HT1A agonist, 8-OH-DPAT, was coadministered with CP-15

18 rats (N = 10) received either saline or the 5-HT1A agonist, 8-OHDPAT (0.05 mg/kg) 20 min prior to a

19 ns DA release, rats were pretreated with the 5-HT1A agonist, 8-OHDPAT.

20 Microinjections of the selective 5-HT1A agonist, (+)-8-hydroxy-dipropylaminotetralin hydr

21 The combined treatment with FGF-2 and the 5-HT1A agonist also synergistically increased FGFR1 and

22 se autoreceptor preferring treatments with a 5-HT1A agonist and antagonist can strongly modify the be

23 in response to intravenous administration of 5-HT1A agonist and antagonist drugs.

24 with fibroblast growth factor 2 (FGF2) and a 5-HT1A agonist, and dependent on the heteroreceptor inte

25 nding autoradiography using [3H]8-OH-DPAT, a 5-HT1A agonist, and in situ hybridization using radiolab

26 effect of a novel, high affinity serotonin (5-HT1A) agonist, BAY X3702, in a rat model of acute subd

27 Systemic application of the somatodendritic 5-HT1A agonist BMY 7378 had a significantly greater supp

28 se results imply a potential clinical use of 5-HT1A agonists for post-SCI respiratory disorders.

29 -hydroxy-dipropylaminotetralin (8-OHDPAT), a 5-HT1A agonist, has anxiolytic properties.

30 mic administration of either full or partial 5-HT1A agonists increases neuroendocrine responses and t

31 In vitro and in vivo studies also revealed a 5-HT1A agonist induced phosphorylation of FGFR1 and extr

32 However, 3a and 3b fully substituted for the 5-HT1A agonist LY293284 ((-)-(4R)-6-acetyl-4-(di-n-propy

33 he [35S]GTPgammaS labeling stimulated by the 5-HT1A agonist (R)-8-hydroxy-2-dipropylaminotetralin [(R

34 ed when the 5-hydroxytryptamine 1A receptor (5-HT1A) agonist (R)-(+)-8-hydroxy-2(di-n-propylamino)tet

35 78 alone and in combination with these other 5-HT1A agonists reduced copulatory behavior, though not

36 Hypothalamic injection of a 5-HT1a agonist stimulated aggression in female hamsters

37 tration of either azapirone or aminotetralin 5-HT1A agonists, such as 8-hydroxy-2-dipropylaminotetral

38 r disruption compromises the ability of this 5-HT1A agonist to antagonize the feeding suppressant act

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