1             Pretreatment of ganglia with the 5-HT3 receptor agonist 1-m-(chlorophenyl) biguanide (m-C

2 ione (DNQX) on the excitatory actions of the 5-HT3 receptor agonist 1-phenylbiguanide (PBG) were stud

3    This initial response was mimicked by the 5-HT3 receptor agonist, 2-methyl-5-HT, whereas 5-methoxy

4         The effect was mimicked by two other 5-HT3 receptor agonists, 2-methyl-5-HT and m-chloropheny

5 data are consistent with the hypothesis that 5-HT3 receptor agonists activate DVMN neurones partly by

6                                     Only the 5-HT3 receptor agonist altered the quality of the lordos

7                                            A 5-HT3 receptor agonist CPBG (1 microM), mimicked the unm

8 urrent by low concentrations of bath-applied 5-HT3 receptor agonists is compatible with the cyclic mo

9 sed with the 5-HT1A receptor agonist and the 5-HT3 receptor agonist, m-chlorophenyl-biguanide (mCPBG;

10                                          The 5-HT3 receptor agonist, m-chlorophenylbiguanide (mCPBG),

11                               The effects of 5-HT3 receptor agonists on cortical circuit response pro

12 retic application of PBG, a highly selective 5-HT3 receptor agonist, significantly increased activity

13  that phenyldiguanide (later recognized as a 5-HT3 receptor agonist) stimulated the firing of C-fibre

14 2-methylserotonin (100 microg kg-1, i.a.), a 5-HT3 receptor agonist, stimulated eleven of twelve affe

15         The rank order of potency of all the 5-HT3 receptor agonists tested in the PI assay as compar

16  by white matter stimulation were reduced by 5-HT3 receptor agonists, whereas the frequency of sponta