ライフサイエンスコーパス: 8-MOP

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1 scintillation counting, indicated that [14C]-8-MOP binding was specific to the apoprotein of P450 2B1

2 After exposure to [14C]-8-MOP and [14C]-5-MOP, SDS-PAGE and HPLC experiments, fo

3 ed reconstituted cytochrome P450 (P450) 2B1, 8-MOP was found to be the most potent (KI, kinact, and p

4 and MAs in repair-competent cells 24 h after 8-MOP/UVA treatment, there was little repair of 8-MOP-IC

5 le/cm2 UVA, the lowest dose of S-59, AMT and 8-MOP required to reduce the number of T cells to the li

6 , three and five products were identified as 8-MOP- and S59-MAs, respectively, and the yields of MAs

7 Although the yields of MAs induced by 8-MOP and S59 were lower than those of the respective IC

8 oximately 100 fold more than that induced by 8-MOP.

9 xypsoralen (8-OH-P), 4',5'-dihydro-8-MOP (DH-8-MOP), and psoralen (P)] tested as mechanism-based inac

10 ), 8-hydroxypsoralen (8-OH-P), 4',5'-dihydro-8-MOP (DH-8-MOP), and psoralen (P)] tested as mechanism-

11 the level of adduct formation (S-59 > AMT > 8-MOP).

12 After treatment with 75 micromol/L 8-MOP and 1.9 Joules/cm2 UVA, only low levels of IL-8 we

13 is incorporated into the 8-methoxypsoralen (8-MOP) and psoralen (P) dihydrodiol metabolites when the

14 the psoralen derivatives, 8-methoxypsoralen (8-MOP) is conventionally applied for psoriasis therapy,

15 Three psoralens, 8-methoxypsoralen (8-MOP), 4'-aminomethyl 4,5', 8-trimethylpsoralen (AMT),

16 -methoxypsoralen (5-MOP), 8-methoxypsoralen (8-MOP), 5-hydroxypsoralen (5-OH-P), 8-hydroxypsoralen (8

17 r assessing the repair of 8-methoxypsoralen (8-MOP)-induced DNA ICLs, as well as monoadducts (MAs), i

18 n experiments supported initial oxidation of 8-MOP and P to an epoxide which can react with some nucl

19 OP/UVA treatment, there was little repair of 8-MOP-ICLs and -MAs in xeroderma pigmentosum, complement

20 les/cm2 UVA and 150 micromol/L S-59, AMT, or 8-MOP induced 12.0 +/- 3.0, 6.0 +/- 0.9, and 0.7 psorale

21 The addition of psoralen (8-MOP) prior to administration of UVA results in substan

22 ocal evidence supporting the notion that the 8-MOP photoadducts are substrates for nucleotide excisio

23 The yields of the three 8-MOP-MAs were 7.6-2.2, 1.9-9.9, and 7.2-51 per 10(6) nu

24 and MAs induced in human cells exposed with 8-MOP or S59 and UVA light.

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