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1 hemotherapy was applied after oral intake of 8-methoxypsoralen (0.6 mg/kg body weight) 5x weekly duri
3 t a single 18O atom is incorporated into the 8-methoxypsoralen (8-MOP) and psoralen (P) dihydrodiol m
6 furanocoumarins [5-methoxypsoralen (5-MOP), 8-methoxypsoralen (8-MOP), 5-hydroxypsoralen (5-OH-P), 8
7 ution technique, for assessing the repair of 8-methoxypsoralen (8-MOP)-induced DNA ICLs, as well as m
8 ponse to 4-nitroquinoline-N-oxide (4NQO) and 8-methoxypsoralen (8MOP), compared with WRN-proficient c
10 s a 4-log reduction in PCR sensitivity, with 8-methoxypsoralen activity facilitated by UV there was b
11 variation in bioavailability of Methoxsalen (8-methoxypsoralen) after ingestion of the standard dose
13 ovel photocross-linking technique using free 8-methoxypsoralen and DNA furan-side monoadducts plus lo
15 s cognate sequence (E-box) was unimpaired by 8-methoxypsoralen and that cross-linking occurred in nor
16 ubated with serial dilutions of hypericin or 8-methoxypsoralen and then stimulated with the mitogen C
17 olated white blood cells to photoactivatable 8-methoxypsoralen and ultraviolet A radiation, is used c
19 ondrial membranes, X-rays, UVB (290-320 nm), 8-methoxypsoralen and UVA, vitamin K3, anti-Fas antibody
24 ivalent, psoralen plus ultraviolet A-induced 8-methoxypsoralen-DNA adduct formation and p53 protein a
25 here is limited information on the effect of 8-methoxypsoralen dose alterations on phototoxicity with
27 ) were significantly reduced with increasing 8-methoxypsoralen dose from conventional dose minus 10 m
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