ライフサイエンスコーパス: A(1) agonist

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1 ty and an enhanced anti-adrenergic effect by A1 agonist.

2 lication of cannabinoid but not by adenosine A1 agonists.

3 inating effects of cannabinoid, GABA(B), and A(1) agonists.

4 n the ocular hypotensive action of adenosine A(1) agonists.

5 reconditioning of rabbits with the selective A(1) agonist 2-chloro-N(6)-cyclopentyladenosine (CCPA; 1

6 clopentyladenosine (CCPA) (10 muM; selective A1 agonist), 5'-N-ethylcarboxamide adenosine (NECA) (10

7 electivity of interactions between adenosine A1 agonist and antagonist and ethanol was further confir

8 DAMGO (mu-agonist) antinociception and CPA (A1-agonist) antinociception were blocked not only by nal

9 ler boutons were more sensitive to adenosine A1 agonist but less sensitive to CB1 agonist, while bout

10 Studies have shown that adenosine A(1) agonists can lower IOP in rabbits, mice, and monkey

11 Both the selective A1 agonist CCPA (1 microm) and the selective A2A agonist

12 -fibres was mimicked by either the selective A1 agonist CCPA (1 microm) or the selective A2A agonist

13 the increase in FVC evoked by the selective A1 agonist CCPA (2-chloro-N6-cyclopentyladenosine; i.a.

14 e exposure to ischemia, adenosine, adenosine A1 agonist CCPA, or A3 agonist resulted in less myocyte

15 c cells killed in A3 agonist-treated than in A1 agonist (CCPA, 10 nM)-treated myocytes (ANOVA and pos

16 The addition of the adenosine A(1) agonist CHA stimulates the secretion of MMP-2 from

17 ated topically in one eye with the adenosine A(1) agonist CHA.

18 with the transient receptor potential (TRP) A1 agonist, cinnamaldehyde, with ozone having no effect

19 )-cyclopentyl adenosine (CCPA), an adenosine A(1) agonist, does not.

20 ic transmission by adenosine or an adenosine A(1) agonist in rat hippocampal slice.

21 ge/reperfusion injury, but not the adenosine A(1) agonist, increased cAMP levels in cultured sinusoid

22 A3 agonist, and it completely abrogated the A1 agonist-mediated cardioprotection.

23 to investigate the actions of the adenosine A(1) agonist N(6)-cyclohexyladenosine (CHA) on conventio

24 In contrast, neither the A(1) agonist N(6)-cyclopentyladenosine nor the A(2A) ago

25 erioles from mouse kidney, adenosine and the A1 agonist N(6)-cyclohexyladenosine, when added to the b

26 d in the presence of EtOH, adenosine, or the A1 agonist N(6)-cyclopentyladenosine.

27 MTX at 200-2,000 nM or the adenosine A1 agonist N5-cyclopentyl adenosine (CPA) (10(-12) to 10

28 ition of the evoked EPSC was mimicked by the A(1) agonist N6-cyclohexyladenosine (100 nm) but not by

29 The selective A(1) agonist N6-cyclopentyladenosine (CPA, 30.0 micromol

30 dicated by results from experiments with the A1 agonist N6-cyclopentyladenosine (1 microM) which caus

31 effects were reversed by selective adenosine A1 agonist N6-cyclopentyladenosine (CPA) or A2A agonist

32 The A1 agonist N6-cyclopentyladenosine (CPA), the A2a agonis

33 nephrons with adenosine A1 receptor blocker, A1-agonist, or 5'-nucleotidase inhibitor.

34 CV-1808, the A2a agonist CGS-21680, and the A1 agonist R-PIA were evaluated.

35 antinociception induced by mu, alpha 2, and A1 agonists requires the physical presence of multiple r

36 e diacylglycerol accumulation induced by the A1 agonist than by the A3 agonist, and it completely abr

37 by combining 5'-nucleotidase inhibitor with A1-agonist to determine whether TGF requires adenosine t

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