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1 tric oxide synthase inhibitors and adenosine A1 receptor agonists.
3 te receptor antagonist MK-801; the adenosine A1 receptor agonist 2-chloro-N6-cyclopentyladenosine (CC
7 stantial increase in CoRCF and CoVC, but the A1 receptor agonist 2-chloro-N6-cyclopentyladenosine had
8 ) receptor agonist baclofen or the adenosine A1 receptor agonist 2-chloroadenosine, short-term synapt
10 ould be demonstrated only with the selective A1-receptor agonist 2-chloro-N6-cyclopentyladenosine and
11 ischemic preconditioning, but the adenosine A(1) receptor agonist, 2-chloro-N(6)-cyclopentyladenosin
12 cked by exogenous adenosine or the selective A1 receptor agonist, 2-chloro-N6-cyclopentyl adenosine.
13 5.5% with SfA; P<0.001), CCPA (the adenosine A1-receptor agonist, 200 nmol/L) (24.9+/-4.5% versus 54.
14 These data are consistent with adenosine A1 receptor agonist actions on REM sleep mediated throug
15 the antiinflammatory effects of intrathecal A1 receptor agonists and AP-5 were reversed by an adenos
16 ine, N(6)-cyclopentyladenosine (an adenosine A(1) receptor agonist), and 2-[p-(2-carboxyethyl)]phenyl
19 or sodium nitroprusside (SNP), the adenosine A1 receptor agonist CCPA (2-chloro-N6-cyclopentyladenosi
20 receptor agonist APEC, but not the adenosine A1 receptor agonist CHA, attenuated c-Fos induction in c
21 lices containing the PnOc incubated with the A1 receptor agonist, cyclohexaladenosine (10(-8) M).
22 rats were treated IT with the selective Ado A1 receptor agonist cyclohexyladenosine (CHA) or vehicle
23 d adult rat ventricular myocytes (ARVM), the A(1) receptor agonist cyclopentyladenosine (CPA) inhibit
26 lts support development of partial adenosine A1-receptor agonists for the treatment of chronic heart
28 nistration of the highly selective adenosine A1 receptor agonist, GR79236 (3-100 microg/kg) had a dos
30 sites was signficantly reduced by adenosine A1 receptor agonists injected through intrathecal cathet
32 derable literature to suggest that adenosine A1 receptor agonists may have anti-nociceptive effects,
34 hwork cells to the addition of the adenosine A(1) receptor agonist N(6)-cyclohexyladenosine (CHA).
35 rived from the IMHV of untrained chicks, the A(1) receptor agonist N(6)-cyclohexyladenosine (CHA; 10
36 ylxanthine (CPDX; 5 microM and 50 microM) or A(1) receptor agonist, N(6)-cyclopentyladenosine (CPA; 5
37 ystemic injection of the selective adenosine A1 receptor agonist, N(6)-cyclohexyladenosine (CHA; 0.3
38 Intrastriatal microinfusion of adenosine A1 receptor agonist N6-cyclohexyladenosine (CHA) and ant
40 cholinergic neurons, the selective adenosine A1 receptor agonist N6-cyclohexyladenosine, administered
41 was mimicked by perfusion with the adenosine A1 receptor agonist N6-cyclopentyladenosine and prevente
43 tudied with the use of a selective adenosine A1 receptor agonist, N6-cyclohexyl-2'-O-methyladenosine
44 In Experiment 1, the selective adenosine A1 receptor agonist, N6-cyclopentyladenosine (CPA), or s
45 cement of [3H]DPCPX binding by the selective A1-receptor agonist, N6-p-sulfophenyladenosine (SPA), yi
46 ts of capadenoson (CAP), a partial adenosine A1-receptor agonist, on left ventricular (LV) function a
48 )-adenosine (R-PIA [50 nmol/L], an adenosine A1 receptor agonist), or 4 beta-phorbol 12-myristate 13-
52 tion were reduced in mutants treated with an A(1) receptor agonist, whereas an A(2A) receptor agonist
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