ライフサイエンスコーパス: A-cell

1 A cell (325 muL) with distant side walls was formed by p

2 A cell adhesion assay was used to determine the proporti

3 A cell adhesion assay, a cell-binding assay, receptor bl

4 A cell autonomous mechanism with sustained cAMP/PKA acti

5 A cell autonomous molecular oscillator, consisting of in

6 A cell autonomous role for Klf6 in endoderm and hepatic

7 A cell becomes senescent when one or more of its telomer

8 A cell can be thought of as a highly sophisticated micro

9 A cell cluster at the anterior tip of planarian head bla

10 A cell culture model demonstrates that cells on chemFN a

11 A cell culture model of freshly isolated corneal stromal

12 A cell culture model of freshly isolated rabbit corneal

13 A cell culture model was used to investigate the role of

14 A cell culture study evaluated the effect of MC on MMP p

15 A cell culture system consisting of mouse S17 stromal ce

16 A cell culture system has been established to detect the

17 A cell culture system has been used to determine the rel

18 A cell cycle analysis revealed that the cellular toxicit

19 A cell cycle regulatory module in islets predicts diabet

20 A cell death assay revealed that apoptotic-related DNA f

21 A cell death detection ELISA kit was used to evaluate HR

22 A cell delivery strategy was investigated that was hypot

23 A cell distribution experiment showed that flk-1:fas fus

24 A cell dose escalation was conducted, starting at 10(7)

25 A cell envelope-associated proteinase gene (prtH) was id

26 A cell fraction separated by density gradient centrifuga

27 A cell fraction that would today be termed "the nuclear

28 A cell fractionation experiment using cisplatin-treated

29 A cell free system was developed, and a succession of co

30 A cell fusion assay was used to assess functional activi

31 A cell fusion complementation assay in fibroblasts from

32 A cell growth inhibition assay was used to measure varia

33 A cell is a minifactory in which structures and molecule

34 A cell kinetics model is developed to describe the evolu

35 A cell line (410.4) derived from a mouse mammary adenoca

36 A cell line (AmE-711) has been established that is compo

37 A cell line (EKT2) was established from an affected kidn

38 A cell line derived from one such tumor expresses vGPCR

39 A cell line expressing the membrane-bound ADAM15 without

40 A cell line lacking functional copies of both NDUFA9 all

41 A cell line representative of human high-grade serous ov

42 A cell line selected for stable resistance to geldanamyc

43 A cell line that is adherent in serum-containing media a

44 A cell line that is established from lung metastases rel

45 A cell line was cloned from a single progenitor cell.

46 A cell line, KG-1, behaves similarly.

47 A cell maintains normal centrosome numbers by coupling c

48 A cell may arrest division via checkpoint activation to

49 A cell membrane can be considered a liquid-phase plane i

50 A cell membrane protein named ATP-binding cassette trans

51 A cell membrane-permeable fusion peptide derived from th

52 A cell metabolic labeling experiment can be completed in

53 A cell permeant peptide derived from this sequence displ

54 A cell plated on a two-dimensional substrate forms adhes

55 A cell population with progenitor-like phenotype (CD45-C

56 A cell printer that can print gels, single cells and cel

57 A cell proliferation assay revealed potent functional ac

58 A cell proliferation assay revealed that Notch signaling

59 A cell regulates the number, size, and kind of each orga

60 A cell responds to a chemotactic signal by activating ac

61 A cell slurry containing both cell types added to silico

62 A cell surface biotinylation assay and live cell confoca

63 A cell surface cytokine CD40 ligand (CD40L) enhances TF

64 A cell surface fraction from S. epidermidis 0-47 grown i

65 A cell suspension model was designed to validate the imp

66 A cell synchronization protocol was established in which

67 A cell system in which the impact on transcription can b

68 A cell therapeutic approach with a tissue-engineered bio

69 A cell tracing experiment was included to observe the pr

70 A cell type expressing this receptor is the osteoblast,

71 A cell type-centric interface allows the identification

72 A cell undergoing mitosis is presented with a potentiall

73 A cell viability assay showed that alpha-tocopheryl succ

74 A cell viability assay showed that IC(50)s for human col

75 A cell viability assay was used to determine the sensiti

76 A cell viability screen identified Abeta(31-42) as the m

77 A cell voltage of 1.035V and power density of 3.154mWcm(

78 A cell wall-associated beta-glucosidase activity that re

79 A cell's ability to regulate gene transcription depends

80 A cell's ability to tolerate DNA damage is directly conn

81 A cell's decision to growth arrest, apoptose, or differe

82 A cell's decision to undergo meiosis is regulated by mul

83 A cell's epigenome arises from interactions among regula

84 A cell's perception of morphogen concentration is though

85 A cell's structural and functional characteristics are d

86 A cell-based assay confirmed that all 4 patients, but no

87 A cell-based assay was developed by fusing a green fluor

88 A cell-based bioassay was generated that detects SMN2 pr

89 A cell-based biological pacemaker is based on the differ

90 A cell-based fluorometric assay was used to measure intr

91 A cell-based functional assay suggests that S100A6 modul

92 A cell-based high-throughput screen to identify small mo

93 A cell-based method for the detection of nanomolar conce

94 A cell-based mortality model was developed and refined.

95 A cell-based MTT (3-[4,5-dimethylthiazol-2-yl]-2,5-diphe

96 A cell-based pharmacodynamic explanation was suggested b

97 A cell-based phenotypic screen for inhibitors of biofilm

98 A cell-based readout system was created to functionally

99 A cell-based receptor trafficking assay was used to furt

100 A cell-based reglucosylation assay was used to investiga

101 A cell-based reporter assay linking LasR function with b

102 A cell-based screen of chemical libraries was carried ou

103 A cell-based screen performed in our laboratories identi

104 A cell-based standard was developed to compare the COBAS

105 A cell-based, drug-screening assay is also described, wh

106 A cell-based, functional high-throughput screen of appro

107 A cell-binding assay and fluorescence cell staining were

108 A cell-binding assay was performed to quantify the abili

109 A cell-cell fusion assay revealed that representative en

110 A cell-cell signalling system mediated by the fatty acid

111 A cell-coupling analysis for Pinus sylvestris showed hig

112 A cell-culture system was developed that allows identifi

113 A cell-culture-based system was designed previously to m

114 A cell-cycle-dependent shuffling of multiple centromeric

115 A cell-free (axenic) culture system was developed, which

116 A cell-free assay of prion protein misfolding was used t

117 A cell-free assay that reproduces transport between the

118 A cell-free assay was developed for the orphan nuclear r

119 A cell-free cellulosomal GH25 lysozyme may provide a bac

120 A cell-free conversion assay was used to measure the int

121 A cell-free conversion reaction approximating physiologi

122 A cell-free ECM was prepared from cultured femoral marro

123 A cell-free in vitro translation assay containing human

124 A cell-free membrane-fusion system demonstrates that eng

125 A cell-free mRNA decay system reconstitutes TPA-induced

126 A cell-free plasma gap representing the ECG could be ima

127 A cell-free Ser(20) phosphorylation site assay was used

128 A cell-free system depleted of intersectin failed to sup

129 A cell-free system derived from Xenopus eggs has been es

130 A cell-free system that reconstructs these events has re

131 A cell-free system was used to evaluate the activation o

132 A cell-free system was used.

133 A cell-free translation system showed that the mutated p

134 A cell-free translation-based small molecule screen was

135 A cell-free, mate-paired library provided single DNA mol

136 A cell-impermeable peptide PC inhibitor blocks the appea

137 A cell-intrinsic timer helps control when rodent oligode

138 A cell-matrix composite was constructed by applying fibr

139 A cell-mediated Th1 pathway of cytokine expression is ev

140 A cell-membrane fraction isolated from Escherichia coli

141 A cell-penetrant peptide that disrupts this interaction

142 A cell-penetrating pepducin, X1/2pal-i3, which targets t

143 A cell-penetrating peptide (CPP)-morpholino oligonucleot

144 A cell-penetrating peptide containing this sequence comp

145 A cell-penetrating peptide-modified fusogenic liposomal

146 A cell-penetrating, anti-DNA, lupus autoantibody, 3E10,

147 A cell-permeable alpha7 helix peptide competitively inhi

148 A cell-permeable decoy peptide corresponding to the same

149 A cell-permeable ester of alpha-ketoglutarate reversed t

150 A cell-permeable Grb10 SH2 domain similarly interfered w

151 A cell-permeable NBD peptide blocked association of NEMO

152 A cell-permeable peptide aminofluorocoumarin substrate d

153 A cell-permeable peptide conjugated to the Akt-binding d

154 A cell-permeable peptide corresponding to this region di

155 A cell-permeable peptide inhibitor of GSK-3beta, and lit

156 A cell-permeable peptide inhibitor specific for the zeta

157 A cell-permeable peptide that contained a polyproline se

158 A cell-permeable peptide that selectively disrupted NF-k

159 A cell-permeable peptide that uncouples G alpha(s) from

160 A cell-permeable PKCzeta pseudosubstrate peptide inhibit

161 A cell-permeable TIRAP blocking peptide attenuated LPS-i

162 A cell-permeant Ca(2+) chelator reduced the CDDO-induced

163 A cell-permeant inhibitor based on the PKC-binding seque

164 A cell-permeant inhibitor of inositol trisphosphate rece

165 A cell-permeant peptide that blocks binding of PAK to PI

166 A cell-permeant TAT-tagged peptide encompassing the seco

167 A cell-permeant version of the peptide encompassing amin

168 A cell-permeant, bioactivatable Ins(3,4,5,6)P4 analog el

169 A cell-permeant, phosphoinositide-binding peptide attenu

170 A cell-proliferation assay was used to determine the res

171 A cell-rounding phenotype associated with the transient

172 A cell-scaled microbead system was used to analyze the f

173 A cell-sparse gap, observed within layer III, was not fi

174 A cell-specific regulation of redox state thus balances

175 A cell-specific targeting peptide, RGD-4C which binds al

176 A cell-surface protein of approximately 82 kd recognized

177 A cell-to-surface attachment assay shows that addition o

178 A cell-type-specific deletion of Tbx1 in osteochondro-pr

179 A cell-wall/membrane (CW/M) antigen from the vaccine str

180 A cells contain heavy chain DJ rearrangements.

181 Extracts of hPMS2-deficient HEC-1-A cells, which express hMLH1, hMLH3, and hPMS1, are only

182 as agonistic with ERalpha in (uterine) HEC-1-A cells.

183 nd cell death caused by tBHP in neuronal N(2)A cells (EC(50) in nm range).

184 ely bound to TNFR1 when overexpressed in N(2)A cells, with DENN/MADD abrogating TNFR1 binding to TRAD

185 inoculated with the nontumorigenic R-3327-5'A cells but a robust growth pattern in sponges inoculate

186 G hepatocellular carcinoma cell lines than A/A cell lines.

187 uced NFkappaB activation is delayed in MEF(A/A) cells and in an MCF-7 cell line that is stably transf

188 d that this inhibition is prevented in MEF(A/A) cells in which the phosphorylation site, Ser-51 in th

189 Their islets show marked alpha (A)-cell hyperplasia, suggesting a possible defect in glu

190 ned the proliferation dynamics of B1, C, and A cells in vivo.

191 of B1 cells, C cells divide three times and A cells once, possibly twice.

192 until they encounter and attach to anterior A cells.

193 (P) compartment cells and induces Anterior (A) cells to create a developmental organizer at the AP c

194 experimental models were used: model A, B10.A cells presensitized to B6 (a anti-b cells) were inject

195 long intervals, we found a richer behavior: A cell swimming in the dark stochastically switches betw

196 uptake and content were reduced in betaIRS1-A cells.

197 tative Hh receptor expressed specifically by A cells, reduced Fu*.

198 Following delivery of E6 siRNA to C33-A cells using oligofectamine or His6 reducible polymers,

199 inar (chymotrypsin), and islet (chromogranin A) cell markers was performed to analyze the tissues.

200 t Madin-Darby canine kidney, HT29, and Clone A cells and from solutions of purified spectrin and alph

201 a dominant negative RhoA (N19RhoA) in clone A cells inhibited alpha6beta4-dependent membrane rufflin

202 d its enhanced detergent solubility in Clone A cells that harbor a defective alpha-catenin.

203 or hydrolysis of the carboxyl ester in clone A cells, although in primary human hepatocytes, both Cat

204 Time-lapse videomicroscopy of clone A cells on laminin-1 revealed that their migration is ch

205 he alpha6beta4-mediated interaction of clone A cells with laminin promoted the translocation of RhoA

206 After C cells, Doublecortin(+) A cells were the second-most common dividing cell type i

207 Ege A overexpressing and ege A(-) cells display similar phenotypes, suggesting that a

208 n of a fully functional cAR1-YFP enables ege A(-) cells to form loose aggregates, but cAR1-YFP/ege A(

209 s to form loose aggregates, but cAR1-YFP/ege A(-) cells are still unable to express GP80, suggesting

210 the present study the association of ephrin-A cell surface ligands and EphA receptor tyrosine kinase

211 ly ET(A)(-/-) --> (+/+) chimeric embryos, ET(A)(-/-) cells are excluded from the caudoventral aspects

212 cardiac outflow tract and great vessels, ET(A)(-/-) cells are excluded from the walls of the develop

213 HbF together with adult hemoglobin (HbA) (F+A+ cells).

214 sults were obtained upon correction of an FA-A cell line, with a reciprocal increase in the half-life

215 d cross-links and shows reduced levels in FA-A cell nuclei.

216 FA-A cells also showed defects in both cell-cell and cell-m

217 ma* are not only significantly reduced in FA-A cells but also in FA-B, FA-C and FA-D cells (i.e. in a

218 toration of alphaIISp levels to normal in FA-A cells corrects these deficiencies.

219 Knocking down mu-calpain by siRNA in FA-A cells restored levels of alphaIISp to normal and rever

220 entation group D) cells but not in mutant FA-A cells.

221 was able to rescue the adhesion defect of FA-A cells.

222 ion of FANCA readily restored adhesion of FA-A cells.

223 Fanconi anemia, complementation group A (FA-A) cells, there is a deficiency in these incisions.

224 Fanconi anemia, complementation group A (FA-A) cells, where alphaIISp levels are 35-40% of normal, I

225 lls (AA4.1(+)/natural killer 1.1(-) Fraction A cells) and cells capable of generating pro-B colonies

226 Wnt1-Cre-activated diphtheria toxin fragment-A cell-killing system was employed.

227 A expression in inflammatory cells and FXIII-A(+) cell numbers were significantly increased in NP.

228 , CD19+, CD3+, CD34+, CD33+, and glycophorin A+ cells and granulocytes were isolated from the periphe

229 A special target of the granzyme A cell death pathway is an endoplasmic reticulum-associa

230 on group O or B cells of infants or on group A cells of adults.

231 syndrome but not Xeroderma pigmentosum group A cells providing evidence that ssDNA regions generated

232 In Group A, cell-free polymerase assays confirm that mutations in

233 ranules in the hyperplastic and hypertrophic A-cells, along with morphological evidence of high rates

234 yed-type hypersensitivity was dependent on I-A+ cells and was genetically restricted.

235 produced if mice were deficient in CD4+ or I-A+ cells during the early interval (< or =30 h) followin

236 bly caused by a reduction in Slit1 ligand in A cells.

237 t into A cells of the same segment, but into A cells of the same parasegment (segment A6).

238 of, say, segment A5) they transform not into A cells of the same segment, but into A cells of the sam

239 Notably, prolonged stimulation of R-/IR-A cells with insulin, but not with IGF-II, targeted the

240 Using R-/IR-A cells, we discovered that insulin evoked significant I

241 for the production of glucagon in the islet A-cells.

242 replication that involves the protein kinase A cell-signaling pathway and a possible down-regulation

243 and viability of TNBC cells, but not Luminal A cells, and (3) is associated with a poor prognosis in

244 In MAT A: cells responding to alpha-factor, we found that Dnf1,

245 to Balb/C mice inoculated with syngenic meth/A cells demonstrated statistically significant dose-depe

246 lished in mechanically stimulated mouse MRTF-A(-/-) cells or upon inhibition of CREB-binding protein

247 ecific feature of MYCN-amplified cells (MYCN-A cells) that represent a particularly aggressive subcla

248 ed in motility (myosin light chain 1, myosin A), cell morphology (PhIL1), and host cell invasion (api

249 iate progenitors (C cells), and neuroblasts (A cells) in the V-SVZ and the number of times these cell

250 ing (type C) cells, chains of young neurons (A cells), and blood vessels.

251 ere it induces engrailed in a narrow band of A cells.

252 under conditions that inhibit the growth of A cells and normal hepatocytes.

253 A 30% cell suspension of A-cells or B-cells was used for haemagglutination on the

254 PC-A cells were capable of further differentiation to periv

255 PC-A cells were not osteogenic or adipogenic under standard

256 kinase pathway when transfected into Phoenix-A cells (derivatives of 293 cells), similar to many GCKs

257 s (Y iB), (iv) stable PR-B plus inducible PR-A cells (B iA), and (v) stable PR-A plus inducible PR-B

258 e control cells (Y iNull), (ii) inducible PR-A cells (Y iA), (iii) inducible PR-B cells (Y iB), (iv)

259 comB has a cell-autonomous role in prestalk A cell differentiation.

260 Activated CD11b+SRA+ (scavenger receptor A) cells were found in blood and cervical lymph node and

261 In ngn3(-/-) mice, glucagon secreting A-cells, somatostatin secreting D-cells, and gastrin sec

262 In contrast, all serotype A cells harvested from brain tissue bound the same O-ace

263 erall rate of accumulation of C3 on serotype A cells that was strongly correlated with increasing ant

264 In phagocytosis assays, using serotype A cells and resident murine peritoneal macrophages, rim-

265 tributes to CAG*CTG tract instability in SVG-A cells.

266 In contrast, the A cell population expands precociously, mostly fails to

267 n that localizes to the cell membrane in the A cell and physically interacts with the Notch receptor.

268 The A cells play a crucial role in night-time (scotopic) vis

269 boundaries, organises the pattern of all the A cells.

270 e present study we aimed to characterize the A cells in human retina.

271 tentials and on the postsynaptic IPSP in the A-cell was occluded by previous paired (but not unpaired

272 acellular Ca2+ in the sensitization of toxin A/cell-surface interactions.

273 Type A cells appear to correspond to type II neurones of Namb

274 Type A cells expressed essentially pure TTX-sensitive (TTX-S)

275 Type A cells were readily detected only in animals in which t

276 over the overall numerically dominating Type A cells, which in turn leads to an acceleration of the o

277 e number of action potentials evoked in type A cells by AF test stimulation and a concomitant increas

278 zole, seven of ten breast cancer lines (type A cells) arrested in mitosis, whereas the other three (t

279 In vivo, migratory neuroblasts (type A cells) and putative precursors (type C cells) are in i

280 e conductances of type B cells, but not type A cells.

281 ver, Ba2+ increased the excitability of type A cells but not B cells.

282 The somata of type A cells were variable in shape (20 x 12 micrometer) whil

283 may be necessary for the production of type A cells.

284 Pure type A cells could migrate in chains but did not give rise to

285 interactions among the more stationary Type A cells, but unexpectedly accelerates again when these A

286 driven synaptically by AF stimulation (type A cells) were concentrated in the deep half of the GCL a

287 ge specific and differ in the number of UCYN-A cells involved.

288 repair-deficient xeroderma pigmentosum (XP)-A cells that stably express photoproduct-specific photol

289 XP-A cells are defective in overall genome NER while CS-B c

290 XP-A cells were unable to repair these oxidative DNA base l

291 mplex formation was similar in normal and XP-A cells, but was reduced in CS-B cells.

292 PCNA relocated as compared to normal and XP-A cells.

293 ulation of active p53 in repair-deficient XP-A cells occurred despite the lack of DNA strand break in

294 icient PCNA complex formation observed in XP-A cells following oxidative damage suggests that formati

295 e repair of oxidative DNA base lesions in XP-A cells in vivo and in vitro.

296 significantly following UV irradiation in XP-A cells in which sumoylation of XPC does not occur.

297 However, unlike in XP-A cells, the absence of pol eta expression resulted in a

298 zation observed was comparable to that of XP-A cells deficient in nucleotide excision repair, a recog

299 normal and xeroderma pigmentosum group A (XP-A) cells.

300 in nucleotide excision repair-deficient (XP-A) cells but were not found in repair-proficient cells.