ライフサイエンスコーパス: A-fiber

1 A fiber adapter assembly and gas filling setup was desig

2 A fiber optic bead-based sensor array platform has been

3 A fiber optic microsphere-based oligonucleotide array is

4 A fiber optic particle plasmon resonance (FOPPR) immunos

5 A fiber optic probe consisting of a 1.0 mm coherent imag

6 A fiber optic salivary cortisol sensor using a contempor

7 A fiber optic with a ring electrode can also be used to

8 A fiber-based laser with a pulse duration of 435 fs and

9 A fiber-based subdivision successfully separates lobar r

10 A fiber-deleted, propagation-defective rescue plasmid ha

11 A fiber-elastomer composite design with a vastly improve

12 A fiber-optic assay for amplified DNA products has been

13 A fiber-optic biosensor array is described for the simul

14 A fiber-optic biosensor using an aptamer receptor has be

15 A fiber-optic bundle was used for the gas detection and

16 A fiber-optic bundle, positioned approximately 2 mm from

17 A fiber-optic coupled attenuated total reflection (ATR)-

18 A fiber-optic microbial biosensor suitable for direct me

19 A fiber-optic OCT imaging system was used to image the m

20 A fiber-optic periodontal endoscope was developed to aid

21 In addition, the role of afferent A-fiber and C-fiber activation of CVNs was examined.

22 data show, therefore, that primary afferent A-fiber activity can cause neuronal cell death in the do

23 rs, was used to examine the role of afferent A-fibers and C-fibers in the synaptic activation of CVNs

24 ed whether activity in myelinated afferents (A fibers), which use glutamate as a transmitter, can ind

25 been implicated in mechanical allodynia, an A-fiber-selective pharmacological blocker is still lacki

26 data provide evidence that both C-fiber and A-fiber nociceptors may encode high-intensity mechanical

27 conditioning stimulation was as effective as A-fiber stimulation alone.

28 onduction failure in large myelinated axons (A fibers) mirrored the time course of glycogen loss.

29 h force over a large stimulus range for both A-fiber and C-fiber nociceptors.

30 ons in phenotype includes the acquisition by A fibers of neurochemical features typical of C fibers,

31 OF mice) displayed only a mild decrease in C:A-fiber ratio compared with wild-type mice (4.42 compare

32 eficit manifested as a substantially lower C:A-fiber ratio compared with other mammals.

33 nerves, the naked mole-rat had the lowest C:A-fiber ratio ( approximately 1.5:1 compared with approx

34 le P2ry1 neurons are largely fast-conducting A fibers that contact pulmonary endocrine cells (neuroep

35 sed with neurofilament-200 in large-diameter A-fiber neurons in the dorsal root ganglion (DRG).

36 locus coeruleus neurons consist of an early (A-fiber mediated) component and a previously undescribed

37 iber-driven spinal hyperexcitability enables A fibers to gain access to specific spinal circuitry, vi

38 For A-fiber and C-fiber nociceptors, we systemically measure

39 Although large, myelinated group A fibers, in particular Abeta-fibers, have previously be

40 However, A fibers, but not C fibers, in the injured L5 spinal ner

41 iii), A fiber conformation can stabilize a regular spacing of

42 accelerated CAP failure during aglycemia in A fibers, but not in C fibers.

43 ensory neurons including novel expression in A fibers, has a role as a central modulator of tactile s

44 lockade of sodium currents, predominantly in A-fiber neurons of mouse DRGs.

45 Elimination of large A-fiber carotid baroreceptor afferents, during similar c

46 ompared to that during anodal block of large A-fibers in the carotid sinus nerve.

47 ese data indicate that projections of larger A-fiber (type I) carotid baroreceptors are localized pri

48 At both levels, A fibers projected to deeper layers of the dorsal horn t

49 alized in 90% of DRG neurons, including most A-fibers (identified by the presence of neurofilament 20

50 Here we identified two populations of murine A fiber-type sensory neurons that display markedly diffe

51 cholera toxin subunit B (CTB) for myelinated A fibers and isolectin B4 (IB4) for unmyelinated C fiber

52 expression was increased in large myelinated A-fiber DRG neurons, whereas it was decreased in small u

53 nduced ultrastructural changes in myelinated A-fibers.

54 inated C-fibers usually outnumber myelinated A-fibers.

55 psaicin persistently reduces C-fiber but not A-fiber compound action potentials and this effect does

56 trocutaneous stimulation at C-fiber, but not A-fiber, strength produced behavioral signs of secondary

57 This novel A fiber input was polysynaptic in nature and required NM

58 in detectable neuron loss, but activation of A fibers in a previously sectioned sciatic nerve did cau

59 approximately 10%) colabeled with markers of A-fiber neurons.

60 itive cells and a more diverse population of A-fiber neurons, some of which exhibit T-type Ca2+ chann

61 , particularly relating to the processing of A-fiber nociceptive information.

62 Because slowing of A-fiber conduction velocities had also been demonstrated

63 The expansion of the territory of A-fiber afferent-evoked cell death is likely to reflect

64 .3-1.5 times the threshold for excitation of A-fibers.

65 roots and reduced the thermal sensitivity of A-fibers.

66 eport a new method for targeted silencing of A-fibers in neuropathic pain.

67 copic sections, we found that stimulation of A-fibers in an intact sciatic nerve at 10 Hz, 20 Hz, and

68 In addition, mean conduction velocities of A-fibers and C-fibers in vincristine-treated rats were s

69 Low-intensity stimuli or A-fiber input had no effect.

70 g NK1 receptor (NK1R-) received polysynaptic A fiber input.

71 Neurofilament-positive A-fiber neurons innervating the distal urethra had a lar

72 determined via Hoffmann's reflex (H-reflex) (A-fiber), was decreased in diabetic compared with contro

73 nding that a subgroup of capsaicin-sensitive A-fiber nociceptors are insensitive to heat predicts the

74 (300 nm), respectively, revealed significant A fiber input to lamina I NK1R+ neurons that was predomi

75 ntial (AP) activity was recorded from single A-fiber nociceptors that innervated the hairy skin in mo

76 hus, glycine may serve to facilitate tactile A-fiber-mediated information and enhance activity-depend

77 These results suggest that A-fiber nociceptors play a role in the pain and hyperalg

78 response of LC neurons without affecting the A-fiber response component.

79 to the afferent stimulation of CVNs, and the A-fiber GABAergic pathway to CVNs may be more complex th

80 ty is dominated by inputs from low threshold A fibers, whereas nociceptive C-fiber inputs mature grad

81 nce of monosynaptic input from low-threshold A-fibers when preceded by early tissue damage.

82 itude resulted from altering the response to A-fiber inputs to the trigeminal nerve because all stimu

83 itch sensation differs between subjects with A-fiber- versus C-fiber-dominated itch, (3) cowhage acti

84 ime of peak itch sensation for subjects with A-fiber-dominated itch matches the time for peak respons

85 reduces itch in a subgroup of subjects with A-fiber-dominated itch, (2) the time course of itch sens