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1 tential mediator of angiogenesis through the A2B receptor.
2 eptor other than A1, A2A or A3, possibly the A2B receptor.
3 cutely isolated astrocytes is coupled to the A2B receptor.
4 t the effect is likely to be mediated by the A2b receptor.
5 which was prevented by blockade of adenosine A2B receptors.
6  receptors and HMEC-1 preferentially express A2B receptors.
7 xanthine 3 was >13-fold more potent at human A2B receptors.
8 a pA2 value of 8.0 but was not selective for A2B receptors.
9 d A3 receptors, retained moderate potency at A2B receptors.
10 c fibroblast proliferation via activation of A2B receptors.
11 A2A receptors and suggested a major role for A2B receptors.
12 -tertiary butyl phenol was identified as the A2b receptor (a P1 receptor for adenosine).
13                   Adenosine acts through the A2b receptor, a G protein-coupled receptor that couples
14 y of p73 to upregulate the expression of the A2B receptor, a recently characterized p53 target that e
15 ating selective induction of human adenosine A2B receptor (A2BR) by hypoxia.
16                          On binding ADO, the A2B receptor (A2BR) stimulates cAMP production to activa
17                 After myocardial infarction, A2b receptor (A2bR) transcription was induced in both T
18                 Acting through the adenosine A2b receptor (A2bR), the luminally derived adenosine ind
19 ular permeability occurred through adenosine A2B receptor activation and was accompanied by a paralle
20                              Because luminal A2B receptor activation does not raise total cellular cA
21 g of axon growth and guidance independent of A2B receptor activation.
22 roarray analysis revealed that the adenosine A2B receptor (AdoRA2B) is selectively up-regulated by hy
23                                          The A2B receptor affinity-enhancing effects of 7-(2-chloroet
24  the adenosine receptor agonist NECA and the A2B receptor agonist BAY60-6583.
25             The work suggests that adenosine A2B receptor agonists and inhibition of equilibrative nu
26           Cyclic AMP elevators and adenosine A2b receptor agonists rejuvenated the behavior of old gr
27 eptors were activated, whereas activation of A2B receptor alone was less effective.
28 ing pathway: both treatments act through the A2b receptor and specifically activate the Epac1/RapGef3
29 ol-induced hepatic steatosis via both A1 and A2B receptors and suggest that targeting adenosine recep
30 human mast cell line HMC-1 expresses A2A and A2B receptors, and that both receptors activate adenylat
31  was blocked by adenosine A2A, but not A1 or A2B, receptor antagonism.
32 ne mimicked the effect of MTX, and adenosine A2b receptor antagonist (3,7-dimethyl-1-propargylxanthin
33                                          The A2B receptor antagonist alloxazine, however, was able to
34 effect of NECA was reversed by the selective A2B receptor antagonist N-(4-cyanophenyl)-2-[4-(2,3,6,7-
35 ar accumulation, which were inhibited by the A2B receptor antagonist PSB603 [(8-[4-[4-((4-chlorophenz
36 2A receptor antagonist), but not by MRS1754 (A2B receptor antagonist), and it was mimicked by the A2A
37  mice treated with either an adenosine A1 or A2B receptor antagonist.
38                         Neither A1, A2A, nor A2B receptor antagonists affected the capacity of MTX to
39 tial therapeutic implications given that A2A/A2B receptor antagonists have already entered clinical t
40          In this study, we revealed that A2A/A2B receptor antagonists were effective in reducing the
41 cked by theophylline, selective A1, A2A, and A2B receptor antagonists were tested as well.
42 enylate cyclase via Gs-protein but that only A2B receptors are also coupled to phospholipase C via Gq
43 al cancer (DCC), neogenin, and the adenosine A2b receptor] are expressed by the fetal mucosal epithel
44                 Furthermore, we identify the A2b receptor as a central player in this process.
45 the 3-position favored affinity at the human A2B receptor, as indicated by 1-allyl-3-methyl-8-phenylx
46 that an electron-rich ring was preferred for A2B receptor binding.
47  a luciferase reporter in HMEC-1 showed that A2B receptors, but not A1, A2A, or A3, activated IL-8 an
48 athophysiological conditions, the purinergic A2b receptor can regulate cell motility, but the underly
49 er ovary cells stably transfected with human A2B receptor cDNA, inhibition of agonist-induced cyclic
50                  A molecular model of the 27-A2B receptor complex based on the structure of rhodopsin
51 e on their luminal surface through adenosine A2B receptors coupled to adenylyl cyclase.
52 ntracellular signaling, we demonstrated that A2B receptor-dependent accumulation of JunB protein and
53 PK pathways are essential steps in adenosine A2B receptor-dependent stimulation of IL-8 production in
54 s, our data suggest an important role of the A2B receptor-dependent upregulation of JunB in VEGF prod
55                             Mice lacking the A2b receptor display increased sensitivity to IgE-mediat
56  of extracellular adenosine or inhibition of A2B receptors enhanced pro-fibrotic ATP signaling.
57 tative trait locus upstream of the adenosine-A2B receptor gene (ADORA2B).
58   To better understand the regulation of the A2b receptor in intestinal epithelia, we studied the eff
59 report increased expression of the adenosine A2b receptor in meniscus cells after stimulation at the
60  and provide further support for the role of A2B receptor in retinal angiogenesis.
61 rs in promoting fatty acid synthesis and for A2B receptors in decreasing fatty acid metabolism.
62  adenosine agonist compounds for the A2A and A2B receptors in human cells.
63              The lack of a potent, selective A2B receptor inhibitor has hampered its characterization
64 otective effects of HPC are negated when the A2B receptor is nonfunctional, the hypoxia-->adenosine--
65 enal perfusion by endothelial cell adenosine A2B receptors is antagonized by adenosine reuptake into
66                         While involvement of A2B receptors is not excluded, adenosine may activate af
67 f Sca-1(+)CD31(-) cells generated from WT or A2B receptor knockout (A2BKO) mice or the same volume of
68                                              A2B receptors mediate VEGF and IL-8 secretion because ne
69 ther apical or basolateral adenosine induces A2b receptor-mediated increase in IL-6 secretion, which
70 indings demonstrate that inosine acts via an A2B receptor-mediated pathway that impinges on specific
71 phenyl)-5-(4-pyridyl)1H- imidaz ole) blocked A2B receptor-mediated production of IL-8.
72                                      A2a and A2b receptor messenger RNA expression was elevated (p =
73  ribozyme that would specifically cleave the A2B receptor mRNA and examine its effect on retinal angi
74  the plasmid expressing the ribozyme reduced A2B receptor mRNA levels by 45+/-4.8% (P=5.1x10(-5)).
75 lls were transfected with these plasmids and A2B receptor mRNA levels were determined by quantitative
76   Ribozymes specific for the mouse and human A2B receptor mRNAs were designed and cloned in expressio
77 A1, A2A, A3, and barely detectable levels of A2B receptor mRNAs.
78                           Stimulation of the A2b receptor on FLS decreases collagenase gene expressio
79  DNA transfection, we also developed A2A and A2B receptor over-expressing lines.
80  is nonfunctional, the hypoxia-->adenosine-->A2B receptor pathway plays a critical role in the preven
81                                     Although A2B receptors play a major role in the regulation of BR
82                                              A2B receptors, predominantly expressed in human microvas
83                  Activation of the adenosine A2B receptor, proposed to contribute to netrin effects o
84                            Activation of the A2b receptor results in cyclic AMP-dependent activation
85                 Our results suggest that the A2B receptor ribozyme will provide a tool for the select
86 ituted in the 1-, 3-, 7-, and 8-positions as A2B receptor-selective antagonists have been identified.
87 in expression or membrane recruitment of the A2b receptor, shown to be essential for its function.
88 actors, we hypothesized that ablation of the A2B receptor signaling in these cells would reduce their
89        Thus, our study demonstrated that the A2B receptor signaling linked to up-regulation of pro-an
90 sine agonist, the desensitization of A2a and A2b receptor-stimulated adenylyl cyclase in dominant neg
91 lls, the rates of desensitization of A2a and A2b receptor-stimulated adenylyl cyclase were markedly s
92 timulation predominantly mediated by the Ado A2B receptor subtype.
93 oalkylacyl derivatives demonstrated that for A2B receptors the optimal chain length occurs with three
94  Caco2-BBE cells stably transfected with GFP-A2b receptor to study the intestinal A2bR.
95 ses (PDEs) confines cAMP generated by apical A2B receptors to a microdomain that includes the CFTR ch
96 product of ATP hydrolysis by ENTPD, acts via A2B receptors to counterbalance the pro-fibrotic respons
97 ion of phospholipase C indicated coupling of A2B receptors to G(q) proteins in HMEC-1.
98              In LLC cells expressing A2A and A2B receptor transcripts, only the nonselective adenosin
99 current study, we tested the hypothesis that A2B receptors upregulate JunB, which can contribute to s
100 king ecto-5'-nucleotidase or adenosine A1 or A2B receptors were protected from developing fatty liver
101 osine A2A receptor; 12-fold versus adenosine A2B receptor) with improved oral efficacy in the rat diu

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