ライフサイエンスコーパス: ABA

1 ABA deficiency also consistently resulted in differences

2 ABA effector loci were identified even when each one was

3 ABA-induced ROS sensor fluorescence accumulates in the n

4 zoylacetate, suggesting that the substrate 2-ABA itself supplies the asparagine-equivalent amino func

5 agonistically acting hormones abscisic acid (ABA) and gibberellin (GA).

6 The plant hormone abscisic acid (ABA) and the protein DELAY OF GERMINATION 1 (DOG1) are e

7 noid, jasmonic acid (JA), and abscisic acid (ABA) biosynthesis as well as enhanced expression of MYC2

8 a process known to depend on abscisic acid (ABA) but whose molecular and cellular basis remains uncl

9 P2 mimics the well-documented abscisic acid (ABA) effect on the plant dehydration response.

10 ulation of the stress hormone abscisic acid (ABA) in response to drought and low water-potential cont

11 howed an enhanced response to abscisic acid (ABA) in the seed germination and seedling growth stages,

12 The role of abscisic acid (ABA) in VPD-induced stomatal closure has been studied us

13 t not salicylic acid (SA) and abscisic acid (ABA) increased in the inoculated tissues.

14 Abscisic acid (ABA) increases reactive oxygen species (ROS) in guard ce

15 ulation of the stress hormone abscisic acid (ABA) induces many cellular mechanisms associated with dr

16 Abscisic acid (ABA) is a phytohormone involved in pivotal physiological

17 Abscisic acid (ABA) is a plant hormone that mediates abiotic stress tol

18 The phytohormone abscisic acid (ABA) is critical to plant development and stress respons

19 The phytohormone abscisic acid (ABA) is induced in response to abiotic stress to mediate

20 biotic stress-related hormone abscisic acid (ABA) is known to up-regulate wax accumulation in respons

21 Abscisic acid (ABA) is the key signal in stress-induced stomatal closur

22 Soil flooding reduces root abscisic acid (ABA) levels in citrus, conversely to what happens under

23 WT) behaviour when exposed to abscisic acid (ABA) or CaCl2 .

24 E-lincRNAs either after salt, abscisic acid (ABA) or cold treatments.

25 Abscisic acid (ABA) plays a fundamental role in plant response and adap

26 The plant hormone abscisic acid (ABA) plays an important role in the plants' response to

27 oms in the benzyl ring of the abscisic acid (ABA) receptor agonist AM1 optimizes its binding to ABA r

28 ater deficiency by increasing abscisic acid (ABA) sensitivity.

29 Abscisic acid (ABA) signaling plays a major role in root system develop

30 esponse to drought stress and abscisic acid (ABA) treatment.

31 We show that abscisic acid (ABA), an antagonist of GA, is also transported by NPF3 i

32 af water potential and foliar abscisic acid (ABA), during drought and through the subsequent rehydrat

33 response to the plant hormone abscisic acid (ABA), elevated CO2 concentration, and reduced air humidi

34 regulating the plant hormone abscisic acid (ABA)-independent drought response.

35 he core signaling pathway for abscisic acid (ABA)-induced stomatal closure involves perception of the

36 rough jasmonic acid (JA)- and abscisic acid (ABA)-mediated pathways.

37 mediated by the plant hormone abscisic acid (ABA).

38 ht and the associated hormone abscisic acid (ABA).

39 o drought by the phytohormone abscisic acid (ABA).

40 tomata to the drought-hormone abscisic acid (ABA).

41 omatal closure in response to abscisic acid (ABA).

42 but involve the phytohormone abscisic acid (ABA).

43 aCl stress and in response to abscisic acid (ABA).

44 successful in identifying genes that affect ABA levels and may act in upstream drought-related sensi

45 es encoding ABA signaling proteins to affect ABA sensitivity.

46 erally, PL inactivation specifically affects ABA renewal by reducing responding in the conditioning c

47 nt increased and reached a peak at 8 h after ABA treatment and then significantly decreased at latter

48 cant increase during the first 8 hours after ABA treatment, which then significantly decreased at 12

49 tially expressed TFs increased rapidly after ABA treatment.

50 All ABA-deficient lines closed their stomata rapidly and ext

51 ng ethylene effects; and the interplay among ABA, ethylene, cytokinin and auxin is tissue-specific, a

52 atal responsiveness is also controlled by an ABA action on leaf water supply upstream from stomata.

53 or transcriptional modulation mediated by an ABA receptor different from the core ABA signaling pathw

54 sociated to the upregulation of CsBGLU18 (an ABA beta-glycosidase) that cleaves ABAGE.

55 erate induction of catabolism (CsCYP707A, an ABA 8'-hydroxylase) and buildup of dehydrophaseic acid (

56 ; PIN1 levels are reduced under stress in an ABA-dependent manner, overriding ethylene effects; and t

57 emicals with transgenic overexpression of an ABA receptor is very effective in helping plants combat

58 guard cells or phloem companion cells of an ABA-deficient mutant.

59 was impaired in the ABA-deficient aba2-3 and ABA-insensitive abi4-1 mutants.

60 lating ABA biosynthesis and accumulation and ABA-dependent signalling, but also by ABA independent pa

61 involved in repressing meristem activity and ABA-mediated dehydration pathways.

62 ental analysis shows: that ABA-dependent and ABA-independent stress responses increase under osmotic

63 Drought and ABA treatment stimulates the degradation of OsOTS1 prote

64 ole-plant wilting in response to drought and ABA.

65 e expression is induced by salt, drought and ABA.

66 tional analysis of ABA metabolic enzymes and ABA-responsive promoters, have all contributed to curren

67 These data, gene expression and ABA signalling transduction were compared in wild-type C

68 ypes suggest the cell cycle uncouples GA and ABA responses in germinating Arabidopsis seeds, and that

69 The responses to both GA and ABA were found to occur within distinct cell types, sugg

70 lay opposite roles in regulating glucose and ABA signaling in Arabidopsis during seed germination and

71 lay opposite roles in regulating glucose and ABA signaling in Arabidopsis during seed germination and

72 tight correlation between sequential SA and ABA signaling and dynamic changes in NPR1 protein levels

73 opsis (Arabidopsis thaliana), the stress and ABA-induced Delta1-PYRROLINE-5-CARBOXYLATE SYNTHETASE1 (

74 NCED3 were studied after feeding sulfate and ABA to detached poplar leaves and epidermal peels of Ara

75 stress to investigate xylem sap sulfate and ABA, stomatal conductance, and sulfate transporter (SULT

76 High temperature and ABA treatments led to greater magnitudes of change durin

77 ch-lock mechanism resembling the Arabidopsis ABA receptors, but the ABA binding pocket in FePYR1 show

78 indicating that these effects of S-della are ABA dependent.

79 PYR1/PYL/RCAR proteins are ABA receptors that function by inhibiting PP2Cs to activ

80 ties to regulating the ABA response at basal ABA levels when efficiently expressed.

81 al ABA receptors exist in mature Mks because ABA induces an intracellular Ca(2+) increase ([Ca(2+)] i

82 biosynthesis is target of cross talk between ABA signaling and regulation of phosphate homeostasis th

83 grass Festuca elata was demonstrated to bind ABA as a monomer.

84 is revealed that FePYR1 recognizes and binds ABA by the common gate-latch-lock mechanism resembling t

85 Drought strongly induced both ABA biosynthesis and catabolism (CsNCED1, 9-cis-neoxanth

86 gnal in stress-induced stomatal closure, but ABA as an early xylem-delivered signal is still a matter

87 erexpressing lines are drought sensitive but ABA insensitive.

88 on and ABA-dependent signalling, but also by ABA independent pathways.

89 cating regulation of cuticle biosynthesis by ABA.

90 estigating the functions of genes induced by ABA and will help to develop a better understanding of t

91 t OsREM4.1 is transcriptionally regulated by ABA and functions as an OsBRI1 substrate and OsSERK1-int

92 OsREM4.1, whose expression is upregulated by ABA through the transcriptional activator OsbZIP23.

93 latory characteristics of observed canonical ABA pathway components, we identified a new family of tr

94 ELLA had no effect on ABA levels, guard cell ABA responsiveness was increased in S-della and reduced

95 l closure via QUAC1/ALMT12 and/or guard cell ABA synthesis.

96 itions and explain the expression of certain ABA-responsive genes.

97 d ZmXerico2 in Arabidopsis and maize confers ABA hypersensitivity and improved water use efficiency,

98 , and a putative mechanistic link connecting ABA and JA signaling pathways.

99 context after extinction in another context (ABA renewal).

100 ts of flooded plants, restoration of control ABA levels after stress release was associated to the up

101 However, mechanisms controlling ABA accumulation itself are less known.

102 d by an ABA receptor different from the core ABA signaling pathway, and a putative mechanistic link c

103 ted flavonols, ethylene treatments decreased ABA-induced stomatal closure in the wild type, but not N

104 tress induced by rTpo and serum deprivation, ABA stimulates, in a PKA- and cADPR-dependent fashion, t

105 ALMT4 likely mediates Mal(2-) efflux during ABA-induced stomatal closure and its activity depends on

106 eatment of nonmycorrhized plants with either ABA or JA induced the up-regulation of MYC2, but only JA

107 y affecting the expression of genes encoding ABA signaling proteins to affect ABA sensitivity.

108 controlling the expression of genes encoding ABA signaling proteins.

109 ABs transitioned from para- to endodormancy, ABA and PA levels decreased, whereas IAA levels were mai

110 Endogenous ABA content increased and reached a peak at 8 h after AB

111 Our data indicated the endogenous ABA of seeds was rich.

112 ated improved shoot growth, whereas enhanced ABA biosynthesis and JA-regulated flavonoid and terpenoi

113 difference was again countered by exogenous ABA, further indicating regulation of cuticle biosynthes

114 ance (Kleaf) was down-regulated by exogenous ABA, with strong variations depending on the genotype.

115 Here we investigated the role of exogenous ABA in modulating thrombopoiesis, the process of platele

116 cs in P. hopeiensis in response to exogenous ABA using Illumina RNA-sequencing.

117 are compared to a commonly used experimental ABA, in an in vitro THP-1 cell model.

118 ) inhibit ABA response, resulting in extreme ABA resistance in transgenic Arabidopsis overexpression

119 F binding proteins (AFPs) results in extreme ABA resistance of seeds via multiple mechanisms repressi

120 F binding proteins (AFPs) results in extreme ABA resistance of seeds via multiple mechanisms repressi

121 ulin genes revealed the transcription factor ABA-insensitive 5 (ABI5) as a highly connected hub.

122 lty and tomato (Solanum lycopersicum) flacca ABA-deficient mutants had higher stomatal conductance co

123 nes in leaf water potential, elevated foliar ABA concentrations and reduced stomatal conductance and

124 These results suggest dual roles for ABA in regulating leaf cuticle formation: one that is fu

125 Functional ABA receptors exist in mature Mks because ABA induces an

126 NPF3 acting as an influx carrier and that GA-ABA interaction may occur at the level of transport.

127 s of amino acid residues in FePYR1 generated ABA receptor variants with significantly increased ABA b

128 veal a mechanism in which rice plants govern ABA-dependant drought responsive gene expression by cont

129 Guard cells of are show greater ABA-induced closure than the wild type, reduced light-de

130 At exogenous high ABA levels, the RCARs regulated most PP2Cs and activated

131 re in response to the drought stress hormone ABA and increased whole-plant wilting in response to dro

132 addition to behaving like an animal hormone, ABA also holds promise as a nutraceutical plant-derived

133 We have identified the hot ABA-deficiency suppressor1 mutant, which has a stomatal

134 This SnapShot explores how ABA signaling operates and coordinates resistance during

135 ed to the upregulation of CsAOG, involved in ABA glycosyl ester (ABAGE) synthesis, and to a moderate

136 se to ABA, showing that they are involved in ABA responses.

137 led abnormal expression of genes involved in ABA sensing, signaling, and response.

138 A6b is a positive regulator participating in ABA-mediated salt and drought resistance.

139 of Actinidia chinensis SVP2 confirm roles in ABA- and dehydration-mediated growth repression and reve

140 There was a 10-fold range of variation in ABA levels among nearly 300 Arabidopsis thaliana accessi

141 d soil and leaf water contents and increased ABA levels.

142 and ZmXerico2 in maize results in increased ABA levels and decreased levels of ABA degradation produ

143 ceptor variants with significantly increased ABA binding affinity.

144 Nitrate treatment increased ABA levels in root tips; this stimulation requires the a

145 Both stress conditions induced ABA-responsive genes CsABI5 and CsDREB2A similarly, sugg

146 ral variation in low-water-potential-induced ABA accumulation and was successful in identifying genes

147 ral ABI5/ABF binding proteins (AFPs) inhibit ABA response, resulting in extreme ABA resistance in tra

148 This study shows that the AFPs that inhibit ABA response have intrinsic repressor activity in a hete

149 act with the co-repressor TOPLESS to inhibit ABA-regulated gene expression.

150 ductance, stunted growth phenotype, and leaf ABA level were restored to wild-type values, pointing to

151 ABA sources and to the effectiveness of leaf ABA transport.

152 Organ and tissue-level ABA measurements, as well as indirect in vivo measuremen

153 vity of the endoplasmic reticulum-localized, ABA-GE-deconjugating enzyme b-GLUCOSIDASE1, but not de n

154 ibited DeltaNHAB1 activity in vitro at lower ABA concentrations than CsPYL8 or CsPYL1, suggesting its

155 s in turfgrass that are important to mediate ABA signaling and drought stress response.

156 articipate in multiple mechanisms modulating ABA response, including both TOPLESS-dependent and -inde

157 ily of transcriptional regulators modulating ABA and salt responsiveness and demonstrated their utili

158 1 depleted transgenic plants accumulate more ABA and exhibit more productive agronomic traits during

159 ine hematopoietic progenitor cells to 10 mum ABA does not increase recombinant thrombopoietin (rTpo)-

160 ating enzyme b-GLUCOSIDASE1, but not de novo ABA biosynthesis.

161 imulus, but is only active in the absence of ABA.

162 as cell-specific transcriptional analysis of ABA metabolic enzymes and ABA-responsive promoters, have

163 Analysis of ABA responses in seed germination, cotyledon greening, a

164 Exogenous application of ABA partially rescued these phenotypes, confirming that

165 A putative binding of ABA to GRP78, a key regulator of endoplasmic reticulum s

166 being distributed among the many branches of ABA metabolism or mediated by genes with partially redun

167 assembly and gelation behavior of a class of ABA triblock copolymers containing a central poly(ethyle

168 ified genomic regions containing clusters of ABA-associated SNPs.

169 The comparison of ABA metabolism and signaling in roots of flooded and wat

170 ling chain has been elucidated consisting of ABA binding to receptors, which alleviates negative regu

171 cremental effect, consistent with control of ABA accumulation being distributed among the many branch

172 hat ZmXerico1 plays a role in the control of ABA homeostasis through regulation of ABA 8'-hydroxylase

173 organ development, we assessed the effect of ABA deficiency on cuticle formation in three ABA biosynt

174 e genetic analysis, which found effectors of ABA accumulation.

175 osphorylation of ABFs and other effectors of ABA response pathways.

176 ench data regarding combinatorial effects of ABA and internal node activation, we experimentally conf

177 f this network and elucidated the effects of ABA plus knockout or constitutive activity of 79 nodes o

178 ddition, stomatal behavior and expression of ABA receptors, drought-responsive genes, transcription f

179 The function of ABA in dormancy is rather well understood, but the role

180 Many biological functions of ABA in mammals are mediated by its binding to the LANCL-

181 The importance of ABA biosynthesis in guard cells versus vasculature for w

182 and Pi-treated plants, whereas the level of ABA was increased specifically in AM leaves.

183 increased ABA levels and decreased levels of ABA degradation products diphaseic acid and phaseic acid

184 lts indicated that relatively high levels of ABA, the ABA metabolite PA, and IAA were found in parado

185 transpiration, and transgenic modulation of ABA levels therefore represents an attractive avenue to

186 EB2A similarly, suggesting the occurrence of ABA signaling in roots of flooded citrus seedlings.

187 e lateral root elongation in the presence of ABA.

188 Transcriptional profile of ABA receptor genes revealed a different induction in res

189 d-type values, pointing to the redundancy of ABA sources and to the effectiveness of leaf ABA transpo

190 rol of ABA homeostasis through regulation of ABA 8'-hydroxylase protein stability, representing a nov

191 nificantly along with the down-regulation of ABA biosynthesis genes, ABA2 and NCED3.

192 l closure and the stomatal VPD regulation of ABA-deficient mutants may be conditional on the initial

193 To characterize the role of ABA production in different cells, we generated transgen

194 lated the expression of NCED3, a key step of ABA synthesis, in guard cells.

195 nes carrying mutations in different steps of ABA biosynthesis as well as pea (Pisum sativum) wilty an

196 eed plants through activation of a subset of ABA receptors.

197 This highly buffered system of ABA metabolism represents both a challenge and opportuni

198 rent views of the localization and timing of ABA accumulations.

199 While DELLA had no effect on ABA levels, guard cell ABA responsiveness was increased

200 nd possible effect of tonoplast transport on ABA accumulation.

201 ions included few genes with known stress or ABA-related function.

202 by the plant-specific miR536 and that other ABA-relevant genes are regulated by miRNAs and ta-siRNAs

203 e of Kleaf to ABA may be part of the overall ABA regulation of leaf water status.

204 to clade A of type 2C protein phosphatases: ABA-HYPERSENSITIVE GERMINATION 1 (AHG1) and AHG3.

205 of Rapamycin (TOR) kinase phosphorylates PYL ABA receptors at a conserved serine residue to prevent a

206 Recently, ABA has been proven to be also secreted and active in ma

207 The impact of reduced ABA levels in flooded roots on CsPYL5 expression along w

208 ming that they were a consequence of reduced ABA levels.

209 hundreds of disparate observations regarding ABA signal transduction responses underlying stomatal cl

210 combinations for their capacity to regulate ABA signaling by transient expression in Arabidopsis pro

211 of plant responses to drought by regulating ABA biosynthesis and accumulation and ABA-dependent sign

212 f BRM restores the ability of BRM to repress ABA response.

213 Thus, TOR signaling represses ABA signaling and stress responses in unstressed conditi

214 domain is also not essential for repressing ABA response in transgenic plants, but does contribute t

215 of seeds via multiple mechanisms repressing ABA response, including interactions with histone deacet

216 of seeds via multiple mechanisms repressing ABA response, including interactions with histone deacet

217 alyses suggested the existence of a residual ABA signaling in roots of flooded Carrizo citrange seedl

218 sistance in turfgrass, we identified several ABA receptors in turfgrass that are important to mediate

219 ARs, and the ABA receptor RCAR4/PYL10 showed ABA-dependent PP2C regulation.

220 ng reveals that miRNA-mediated SA signaling, ABA-dependent, and ROS response pathways are involved in

221 y metabolites were found to be significantly ABA responsive, including carbohydrates, fatty acids, gl

222 ABA-insensitive mutant, defective in the six ABA PYR/RCAR receptors, responded to changes in VPD in b

223 design of small molecules targeting specific ABA receptors in economically important plant species.

224 Under stress, ABA-activated SnRK2s phosphorylate Raptor, a component o

225 enic plants, but does contribute to stronger ABA resistance.

226 Another strongly ABA-insensitive mutant, defective in the six ABA PYR/RCA

227 e-assisted engineering could create superior ABA receptors for improving plant drought resistance.

228 In conclusion, we demonstrate that ABA is a prosurvival factor for Mks in a Tpo-independent

229 Immunogold labeling demonstrated that ABA is associated with cytoplasmic structures near, but

230 tomato (Solanum lycopersicum), we find that ABA-increased ROS is followed by stomatal closure and th

231 We found that ABA accumulated in the endodermis and quiescent center o

232 To address the hypothesis that ABA also mediates cuticle formation during organ develop

233 lysis with network construction reveals that ABA regulates root growth under osmotic stress condition

234 Experimental analysis shows: that ABA-dependent and ABA-independent stress responses incre

235 ected than in leaf cuticles, suggesting that ABA action influences cuticle formation in an organ-depe

236 ected in the aba2-3 mutants, suggesting that ABA is implicated in growth retardation in such nutritio

237 The ABA biosynthesis inhibitor fluridone rescued the mybs1 g

238 The ABA mutants also showed reduced expression of genes invo

239 The ABA receptor FePYR1 from turfgrass Festuca elata was dem

240 ated that relatively high levels of ABA, the ABA metabolite PA, and IAA were found in paradormant bud

241 RCARs regulated most PP2Cs and activated the ABA response to a similar extent.

242 to HAI3 were regulated by all RCARs, and the ABA receptor RCAR4/PYL10 showed ABA-dependent PP2C regul

243 players of the ABA-signaling pathway are the ABA-binding receptors (RCAR/PYR1/PYL), which, together w

244 nstrated that PL inactivation attenuated the ABA renewal effect in the same animals, replicating earl

245 bling the Arabidopsis ABA receptors, but the ABA binding pocket in FePYR1 shows discrepant residues r

246 it cuticles were affected differently by the ABA-associated mutations, but in general were thicker.

247 are consistent with flavonols dampening the ABA-dependent ROS burst that drives stomatal closure and

248 transitioned from endo- to ecodormancy, the ABA metabolites PA and DPA decreased significantly along

249 Plants expressing the ABA receptors RCAR6/PYL12 combined up to 40% increased W

250 howed that BRM resides at target loci in the ABA pathway in the presence and absence of the stimulus,

251 a number of candidate genes involved in the ABA signaling pathway, as well as transcription factors,

252 1 and phl1 mutations and was impaired in the ABA-deficient aba2-3 and ABA-insensitive abi4-1 mutants.

253 d water loss were strongly suppressed in the ABA-deficient mutant sitiens, indicating that these effe

254 Several steps in the ABA-signaling pathway are controlled by ubiquitination i

255 egrating fluctuating hormone levels into the ABA-response pathway.

256 her regulators are suggested to modulate the ABA signaling pathway, including the protein ENHANCED RE

257 novel control point in the regulation of the ABA pathway.

258 6b operates as a downstream regulator of the ABA-mediated stress response and is required for heat st

259 Key players of the ABA-signaling pathway are the ABA-binding receptors (RCA

260 dies reveal that hydrotropism depends on the ABA signalling kinase SnRK2.2 and the hydrotropism-speci

261 ed different sensitivities to regulating the ABA response at basal ABA levels when efficiently expres

262 at OsOTS1 SUMO protease directly targets the ABA and drought responsive transcription factor OsbZIP23

263 We propose that the ABA and DOG1 dormancy pathways converge at clade A of ty

264 ic may have different sensitivities in these ABA responses.

265 hormone affinity reinforce the role of this ABA receptor under soil-flooding conditions and explain

266 esis genes, ABA1, NCED9, and AAO3, and three ABA signaling genes, ABI3, ABI4, and ABI5, were increase

267 ABA deficiency on cuticle formation in three ABA biosynthesis-impaired tomato mutants.

268 Moreover, the mRNA levels of three ABA biosynthesis genes, ABA1, NCED9, and AAO3, and three

269 ropose a model in which drought acts through ABA to increase ABIG1 transcription which in turn restri

270 Thus, ABA signaling is required for proper HSFA6b expression.

271 s resulting in different binding affinity to ABA.

272 riments and theory expose close analogies to ABA' triblock copolymer phase behavior, collectively sug

273 eceptor agonist AM1 optimizes its binding to ABA receptors by increasing the number of hydrogen bonds

274 The era1 mutant is hypersensitive to ABA during seed germination and shows a more closed stom

275 s in addition to conferring insensitivity to ABA.

276 opose that the observed response of Kleaf to ABA may be part of the overall ABA regulation of leaf wa

277 vulgare VIVIPAROUS1 (HvVP1), orthologous to ABA-INSENSITIVE3 from Arabidopsis thaliana HvVP1 transcr

278 ata of two temperate fern species respond to ABA and CO2 and that an active mechanism of stomatal reg

279 hand, active stomatal closure in response to ABA and CO2 was found in several moss, lycophyte, and fe

280 required for stomatal closure in response to ABA and environmental factors.

281 identify metabolic signatures in response to ABA in B. napus guard cell protoplasts.

282 ins led to faster germination in response to ABA, showing that they are involved in ABA responses.

283 oles in plant cells that affect responses to ABA.

284 abidopsis hydathode pores were responsive to ABA and light similar to stomata.

285 fficient to increase stomatal sensitivity to ABA and to reduce water loss under water deficit stress

286 RNA11195, mutants had reduced sensitivity to ABA as demonstrated by longer roots and higher shoot bio

287 in the guard cells, increased sensitivity to ABA, and a reduction in stomatal apertures.

288 sohydry correlated with Kleaf sensitivity to ABA, with Kleaf in the most anisohydric genotypes being

289 exhibited slower growth at germination under ABA or alkaline conditions, while maintaining very high

290 Overall, using ABA accumulation as a basis for a combined GWAS-reverse

291 uced stomatal closure has been studied using ABA-related mutants that respond to VPD in some studies

292 cells, we generated transgenic plants where ABA biosynthesis was rescued in guard cells or phloem co

293 ocalized in the endoplasmic reticulum, where ABA 8'-hydroxylases have been shown to be localized, and

294 responses in unstressed conditions, whereas ABA signaling represses TOR signaling and growth during

295 ort, and expression of genes associated with ABA signalling in the nia1nia2 mutant.

296 hosphorylation disrupts PYL association with ABA and with PP2C phosphatase effectors, leading to inac

297 eatments containing pyrabactin-combined with ABA or alone-diminished protein content, thus partially

298 ined with assays on detached leaves fed with ABA.

299 When treated with ABA, abig1 mutants remain greener and produce more leave

300 both at homeostasis and after treatment with ABA.