ライフサイエンスコーパス: ABR

1 ABR gene profiles were maintained in recipients for up t

2 ABR shifts after combination treatment were similar to t

3 ABR thresholds develop rapidly in normal, euthyroid anim

4 ABR thresholds differed between 129S-Cdh23(c.753A) SNV a

5 ABR thresholds increased maximally immediately after exp

6 ABR-217620 was well tolerated with evidence of immunolog

7 ABRs in AQP4 null CD1 mice measured in response to tone

8 ABRs were obtained by using 4-, 8-, 16-, and 32-kHz tone

9 ata demonstrate a critical role for the ZO-1 ABR in barrier function and suggest that MLCK-dependent

10 The identification of BCL-6 ABR provides new tools for the diagnosis of lymphomas ca

11 our of eight Coch(G88E/G88E) mice had absent ABRs at all frequencies tested and two of three Coch(G88

12 two of three Coch(G88E)(/+) mice had absent ABRs at three of four frequencies tested.

13 in the same Coch(G88E/G88E) mice with absent ABRs.

14 These changes are associated with altered ABR latency during development.

15 An ABR was obtained after removal of the probe to assess lo

16 er loci in Atp6v1b1vtx/vtx mice, we analysed ABR thresholds of progeny from a backcross segregating M

17 ion of auditory brainstem response analyses (ABR) and electron microscopy, we identified an unexpecte

18 ADPHd expression increased with both age and ABR thresholds in the medial superior olive but not in e

19 holds had recovered, but synaptic counts and ABR amplitudes at high frequencies were reduced by up to

20 DPOAE and ABR exhibited an increasing threshold at high frequencie

21 nificant changes in Aqp4 gene expression and ABR and DPOAE hearing status in the cochlea and auditory

22 MRE prevented the inflammation and ABR by reducing TNF-alpha and IL-1beta, preventing the o

23 While acoustic PPI remained intact and ABR thresholds recovered, the ABR wave P1-N1 amplitude r

24 ssion of immediately adjacent genes, such as ABR, and could be a common mechanism in the causation of

25 5-week-old mice in a CD1 genetic background, ABR thresholds in response to a click stimulus were rema

26 ods applied to evaluate arterial baroreflex (ABR) function provide the same information as the modell

27 We found significant correlations between ABR thresholds and the loss of outer hair cells and spir

28 reflects the difference between the binaural ABR and the sum of the monaural ABRs (i.e., binaural - (

29 from noise-induced hearing loss (NIHL): both ABR threshold shifts and hair cell death were significan

30 Both ABRs and IPM-FRs were observed most clearly from similar

31 At high frequencies, both ABRs and DPOAEs were attenuated by loss of M2 and/or M4,

32 xposure in a mouse model system, measured by ABR.

33 Although all subjects displayed clear ABRs, the BIC was not reliably observed.

34 We compared ABR thresholds and cochlear pathologies of these SNV mic

35 7620 followed by docetaxel in 21-day cycles (ABR-217620 dose escalation combination with docetaxel [C

36 Older adults had delayed ABRs, especially in response to the rapidly changing for

37 ely 70% of Clrn1(-/-) mice had no detectable ABR and by P30 these mice were deaf.

38 with similar ABR threshold, and 6) elevated ABR thresholds and deceased wave I amplitudes at high fr

39 normal VsEPs with normal to mildly elevated ABR thresholds.

40 at this locus (genotype ahl2/ahl2) exhibited ABR thresholds that were on average 26 decibels above th

41 The free ABR also prevented increases in barrier function followi

42 Moreover, the free ABR interfered with full-length ZO-1 exchange and reduce

43 ake no longer than a few minutes, apart from ABR testing which takes upto 3.5 h per mouse.

44 ss of ribbons in both regions and changes in ABR sensitivity and dynamic responsiveness.

45 he more robust ABR wave-V mirrors changes in ABR wave-I amplitude.

46 with ABRs during development, a decrease in ABR thresholds between P13 and P15, and anatomical resul

47 t VsEPs) with mild to moderate elevations in ABR thresholds.

48 creased noise vulnerability was seen only in ABRs, consistent with a role for dopaminergic signaling

49 fluoroquinolone, and antibiotic inactivation ABR genes were increased in RCDI patients, although dono

50 T-dfw2J/ + heterozygotes displayed increased ABR thresholds, suggesting that a second locus, controll

51 etermined by showing significantly increased ABR thresholds 6 weeks after adoptive transfer of peptid

52 We investigated ABR S. aureus nasal carriage prevalence among adults wit

53 nnual bleeding rate (ABR), spontaneous joint ABR, the number of infusions and dose required to resolv

54 y and in the control of bleeding events, low ABR in patients on prophylaxis, and a favorable safety p

55 fe compared with rFVIII, and resulted in low ABRs when dosed prophylactically 1 to 2 times per week.

56 stablish normative values of forward-masking ABRs and investigate the development of auditory tempora

57 The median ABR was 2.0 (0.0-3.1) overall and 0.0 (0.0-0.0) for spon

58 Median ABRs observed in arms 1, 2, and 3 were 1.6, 3.6, and 33.

59 the binaural ABR and the sum of the monaural ABRs (i.e., binaural - (left + right)).

60 This QTL is responsible for 57 and 43% of ABR threshold variance, respectively, in each strain com

61 ed statistically significant associations of ABR thresholds with markers on chromosome 5, with a peak

62 Two phase I studies were conducted of ABR-217620 alone or in combination with docetaxel.

63 tly with a decreased number and diversity of ABR genes and increased Bacteroidetes and Firmicutes wit

64 tients had a greater number and diversity of ABR genes compared with donors and healthy controls.

65 NSCLC received 4 daily, escalating doses of ABR-217620 followed by docetaxel in 21-day cycles (ABR-2

66 iotic-resistant organisms and elimination of ABR genes.

67 The extent of ABR S. aureus exposure in IHO workers and children livin

68 est that measures of the effects of noise on ABR wave-V latency can be used to diagnose cochlear syna

69 ve treatment with the Q compound paquinimod (ABR-215757) during the first 5 days after induction of e

70 Division of the American Board of Radiology (ABR) approved the studies (human subjects and HIPAA comp

71 The American Board of Radiology (ABR) has provided certification for diagnostic radiologi

72 ncology and the American Board of Radiology (ABR) Written Radiation Oncology Board Examination.

73 the median overall annualized bleeding rate (ABR) was 1.14 (Q1, Q3: 0.0, 4.2).

74 nd points included annualized bleeding rate (ABR), inhibitor development, and adverse events.

75 were pharmacokinetics, annual bleeding rate (ABR), spontaneous joint ABR, the number of infusions and

76 t carotid, the arterial-wall-to-blood ratio (ABR) 4.5 h post-infusion was 2.13 +/- 0.35 in patients v

77 To assess this hypothesis, we recorded ABRs to the speech syllable /da/ in normal hearing young

78 bons correlates with the presence of reduced ABR peak one amplitudes in both levels of noise exposure

79 Tempol alone significantly reduced ABR threshold shifts on day 10 but not on day 1.

80 ing a similar N-terminal APC binding region (ABR) and Src-homology 3 (SH3) domain.

81 lts indicate that this actin-binding region (ABR) is both necessary and sufficient for binding to F-a

82 Substitutions in the YFV Ag-binding region (ABR) occur at four of the eight highly conserved residue

83 itro, ZO-1 lacking the actin binding region (ABR) was not stabilized by MLCK inhibition, either in th

84 ly identified alternative breakpoint region (ABR), showed a rearrangement.

85 we called the alternative breakpoint region (ABR), were found to be recurrent in lymphomas carrying t

86 oarray containing 354 antibiotic resistance (ABR) genes.

87 pport the emergence of antibiotic-resistant (ABR) Staphylococcus aureus.

88 d its influence on alveolar bone resorption (ABR) in rats.

89 blink activity, auditory brainstem response (ABR) amplitudes, and tail-flick latency.

90 es, such as the auditory brainstem response (ABR) and distortion product otoacoustic emissions (DPOAE

91 measurements of auditory brainstem response (ABR) and distortion product otoacoustic emissions (DPOAE

92 neration of the auditory brainstem response (ABR) and receives direct innervation from the cochlea, i

93 The auditory-evoked brainstem response (ABR) in Thrb-/- mice, although greatly diminished, displ

94 sured using the auditory brainstem response (ABR) or distortion product otoacoustic emissions (DPOAE)

95 elevated auditory-evoked brainstem response (ABR) thresholds and prolonged peak and interpeak latenci

96 ion (DPOAE) and auditory brainstem response (ABR) thresholds during and after middle ear infusion of

97 GLUT3 delivery, auditory brainstem response (ABR) thresholds normalize, along with partial rescue of

98 Auditory brainstem response (ABR) thresholds of Coch(G88E/G88E) mice were substantial

99 study that used auditory brainstem response (ABR) thresholds to identify QTLs affecting HFHL.

100 Auditory brainstem response (ABR) thresholds were compared to VsEP thresholds for 14

101 Auditory brainstem response (ABR) thresholds, peak latencies, and amplitude growth we

102 TL analysis for auditory brainstem response (ABR) thresholds, which indicates hearing ability, was pe

103 neural output [auditory brainstem response (ABR) wave 1] and in outer hair cell function [distortion

104 icantly delayed auditory brainstem response (ABR) wave I latencies at low and middle frequencies comp

105 he amplitude of auditory brainstem response (ABR) wave-I.

106 was analyzed by auditory brainstem response (ABR) which confirmed that severe age-related hearing los

107 The auditory brainstem response (ABR), a scalp-recorded electrical potential, is known fo

108 changes in the auditory brainstem response (ABR).

109 ssay and acoustic brainstem evoked response (ABR) threshold analysis were carried out to evaluate the

110 le in hearing, auditory brain stem response (ABR) thresholds were compared in wild-type mice and tran

111 re measured by auditory brain stem response (ABR).

112 nd frequency (auditory brainstem response -- ABR thresholds, and distortion-product otoacoustic emiss

113 ts (measured by auditory brainstem response, ABR) of up to 73 dB (16 kHz) on day 1, and up to 50 dB (

114 ectorin 71-90, auditory brainstem responses (ABR) showed significant hearing loss at all frequencies

115 he analysis of auditory brainstem responses (ABR) showed that mtl and bsd homozygotes are deaf, where

116 no discernible auditory brainstem responses (ABR) to sound pressure level stimuli up to 100 dB, indic

117 , we collected auditory brainstem responses (ABRs) and determined cochlear hair cell loss in 13-month

118 Auditory brainstem responses (ABRs) and distortion product otoacoustic emissions (DPOA

119 is we obtained auditory brainstem responses (ABRs) and micro-CT x-ray scans to measure changes in the

120 , we collected auditory brainstem responses (ABRs) from rhesus monkeys spanning in age from 10 to 35

121 Auditory brainstem responses (ABRs) were obtained in anesthetized animals for click an

122 present study, auditory brainstem responses (ABRs) were recorded in a forward-masking paradigm in hea

123 y by recording auditory brainstem responses (ABRs)-the BIC reflects the difference between the binaur

124 tion (PPI) and auditory brainstem responses (ABRs).

125 emissions and auditory brainstem responses (ABRs).

126 rmally delayed auditory brainstem responses (ABRs).

127 , we measured auditory brain stem responses (ABRs), hair cell loss, and free radical activity in the

128 king noise on the latency of the more robust ABR wave-V mirrors changes in ABR wave-I amplitude.

129 cies compared with control mice with similar ABR threshold, and 6) elevated ABR thresholds and deceas

130 e-treated subjects had significantly smaller ABR threshold shifts on day 1 and on day 10.

131 ow that the device is capable of stimulating ABRs in vivo with latencies and growth functions compara

132 The ABR is located 200-270 kb telomeric to MBR.

133 A primary tool used by the ABR in fulfilling this responsibility is the secure proc

134 For the ABR MOC examinations, 1280 x 1024 displays should be use

135 omen, 1.95:1) had an initial sitting for the ABR written examination.

136 Furthermore, the ABR is required for localization to a novel actin-rich p

137 urements), and explains in each case how the ABR implements that standard.

138 Interestingly, the ABR in CBy-dfw2J/+ heterozygotes is also abnormal, showi

139 Translocations involving the ABR may juxtapose BCL-6 to distantly acting, heterologou

140 VSVG-tagged transgenes that lack the ABR still accumulate at both early and late cell-cell co

141 ever, accumulation of constructs lacking the ABR is markedly reduced at tight junctions in confluent

142 Moreover, the ABR of Asef2 did not function independently but acted in

143 Transport of the ABR-anchored exchangeable pool is regulated by MLCK.

144 In patients, the ABR correlated with the TBR of the corresponding vessel

145 ned intact and ABR thresholds recovered, the ABR wave P1-N1 amplitude reduction persisted in all blas

146 ions in confluent cells, suggesting that the ABR does play an important role in the localization of Z

147 contacts in MDCK cells, suggesting that the ABR is not required for tight junction localization.

148 Our request to the ABR to permit us to study performance on their examinati

149 lymphomas carrying rearrangements within the ABR showed that the breakpoints cluster within a 20-kb d

150 This ABR specialization indicates that even though YFV is pol

151 valence and potential sources of exposure to ABR S. aureus among children living with IHO workers.

152 Wistar rats were subjected to ABR by ligature with nylon thread in the second upper-le

153 ively, elevations in 4-, 8- and 20 kHz tonal ABR thresholds and reduced dynamic output at higher inte

154 loci account for more than 70% of the total ABR threshold variation among the backcross mice at all

155 Unfortunately, ABR wave-I is difficult to measure in humans, limiting i

156 red with wild-type mice (p < 0.001), whereas ABR thresholds were not affected by AQP1, AQP3, or AQP5

157 nearly all AQP4 null mice were deaf, whereas ABRs could be elicited in wild-type controls.

158 While ABR wave latencies and interpeak intervals decreased ear

159 Conversely, PV expression correlated with ABR amplitudes and outer hair cell loss in the cochlea,

160 The strongest correlation with ABR thresholds was the number of different pathologies p

161 und highly significant linkage of Mass1 with ABR threshold variation among mice from two backcrosses

162 Regression analyses of these values with ABR properties and cochlear histopathologies revealed re

163 ng changes in the percentage of animals with ABRs during development, a decrease in ABR thresholds be

164 , it is unlikely that Ag is bound in the YFV ABR in the manner typical of class Ia molecules.