ライフサイエンスコーパス: ACC

1 ACC inhibition with a single dose of NDI-010976 is well

2 ACC is a rare but aggressive tumor.

3 ACC molecular changes in chronic pain-induced depression

4 ACC-ITE scores from third-year fellows from 2011 to 2014

5 ACC-ITE scores were the strongest predictor of ABIM CVD

6 with mood symptoms (PFC: r = .52, p = .005; ACC: r = .53, p = .004) and suicidality (PFC: r = .40, p

7 04) and suicidality (PFC: r = .40, p = .037; ACC: r = .38, p = .046), while hippocampus MAO-A VT was

8 arts Tool reinforces and builds on the "2013 ACC/AHA Guideline on the Assessment of Cardiovascular Ri

9 r ASCVD risk was estimated by using the 2013 ACC/AHA ASCVD risk calculator.

10 the 2016 USPSTF recommendations vs the 2013 ACC/AHA cholesterol guidelines among a nationally repres

11 Adoption of the 2013 ACC/AHA Cholesterol Management Guideline in cardiology p

12 The 2013 ACC/AHA guideline advises consideration of statin therap

13 4, to April 1, 2015) publication of the 2013 ACC/AHA guideline among 4 mutually exclusive risk groups

14 To examine adoption of the 2013 ACC/AHA guideline in US cardiology practices.

15 Assuming full implementation, the 2013 ACC/AHA guideline would identify approximately 13 millio

16 use before and after publication of the 2013 ACC/AHA guideline.

17 The 2013 ACC/AHA guidelines increase statin eligibility most amon

18 tment disparities remain unchanged, the 2013 ACC/AHA guidelines increase the number of US adults who

19 gibility were more pronounced under the 2013 ACC/AHA guidelines than under third adult treatment pane

20 s for statin therapy, compared with the 2013 ACC/AHA guidelines, could lead to a lower number of indi

21 iation under full implementation of the 2013 ACC/AHA guidelines.

22 adult treatment panel criteria and the 2013 ACC/AHA guidelines.

23 udy was to evaluate the accuracy of the 2013 ACC/AHA Pooled Cohort Risk Equation within a large, mult

24 The 2016 USPSTF recommendations vs 2013 ACC/AHA guidelines.

25 ove the treatment goal according to the 2017 ACC/AHA and JNC7 guidelines, respectively.

26 ove the treatment goal according to the 2017 ACC/AHA and JNC7 guidelines, respectively.

27 According to the 2017 ACC/AHA and JNC7 guidelines, the crude prevalence of hyp

28 According to the 2017 ACC/AHA and JNC7 guidelines, the crude prevalence of hyp

29 among US adults using criteria from the 2017 ACC/AHA guideline and the Seventh Report of the Joint Na

30 ong U.S. adults using criteria from the 2017 ACC/AHA guideline and the Seventh Report of the Joint Na

31 Compared with the JNC7 guideline, the 2017 ACC/AHA guideline results in a substantial increase in t

32 Compared with the JNC7 guideline, the 2017 ACC/AHA guideline results in a substantial increase in t

33 ypertensive medication according to the 2017 ACC/AHA guideline.

34 ypertensive medication according to the 2017 ACC/AHA guideline.

35 IntPred predictor performs well (MCC=0.370, ACC=0.811, SPEC=0.916, SENS=0.411) and compares favourab

36 ghtly better overall performance (MCC=0.410, ACC=0.759, SPEC=0.783, SENS=0.676), but considerably wor

37 hereafter referred to using the abbreviation ACC for the first 3 letters of Accessibility) is defined

38 on is 1-aminocyclopropane-1-carboxylic acid (ACC) synthases (ACS), generally the rate-limiting step i

39 ) and 1-aminocyclopropane-1-carboxylic acid (ACC) were ineffective.

40 ne to 1-aminocyclopropane-1-carboxylic acid (ACC)] and PsACO (encode enzymes that convert ACC to ethy

41 with M/G ratio increased in the order ADM - ACC - APP.

42 he protein level and thus potentially affect ACC tumorigenesis.

43 ations [rpoB 531 (TCG/TTG) and katG 315 (AGC/ACC)] causing MDR-TB and verification of loss of the res

44 OTCH1 mutations define a distinct aggressive ACC subgroup with a significantly higher likelihood of s

45 h Thoracic Aortic Disease" and the "2014 AHA/ACC Guideline for the Management of Patients With Valvul

46 Data from the AHA/ACC/HRS guidelines on AF from 2001, 2006, 2011, and 2014

47 idence base and changes over time in the AHA/ACC/HRS guidelines on AF with respect to the distributio

48 ts of the quality of evidence underlying AHA/ACC/HRS AF guidelines have improved over time.

49 ltaneously in mice, we found that almost all ACC neurons showed increased activity before hippocampal

50 gulation is potentially reflected by altered ACC maturation.

51 t into downstream MYB functions in alternate ACC lineages.

52 fixed amino acid at the P1 position, and an ACC (7-amino-4-carbamoylmethylcoumarin) fluorescent tag

53 cose and fatty acid metabolism identified an ACC necessary for satiety quiescence in C. elegans.

54 al cell carcinoma (3 of 4 tumors [75%]), and ACC (5 of 36 tumors [14%]).

55 ions between hippocampal ripple activity and ACC neural firings.

56 ffect was observed for the extracts; ADM and ACC exhibited the highest inhibition of gastric lesions,

57 We find that FEF and ACC showed prominent synchronization at a 3-9 Hz theta a

58 le because they confirm that current FHS and ACC/AHA risk algorithms work well in black individuals a

59 ming of cognitive processing by the LPFC and ACC in normal and pathological brain states.

60 d penetrance, including mirror movements and ACC associated with a favorable developmental prognosis.

61 nding and descending fibers from the OFC and ACC.

62 vere BPD was associated with greater PFC and ACC MAO-A VT compared with moderate BPD, MDE, and health

63 In BPD, PFC and ACC MAO-A VT were positively correlated with mood sympto

64 ured with the Repetitive Behavior Scale) and ACC glutamate (rho=0.24, p=0.03).

65 ge of Cardiology/American Heart Association (ACC/AHA) cholesterol guideline advocated several changes

66 ge of Cardiology/American Heart Association (ACC/AHA) cholesterol guidelines and 2014 US Department o

67 ge of Cardiology/American Heart Association (ACC/AHA) Cholesterol Management Guideline recommends mod

68 ge of Cardiology/American Heart Association (ACC/AHA) CVD risk algorithm and the Framingham Risk Scor

69 ge of Cardiology/American Heart Association (ACC/AHA) guideline expands recommended statin use, but i

70 ge of Cardiology/American Heart Association (ACC/AHA) Guideline for the Prevention, Detection, Evalua

71 ge of Cardiology/American Heart Association (ACC/AHA) guidelines expanded eligibility criteria for st

72 ge of Cardiology/American Heart Association (ACC/AHA) guidelines.

73 ge of Cardiology/American Heart Association (ACC/AHA) Pooled Cohort Risk Equation for atherosclerotic

74 f Cardiology and American Heart Association (ACC/AHA) recommend lipid screening in all adults older t

75 ge of Cardiology/American Heart Association (ACC/AHA) recommendations in identifying African American

76 th of theta- and beta-band coherence between ACC and FEF but not variations in power predict correct

77 med significant differences in the bilateral ACC and right SFG, where GMV was significantly lower in

78 GMV was significantly lower in the bilateral ACC, median cingulate cortex (MCC) and right SFG.

79 despite an increasing tendency to stop, but ACC activity did not change in proportion with subjectiv

80 mong individuals who were statin eligible by ACC/AHA guidelines, the 10-year ASCVD incidence per 1000

81 novel mode of prediction error signaling by ACC neurons that is associated with the generation of an

82 tion who would be recommended for statins by ACC/AHA guidelines but not by USPSTF recommendations, 55

83 American individuals eligible for statins by ACC/AHA guidelines with vascular calcification and at lo

84 th isolated agenesis of the corpus callosum (ACC) without intellectual disability.

85 stologically verified adrenocortical cancer (ACC) were first screened by Taqman Human Microarray A-ca

86 comprises 7-amino-4-carbamoylmethylcoumarin (ACC) as the fluorophore and 2,4-dinitrophenyl-lysine (Ly

87 sient phases of amorphous calcium carbonate (ACC) in the construction of crystalline shells.

88 d one anhydrous amorphous calcium carbonate (ACC); that these are formed in the tissue as 400-nm part

89 d phosphorylation of acetyl-CoA carboxylase (ACC) 1 and/or ACC2 at the AMPK sites.

90 specific knockout of acetyl-CoA carboxylase (ACC) genes and treat the mice with the hepatocellular ca

91 nduced both AMPK and acetyl-CoA carboxylase (ACC) phosphorylation.

92 rate-limiting enzyme acetyl-CoA carboxylase (ACC), an attractive but traditionally intractable drug t

93 cid synthase (FASN), acetyl-CoA carboxylase (ACC), ATP citrate lyase (ACLY)].

94 rylates and inhibits acetyl-CoA carboxylase (ACC), which catalyzes carboxylation of acetyl-CoA to mal

95 itor of acetyl coenzyme A (CoA) carboxylase (ACC), the enzyme that controls the first rate-limiting s

96 inhibitor of acetyl-coenzyme A carboxylases (ACC) ACC1 and ACC2, reduces hepatic de novo lipogenesis

97 Adrenocortical carcinoma (ACC) is a rare but aggressive endocrine tumor, and the p

98 role of RARRES2 in adrenocortical carcinoma (ACC), a rare lethal malignancy in which aberrant Wnt/bet

99 intent surgery for adrenocortical carcinoma (ACC).

100 Pancreatic acinar cell carcinoma (ACC) is an aggressive exocrine tumor with largely unknow

101 carcinoma (HNSCC), adenoid cystic carcinoma (ACC), and other salivary and cutaneous cancers, whose tu

102 carcinoma (HNSCC), adenoid cystic carcinoma (ACC), and other salivary and cutaneous cancers, whose tu

103 Adenoid cystic carcinomas (ACC) are rare salivary gland cancers with a high inciden

104 of FALD, the American College of Cardiology (ACC) sponsored a stakeholders meeting on October 1 to 2,

105 The joint American College of Cardiology (ACC), American Heart Association (AHA), and Heart Rhythm

106 nes from the American College of Cardiology (ACC), the American Heart Association (AHA), and collabor

107 The 2013 American College of Cardiology (ACC)/American Heart Association (AHA) guidelines recomme

108 In the 2013 American College of Cardiology (ACC)/American Heart Association Guideline (AHA) on the T

109 orbitofrontal (OFC) and anterior cingulate (ACC) cortices has been linked with several psychiatric d

110 /mol for alginate from Cystoseira compressa (ACC) and 58,000g/mol for alginate from Dictyopteris memb

111 , we demonstrated that substrates containing ACC/Lys(DNP) exhibit 7 to 10 times higher sensitivity th

112 High available chlorine content (ACC) and long treatment time of EO water resulted in hig

113 Conversely, ACC dopamine release by progressive ratio responding to

114 ACC)] and PsACO (encode enzymes that convert ACC to ethylene), and ethylene evolution were developmen

115 tal cortex (mPFC)/anterior cingulate cortex (ACC) and brainstem pons region.

116 with overlapping anterior cingulate cortex (ACC) and insular volumetric reductions, and that FND and

117 circuit: midline anterior cingulate cortex (ACC) and left dorsal striatum.

118 the right dorsal anterior cingulate cortex (ACC) and left rostral ACC.

119 in volume in the anterior cingulate cortex (ACC) and lower structural covariance between the ACC and

120 he volumes of the anterior cingulate cortex (ACC) and periaqueductal grey, areas involved in pain pro

121 ivity between the anterior cingulate cortex (ACC) and posterior cingulate cortex (PCC) regions of the

122 tions in both the anterior cingulate cortex (ACC) and the DLPFC during REM sleep.

123 in the bilateral anterior cingulate cortex (ACC) and the right superior frontal gyrus (SFG).

124 ds (FEFs) and the anterior cingulate cortex (ACC) are commonly coactivated for cognitive saccade task

125 emerged first in anterior cingulate cortex (ACC) before dorsolateral prefrontal cortex (dlPFC), foll

126 activities in the anterior cingulate cortex (ACC) correlated with noxious intensities, and optogeneti

127 The anterior cingulate cortex (ACC) has been shown to be crucial for expression and lik

128 The anterior cingulate cortex (ACC) has shown decreased glutamate levels in patients wi

129 rtex (PCC) to the anterior cingulate cortex (ACC) in the 10-Hz range.

130 d activity of the Anterior Cingulate Cortex (ACC) in the low frequency band suggesting the presence o

131 Anterior cingulate cortex (ACC) is thought to control a wide range of reward, punis

132 nce suggests that anterior cingulate cortex (ACC) maturation during adolescence contributes to or und

133 cortex (PFC) and anterior cingulate cortex (ACC) occur in rodent models of depressive behavior and h

134 cortex (LPFC) and anterior cingulate cortex (ACC) of the primate play distinctive roles in the mediat

135 cortex (LPFC) and anterior cingulate cortex (ACC) play temporally distinct roles during the execution

136 that dopamine in anterior cingulate cortex (ACC) plays a role in evaluating effort cost for engaging

137 ctivity in dorsal anterior cingulate cortex (ACC) reflected individual choice tendencies by showing i

138 e function of the anterior cingulate cortex (ACC) remains controversial, yet many theories suggest a

139 d function of the anterior cingulate cortex (ACC), biasing the ACC to favor goal-directed behaviors t

140 thms in the mouse anterior cingulate cortex (ACC), by using optogenetics to induce oscillations in ac

141 C levels from the anterior cingulate cortex (ACC), caudate and putamen of 16 RC BD-I, 34 non-RC BD-I

142 l hippocampus, or anterior cingulate cortex (ACC), during consolidation and retrieval of context fear

143 ailability in the anterior cingulate cortex (ACC), prefrontal cortex, and insula between 14 medicatio

144 rtical areas, the anterior cingulate cortex (ACC, areas 24a and 24b) appears to be important for mood

145 udies targeting Notch1 in a genotype-defined ACC subgroup are warranted.

146 ptake in the orbitofrontal cortex and dorsal ACC.

147 However, dorsal ACC had different connectivity profiles across species.

148 th decreased connectivity between the dorsal ACC and bilateral caudate.

149 d alpha2 (10.5-12 Hz) activity in the dorsal ACC, and increased alpha2 activity in the right dlPFC.

150 everity was inversely associated with dorsal ACC volume, and the magnitude of lifetime adverse events

151 endent on translation of the plastid-encoded ACC beta-carboxylase subunit.

152 ican College of Cardiology In-Training Exam (ACC-ITE) is incorporated into most U.S. training program

153 There was a main group effect for ACC glutamate (p=0.019).

154 icipants was 69 years, and 86% qualified for ACC/AHA risk-based statin therapy, with high sensitivity

155 th DEN, and identifies an important role for ACC enzymes in redox regulation and cell survival.

156 fies RARRES2 as a novel tumor suppressor for ACC, which can function through an immune-independent me

157 ts who underwent curative-intent surgery for ACC at 13 major institutions in the United States betwee

158 rence risk after curative-intent surgery for ACC were selected to create nomograms predicting RFS and

159 th mitotane, the gold-standard treatment for ACC.

160 treatment to a level indistinguishable from ACC-deficient primary hepatocytes.

161 with a strong Granger-causal influence from ACC to FEF.

162 Sebaceous gland ACC represents an attractive therapeutic target given it

163 ine) via a single nucleotide mutation (AAC-->ACC).

164 The current work aimed at identifying ACC molecular factors subserving depression.

165 ression of hsa-miR-101 and hsa-miR-483-5p in ACC relative to ACA samples has been validated.

166 ompared to healthy controls (P<0.005) and in ACC compared to non-RC BD-I patients (P<0.05).

167 The change in ACC-ITE scores from first to third year was a strong pre

168 Glutamate concentration in ACC seems to be associated with the severity of compulsi

169 and structurally more robust and diverse in ACC than in LPFC, resulting in a lower excitatory: inhib

170 that RARRES2 expression is downregulated in ACC as compared with normal and benign adrenocortical ti

171 underlie the distinctive network dynamics in ACC and LPC during normal and pathological brain states.

172 Bromodomain inhibitors slow tumor growth in ACC primagraft models in vivo.

173 tory inputs are also significantly higher in ACC compared with LPFC neurons.

174 vel of synaptic inhibition is much higher in ACC than LPFC layer 3 pyramidal neurons, with a signific

175 natomic cases with anterograde injections in ACC/OFC to determine the extent to which corticostriatal

176 ound significantly elevated GPC+PC levels in ACC, putamen and caudate of RC BD-I patients compared to

177 RARRES2 treatment, RARRES2 overexpression in ACC cell lines not only reduced cell proliferation, cell

178 Mechanistically, RARRES2 overexpression in ACC cells inhibited Wnt/beta-catenin pathway activity by

179 ail-suspension triggered dopamine release in ACC of controls, which was severely attenuated in the mu

180 identifies promising therapeutic targets in ACC for drugs recently approved for precision cancer the

181 extracts showed that OG treatment increased ACC levels and the ratio of acetyl-CoA to free CoA in th

182 stablished cell cultures from two individual ACC PDX tumors that maintain the characteristic MYB tran

183 Measurements of internal ACC and JA concentrations in wild-type and ET-insensitiv

184 y inflexibility in local efficiency of a key ACC node in the CEN: right dorsal anterior cingulate cor

185 y, after 15min EO water treatment at 120mg/l ACC.

186 binding in the anterior striatum and limbic ACC.

187 the DNA site methylated by ModH5 as 5'-G(m6)ACC-3'.

188 rtainty-related behaviors, we show that many ACC-neurons represent expected value and uncertainty in

189 In OA conditions, Mytilus edulis has more ACC at crystalisation sites.

190 en lower-frequency oscillations in the mouse ACC and the expression of anxiety-related behaviors, pot

191 remediation therapy is shown to enhance mPFC/ACC activity even at resting state and improve cognitive

192 s significant increased activity in the mPFC/ACC in CCRT but not TAU group.

193 hermore, an index patient with NOTCH1-mutant ACC had a partial response to brontictuzumab.

194 The ability of ACC to stimulate tyloses formation when combined with JA

195 This postripple activation of ACC neurons correlated positively with ripple amplitude,

196 Interestingly, the preripple activation of ACC neurons was present during the sleep state, but not

197 Metabolomics analysis of ACC-deficient liver identifies a marked increase in anti

198 rulites that crystal growth by attachment of ACC particles is more than 100 times faster than ion-by-

199 e gene expression data from three cohorts of ACC patients to identify relevant alterations.

200 rt with little benefit, through a deficit of ACC dopamine release triggered by high-effort cost behav

201 e biomarker in the preoperative diagnosis of ACC but needs further validation in larger cohorts of pa

202 ges in hepatic mRNA or protein expression of ACC-1, FASN, PPARalpha, MCAD, ADIPOR-1, and mCPT-1.

203 Inhibition of ACC activity in the sebaceous glands is designed to subs

204 mRNA levels of ACC oxidase and ethylene levels also increased in rice r

205 In addition, the majority of ACC neurons are activated just before ripple activity du

206 s intensities, and optogenetic modulation of ACC neurons showed bidirectional control of the aversive

207 increases the inhibitory phosphorylation of ACC and decreases the conversion of acetyl-CoA to malony

208 Because phosphorylation of ACC by AMPK is known to lower liver lipid content, these

209 ed liver AMPK activation, phosphorylation of ACC, and plasma beta-hydroxybutyrate concentrations.

210 o regulate activity of a small population of ACC neurons, a process that may play a critical role in

211 edict RFS and OS after surgical resection of ACC were proposed using a multi-institutional cohort of

212 aken together, the results support a role of ACC in cognitive control of frontoparietal networks and

213 ence representations may support the role of ACC in many behavioral states that are either enhanced b

214 specificity in two distinct subdivisions of ACC with different AMPA/N-methyl-D-aspartate receptor pr

215 Additional mutations found in one ACC culture also seen in the PDX tumor.

216 00 patient-years, those indicated under only ACC/AHA guidelines were at low to intermediate risk (4.1

217 ictable chronic mild stress, and optogenetic ACC stimulation and were evaluated using novelty suppres

218 dorsal hippocampal, ventral hippocampal, or ACC infusions immediately 'before' testing (retrieval) d

219 Other ACC-neurons signal prediction information about rewards

220 both highlights the functional roles of PCC-ACC connectivity in the resting state and provides insig

221 This study sought to personalize ACC/AHA risk-based statin eligibility using noninvasive

222 structure-to-function correlation in the PFC/ACC in the MDD group.

223 ethods and modalities alterations in the PFC/ACC were a common finding, each modality and method dete

224 red glutamate and glutamine in the pregenual ACC (pgACC) and the anterior midcingulate cortex at 1 an

225 the ACC enzymes ACC1 and ACC2 that prevents ACC subunit dimerization-to suppress fatty acid synthesi

226 jections of an anterograde tracer in the rat ACC and OFC.

227 DCA storage reduces ACC (1-aminocyclopropane-1-carboxylate) oxidase activity

228 predictable chronic mild stress and repeated ACC optogenetic stimulation and is reversed by fluoxetin

229 rior cingulate cortex (ACC) and left rostral ACC.

230 PPARG2, and SIRT1) and lipogenesis (SREBP1c, ACC, LPL, and FASN).

231 (STS/ACC Transcatheter Valve Therapy Registry [TVT Registry];

232 ing TAVR in centers participating in the STS/ACC TVT Registry from November 1, 2011, to February 28,

233 Cardiology Transcatheter Valve Therapy (STS/ACC TVT) Registry.

234 Based on this European population study, ACC/AHA and ESC prevention guidelines often did not alig

235 occipital/fusiform gyrus and left subgenual ACC.

236 Compared with LPFC-mediated tasks, ACC-mediated tasks can span longer timescales and requir

237 ortem dissolution, but the finding here that ACC particles are formed inside tissue may make coral sk

238 usly with these two compounds indicated that ACC and JA regulate each other's concentration in an ET-

239 certainty signals in the brain suggests that ACC could be a target for the treatment of uncertainty-r

240 The ACC is a single-value measure that captures an individua

241 The ACC is a structure that sits at the intersection of seve

242 The ACC represents an individual's access to text across the

243 The ACC-ITE is strongly associated with performance on the A

244 The ACC/AHA guideline would potentially result in up to 12.3

245 (55.2%) with a CAC score greater than 0; the ACC/AHA guidelines identified 507 individuals (69.3%) (r

246 Since the DLPFC and the ACC are implicated in memory and emotional regulation, a

247 in memory and emotional regulation, and the ACC has motor areas and is thought to be important for e

248 The correlation between the ACC and Early Treatment Diabetic Retinopathy Study visua

249 ctivity (termed 'inflexibility') between the ACC and task-negative networks, no study has used networ

250 and lower structural covariance between the ACC and the cerebellum were observed in the MDD group.

251 anterior cingulate cortex (ACC), biasing the ACC to favor goal-directed behaviors that converge on dr

252 the LPFC, integration of information by the ACC can span longer timescales and requires stronger eng

253 l integration and network oscillation by the ACC.

254 rd atherosclerotic CVD (ASCVD) following the ACC/AHA guideline, 10-year risk of CVD mortality followi

255 By recording from the ACC and hippocampal CA1 simultaneously in mice, we found

256 We recorded from the ACC as rats performed a task identical to one that relia

257 red with those without, most robustly in the ACC (p = .008) and insula (p = .023).

258 of large effect size and significant in the ACC (p = .022, Cohen's d = 0.95), with smaller nonsignif

259 the presence of low gamma modulations in the ACC and Superior Temporal Gyrus may associate with incre

260 that these theta and beta activities in the ACC and the DLPFC, two relatively distant but reciprocal

261 r modulating reward-related signaling in the ACC can reverse the atypical valuation of nondrug and dr

262 predominantly microglial activation, in the ACC during a moderate to severe major depressive episode

263 d depression, gene expression changes in the ACC highlighted the overexpression of a regulator of the

264 ted a single-cell correlate of the FN in the ACC of rodents.

265 ontictuzumab was observed exclusively in the ACC patient-derived xenograft model that harbored a NOTC

266 aling is much more robust and diverse in the ACC than in the LPFC.

267 d acute pain intensity representation in the ACC to increase the aversive response to noxious stimuli

268 Global connectivity in the ACC was negatively correlated with reported depressive s

269 , respectively, and dendritic changes in the ACC were evident.

270 integration and oscillatory behavior in the ACC.

271 the phase of the 3- to 4-Hz activity in the ACC.

272 Despite differences in the ACC/AHA and EAS/ESC guidelines, the numbers of adults ag

273 associated with the power alteration in the ACC: strong influence corresponded with a decrease of po

274 Post-training infusions of CGP into the ACC, however, did not promote generalized fear.

275 At a population level, the ACC/AHA guideline for expanded statin use for primary pr

276 rk-level topology in eight subregions of the ACC (spanning three task-positive networks) in 42 unmedi

277 ity of ND-646-an allosteric inhibitor of the ACC enzymes ACC1 and ACC2 that prevents ACC subunit dime

278 proaches to investigate inflexibility of the ACC in task-positive networks to understand adolescent M

279 OG is a pro-drug of the ACC inhibitor 5-(tetradecyloxy)-2-furoic acid and was de

280 architectonical receptor fingerprints of the ACC subregions.

281 signal believed to index sensitivity of the ACC to rewards-while participants engaged in two feedbac

282 Here, a new bentho-pelagic version of the ACC-HUMAN model was evaluated for polychlorinated biphen

283 el to estimate the cost-effectiveness of the ACC/AHA guideline relative to current use, Adult Treatme

284 S) to investigate the influence of OA on the ACC formation in the shells of adult Mytilus edulis.

285 -related negativity (FN), is evoked over the ACC whenever expectations are violated.

286 is recognized and cleaved by a protease, the ACC is released and produces a readable fluorescence sig

287 t in 28.9 million more statin users than the ACC/AHA guideline, with a marginal number needed to trea

288 djustments in executive control and that the ACC might be just one component of overlapping distribut

289 We therefore propose that the ACC/Lys(DNP) pair can be considered a novel and sensitiv

290 ysis examined fellows (n = 511) who took the ACC-ITE in the first and third years.

291 4 mutually exclusive risk groups within the ACC Practice Innovation and Clinical Excellence Registry

292 These data point to ACC MKP-1 as a key factor in the pathophysiology of depr

293 ould be recommended for statin therapy under ACC/AHA guidelines compared with 47.6% (95% CI, 44.6%-50

294 ary arginine level significantly upregulated ACC, FAS and G6PDH mRNA levels in the fat synthesis path

295 shed the induction of tyloses by JA, whereas ACC was synergistic with JA.

296 Here we test whether ACC and FEF circuits coordinate through phase synchroniz

297 These changes are consistent with ACC inhibition.

298 from patients with ACC and in a patient with ACC who was enrolled in a phase I study.

299 atient-derived xenografts from patients with ACC and in a patient with ACC who was enrolled in a phas

300 DFS estimates for patients with ACC improved dramatically over time, in particular among