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1 ke aneuploid mammary cancers in ovary-intact ACI rats.
2 zymes in the striatum of male but not female ACI rats.
3 P5 and injected intravenously into syngeneic ACI rats.
4 ations in corticosterone compared to LEW and ACI rats.
5 induced solely by hormones in ovariectomized ACI rats.
6 tolerant state was transferred to irradiated ACI rats (400 rad) with either purified T cells (4-10 x
7 actotroph number, was increased 10.6-fold in ACI rats and 4.5-fold in COP rats.
8                     August-Copenhagen-Irish (ACI) rats are unique in that the ovary-intact females de
9 eterotopically into the abdomen of untreated ACI rats as controls (group 1).
10 to cardiac and islet allografts in the WF-to-ACI rat combination and therefore hypothesized that iden
11 5+/-1.0 days to 18.6+/-1.8 days in the WF-to-ACI rat combination.
12  (thymic) DCs on islet survival in the WF-to-ACI rat combination.
13  DC on islet allograft survival in the WF-to-ACI rat combination.
14 manent islet allograft survival in the WF-to-ACI rat combination.
15 tolerance to cardiac allografts in the WF-to-ACI rat combination.
16  of pure WF-derived peptide u-5 in the WF-to-ACI rat combination.
17 on and found this to be true in the Lewis-to-ACI rat combination.
18                      In BN-to-LEW and GFP-to-ACI rat combinations, donor bone marrow (BM) infusion to
19                   Whereas nearly 100% of the ACI rats developed mammary cancer when treated continuou
20 12 wk of E2 treatment, the mammary glands of ACI rats exhibited a significantly greater proliferative
21 Two-thirds of the induced mammary cancers in ACI rats exhibited aneuploidy.
22 continuously with 17beta-estradiol (E2), the ACI rat exhibits a genetically conferred propensity to d
23 easured in brain of August-Copenhagen Irish (ACI) rats exposed chronically to low doses of estradiol
24      In vivo primed thymic DCs isolated from ACI rats given intrathymic inoculation of P5 for 2 days
25                                       PVG-to-ACI rat heterotopic cardiac transplantation procedures w
26                                       PVG-to-ACI rat heterotopic cardiac transplantations were perfor
27                                     A PVG to ACI rat heterotopic heart transplantation model was used
28                                              ACI rat intestinal grafts transplanted into Lewis rat re
29            Our laboratory has shown that the ACI rat is uniquely susceptible to 17beta-estradiol (E2)
30 ed a high degree of homology with a 99 amino acid rat liver-kidney perchloric acid-soluble protein (L
31 in and centrin, rose significantly in female ACI rat mammary glands and remained elevated in mammary
32 ransplantations were performed in the PVG-to-ACI rat model and followed over the course of 120 days.
33 vious studies in the intrahippocampal kainic acid rat model of chronic epilepsy that provide evidence
34  behavioral deficits in the 3-nitropropionic acid rat model of HD.
35  WF donor a.a.) injected subcutaneously into ACI rats modestly prolonged the survival of WF hearts to
36 eviously identified in (DA x F344) and (DA x ACI) rats, only Cia1 in the major histocompatibility com
37 n of splenic T-cells obtained from syngeneic ACI rats primed with intravenous injection of P5-pulsed
38                                 WF hearts in ACI rats receiving 7 days of CsA exhibited myocardial fi
39  expression, grafts were retransplanted into ACI rat recipients and reperfused for 4 or 8 hours or 90
40     Mammary carcinogenesis in ovariectomized ACI rats requires continuous exposure to high concentrat
41 rm graft recipients into naive unmanipulated ACI rats resulted in indefinite survival of secondary Wi
42 Sensitized WF rats rejected VCTA grafts from ACI rats significantly faster (P<0.05) than unsensitized
43 ry cancer in crosses between the susceptible ACI rat strain and the genetically related, but resistan
44 ion H4IIE cells into the liver parenchyma of ACI rats, they typically form a 1-cm tumor.
45 te mismatch (AKR to C57BL/6), and xenograft (ACI rat to B6AF1).
46                    The susceptibility of the ACI rat to E2-induced mammary cancer appears to segregat
47 esis, six-week-old intact and ovariectomized ACI rats were continuously exposed to low- and high-dose
48 imental model, streptozocin-induced diabetic ACI rats were grafted with an average of 1200 syngeneic
49 r hearts obtained from either Lewis (LEW) or ACI rats were heterotopically transplanted in recipient
50          In contrast, kidney allografts from ACI rats were hyperacutely rejected within 30 min by sen
51                                    Recipient ACI rats were pretreated with M40401 or vehicle control.
52                                              ACI rats were selected for study because this strain is
53                                              ACI rats were subjected to 75 min SMA clamp-induced isch
54                                              ACI rats were subjected to small bowel manipulation, aft
55                          Male LEW, F344, and ACI rats were surgically prepared with indwelling jugula
56                         Thoracic aortas from ACI rats were transplanted heterotopically into the abdo
57                         Also, ovariectomized ACI rats were treated with high-dose estrogen plus proge
58                                              ACI rats were treated with PY, whereas control animals r
59                                   AxC-Irish (ACI) rats were given intrahepatic injections of rat hepa
60 trogen plus progesterone, and ovariectomized ACI rats with high-dose estrogen plus progesterone.
61 injection in BALB/c mice; acute rejection in ACI rats with transplanted heterotopic PVG cardiac allog
62                        The i.p. injection of ACI rats with Wistar-Furth rat splenocytes displaying SA
63 elf-administration more rapidly than F344 or ACI rats, yet LEW rats display reduced corticosterone re

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