ライフサイエンスコーパス: AHI

1 AHI was not associated with cancer incidence in the adju

2 14.9, and > or =15, respectively (P=0.0049, AHI > or =15 versus <1).

3 I, 55 +/- 6/h) and control subjects (n = 14; AHI, 4 +/- 1/h) were extracted from the genioglossus mus

4 1,655 MUPTs from patients with OSA (n = 17; AHI, 55 +/- 6/h) and control subjects (n = 14; AHI, 4 +/

5 ction (AHI) detection was evaluated using 21 AHI specimens (range, 1,520 to 500,000 copies/ml) previo

6 I 15 to 29.9, OR=2.38 [95% CI 1.30 to 4.38]; AHI > or =30, OR=2.24 [95% CI 1.10 to 4.54]).

7 edominant obstructive apneas (OA) was 12.8% (AHI >/=5 events/h with OAs >50%; nighttime: 14.7%; dayti

8 requires symptoms in addition to an abnormal AHI (>/=5/h with symptoms, prevalence 2%-4%).

9 f this study were to determine how OA alters AHI and four phenotypic traits (upper-airway anatomy/col

10 1 event/h, and severe OSAS was defined by an AHI greater than or equal to 5 events/h.

11 Sleep-disordered breathing was defined by an AHI of at least 10 (mild to moderate) or of at least 15

12 In adolescents free of known CVD, an AHI > or =5 is associated with increasing levels of CRP,

13 dy mass index (BMI) and percent body fat, an AHI gt-or-equal, slanted 5 events/h was associated with

14 the treatment group (35 [51%] of 68) had an AHI reduction from baseline of 50% or greater at 6 month

15 ignificance of mild SDB, we estimate that an AHI of 15 is equivalent to the decrement in psychomotor

16 litus with an AHI of 15 or greater versus an AHI of less than 5 was 2.30 (95% confidence interval, 1.

17 In participants with an AHI > or = 1, there was no independent association betwe

18 ke systolic blood pressure in people with an AHI < 1; a 2 standard deviation increase in the SFI was

19 mprised the control group, and those with an AHI greater than or equal to 10 were diagnosed with OSA

20 Fifteen patients (10 men) with an AHI greater than or equal to 20 (10 OSA; 5 CSA) particip

21 re 1.00, 1.30, and 1.65 for patients with an AHI of 10 to 29.9, 30 to 59.9, and >/= 60 events/h, resp

22 ician diagnoses of diabetes mellitus with an AHI of 15 or greater versus an AHI of less than 5 was 2.

23 loping diabetes mellitus within 4 yr with an AHI of 15 or more compared with an AHI of less than 5 wa

24 r with an AHI of 15 or more compared with an AHI of less than 5 was 1.62 (95% confidence interval, 0.

25 betes compared with 2.8% of subjects with an AHI of less than 5.

26 By ordinal logistic regression analysis, AHI was significantly associated with age (odds ratio [O

27 ter adjusting for percent body fat, BMI, and AHI.

28 an snoring sound intensity (301-850 Hz), and AHI could significantly predict the surgical response.

29 In severe OSA subjects (n = 22), NFI and AHI had a Spearman correlation coefficient of 0.44 (p =

30 In the total sample, NFI and AHI were moderately correlated (r = 0.358; p = 0.001).

31 ty, specificity, area under the curve (AUC), AHI, Epworth Sleepiness Scale (ESS) scores, blood pressu

32 ex with men (MSM) undergoing community-based AHI screening in San Diego, California.

33 man correlation coefficient of 0.246 between AHI severity and periodontal disease severity categories

34 , there was a consistent association between AHI and all-cause mortality.

35 r =60 years, no adjusted association between AHI and systolic/diastolic hypertension was found.

36 indicated a significant association between AHI severity categories and percentage of sites with pla

37 The correlations between AHI and per cent predicted MIP and VC were less strong (

38 rity of SDB (P value for interaction between AHI and FEV1, 0.004).

39 The relationship between AHI and RNFL parameters was also evaluated.

40 involving one or more genes regulating both AHI and BMI levels.

41 index was significantly associated with both AHI and hypoxemia index after adjustment for age, sex, e

42 1-4.6) when the diagnosis was established by AHI >/=10/h).

43 ABA levels, but not Glx, and SDB severity by AHI (r = -0.68, P < 0.0001), and a positive correlation

44 This study is the first to characterize AHI among Nigerians identified as HIV infected before se

45 Under each condition, AHI and the phenotypic traits were assessed.

46 e efficacious than MAD in reducing AHI (CPAP AHI, 4.5 +/- 6.6/h; MAD AHI, 11.1 +/- 12.1/h; P < 0.01)

47 ted annual costs of 3 algorithms that detect AHI based on HIV nucleic acid amplification testing (Ear

48 The change in AHI with the device (AHI without device - AHI with device) was directly relat

49 in AHI with the device (AHI without device - AHI with device) was directly related to the AHI without

50 During AHI, a viral latent pool reservoir develops, the immune

51 nst heterologous T/F viruses appeared during AHI at the first time point sampled in each of the 5 don

52 ecificity of antiviral CD8(+) T cells during AHI.

53 mpen leukocyte extravasation into CNS during AHI.

54 assays, and a model of viral dynamics during AHI.

55 that were shown to drive HIV-1 escape during AHI.

56 unctions were assessed longitudinally during AHI in five donors from the CHAVI 001 cohort using a CD8

57 he faster and stronger cytokine storm during AHI could promote disease progression.

58 After adjustment for AHI, the primary linkages to BMI remained suggestive but

59 ients younger than 65 years (adjusted HR for AHI >43 vs. <18.7, 1.66; 95% CI, 1.04-2.64).

60 demonstrated improved efficiency and PPV for AHI case detection compared to individual NAAT.

61 ficiency and test performance of testing for AHI, but optimizing the pooling algorithm can be difficu

62 termined the ability of circulating IgG from AHI to bind infectious versus noninfectious virions.

63 us control groups achieving a 50% or greater AHI reduction from baseline to 6 months, measured by a f

64 retic peptide level, renal dysfunction, 24-h AHI, CAI, and time with oxygen saturation of <90% were i

65 A higher AHI and higher hypoxemia index were also associated with

66 Children with higher AHI (>5 per hour of total sleep time) were significantly

67 oss the groups, with individuals with higher AHI showing worse performance.

68 her odds of systolic/diastolic hypertension (AHI 15 to 29.9, OR=2.38 [95% CI 1.30 to 4.38]; AHI > or

69 ary outcome was baseline to 2-year change in AHI on diagnostic polysomnography scored by staff blinde

70 The change in AHI with the device (AHI without device - AHI with devic

71 There were significant improvements in AHI (30 +/- 2/h versus 14 +/- 2/h, p < 0.0001), MinSa(O(

72 terval 1.02 to 1.26] per 10-unit increase in AHI).

73 uction in LG and the percentage reduction in AHI (r =0.660, P =0.05).

74 e independent predictors of the reduction in AHI (r(2) = 0.70; P = 0.001).

75 The reduction in AHI correlated with the reduction in total body ECFV (r

76 sult in a statistically greater reduction in AHI despite major differences in weight loss.

77 This reduction in AHI was accompanied by a virtual elimination of snoring,

78 l airway had a greater than 80% reduction in AHI with the device.

79 bjects (68%) had at least a 50% reduction in AHI with the device.

80 l response (PR) as a > or = 50% reduction in AHI, but remaining > or = 5/h.

81 e independent predictors of the reduction in AHI.

82 tions aimed at controlling cytokine storm in AHI may be beneficial to slow eventual disease progressi

83 , 0.3-0.6 SDs), all favoring CPAP, including AHI (15 +/- 16 and 8 +/- 6/hour, respectively), effectiv

84 rachycephaly is associated with an increased AHI in whites but not in African-Americans.

85 eased chemoreflex sensitivity, and increased AHI (6 +/- 2/h vs. 14 +/- 3/h), RRVI (5 +/- 3/h vs. 20 +

86 The OR for increased AHI per 1-unit increase in BMI decreased from 1.21 (95%

87 The OR for increased AHI per 10-year age increase was 2.41 in women (95% CI,

88 Increasing AHI was significantly associated with decreasing AVR (P

89 tionship was demonstrated between increasing AHI and attenuated retinal vascular pulsation amplitude

90 regression analyses revealed that increasing AHI was associated with worsening insulin resistance ind

91 l participants with an apnea-hypopnea index (AHI) > or = 1 were analyzed separately.

92 cclusion increased the apnea hypopnea index (AHI) (occlusion mean = 6.6 +/- 8.0 versus baseline mean

93 uantitative phenotypes apnea-hypopnea index (AHI) and body mass index (BMI).

94 or association between apnea-hypopnea index (AHI) and health outcomes among community-based participa

95 mnographically derived apnea-hypopnea index (AHI) and hypoxemia index (percent of sleep time with oxy

96 as quantified with the apnea hypopnea index (AHI) and oxygen desaturation measures.

97 Apnea-hypopnea index (AHI) and percent nighttime with oxygen saturation less t

98 ationships between REM apnea-hypopnea index (AHI) and prevalent hypertension.

99 categorized using the apnea-hypopnea index (AHI) and the hypoxemia index (percent sleep time below 9

100 rtality, with FEV1 and apnea-hypopnea index (AHI) as the primary exposure indicators along with sever

101 hing, depending on the apnea-hypopnea index (AHI) cutoff, ranged from 40 to 60%.

102 , defined as having an Apnea-Hypopnea Index (AHI) greater than 15 as assessed by polysomnography, and

103 models, patients with apnea-hypopnea index (AHI) greater than 30 had a 30% higher hazard of developi

104 OSAS was defined by an apnea/hypopnea index (AHI) greater than or equal to 1 event/h, and severe OSAS

105 day, subjects with an apnea-hypopnea index (AHI) greater than or equal to 20 had fluid removed by UF

106 al disease between the apnea-hypopnea index (AHI) groups, with a negligible Spearman correlation coef

107 d facial form with the apnea hypopnea index (AHI) in 364 white individuals and 165 African-Americans.

108 Women with an apnea-hypopnea index (AHI) less than 10 comprised the control group, and those

109 Women with an apnea-hypopnea index (AHI) less than 10 were the control group.

110 Patients with an apnea-hypopnea index (AHI) less than 15 were the control group.

111 nt hypertension and an apnea-hypopnea index (AHI) of 15 or higher.

112 ion of 45% or less, an apnea-hypopnea index (AHI) of 15 or more events (occurrences of apnea or hypop

113 7% of subjects with an apnea-hypopnea index (AHI) of 15 or more had a diagnosis of diabetes compared

114 (<6 months) OSA and an apnea-hypopnea index (AHI) of 20 events/hour or more.

115 ce varies based on the apnea-hypopnea index (AHI) threshold used for the evaluation (>/=5 events/h, p

116 oms and a reduction in Apnea/Hypopnea Index (AHI) to < 5/h, and partial response (PR) as a > or = 50%

117 Mean apnea-hypopnea index (AHI) was 52.6 +/- 28.2 (SD) events/h without the device

118 The apnea-hypopnea index (AHI) was derived from standardized sleep tests; diabetes

119 cal data including the apnea-hypopnea index (AHI), and nocturnal oxygen saturation (O2sat) parameters

120 uccess at reducing the apnea-hypopnea index (AHI), and predicting responders is challenging.

121 P 8/14 levels and with apnea-hypopnea index (AHI), BMI z score, and apolipoprotein B levels.

122 s were the obstructive apnea-hypopnea index (AHI), central apnea index (CAI >/= 5), and Cheyne-Stokes

123 groups on the basis of apnea-hypopnea index (AHI), followed by comparisons of cognitive function, wit

124 In combination with apnea-hypopnea index (AHI), identification of "respiratory events," consisting

125 a 55% reduction in the apnea-hypopnea index (AHI), which decreased from a preflight value of 8.3 +/-

126 was quantified by the apnea-hypopnea index (AHI)-the average number of apneas and hypopneas per hour

127 SAS and calculation of Apnea-Hypopnea Index (AHI).

128 ask leak, and residual apnea-hypopnea index (AHI).

129 ub-groups based on the apnea/hypopnea index (AHI): mild, moderate, or severe OSA.

130 ity was defined by the apnea-hypopnea index (AHI): severe >30, moderate >15-30, mild 5-15, and contro

131 Apnea-hypopnea index (AHI, the average number of apneas plus hypopneas per hou

132 come measures included apnea-hypopnea index (AHI; average number of apneas plus hypopneas per hour of

133 come measures were the apnea-hypopnea index (AHI; the number of apnea or hypopnea events per hour, wi

134 correlated with the apnoea/hypopnoea index (AHI) (P = 0.035, 0.042 and 0.026, respectively).

135 OSA (mean +/-s.e.m., apnoea-hypopnoea index (AHI) 48.9 +/- 5.9 events h(-1)) during non-rapid eye mov

136 as quantified as the apnoea-hypopnoea index (AHI) and respiratory rate variability index (RRVI).

137 ble patients with an apnoea-hypopnoea index (AHI) of at least 20 events per h, tested by a polysomnog

138 The apnoea-hypopnoea index (AHI)was assessed on separate nights using standard polys

139 and clinical criteria (apnea/hypopnea index [AHI] > or = 10 and the presence of daytime symptoms) was

140 741 patients with SDB (apnea-hypopnea index [AHI] >/= 10 events/h).

141 ce of predominant CAs (apnea/hypopnea index [AHI] >/=5 events/h, with CA of >50%) was 64.8% (nighttim

142 s diagnosed with SAHS (apnea/hypopnea index [AHI] >or= 5/hour, and >or= 2 symptoms including sleepine

143 o have normal results (apnea hypopnea index [AHI] <5).

144 /=50% reduction in the apnea-hypopnea index [AHI] and <10 events/hour) in patients with OSAHS.

145 tified as free of OSA (apnea-hypopnea index [AHI] of <5 events/h and not treated) by 2 baseline polys

146 er 10-unit increase in apnea-hypopnea index [AHI]) but not in older men or in women of any age.

147 h moderate-severe OSA (apnea hypopnea index [AHI], 25.6 [SD 12.3]) were randomly assigned to a treatm

148 es, 13 females; median apnea-hypopnea index [AHI], 35) not receiving hypotensive medication.

149 sleep apnea probands (apnea-hypopnea index [AHI]: 43.2 +/- 26.3 events/h), 55 proband siblings (AHI:

150 with positional OSAS (apnea-hypopnea index [AHI]supine:AHInon-supine >/= 2) were prospectively enrol

151 and hypopnea per hour (apnea-hypopnea index, AHI).

152 eas per hour of sleep (apnea-hypopnea index, AHI).

153 CD8(+) T cells during acute HIV-1 infection (AHI) can elucidate the nature of the CD8(+) responses th

154 (Ab) produced during acute HIV-1 infection (AHI) is critical for defining antibody antiviral capabil

155 lly understood during acute HIV-1 infection (AHI).

156 imen pooling scheme for acute HIV infection (AHI) detection was evaluated using 21 AHI specimens (ran

157 ted during acute seronegative HIV infection (AHI) in whom RCI has been stringently quantified, we fou

158 uman immunodeficiency virus (HIV) infection (AHI) can be defined as the time from HIV acquisition unt

159 uman immunodeficiency virus (HIV) infection (AHI) is limited by cost and decision algorithms to bette

160 uman immunodeficiency virus (HIV) infection (AHI) may account for a significant proportion of HIV-1 t

161 cute human immunodeficiency virus infection (AHI); however, the cost of nucleic acid amplification te

162 toms of excessive daytime somnolence and low AHI this may help diagnose the UARS and separate it from

163 ificantly more impaired than all three lower AHI groups, indicating a dose-response impact of SDB.

164 d coronary heart disease (upper versus lower AHI quartile) were 2.38 (1.22-4.62), 1.58 (1.02- 2.46),

165 n reducing AHI (CPAP AHI, 4.5 +/- 6.6/h; MAD AHI, 11.1 +/- 12.1/h; P < 0.01) but reported compliance

166 In these patients, the mean AHI (36+/-12 [SD] versus 4+/-3 per hour, P=0.0001), arou

167 e adaptive servo-ventilation group, the mean AHI at 12 months was 6.6 events per hour.

168 In the randomized phase, the mean AHI score did not differ significantly from the 12-month

169 The mean AHI was 40.4 (SD, 18.9) and an average of 3.8 antihypert

170 D], 46 +/- 9 years; Epworth 14 +/- 4; median AHI, 22/hour; interquartile ratio [IQR], 11-43/hour).

171 p was highly prevalent in the sample (median AHI: 4.4; interquartile range: 1.3 to 11.0).

172 The median AHI score at 12 months decreased 68%, from 29.3 events p

173 n a multivariate proportional hazards model, AHI and nocturnal hypoxemia were independent predictors

174 OSA was defined as mild to moderate (AHI, 15-29) or severe (AHI, >/=30).

175 0 or higher (classified as mild to moderate [AHI of 10 to 29] or severe [AHI >/=30]).

176 associated with increased cardiac mortality (AHI of </>/=15 events/h; log-rank: 6.6, 8.7, and 5.3, re

177 5.4] versus 2.0 [1.4-3.5]; P <0.05) and NREM AHI (50 [36-57] versus 24 [13-42] events h-1; P <0.05),

178 s breathing, but not an elevated obstructive AHI, is significantly associated with increased risk of

179 Cheyne-Stokes breathing but not obstructive AHI were significant predictors of incident heart failur

180 criteria, the prevalence of OSA (obstructive AHI > or = 20) showed an age distribution similar to tha

181 lexes (ICs), present in approximately 90% of AHI subjects, and compared the levels and antibody speci

182 ertension, comparing the highest category of AHI (> or = 30 per hour) with the lowest category (< 1.5

183 of a pooling algorithm for the detection of AHI cases in a wide variety of settings.

184 ffectiveness ratios (ICERs) per diagnosis of AHI were calculated for programs with HIV prevalence rat

185 Identification of AHI by the dual EarlyTest screening algorithm is likely

186 pooling with NAAT for the identification of AHI cases; these models incorporate group testing theory

187 97, and 1.66 mg/L for SDB severity levels of AHI <1, 1 to 4.9, 5 to 14.9, and > or =15, respectively

188 eous manifestations of CVD within a range of AHI values that are considered normal or only mildly ele

189 s significantly associated with reduction of AHI (weighted mean difference [WMD], -33.8 [95% CI, -42.

190 (LR, 0.07; 95% CI, 0.03-0.19 at threshold of AHI >/=15/h).

191 Menopausal groups did not differ on AHI, arousal index, or oxygen saturation nadir in either

192 e control plate had no significant effect on AHI and MinSa(O(2)).

193 f asthma with 4-year incidences of both OSA (AHI of >/=5 or positive airway pressure treatment) and O

194 CI and FI differed for whites with OSA (AHI > or = 15) versus those without OSA (AHI < 5) (incre

195 SA (AHI > or = 15) versus those without OSA (AHI < 5) (increased CI and decreased FI in subjects with

196 o, EarlyTest was cost-savings (ie, ICERs per AHI diagnosis less than $13.000) when compared with the

197 Multiple treatments for OSA reduce AHI, ESS scores, and blood pressure.

198 abbits restored KLF2 expression, and reduced AHI (7 +/- 2/h), RSNA (18 +/- 2% max) and arrhythmia inc

199 s programs were also associated with reduced AHI and excessive sleepiness.

200 AP was more efficacious than MAD in reducing AHI (CPAP AHI, 4.5 +/- 6.6/h; MAD AHI, 11.1 +/- 12.1/h;

201 vealed a significant association between REM AHI categories and the development of hypertension (P tr

202 han or equal to 5, a twofold increase in REM AHI was associated with 24% higher odds of hypertension

203 In individuals with non-REM AHI less than or equal to 5, a twofold increase in REM A

204 Non-REM AHI was not a significant predictor of hypertension in a

205 lent hypertension were most evident with REM AHI greater than or equal to 15.

206 t were overweight or obese, and 12% had SDB (AHI > or =5).

207 idence interval [CI], 0.5-2.7) for mild SDB (AHI, 5-14.9), 2.0 (95% CI, 0.7-5.5) for moderate SDB (AH

208 d an age-adjusted prevalence of minimal SDB (AHI >/= 5), moderate SDB (AHI >/= 15), and severe SDB (A

209 ce of minimal SDB (AHI >/= 5), moderate SDB (AHI >/= 15), and severe SDB (AHI >/= 30) of 25.8, 9.8, a

210 .9), 2.0 (95% CI, 0.7-5.5) for moderate SDB (AHI, 15-29.9), and 4.8 (95% CI, 1.7-13.2) for severe SDB

211 8.5 kg/m2) and had evidence of moderate SDB (AHI: 47.6 +/- 29.3 events/h).

212 , and 4.8 (95% CI, 1.7-13.2) for severe SDB (AHI >/= 30) (P-trend = 0.0052).

213 , moderate SDB (AHI >/= 15), and severe SDB (AHI >/= 30) of 25.8, 9.8, and 3.9%, respectively.

214 .9 per 1,000 person-years in those with SDB (AHI >/=5 events/h) and 18.2 per 1,000 person-years in th

215 age +/- SD: 62.3 +/- 1.3 years) without SDB (AHI < 5).

216 as mild to moderate (AHI, 15-29) or severe (AHI, >/=30).

217 ild to moderate [AHI of 10 to 29] or severe [AHI >/=30]).

218 +/- 1.7 events/h), and 55 control siblings (AHI: 4.2 +/- 4.0 events/h).

219 3.2 +/- 26.3 events/h), 55 proband siblings (AHI: 11.8 +/- 16.6 events/h), 55 control subjects (AHI:

220 Sixteen AHIs were identified from 28 655 persons screened.

221 apnea and hypopnea events per hour of sleep (AHI), but were associated with decreased mean lowest oxy

222 e interval [CI], 13%-21%) had a second-study AHI of at least 10 and 29 (10%) participants (95% CI, 7%

223 icipants (95% CI, 7%-14%) had a second-study AHI result of at least 15.

224 1.8 +/- 16.6 events/h), 55 control subjects (AHI: 2.1 +/- 1.7 events/h), and 55 control siblings (AHI

225 r testing for detecting severe OSA syndrome (AHI >/=30 and ESS score >10) was AUC 0.80 (95% CI, 0.78

226 ts experiencing a mild form of the syndrome (AHI < 15, n = 18), symptoms, treatment efficacy and sati

227 epwise multiple regression model showed that AHI was independently associated with FOXP3 DNA methylat

228 These results suggest that AHI, nocturnal hypoxemia, and sleep fragmentation are in

229 The AHI decreased by 14.0 events/hour (95% CI, 3.3 to 24.6 e

230 ass index (BMI), neck circumference, and the AHI.

231 For the AHI results of at least 15, we estimate that about 2.5%

232 ence interval [CI], 20%-45%) increase in the AHI.

233 cted a 26% (95% CI, 18%-34%) decrease in the AHI.

234 e second, third, and fourth quartiles of the AHI (versus the first) were 0.98 (0.77-1.24), 1.28 (1.02

235 e after controlling for the influence of the AHI.

236 OA therapy reduced the AHI (30 +/- 5 vs. 11 +/- 2 events/h; P < 0.05), which wa

237 and 7.2 events per hour, respectively); the AHI score was significantly higher (indicating more seve

238 AHI with device) was directly related to the AHI without the device.

239 lowing removal of 2.17 +/- 0.45 L by UF, the AHI decreased by 36% (43.8 +/- 20.3 to 28.0 +/- 17.7; P

240 structive sleep apnea was diagnosed when the AHI was 10 or higher (classified as mild to moderate [AH

241 d a dose response with SDB above a threshold AHI of 5.

242 g indicated that the evidence for linkage to AHI was effectively removed after adjustment for BMI, wi

243 1.40) gave the most evidence for linkage to AHI.

244 ociation between logarithmically transformed AHI (LogAHI) and psychomotor efficiency score independen

245 Among other OSA-related variables, AHI in rapid eye movement sleep and time spent with oxyg

246 oved very difficult to find individuals with AHI either in longitudinal cohorts of subjects at high r

247 Men with AHI > or =30 were 58% more likely to develop heart failu

248 nd characterized individuals in Nigeria with AHI.

249 ciated with the hypoxemia index but not with AHI.

250 44.1 (SD 9.90) g/m(2.7) in participants with AHI > or = 30 (n=84) events per hour.

251 41.3 (SD 9.90) g/m(2.7) in participants with AHI < 5 (n=957) and 44.1 (SD 9.90) g/m(2.7) in participa

252 Compared with participants with AHI < 5, those with AHI > or = 30 had an adjusted odds r

253 w more widespread detection of patients with AHI and to affect HIV treatment outcomes and transmissio

254 redicting moderate/severe periodontitis with AHI score, age, and smoking status indicated a significa

255 r = -0.51) emerged, but was not present with AHI.

256 The subjects with AHI >/= 20 were accepted as OSAS.

257 d with participants with AHI < 5, those with AHI > or = 30 had an adjusted odds ratio of 1.78 (95% co

258 Among men 40 to 70 years old, those with AHI > or =30 were 68% more likely to develop coronary he

259 ely to develop heart failure than those with AHI <5.

260 velop coronary heart disease than those with AHI <5.

261 azard of developing diabetes than those with AHI less than 5.

262 8.2 per 1,000 person-years in those without (AHI <5 events/h).

263 16 OSA children (8 male, 8.1 +/- 2.2 years, AHI:11.1 +/- 5.9 events/hr), and 200 control subjects (8

264 In those aged<60 years, AHI was significantly associated with higher odds of sys