ライフサイエンスコーパス: AHO

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1 AHO is caused by heterozygous inactivating mutations in

2 from four crosses (B73 x OH43, Mo17 x A632, AHO x A632, Latente x A632) revealed that alleles of the

3 region (Gsalpha R258W) was identified in an AHO patient.

4 d their regulatory mechanisms in control and AHO-treated tobacco leaves.

5 ed equivalent results when comparing another AHO mutation in Galphas (D173N) with a counterpart cance

6 skeletal scintigraphy shows major-long-bone AHO if treatment response is slow.

7 und in 3 (6%) of 48 cases of major-long-bone AHO.

8 ent G proteins appeared associated with both AHO and cancer.

9 that both mRNA and miRNA regulation enhances AHO-induced SAR.

10 3-Acetonyl-3-hydroxyoxindole (AHO) induces systemic acquired resistance (SAR) in Nicot

11 affinity leading to the loss-of-function in AHO whereas Galphao-R243H has a mild decrease in nucleot

12 nism might explain the variable phenotype in AHO.

13 In no instance was the diagnosis of AHO indicated only by MRI.

14 ing 79 (92%) of 86 with a final diagnosis of AHO.

15 which was the other preponderant location of AHO.

16 l scintigraphy because of the possibility of AHO.

17 previously described clinical case report of AHO.

18 at the same mutation can cause either POH or AHO was observed within a single family, in which the ph

19 ion in Albright's hereditary osteodystrophy (AHO) and a recent report of two patients with AHO who ha

20 In Albright Hereditary Osteodystrophy (AHO) heterozygous loss of function mutations of GNAS can

21 esity in Albright hereditary osteodystrophy (AHO) patients, but only when the mutations occur on the

22 s of the Albright hereditary osteodystrophy (AHO) phenotype.

23 Albright's Hereditary Osteodystrophy (AHO) was the first inherited disease associated with los

24 Albright hereditary osteodystrophy (AHO), a disorder characterized by skeletal abnormalities

25 Albright hereditary osteodystrophy (AHO), an autosomal dominant disorder characterized by sh

26 suspected acute hematogenous osteomyelitis (AHO) with nondiagnostic radiographs.

27 to underlie increased aqueous humor outflow (AHO) resistance, which leads to elevated ocular pressure

28 I should be strongly considered after pelvic AHO is detected, and MRI might be substituted diagnostic

29 for suspected radiographically occult pelvic AHO.

30 of the association of abscesses with pelvic AHO, however, the use of MRI should be strongly consider

31 r puncture model, we determined that reduced AHO altered the fate of SC both during development and u

32 It has remained unclear why only some AHO patients present with multihormone resistance and wh

33 n 146 (69%) of all cases and 46 (53%) of the AHO cases.

34 In total, AHO treatment led to 17 up- and 6 down-regulated at leas

35 tal scintigraphy is the first test used when AHO is suspected but radiographs are negative remains hi

36 present with multihormone resistance and why AHO patients demonstrate resistance to some hormones [e.

37 expressed genes (DEGs) at least 2 folds with AHO treatment.

38 so resembles the ossification observed with AHO.

39 HO) and a recent report of two patients with AHO who had atypically extensive heterotopic ossificatio

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