ライフサイエンスコーパス: AI

1 AI algorithm performance was tested on a separate datase

2 AI lesions also attenuated both the development and the

3 AI rd (r = 0.68, P < 0.001) shows a stronger correlation

4 AI was also associated with stroke, pulmonary hypertensi

5 AI, corrected for heart rate, increased more rapidly wit

6 AI-2 mimic activity is induced when epithelia are direct

7 AI-ETD achieves these gains through improved quality of

8 AI-ETD provided the greatest sequence coverage for all f

9 on interval than AI r (r = 0.61, P < 0.001), AI d (r = -0.17, P = 0.06), the central augmentation ind

10 ood pressure, P < 0.001; weight, P = 0.001), AI rd has fewer confounding factors (age, P < 0.001; gen

11 aboratory and abattoir workers involved in 2 AI outbreaks in the Western Cape province: (1) controlli

12 ria, and its signal molecule, autoinducer-2 (AI-2), is synthesized as a product of 1-carbon metabolis

13 crobiota or the host, such as autoinducer-3 (AI-3), epinephrine (Epi), and norepinephrine (NE).

14 cal core-cuts and blood from 60 patients (40 AI treated, 20 controls).

15 Even in the absence of TPs, many (66%) AI salt-responsive type III taste cells still exhibited

16 ription of pi-delocalization and an accurate AI.

17 stains ERalpha signaling and drives acquired AI resistance.

18 ion analysis of cell line models of acquired AI resistance indicated that prostaglandin E2 receptor 4

19 a potential effective treatment of acquired AI-resistant breast cancer.

20 In addition, AI use without tamoxifen therapy was associated with red

21 All AI type III cells had osmotic responses to cellobiose, w

22 no-specific immune responses with alloislet (AI) responses in primates.

23 with iron supplement regimens to ameliorate AI.

24 Among AI-naive patients, serial FSH significantly increased ov

25 Building an AI algorithm can be surprisingly easy.

26 We present Libratus, an AI that, in a 120,000-hand competition, defeated four to

27 h factor receptor 2-targeted therapy plus an AI can be effective for those who are not chemotherapy c

28 We show that mammalian epithelia produce an AI-2 mimic activity in response to bacteria or tight-jun

29 early-stage breast cancer (BC) receiving an AI.

30 in patients with early-stage BC receiving an AI; secondary objectives included evaluation of adverse

31 nd characteristics of CTS in women taking an AI in a placebo-controlled trial.

32 familial AI, and comanifestation of DDD and AI has been reported for decades.

33 an indeed cause a comanifestation of DDD and AI that is likely triggered by predisposing factors for

34 amenorrheic women in their 40s is high, and AI therapy should be avoided in these patients.

35 between altered breast cancer metabolism and AI resistance using AI-resistant and sensitive breast ca

36 e full extent of connections between MGN and AI, R, RT, RTp, and STGr using retrograde and anterograd

37 rosubtypic reactivity to older pandemic- and AI viruses.

38 y the Power Law Exponent (PLE) in PostCG and AI.

39 Moreover, PWV and AI are associated with several SCD clinical complication

40 PWV and AI are deeply modified in SCD patients in comparison wit

41 adjustment for these correlates, cf-PWV and AI were associated with the glomerular filtration rate a

42 cf-PWV and AI were independently associated with age, sex, height,

43 ical and biological correlates of cf-PWV and AI were investigated by using a multivariable multilevel

44 ast cancer cells, patient tumor samples, and AI-sensitive human xenografts.

45 e association between adjuvant tamoxifen and AI therapy and CBC risk within a general community setti

46 e association between adjuvant tamoxifen and AI therapy and CBC risk within a general community setti

47 Adjuvant tamoxifen use and AI therapy were treated as time-dependent exposures, ass

48 Exposures: Adjuvant tamoxifen use and AI therapy were treated as time-dependent exposures, ass

49 ared with 1 hour in both NPI (wild-type) and AIs.

50 rising DMPC, cholesterol, and apolipoprotein AI (423:74:1 mol/mol) forming a discoidal particle 360 A

51 HDL formed spontaneously from apolipoprotein AI, cholesterol, and excess DMPC and isolated to near ho

52 rs of antiparallel helices of apolipoprotein AI circumscribing a phospholipid bilayer.

53 Interaction of apolipoprotein AI with dimyristoyl (DM)PC forms reconstituted (r)HDL, w

54 formed by the interaction of apolipoprotein AI with macrophage and hepatic ATP-binding cassette tran

55 ost efficient when lipid-poor apolipoprotein AI (apoAI) packages raft cholesterol into soluble partic

56 erol efflux to the lipid-poor apolipoprotein AI.

57 arcopallium (AA), intermediate arcopallium (AI), PoA, lateral, caudolateral and central nidopallium,

58 t most have focused on the caudal core areas AI and R.

59 on-lethal symptomatic (NSI) or asymptomatic (AI) cases of diarrhoeal illness in children enrolled in

60 overning these processes are an asynchronous AI-2 uptake, where positive intracellular feedback in Ls

61 One autoinducer, AI-2, is proposed to promote interspecies bacterial comm

62 This AI-2 mimic is detected by the bacterial AI-2 receptor, LuxP/LsrB, and can activate quorum-sensin

63 Efforts to develop better AIs with higher selectivity and lower side effects were

64 by the best correlation coefficient between AI rd and cfPWV.

65 initiated ET, which was evenly split between AI and tamoxifen, and no significant differences were ob

66 s failure to identify a precise link between AIs and AIMSS, underscoring the potential of channel ant

67 then investigated the influence of bilateral AI cortex lesions on the high impulsivity trait, as meas

68 Both AI and R received nearly 90% of their thalamic inputs fr

69 for early-stage breast cancer is limited by AI-associated musculoskeletal symptoms (AIMSS).

70 we introduce a new activation scheme called AI-ETD+ that combines AI-ETD in the high pressure cell o

71 vel heterozygous ENAMp.L31R mutation causing AI in humans.

72 nalyses (reported as hazard ratio [95% CI]), AI-only users had a similar risk of cardiac ischemia (my

73 duced by recombinant strains carrying cloned AI synthase genes, increased survival of V. cholerae and

74 proposed as an important strategy to combat AI.

75 tivation scheme called AI-ETD+ that combines AI-ETD in the high pressure cell of the QLT with a short

76 Conclusion AI using deep learning demonstrates promise for detectin

77 In conclusion, AI rd derives performance improvement in assessing arter

78 Lake et al. suggest that current AI systems lack the inductive biases that enable human l

79 uggest that theoretical accounts of the dACC-AI network as a neural alarm system restricted within th

80 directly tested the hypothesis that the dACC-AI would respond to cues of both inclusion and exclusion

81 However, recent work suggests that this dACC-AI matrix may index any socially pertinent information.

82 ctivated ion electron transfer dissociation (AI-ETD) on a quadrupole-Orbitrap-linear ion trap hybrid

83 Here, we identify the aetiology driving AI in mice carrying a p.S55I mutation in enamelin; one o

84 e the antiestrogenic SERM activity of a dual AI/SERM could act synergistically with AI activity to en

85 issue the estrogenic SERM activity of a dual AI/SERM could attenuate the undesired effects stemming f

86 epletion caused by the AI activity of a dual AI/SERM, while in breast cancer tissue the antiestrogeni

87 further development in order to obtain dual AI/SERM agents for breast cancer treatment.

88 on (ETD) reactions, i.e., activated ion ETD (AI-ETD), significantly increases dissociation efficiency

89 an expected in ER-positive tumors exhibiting AI-resistant proliferation.

90 PSENEN mutations were identified in familial AI, and comanifestation of DDD and AI has been reported

91 Furthermore, the airway field AI included oncogenic gains and tumor suppressor losses

92 Identifying a common aetiology for AI resulting from mutations in two different genes opens

93 likely triggered by predisposing factors for AI.

94 obesity, which are known trigger factors for AI.

95 was associated with reduced CBC risk (RR for AI users compared with nonusers, 0.48; 95% CI, 0.22-0.97

96 rds and quarantined exotic birds in Gauteng, AI outbreaks in poultry in KwaZulu-Natal, and ostriches

97 d served as the catalyst for next generation AI discovery research.

98 typic antibodies to H9 and H7, but not to H5 AI virus.

99 n why a minority of type III cells (34%) had AI salt responses but lacked anion sensitivity.

100 MTN as an excellent candidate gene for human AI.

101 he aetiological factor in this case of human AI as it shared the characteristic phenotype described a

102 hat AMTN mutations cause non-syndromic human AI and explores the human phenotype, comparing it with t

103 e phenotypic analysis, we suggest that human AI resulting from the ENAMp.L31R mutation is another pro

104 utations have yet been associated with human AI.

105 family segregating dominant hypomineralised AI.

106 Using calcium imaging, we identified AI salt-responsive type III taste cells and demonstrated

107 ions relevant to cognitive control (esp. IFG/AI and the dorsal anterior cingulate cortex) were strong

108 omal alterations inducing allelic imbalance (AI) in the airway field of the most common type of lung

109 Amelogenesis Imperfecta (AI) can be caused by the deficiencies of enamel matrix p

110 Amelogenesis imperfecta (AI) is a heterogeneous group of genetic conditions that

111 Amelogenesis imperfecta (AI) is a heterogeneous group of genetic disorders affect

112 Amelogenesis imperfecta (AI) is group of inherited disorders resulting in enamel

113 are associated with Amelogenesis Imperfecta (AI) with gingival hyperplasia and nephrocalcinosis, whil

114 A5 (NCKX5) genes to amylogenesis imperfecta (AI) and non-syndromic oculocutaneous albinism (OCA6), re

115 'Amelogenesis imperfecta' (AI) describes a group of inherited diseases of dental en

116 argeted diagnoses and treatments can improve AI-resistant breast cancer treatment.

117 een spontaneous and task-induced activity in AI was mediated by narcissistic features.

118 nce fields and emphasize current advances in AI that have been inspired by the study of neural comput

119 diac ischemia or stroke) was not elevated in AI-only users compared with tamoxifen users.

120 /EnR calcium ATPase 2b (SERCA2b) function in AI-resistant cells.

121 gher predictive accuracies for generosity in AI, whereas those who strongly relied on cognitive persp

122 at other CVD events combined were greater in AI users requires further study.

123 The extensive generation of fragment ions in AI-ETD+ substantially increases peptide sequence coverag

124 these responses were significantly larger in AI type III cells that did not exhibit the anion effect.

125 es can significantly improve localization in AI-ETD spectra.

126 mp) cells also emerged in vitro, but only in AI-resistant models.

127 omotes local autocrine estrogen signaling in AI-resistant metastatic patients.

128 te that PTGER4 is a potential drug target in AI-resistant cancers.

129 e, synergistically reduced cell viability in AI-sensitive models.

130 This first application of WES in AI bulls with divergent fertility phenotypes has identif

131 cholerae mutant strains carrying inactivated AI synthase genes were significantly more susceptible to

132 ized, double-blind, phase III trial included AI-treated postmenopausal women with early-stage breast

133 This study proposes a novel index AI rd , a combination of AI r and diastolic augmentation

134 ce constants to derive an aromaticity index (AI) that quantitatively determines the degree of pi-delo

135 , P = 0.06), the central augmentation index (AI c ) (r = 0.61, P < 0.001) or AI c normalized for hear

136 on of AI r and diastolic augmentation index (AI d ) with a weight alpha, to achieve better performanc

137 Radial augmentation index (AI r ) can be more conveniently measured compared with c

138 ve velocity (cf-PWV) and augmentation index (AI) at a steady state.

139 locity (PWV) and carotid augmentation index (AI)].

140 rce-time integral [FTI], and ablation index [AI]) and contiguity (automated interlesion distance [ILD

141 bular molar at postnatal day 8 (PN8) induced AI-like pathologies when the enamel development reached

142 e (FeSO4) ameliorated the turpentine-induced AI in mice (indicated by increased haemoglobin level, se

143 ically manifested as anemia of inflammation (AI).

144 HP) and low-pathogenic (LP) avian influenza (AI) H5N2 and H7N1 were investigated during outbreaks in

145 size: 500m x 500m) maps for Avian Influenza (AI) suitability in each of the four North American migra

146 Seropositivity to avian influenza (AI) via low-level antibody titers has been reported in t

147 in combination with the aromatase inhibitor (AI) exemestane.

148 Purpose Adherence to aromatase inhibitor (AI) therapy for early-stage breast cancer is limited by

149 s are selected by prior aromatase inhibitor (AI) therapy in advanced breast cancer.

150 ated with resistance to aromatase inhibitor (AI) therapy in patients with ER+ metastatic breast cance

151 he impact of 2 years of aromatase inhibitor (AI) therapy on the incidence of ovarian function recover

152 4 weeks of neoadjuvant aromatase inhibitor (AI) therapy.

153 the long-term effect of aromatase inhibitor (AI) use on CVD risk in breast cancer survivors.

154 has been to create dual aromatase inhibitor (AI)/selective estrogen receptor modulators (SERMs).

155 patient groups (no ET, aromatase inhibitor [AI], or tamoxifen) were compared by using linear mixed m

156 Aromatase inhibitors (AI) are associated with significant urogenital atrophy,

157 Aromatase inhibitors (AI) have become the first-line endocrine treatment of ch

158 Aromatase inhibitors (AI) induce painful musculoskeletal symptoms (AIMSS), whi

159 postmenopausal women, aromatase inhibitors (AIs) are the preferred first-line endocrine therapy, wit

160 (ER+) breast cancer to aromatase inhibitors (AIs) but have been limited to small biopsies.

161 d clinical trials with aromatase inhibitors (AIs) compared with tamoxifen.

162 alled third generation aromatase inhibitors (AIs) letrozole, anastrozole, and the steroidal exemestan

163 Aromatase inhibitors (AIs) prevent estrogen production and inhibit ER signalin

164 tamoxifen citrate and aromatase inhibitors (AIs) reduces CBC risk.

165 dherence to tamoxifen, aromatase inhibitors (AIs), and overall AET (tamoxifen or AIs) was assessed us

166 s initially respond to aromatase inhibitors (AIs), but eventually acquire resistance.

167 ulators (SERMs) and/or aromatase inhibitors (AIs).

168 roduced by SCNT and artificial insemination (AI) at day (d) 18 (preimplantation) and d 34 (postimplan

169 7,000 bulls used in artificial insemination (AI) were used to identify 160 reliable and divergently f

170 taste in mammals, the amiloride-insensitive (AI) pathway is the least understood.

171 Neural responses in the anterior insula (AI) (but not temporoparietal junction [TPJ]) encoded tri

172 The anterior insula (AI) and mid-anterior cingulate cortex (mACC) have repeat

173 entral striatum (VS) or the anterior insula (AI) during reward anticipation regardless of motivated r

174 cingulate cortex (dACC) and anterior insula (AI), have been shown to be equally sensitive to the dete

175 issistic features, in right anterior insula (AI).

176 ates of impulsivity in the anterior insular (AI) cortex by measuring both the thickness of, and cellu

177 in D intakes below the EFSA Adequate Intake (AI) (<15mug/d vitamin D) in adults across Europe.

178 or exceeded the recommended Adequate Intake (AI).

179 that informatic and artificial intelligence (AI) analytics simply cannot handle.

180 of neuroscience and artificial intelligence (AI) have a long and intertwined history.

181 Despite artificial intelligence (AI) successes in perfect-information games, the private

182 Radiomic artificial intelligence (AI) technology, either based on engineered hard-coded al

183 e performance of an artificial intelligence (AI) tool using a deep learning algorithm for detecting h

184 Artificial intelligence (AI), machine learning, and deep learning are terms now s

185 m dorsale (AD), the arcopallium intermedium (AI), the arcopallium mediale (AM), the arcopallium poste

186 rriers presented with comorbid acne inversa (AI), an inflammatory hair follicle disorder, and had a h

187 We show that the dACC and left AI are commonly activated by feedback cues of inclusion

188 lity-independent activation maps in the left AI and mACC, pointing to common coding of affective unpl

189 or temporal gyrus (STG) and between the left AI/FO and dorsal anterior cingulate cortex correlated po

190 Connectivity between the left AI/FO and left posterior superior temporal gyrus (STG) a

191 Radiology has the chance to leverage AI to become a center of intelligently aggregated, quant

192 s uncover a potential role for the mammalian AI-2 mimic in fostering crosskingdom signaling and host-

193 In patients who completed neoadjuvant AI, stratified Cox modeling was used to assess whether t

194 eatment was randomly assigned to neoadjuvant AI therapy with anastrozole, exemestane, or letrozole.

195 ients with prior sensitivity to nonsteroidal AI and in baseline plasma from the PALOMA3 (Palbociclib

196 ease progresses while receiving nonsteroidal AIs.

197 for beneficiaries, whereas the TPJ (but not AI) predicted the degree of perspective taking.

198 n was increased when wild-type (WT), but not AI-forms of FAM20A was co-transfected.

199 We found that 21.5% of AI-treated, relapsed patients had acquired CYP19A1 (enco

200 poses a novel index AI rd , a combination of AI r and diastolic augmentation index (AI d ) with a wei

201 inhibitory synapses on proximal dendrites of AI gammaACs.

202 Our findings of AI infection in pikas are consistent with previous obser

203 ur data also indicated a spatial gradient of AI in samples at closer proximity to the NSCLC.

204 Here we describe the first implementation of AI-ETD on a quadrupole-Orbitrap-quadrupole linear ion tr

205 Lesions of AI reduced impulsive behavior in HI rats, which were als

206 Chromosome 9 displayed the highest levels of AI and comprised recurrent independent events.

207 insights into the transduction mechanisms of AI salt taste but also have important implications for g

208 term estrogen deprivation (LTED), a model of AI resistance, was associated with increased glycolysis

209 the AI with the harmonic oscillator model of AI, the latter is found to exaggerate the antiaromaticit

210 gic factor contributing to the occurrence of AI.

211 epigenetic regulation in the pathogenesis of AI is yet to be clarified due to a lack of knowledge abo

212 However, the performance of AI r in assessing arterial stiffness is limited.

213 Here we present the performance of AI-ETD for identifying and localizing sites of phosphory

214 We then characterize the performance of AI-ETD using standard peptides in addition to a complex

215 ions due to the infrared photoactivation of AI-ETD and show that modifying phosphoRS (a phosphosite

216 lls, we identify two separate populations of AI salt-responsive type III taste cells distinguished by

217 indings demonstrate highly specific roles of AI for affective empathy and TPJ for cognitive perspecti

218 anted into the risk factors and treatment of AI-induced CTS.

219 Finally, we demonstrate the utility of AI-ETD in localizing phosphosites in alpha-casein, an ap

220 mor Ki67 determination after 2 to 4 weeks of AI.

221 ncers most likely to adapt to and relapse on AI therapy.

222 rine therapy status (tamoxifen citrate only, AI only, both, or neither).

223 the left anterior insula/frontal operculum (AI/FO) and the visual word form area (VWFA).

224 ation index (AI c ) (r = 0.61, P < 0.001) or AI c normalized for heart rate of 75 bpm (r = 0.65, P <

225 emented with exogenous autoinducers CAI-1 or AI-2 produced by recombinant strains carrying cloned AI

226 ctive protein (P = 0.10), PWV (P = 0.30), or AI (P = 0.84).

227 bute to generous behavior (but not in TPJ or AI).

228 ibitors (AIs), and overall AET (tamoxifen or AIs) was assessed using the medication possession ratio

229 ht alpha, to achieve better performance over AI r in assessing arterial stiffness.

230 ysis in plasma after progression after prior AI therapy may help direct choice of further endocrine-b

231 associated with acquired resistance to prior AI.

232 naling indicates that PTGER4 likely promotes AI resistance via ligand-independent activation of the E

233 In PM women with early-stage BC receiving AIs, treatment with a vaginal ring or IVT over 12 weeks

234 of C-type lectin receptor scavenger receptor-AI (SR-AI) is crucial for promoting M2-like Mvarphi acti

235 Given SGK3 inhibition reduces AI-resistant cell survival by eliciting excessive EnR st

236 it was addressed, particularly in the right AI.

237 gh the primary, rostral, and rostrotemporal (AI, R, and RT) core areas on the supratemporal plane, co

238 th the biosynthesis of quorum sensing signal AI-2.

239 4 out of 10 that developed or worsened since AI therapy initiation.

240 SR-AI expression on liver Mvarphi promotes recovery from in

241 pe lectin receptor scavenger receptor-AI (SR-AI) is crucial for promoting M2-like Mvarphi activation

242 Furthermore, in vitro studies using an SR-AI-deficient Mvarphi cell line revealed impeded M2 polar

243 crophages regulate FX plasma levels in an SR-AI-dependent manner.

244 ntial to prevent internalization of FX by SR-AI, and the presence of FX is needed to interfere with i

245 cted mice deficient for the gene encoding SR-AI (msr1).

246 Binding studies showed that FX, SR-AI, and PTX2 independently bind to each other (KD,app: 0

247 Here, we demonstrate that the FX/SR-AI-complex comprises a third protein, pentraxin-2 (PTX2)

248 fied scavenger receptor class A member I (SR-AI) as a receptor for coagulation factor X (FX), mediati

249 d the scavenger receptor type A member I (SR-AI) to be a macrophage-specific receptor for FX.

250 e damage and fibrosis were exacerbated in SR-AI(-/-) mice following hepatic infection and adoptive tr

251 FX levels were correspondingly reduced in SR-AI-deficient mice.

252 3 mug/mL) to the extracellular domain of SR-AI.

253 Liver Mvarphi uniquely up-regulated SR-AI during hepatotropic viral infection and displayed inc

254 both proteins cooperate to prevent their SR-AI-mediated uptake by macrophages.

255 This result was confirmed using SR-AI-deficient mice, which exhibit reduced circulating lev

256 eptor (HR)-positive stage I to III BC taking AIs with self-reported vaginal dryness, dyspareunia, or

257 fPWV and a narrower prediction interval than AI r (r = 0.61, P < 0.001), AI d (r = -0.17, P = 0.06),

258 We previously demonstrated that AI in mice carrying the Amelxp.Y64H mutation is a protei

259 The current data indicate that AI in Enamp.S55I mice is also a proteinopathy, and based

260 ptides using LC-MS/MS, showing not only that AI-ETD can nearly double the identifications achieved wi

261 Results reveal that AI suitability is heterogeneously distributed throughout

262 First, we show that AI-ETD can be implemented in a straightforward manner by

263 Using 90 min analyses we show that AI-ETD can identify 24,503 localized phosphopeptide spec

264 These data suggest that AI treatment itself selects for acquired CYP19A1(amp) an

265 The AI confirms Clar's rule of disjoint benzene units in man

266 showed increased severity over time, and the AI group reported more severe musculoskeletal (P = .02),

267 However, as the AI concentrations required to engage TRPA1 in mice are h

268 Therefore, we attributed the AI primarily to the reduction of MMP20 and KLK4.

269 e survey historical interactions between the AI and neuroscience fields and emphasize current advance

270 ls initially and improved over time, but the AI group had a significantly lower score at 12 months (P

271 from global estrogen depletion caused by the AI activity of a dual AI/SERM, while in breast cancer ti

272 When comparing the AI with the harmonic oscillator model of AI, the latter

273 Few toddlers (<1%) met or exceeded the AI for fiber and potassium.

274 This study thus identifies the AI as a novel neural substrate of maladaptive impulse co

275 ent an account of recent developments in the AI field from the perspective of the enzyme's structure-

276 of, and cellular plasticity markers in, the AI with magnetic resonance imaging and in situ hybridiza

277 both detect and stimulate production of the AI-2 mimic.

278 We demonstrate that the AI cortex causally contributes to individual vulnerabili

279 e hexokinase-2 (HK2) in combination with the AI, letrozole, synergistically reduced cell viability in

280 Kekule benzene as a suitable reference, the AIs of 30 mono- and polycyclic conjugated hydrocarbons a

281 To show how these AI products reduce costs and improve outcomes will requi

282 This AI-2 mimic is detected by the bacterial AI-2 receptor, L

283 (amp) cells showing decreased sensitivity to AI treatment.

284 associated with increased susceptibility to AI.

285 , and blacks had higher odds of adherence to AIs at initiation (OR, 1.27; 95% CI, 1.04 to 1.54) compa

286 cate that multiple mechanisms could underlie AI salt responses in type III taste cells, one of which

287 he most commonly mutated proteins underlying AI in humans.

288 djusted, 1.29 [1.11-1.50]) in women who used AIs only or sequentially after tamoxifen (1.26 [1.09-1.4

289 st cancer metabolism and AI resistance using AI-resistant and sensitive breast cancer cells, patient

290 l, this work highlights the analytical value AI-ETD can bring to both bottom-up and top-down phosphop

291 Our results demonstrate that genome-wide AI is common in the airway field of cancerization, provi

292 Compared with AI r (age, P < 0.001; gender, P < 0.001; heart rate, P <

293 r these findings reflect true infection with AI or cross-reactivity.

294 introduced mutations found in patients with AI and OCA6 into human SLC24A4 (NCKX4) cDNA leading to s

295 dual AI/SERM could act synergistically with AI activity to enhance the antiproliferative effect.

296 alactosyltransferase knockout) compared with AIs.

297 ling])-positive cells) of NPIs compared with AIs.

298 Advanced tumors treated with AIs almost always develop resistance to these drugs via

299 The risk of OFR during treatment with AIs in amenorrheic women in their 40s is high, and AI th

300 tional specialization and integration within AI and mACC, and support a comprehensive role of this ne