ライフサイエンスコーパス: AIDS dementia

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1 ctive neuroprotection against development of AIDS dementia.

2 ured macrophages obtained from patients with AIDS dementia.

3 vant to drug strategies for the treatment of AIDS dementia.

4 by contributing to neuronal deregulation and AIDS dementia.

5 function, contributing to the development of AIDS dementia.

6 understanding mechanisms of pathogenesis in AIDS dementia.

7 e during progression to immunodeficiency and AIDS dementia.

8 unique subset of monocytes in patients with AIDS dementia.

9 ntial therapeutic target for HIV-1 infection/AIDS dementia.

10 d play a pivotal role in the pathogenesis of AIDS dementia.

11 tissue from HIV-1-infected patients who have AIDS dementia.

12 erbating neurological diseases such as HIV-1 AIDS dementia and cerebral ischemia.

13 AIDS dementia and encephalitis are complications of AIDS

14 for the investigation of the pathogenesis of AIDS dementia and for the investigation of drugs with po

15 has been implicated in the neuropathology of AIDS dementia and other neurodegenerative diseases.

16 system (CNS) is a characteristic feature of AIDS dementia, and it represents a common mechanism of n

17 of relative basal ganglia hypermetabolism in AIDS dementia complex (ADC) and to develop clinically us

18 n of the central nervous system (CNS) causes AIDS dementia complex (ADC) in certain infected individu

19 nce indicates that the mechanism for causing AIDS dementia complex (ADC) involves the release of dama

20 AIDS dementia complex (ADC) is the most common presentin

21 with HIV and clinical manifestations of the AIDS dementia complex (ADC), a group of patients (n = 8)

22 ased CSF QUIN levels and the presence of the AIDS dementia complex (ADC), a neurodegenerative conditi

23 d subsequent development of HIV-1-associated AIDS dementia complex are not fully understood, a correl

24 the cognitive and motor complications termed AIDS dementia complex or HIV-associated dementia.

25 ated cognitive-motor disorder, including the AIDS dementia complex, is characterized by brain functio

26 he neurochemical pathologies contributing to AIDS dementia complex, neurotransmitter levels were meas

27 y, and cryptococcal meningitis) but also the AIDS dementia complex, with its characteristic cognitive

28 or disease progression in the early stage of AIDS dementia complex.

29 tion and contributing to the pathogenesis of AIDS dementia complex.

30 e neurological complications associated with AIDS dementia complex.

31 e of the LP-BM5-infected mouse as a model of AIDS dementia complex.

32 vement of glial cells in the pathogenesis of AIDS dementia complex.

33 nt of AIDS-associated encephalopathy and the AIDS dementia complex.

34 p to the acquired immunodeficiency syndrome (AIDS) dementia complex (ADC) and brain pathology remains

35 nesis of acquired immunodeficiency syndrome (AIDS) dementia complex.

36 Alzheimer's disease, multiple sclerosis, the AIDS-dementia complex, and cerebral ischemia.

37 R studies on brain tissue from patients with AIDS dementia demonstrated elevated MIP-1 alpha and MIP-

38 in the United States and the progression to AIDS dementia is accelerated in opiate drug abusers.

39 AIDS dementia is characterized by neuronal loss in assoc

40 he monocyte subset we found in patients with AIDS dementia might enter the brain and expose neural ce

41 gative controls and in AIDS non-dementia and AIDS dementia patients were 6.5% (4), 16% (13), and 37%

42 ed with acquired immune deficiency syndrome (AIDS) dementia, we tested for correlations between MCP-1

43 nd in AIDS pathogenesis in syndromes such as AIDS dementia where HIV-1 gp120 contributes to inappropr

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