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1 e of the LP-BM5-infected mouse as a model of AIDS dementia complex.
2 vement of glial cells in the pathogenesis of AIDS dementia complex.
3 nt of AIDS-associated encephalopathy and the AIDS dementia complex.
4 or disease progression in the early stage of AIDS dementia complex.
5 tion and contributing to the pathogenesis of AIDS dementia complex.
6 e neurological complications associated with AIDS dementia complex.
7 nesis of acquired immunodeficiency syndrome (AIDS) dementia complex.
8 of relative basal ganglia hypermetabolism in AIDS dementia complex (ADC) and to develop clinically us
9 n of the central nervous system (CNS) causes AIDS dementia complex (ADC) in certain infected individu
10 nce indicates that the mechanism for causing AIDS dementia complex (ADC) involves the release of dama
12 with HIV and clinical manifestations of the AIDS dementia complex (ADC), a group of patients (n = 8)
13 ased CSF QUIN levels and the presence of the AIDS dementia complex (ADC), a neurodegenerative conditi
14 p to the acquired immunodeficiency syndrome (AIDS) dementia complex (ADC) and brain pathology remains
16 d subsequent development of HIV-1-associated AIDS dementia complex are not fully understood, a correl
17 ated cognitive-motor disorder, including the AIDS dementia complex, is characterized by brain functio
18 he neurochemical pathologies contributing to AIDS dementia complex, neurotransmitter levels were meas
20 y, and cryptococcal meningitis) but also the AIDS dementia complex, with its characteristic cognitive
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