ライフサイエンスコーパス: AMF

1 AMF activity could not be mimicked by an extensive set o

2 AMF could be detected in serum or urine of cancer patien

3 AMF enhanced the competition but equalized growth of leg

4 AMF hyphae were either allowed (AMF) or prevented (nonAM

5 AMF promoted marked rearrangement of focal adhesion plaq

6 AMF stimulation of human melanoma cells resulted in stre

7 AMF was delivered 72 h after bioprobe injection at ampli

8 AMF was fractionated at 21 degrees C to obtain stearin a

9 * AMF were characterized in colonized roots of thermal soi

10 AMF/PGI acts extracellularly as a potent mitogen/cytokin

11 AMF/PGI is also a multifunctional protein that displays

12 AMFs have been measured in unventilated chambers or stea

13 In conclusion, (1) AMF mediate plant interaction through directly influenci

14 In total, 147 AMF virtual taxa (VT) were detected, including 22 VT new

15 Because little is known about AMF-dependent signaling, we sought to study whether AMF

16 Because little is known about AMF/PGI-dependent signaling in general and during tumori

17 comparable with a known G protein activator, AMF.

18 cell death by necrosis at 24 and 48 h after AMF.

19 AMF hyphae were either allowed (AMF) or prevented (nonAMF) access to a compartment conta

20 red to the lung of neonatal mice acquired an AMF phenotype via defined developmental stages over the

21 e optimal characteristics of the MNPs and an AMF for effective magneto-mechanical actuation of single

22 tial link between carbon (C) release from an AMF and phosphorus (P) availability via a phosphate-solu

23 ions, yet they are typically performed in an AMF device with non-adiabatic conditions.

24 Our results suggest that an AMF and a free-living PSB interacted to the benefit of e

25 We tested whether an AMF modifies the soil microbial community and nitrogen c

26 , and combination studies of antibiotics and AMF demonstrate a 5-log increase in the sensitivity of P

27 rthermore, we found that HER2 expression and AMF secretion were inversely related in breast carcinoma

28 f legumes was higher than that of grass, and AMF benefited the plant biomass of legumes but had no ef

29 inherent relationship between host plant and AMF community structures, although pH-based models were

30 ogs could moreover be identified for another AMF, Gigaspora margarita, and surprisingly, also the non

31 partially suppressed by inclusion of an anti-AMF antibody to breast cancer cells, suggesting that a H

32 As AMF acquire N, it was hypothesized that AMF hyphae may r

33 The new chemicals, known as AMFs, have long-lasting effects in promoting stomatal cl

34 atment of the cells with C3 exoenzyme before AMF stimulation inhibited both the formation of stress-f

35 ose per gram; this concentration 24 h before AMF treatment was used to calculate THD.

36 g had shown a partial colocalization between AMF and its receptor (Mr 78,000 glycoprotein), especiall

37 Reported as a cancer biomarker, AMF secretion into body fluids might be closely related

38 transcription, while cotransfection of both AMF-1 and p300 showed an additive effect.

39 al kinase 2 were simultaneously activated by AMF, supporting the notion that they are involved in the

40 ecular mechanisms of atrazine degradation by AMF.

41 and in vitro nanoparticle heat induction by AMF, correlated with tumor growth delay.

42 sm would occur also between plants linked by AMF.

43 over, acute disruption of the MAPK module by AMF, a selective inhibitor of MEK1, suppressed both basa

44 by cell contact whereas in transformed cells AMF-R is constitutively expressed irrespective of cell d

45 we investigated these issues with a chimeric AMF that is secreted at high levels through a canonical

46 eate a root-free soil environment to control AMF access to (13) C- and (15) N-labelled root litter.

47 munohistochemical visualization has depicted AMF/NLK/PHI/MF to be localized into tubular-like vesicle

48 ated motility and responded to tumor-derived AMF locomotory stimulus as compared with the nonmetastat

49 plant community situations harbored distinct AMF communities with few fungal taxa occurring in both c

50 populations, each associated with divergent AMF hosts.

51 Forested areas supported more diverse AMF communities than savannas and grassland.

52 We found that each AMF species harbored a genetically distinct group of MRE

53 Early AMF commitment from fetal monocytes was absent in GM-CSF

54 uced following nitrate application to either AMF treatment.

55 increase soil available P, the PSB enhanced AMF hyphal growth, and PSB activity was also stimulated

56 Conversely, exogenous AMF overcame the inhibitory effect of EGF receptor inhib

57 The results suggest that extracellular AMF activity may be a result of the product of intracell

58 ity, designated autocrine macrophage factor (AMF), elicited a change in C/EBPbeta localization from a

59 ere, we show that autocrine motility factor (AMF) binds to HER2 and induces cleavage to the ectodomai

60 ties that include autocrine motility factor (AMF) eliciting mitogenic, motogenic, differentiation fun

61 Autocrine motility factor (AMF) enhances invasion by breast cancer cells, but how i

62 Autocrine motility factor (AMF) is a tumor-secreted cytokine that acts as a motogen

63 Autocrine motility factor (AMF) is a tumor-secreted cytokine that stimulates tumor

64 ties that include autocrine motility factor (AMF) regulating tumor cell motility.

65 Autocrine motility factor (AMF), a tumor-associated C-X-X-C cytokine, the ligand fo

66 ve suggested that autocrine motility factor (AMF), a tumor-secreted Mr 55,000 cytokine that regulates

67 ties that include autocrine motility factor (AMF)-eliciting mitogenic, motogenic, and differentiation

68 d function of the autocrine motility factor (AMF).

69 Autocrine motility factor (AMF)/phosphoglucose isomerase (PGI) is a ubiquitous cyto

70 Autocrine motility factor (AMF)/phosphoglucose isomerase (PGI; EC 5.3.1.9) is a hou

71 ts as a cytokine [autocrine motility factor (AMF)] eliciting mitogenic, motogenic, and differentiatio

72 the solubility of CO2 in anhydrous milk fat (AMF) as functions of partial pressure, temperature, chem

73 r unsaturation degree on anhydrous milk fat (AMF) crystallization was evaluated.

74 ce area under an alternating magnetic field (AMF) are calculated and compared through the concentrati

75 ternally applied alternating magnetic field (AMF).

76 ency (>100 kHz) alternating magnetic fields (AMF).

77 d low frequency alternating magnetic fields (AMFs) and its possible use for remote control of nanomed

78 when exposed to alternating magnetic fields (AMFs), making them suitable for cancer hyperthermia.

79 ntial for symbiosis, D14L is dispensable for AMF-induced root architectural modulation, which convers

80 ves from seven research groups using NGS for AMF community ecology gathered to discuss common challen

81 during pre-symbiosis with CERK1 required for AMF-induced root architectural changes.

82 wth factor and suggests a potential role for AMF in HRG regulation of breast cancer cell motility and

83 an unexpected plant recognition strategy for AMF and a previously unknown signaling link between symb

84 This is particularly true for AMF community studies, because much remains to be known

85 The aerosol mass fraction (AMF), a.k.a. SOA yield, quantifies the SOA forming poten

86 omes harbor genes horizontally acquired from AMF.

87 on symbiotic arbuscular mycorrhizal fungal (AMF) communities is difficult to study in situ as both s

88 rmation about arbuscular mycorrhizal fungal (AMF) communities.

89 ctions between arbuscular mycorrhizal fungi (AMF) and land plants to at least 400 Ma, but the functio

90 d diversity of arbuscular mycorrhizal fungi (AMF) and the rules that govern AMF assemblages has been

91 Arbuscular mycorrhizal fungi (AMF) are significantly depleted in H. forsteriana on vol

92 stimulation of arbuscular mycorrhizal fungi (AMF) by elevated atmospheric carbon dioxide (CO(2)) has

93 feedback with arbuscular mycorrhizal fungi (AMF) collected from soils of conspecific plants, and fee

94 Arbuscular mycorrhizal fungi (AMF) form a mutualistic symbiosis with two-thirds of lan

95 million years, arbuscular mycorrhizal fungi (AMF) have formed intimate, mutualistic symbioses with th

96 g the roles of arbuscular mycorrhizal fungi (AMF) in plant interaction is essential for optimizing pl

97 y structure of arbuscular mycorrhizal fungi (AMF) is important for potentially optimizing their role

98 Arbuscular mycorrhizal fungi (AMF) perform an important ecosystem service by improving

99 These arbuscular mycorrhizal fungi (AMF) perform vital functions in the phosphorus cycle tha

100 Arbuscular mycorrhizal fungi (AMF) protect host plants against diverse biotic and abio

101 Arbuscular mycorrhizal fungi (AMF) transfer plant photosynthate underground which can

102 Perception of arbuscular mycorrhizal fungi (AMF) triggers distinct plant signalling responses for pa

103 symbiotic with arbuscular mycorrhizal fungi (AMF), which take up mineral nutrients from the soil and

104 sociation with arbuscular mycorrhizal fungi (AMF).

105 sociation with arbuscular mycorrhizal fungi (AMF).

106 Arbuscular mycorrhizal fungi (AMF, Glomeromycota) colonize roots of the majority of te

107 ssociated with arbuscular mycorrhizal fungi (AMF; phylum Glomeromycota), we proposed that epiparasiti

108 Furthermore, AMF-associated genes exhibit evidence of divergent selec

109 rhizal fungi (AMF) and the rules that govern AMF assemblages has been hampered by a lack of data from

110 gh binding to its cell surface receptor gp78/AMF receptor (AMFR).

111 homology with AMF and has been shown to have AMF activity.

112 Toxicity was observed at the highest AMF amplitude-duty combination of 1,300 Oe and 60% over

113 Miombo woodlands had the highest AMF richness, number of novel VT, and number of exclusiv

114 ure from soil, yet little is known about how AMF influence soil microbial communities during nutrient

115 s a nutrient, very little is known about how AMF take up nitrogen and transfer it to their host plant

116 Taken together, our findings show how AMF modulates EGF-induced invasion while affecting acqui

117 The dissolved CO2 concentration in AMF increased with an increase in CO2 partial pressure (

118 ice, but pro-MMP9 induction was abrogated in AMFs from OPN(-/-) mice.

119 TNF-alpha upregulated pro-MMP9 in AMFs isolated from wild-type (OPN(+/+)) mice, but pro-MM

120 A) simulations demonstrate that intermittent AMF exposures can achieve uniform surface heating of a p

121 Intriguingly, AMF can link neighboring plants, forming common mycorrhi

122 he precise ontogeny of alveolar macrophages (AMFs) is unknown.

123 Mature AMFs were undetectable before birth and only fully colon

124 vation in aortic adventitial myofibroblasts (AMFs) and A7r5 vascular smooth muscle cells (VSMCs).

125 ra margarita, and surprisingly, also the non-AMF Mortierella verticillata.

126 cetyl-lysine failed to detect acetylation of AMF-1 or E2 in complex with p300.

127 RNA interference-mediated attenuation of AMF expression inhibited EGF-induced invasion by suppres

128 significantly amplified the net benefits of AMF and likely selection pressures for establishment of

129 Cotransfection of AMF-1 or p300 stimulated levels of E2-dependent transcri

130 present study is the first demonstration of AMF regulation by a growth factor and suggests a potenti

131 Our data reveal a sharp differentiation of AMF communities between forested areas and periodically

132 to investigate the vertical distribution of AMF in a calcareous field and their temporal structure i

133 e expression, secretion, and distribution of AMF/NLK/PHI/MF in neoplastic and their normal counterpar

134 This study investigated the effects of AMF and the presence of legume or grass herbs on phytore

135 t the background of globally low endemism of AMF, local communities are shaped by regional processes

136 ents demonstrating that CO(2) enhancement of AMF results in considerable soil carbon losses.

137 report that HRG increases the expression of AMF mRNA by 3-8-fold in an actinomycin D-sensitive manne

138 Increased expression of AMF/PGI and its receptor/CXXC-R has been found in a wide

139 The unusual biological features of AMF have long fascinated evolutionary biologists.

140 conducted to determine the direct impact of AMF hyphal exudates on growth of the PSB.

141 mediated, at least in part, via induction of AMF.

142 investigations indicated that interaction of AMF with HER2 triggers HER2 phosphorylation and metallop

143 Here we explored the possible involvement of AMF in the motility-promoting action of HRG in the MCF-7

144 All major lineages of AMF harbor endobacteria classified as Mollicutes, and kn

145 try (NanoSIMS) we visualized the location of AMF-transported (13) C and (15) N in plant roots.

146 oil, with each plant supporting 100-400 m of AMF mycelia.

147 biofilm matrix components within 1 minute of AMF exposure, and combination studies of antibiotics and

148 ction in bacterial counts after 5 minutes of AMF exposure.

149 ion and quantification of a protein model of AMF, namely phosphoglucose isomerase from rabbit muscle

150 Overexpression of AMF-1 stimulated transactivation by both wild-type E2 an

151 , we may conclude that the overexpression of AMF/PGI enhances cell proliferation together with up-reg

152 Ectopic overexpression of AMF/PGI results in its secretion and activation via a co

153 ommunity type was more indicative than pH of AMF community structure.

154 The transformed phenotype of AMF/PGI-transfected cells leads in part resistance to in

155 In the presence of AMF hyphae, N2 O production remained low following ammon

156 ction of N2 O was reduced in the presence of AMF hyphae.

157 Here we have analyzed the regulation of AMF-R expression in a BALB/c angiosarcoma tumor system t

158 ion had no-significant effect on richness of AMF except at greater soil-depths.

159 To directly elucidate the functional role of AMF/PGI on cell motility and neoplastic transformation,

160 EGF enhanced secretion of AMF through its casein kinase II-mediated phosphorylatio

161 The cDNA sequences of AMF/NLK/PHI/MF found in both human cancer and normal cel

162 any researcher undertaking NGS sequencing of AMF communities.

163 The first signs of AMF differentiation appeared around the saccular stage o

164 , chemical composition and physical state of AMF.

165 The HRG-induced stimulation of AMF expression was inhibited by specific inhibitors of p

166 We explored the diversity and structure of AMF communities in grasslands, savannas, dry forests and

167 This marked ecological structure of AMF communities provides the first comprehensive landsca

168 Our results indicate that the structure of AMF community assemblages is correlated with P fertiliza

169 We found a high turnover of AMF with < 12% of VT present in all vegetation types.

170 Application of AMFs or transgenic overexpression of the receptor PYL2 i

171 itive macrophages, as the main precursors of AMFs.

172 In addition, the current knowledge on AMF diversity is biased towards temperate ecosystems, wh

173 e 2 (Nox2) subunits were reduced in OPN(-/-) AMFs.

174 TNF-alpha activation of pro-MMP9 in OPN(-/-) AMFs.

175 PGI/AMF cellular expression in HT1080 human fibrosarcoma was

176 PGI/AMF has been correlated significantly with breast cancer

177 PARP-14-PGI/AMF interaction was confirmed by coimmunoprecipitation a

178 Augmented expressions of both PGI/AMF and AMFR have been implicated in tumor progression a

179 tes the expression level of tumor cells' PGI/AMF.

180 f the housekeeping gene product/cytokine PGI/AMF, and the results depicted here suggest a novel thera

181 an PGI/AMF down-regulated the endogenous PGI/AMF expression and completely extinguished the secretion

182 studies of the significance of exogenous PGI/AMF on tumor progression have been reported.

183 high levels of endogenous and exogenous PGI/AMF, were stably transfected with PGI/AMF small interfer

184 ose isomerase/autocrine motility factor (PGI/AMF) is a housekeeping gene product present in all cells

185 ose isomerase/autocrine motility factor (PGI/AMF) is a housekeeping gene product/cytokine that cataly

186 ose isomerase/autocrine motility factor (PGI/AMF) plays an important role in glycolysis and gluconeog

187 The siRNA targeting human PGI/AMF down-regulated the endogenous PGI/AMF expression and

188 ivities, although it is known that human PGI/AMF is phosphorylated at Ser(185) by protein kinase CK2

189 ent kinase inhibitor was up-regulated in PGI/AMF knockdown cells and that superoxide dismutase is the

190 t the expression of miR-200s was lost in PGI/AMF overexpressing MCF-10A cells and in highly invasive

191 se results suggest a role of miR-200s in PGI/AMF-induced EMT and thus approaches for upregulation of

192 periments revealed that PARP-14 inhibits PGI/AMF ubiquitination, thus contributing to its stabilizati

193 s known of the biochemical regulation of PGI/AMF activities, although it is known that human PGI/AMF

194 c energy generation; thus, inhibition of PGI/AMF expression and activities may provide a new therapeu

195 hich in turn led to the up-regulation of PGI/AMF expression and was specifically inhibited by inhibit

196 Silencing of PGI/AMF expression by RNA interference in MDA-MB-231 cells i

197 Moreover, silencing of PGI/AMF expression in MDA-MB-231 cells led to overexpression

198 breast epithelial cells and reduction of PGI/AMF expression led to MET in aggressive breast cancer ce

199 east epithelial cells, and inhibition of PGI/AMF expression triggered mesenchymal-to-epithelial trans

200 In addition, the hypoxia induction of PGI/AMF expression was suppressed by inhibitors of vascular

201 completely extinguished the secretion of PGI/AMF in a human fibrosarcoma HT1080, whereas the control

202 her, the results show the involvement of PGI/AMF in both EMT and MET: overexpression of PGI/AMF induc

203 or cells, we examined the involvement of PGI/AMF in overcoming cellular senescence in cancer cells.

204 A specific inhibitor of PGI/AMF induced cellular senescence and p21 expression in tu

205 We show here that ectopic expression of PGI/AMF induced epithelial-to-mesenchymal transition (EMT) i

206 F in both EMT and MET: overexpression of PGI/AMF induces EMT in normal breast epithelial cells and re

207 new therapeutic target for inhibition of PGI/AMF inducing tumor cell migration and invasion during me

208 ssist in understanding the regulation of PGI/AMF intracellular function(s) and may provide a new ther

209 and an intracellular binding partner of PGI/AMF is expected to regulate in part its diverse biologic

210 der hypoxic conditions the expression of PGI/AMF is regulated in part by the HIF pathway, which in tu

211 l fibroblasts whereas down-regulation of PGI/AMF leads to mesenchymal-to-epithelial transition in tum

212 Suppression of PGI/AMF led to a contact-dependent inhibition of cell growth

213 reexpression of miR-200 or silencing of PGI/AMF suppressed pulmonary metastases of MDA-MB-231 cells

214 We also report that PGI/AMF degradation is mainly regulated by the ubiquitin-lys

215 h there are many studies indicating that PGI/AMF has been implicated in progression of metastasis, no

216 results suggest for the first time that PGI/AMF is a key gene to both EMT in the initiating step of

217 the results presented here suggest that PGI/AMF is involved in oxidative stress-induced cellular sen

218 Here we show, for the first time, that PGI/AMF overexpression led to an increase in the DNA-binding

219 Molecular analysis showed that PGI/AMF suppressed epithelial marker expressions and enhance

220 hat hypoxia-inducible VEGF regulates the PGI/AMF expression.

221 The PGI/AMF siRNA caused cells to change shape dramatically and

222 e effects were strongly inhibited by the PGI/AMF, VEGF, or VEGF receptor inhibitors.

223 Those PGI/AMF siRNA-transfected cells showed epithelial phenotype.

224 owever, the molecular mechanism by which PGI/AMF regulates EMT is not known.

225 Furthermore, tumor cells with PGI/AMF deficiency lost their abilities to form tumor mass.

226 us PGI/AMF, were stably transfected with PGI/AMF small interfering RNA (siRNA).

227 4 (PARP-14) to be a binding partner with PGI/AMF.

228 The cellular protein AMF-1 (Gps2) positively modulates gene expression by the

229 Our results not only prove AMF has important ecological significance on atrazine de

230 Fs increased with the amount of ROG reacted; AMFs also increased with decreasing AERs and increasing

231 Expression of the AMF receptor (AMF-R) in normal cells is regulated by cell contact wher

232 at HRG does indeed significantly up-regulate AMF expression.

233 ion differed and cell contact down-regulated AMF-R expression in the normal but not the transformed c

234 inatal intrapulmonary GM-CSF therapy rescued AMF development for weeks, although the resulting AMFs d

235 evelopment for weeks, although the resulting AMFs displayed an immature phenotype.

236 netic hyperthermia in a radio frequency (RF) AMF.

237 On a global scale AMF form the most widespread mycorrhizae, thus the abili

238 fferences in the community structure of soil AMF were observed between the controls and P treatments

239 es contributed minimally to the steady-state AMF pool.

240 ntial missing functional evidence supporting AMF symbionts as drivers of plant terrestrialization in

241 guttatus in both isolated plants supporting AMF for only a few months of the growing season and plan

242 The obligate plant-symbiotic AMF host additional symbionts, so-called Mollicutes-rela

243 phaGTPgammaS, weaker affinity for TDalphaGDP*AMF, and weakest affinity for TDalphaGDP.

244 ly a comparable high affinity for TDalphaGDP*AMF.

245 GTPgammaS), the transition state (TDalphaGDP*AMF) and the GDP-bound form (TDalphaGDP) with RGS-r and

246 Our findings challenge the assumption that AMF protect against degradation of organic carbon in soi

247 We thus conclude that AMF signaling shares a similar pathway to previously est

248 On the basis of this evidence that AMF may contribute to HER2-mediated breast cancer progre

249 Using this tool, we found that AMF enhances tumor cell motility by activating AKT/ERK,

250 We show here that AMF-1 also binds the transcriptional coactivator p300 in

251 initial studies support the hypothesis that AMF exposures can eradicate biofilm on metal implants, a

252 It is hypothesized that AMF hyphae may be outcompeting slow-growing nitrifiers f

253 As AMF acquire N, it was hypothesized that AMF hyphae may reduce N2 O production.

254 Phylogenetic analyses indicate that AMF may influence bacterial community assembly processes

255 a LexA fusion to the E2 AD, indicating that AMF-1 is a positive effector of the AD of E2.

256 These results suggest that AMF-1 facilitates the recruitment of p300 and its histon

257 ancer progression, our findings suggest that AMF-HER2 interaction might be a novel target for therape

258 This suggests that AMF mediate significant inter-plant carbon transfer in n

259 The AMF depends on the organic aerosol concentration, as wel

260 The AMF obligate biotrophy is not explained by genome erosio

261 The AMF released substantial C to the environment, triggerin

262 in the dissolved CO2 concentration among the AMF, stearin and olein fractions in their liquid state a

263 d the outcome of the interaction between the AMF and the PSB by conducting a microcosm and two Petri

264 uality and accessibility of NGS data for the AMF research community.

265 organic P, increasing P availability for the AMF.

266 sequenced the genome of DhMRE living in the AMF Dentiscutata heterogama.

267 ent of focal adhesion plaque proteins in the AMF migration-responsive cells exclusively.

268 ons resulted in higher CO2 solubility in the AMF.

269 Expression of the AMF receptor (AMF-R) in normal cells is regulated by cel

270 ilarities to nuclear-encoded proteins of the AMF Rhizophagus irregularis, which itself lacks MRE.

271 Depending on the AMF species, we found a strong asymmetry in the terms of

272 pe ratio mass spectrometry revealed that the AMF exported 4.9% of the litter (15) N to the host plant

273 It has been suggested that the AMF maintain a stable assemblage of several different ge

274 Our results suggest that the AMF primarily took up N in the inorganic form, and N exp

275 of the bacterial community responded to the AMF, Glomus hoi.

276 ailable P was low, the PSB competed with the AMF for P, and its activity was not stimulated by the fu

277 skingdom gene transfer between MRE and their AMF host.

278 is and neoplastic transformation, and, thus, AMF/PGI represents a novel class of oncogenic protein.

279 Antibodies to AMF-1 or E2 immunoprecipitated histone acetylase activit

280 a and Comamonadaceae responded negatively to AMF.

281 from the Firmicutes responded positively to AMF, while taxa from the Actinobacteria and Comamonadace

282 on and premeasured particle heat response to AMF amplitudes.

283 We identified loss of responsiveness to AMF in the rice (Oryza sativa) mutant hebiba, reflected

284 lastic transformation, we stably transfected AMF/PGI cDNA into NIH-3T3 cells.

285 Transient AMFs increased with the amount of ROG reacted; AMFs also

286 Herein, we quantify "transient AMFs" from ozonolysis of pulse-emitted limonene in a ven

287 ich can reach as high as 57.8 degrees C when AMF applied for 300 s.

288 experiments, N2 O production decreased when AMF hyphae were present before inorganic N addition.

289 rease in drought resistance is achieved when AMFs are applied to the PYL2-overexpression transgenic p

290 endent signaling, we sought to study whether AMF signaling involves family members of the Rho-like GT

291 form, and N export is one mechanism by which AMF could modify the soil microbial community and decomp

292 These observations led to a model in which AMF-1 recruits p300 into a complex with E2.

293 hat non-photosynthetic plants associate with AMF and can display the characteristic specificity of ep

294 th net whole-soil feedback and feedback with AMF of conspecifics; conservatism was especially strong

295 Indeed, RmPGI displays high homology with AMF and has been shown to have AMF activity.

296 acquisition, (2) legume tree inoculated with AMF and co-planted with legume herbs provides an effecti

297 ed based on their inability to interact with AMF-1 and were found to be unable to stimulate transcrip

298 edict how a plant species will interact with AMF.

299 We conclude that interaction with AMF-1 is necessary for the transcriptional activation fu

300 st ancient extant clade of land plants, with AMF significantly promotes photosynthetic carbon uptake,

301 lts show a linear dependence of the SLP with AMF frequency, as anticipated by current models.