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2 how conformation regulated interactions with AMPA-type and NMDA-type glutamate receptors (AMPARs/NMDA
5 was markedly attenuated by a Ca2+ permeable AMPA-type (Ca-AMPA) glutamate channel blocker, or by a n
6 single subunit is sufficient to desensitize AMPA-type channels and that receptors with one to four g
7 ion with a Poisson train of fast excitatory (AMPA-type) conductance transients, to simulate independe
8 in, mediates homeostatic synaptic scaling of AMPA type glutamate receptors (AMPARs) via its ability t
9 -hydroxy-5-methyl-4-isoxazolepropionic acid (AMPA) type glutamate receptors mediate most fast synapti
11 alpha-amino-3-hydroxy-5-methyl-4-isoxazole (AMPA)-type glutamate receptor has recently been demonstr
13 -hydroxy-5-methyl-4-isoxazolepropionic acid (AMPA)-type glutamate receptors (AMPA-Rs), which mediate
14 -hydroxy-5-methyl-4-isoxazolepropionic acid (AMPA)-type glutamate receptors (AMPARs) at Schaffer coll
15 hydroxy-5-methyl-4-isoxazole propionic acid (AMPA)-type glutamate receptors (AMPARs) mediate excitato
16 -hydroxy-5-methyl-4-isoxazolepropionic acid (AMPA)-type glutamate receptors (AMPARs) mediate the majo
17 hydroxy-5-methyl-4-isoxazole propionic acid (AMPA)-type glutamate receptors (AMPARs) to synapses is a
18 hydroxy-5-methyl-4-isoxazole propionic acid (AMPA)-type glutamate receptors (but not by blockade of N
19 ydroxy-5-methyl-4-isoaxazole propionic acid (AMPA)-type glutamate receptors (GluR1 and GluR2/3) durin
20 hydroxy-5-methyl-4-isoxazole propionic acid (AMPA)-type glutamate receptors and the function of synap
21 -hydroxy-5-methyl-4-isoxazolepropionic acid (AMPA)-type glutamate receptors and the stabilization of
22 ve IDRA 21 and other positive modulators of (AMPA)-type glutamate receptors are considered potential
23 o-3-hydroxy-5-methyl-4-isoxazole propionate (AMPA)-type glutamate receptors cause the enhanced respon
24 hydroxy-5-methyl-4-isoxazoleproprionic acid (AMPA)-type glutamate receptors during long-term potentia
25 -hydroxy-5-methyl-4-isoxazolepropionic acid (AMPA)-type glutamate receptors have distinct roles in co
26 no 3-hydroxy-5-methyl-4-isoxazolepropionate (AMPA)-type glutamate receptors in rat brain and to test
27 hydroxy-5-methyl-4-isoxazole propionic acid (AMPA)-type glutamate receptors mediate the majority of e
28 -hydroxy-5-methyl-4-isoxazolepropionic acid (AMPA)-type glutamate receptors were studied using equili
29 -hydroxy-5-methyl-4-isoxazolepropionic acid (AMPA)-type glutamate receptors, and thereby enhance fast
30 -hydroxy-5-methylisoxazole-4-propionic acid (AMPA)-type glutamate receptors, which become phosphoryla
36 to spines, reduced the synaptic targeting of AMPA-type glutamate (GluR1) receptors, and decreased AMP
39 taneous synaptic currents mediated by either AMPA-type glutamate or nicotinic acetylcholine receptors
40 on at synapses, which are mediated by either AMPA-type glutamate or nicotinic acetylcholine receptors
43 NMDA-type glutamate receptor (NMDAR) but not AMPA-type glutamate receptor (AMPAR) mediated currents.
45 synaptic strength in brain are dependent on AMPA-type glutamate receptor (AMPAR) recycling, which is
46 ances have been made in our understanding of AMPA-type glutamate receptor (AMPAR) regulation by trans
48 e that the specific intracellular domains of AMPA-type glutamate receptor (AMPAR) subunits are critic
51 ansmission is mediated primarily through the AMPA-type glutamate receptor (AMPAR); the regulation of
52 units for a very different ion channel - the AMPA-type glutamate receptor - prominently regulating ea
54 tamate input is necessary for clustering the AMPA-type glutamate receptor but not for clustering the
59 piny neurons as a primary site of persistent AMPA-type glutamate receptor plasticity by two widely us
60 et neurons of ALa in dorsal pallidum possess AMPA-type glutamate receptor profiles resembling those o
61 ill training induces an increase of synaptic AMPA-type glutamate receptor subunit 1 (GluA1), there is
62 -4-isoxazolepropionic acid receptor (AMPAR) [AMPA-type glutamate receptor subunit 1 (GluR1 subunit)],
64 PICK1 protein interacts in neurons with the AMPA-type glutamate receptor subunit 2 (GluR2) and with
65 AMPA receptor complexes that contain the AMPA-type glutamate receptor subunit 2 (GluR2) are respo
66 h correlates with a significant reduction of AMPA-type glutamate receptor subunit 2 (GluR2) at the sy
67 similar to the decrease in the number of the AMPA-type glutamate receptor subunit 2/3-immunoreactive
68 KAP5 is important for phosphorylation of the AMPA-type glutamate receptor subunit GluA1 on Ser-845 by
69 sociated with enhanced surface levels of the AMPA-type glutamate receptor subunit GluA2, an effect th
70 ctive effect of EphB2 may be mediated by the AMPA-type glutamate receptor subunit GluA2, which can be
71 ever, silenced neurons could not recruit the AMPA-type glutamate receptor subunit GluR1 as efficientl
72 of the presynaptic marker synaptophysin, the AMPA-type glutamate receptor subunit GluR1, and the puta
73 , and PKA form a signalling complex with the AMPA-type glutamate receptor subunit GluR1, which is lin
76 bridization, we show that mRNAs encoding the AMPA-type glutamate receptor subunits (GluRs) 1 and 2 ar
78 rc protein has been demonstrated to regulate AMPA-type glutamate receptor trafficking by recruiting e
80 display a dramatic reduction in frequency of AMPA-type glutamate receptor-mediated miniature excitato
81 ls exhibit a large and selective decrease in AMPA-type glutamate receptor-mediated synaptic transmiss
86 excitatory synapses where it associates with AMPA-type glutamate receptors (AMPAR) and enhances synap
87 molecules to synapses and in endocytosis of AMPA-type glutamate receptors (AMPAR) in the dendrites o
90 ity is the regulated addition and removal of AMPA-type glutamate receptors (AMPARs) at excitatory syn
93 Although the properties and trafficking of AMPA-type glutamate receptors (AMPARs) depend critically
95 ates endocytosis of GluR2 subunit-containing AMPA-type glutamate receptors (AMPARs) in an ATPase-depe
96 ), which activates postsynaptic synthesis of AMPA-type glutamate receptors (AMPARs) in dendrites and
97 /Arg3.1 selectively modulates trafficking of AMPA-type glutamate receptors (AMPARs) in neurons by acc
98 We studied the dynamics of newly synthesized AMPA-type glutamate receptors (AMPARs) induced with lear
101 The synaptic insertion of GluR1-containing AMPA-type glutamate receptors (AMPARs) is critical for s
113 naptic density protein-95 (PSD-95) localizes AMPA-type glutamate receptors (AMPARs) to postsynaptic s
115 xocytic fusion events mediating insertion of AMPA-type glutamate receptors (AMPARs) to the somatodend
117 ynthesis alters endocytosis and recycling of AMPA-type glutamate receptors (AMPARs), implicating PI(3
118 tic strength through changes in postsynaptic AMPA-type glutamate receptors (AMPARs), suggesting the e
120 ransmission in the CNS is mediated mainly by AMPA-type glutamate receptors (AMPARs), whose biophysica
121 ron synapses were dominated by GluA2-lacking AMPA-type glutamate receptors (AMPARs), with little cont
128 turation by recruitment of calcium-permeable AMPA-type glutamate receptors (CP-AMPARs) after drug wit
129 quires opening of calcium (Ca(2+))-permeable AMPA-type glutamate receptors (CP-AMPARs) and signaling
132 p) has been implicated in the aggregation of AMPA-type glutamate receptors (GluR) at excitatory synap
136 city that converge on regulation of NMDA and AMPA-type glutamate receptors (NMDAR, AMPAR), including
137 d of synaptic levels of the GluA1 subunit of AMPA-type glutamate receptors after 48 h silencing with
138 wo glycine receptors, one GABA receptor, two AMPA-type glutamate receptors and one purinergic recepto
139 tentiation was expressed postsynaptically by AMPA-type glutamate receptors and required calmodulin-de
140 oning induce similar changes in postsynaptic AMPA-type glutamate receptors and that occluding these c
141 These results suggest an interaction between AMPA-type glutamate receptors and the gap junction prote
142 EAAT2 buffers basal glutamate activation of AMPA-type glutamate receptors and therefore decreases ba
146 Several studies have implicated a change in AMPA-type glutamate receptors as being responsible for t
148 orylation cascades that alter the density of AMPA-type glutamate receptors at excitatory synapses; ho
149 ely increases the level of GluA1 subunits of AMPA-type glutamate receptors at the synapses of the nuc
150 then the present data suggest that forebrain AMPA-type glutamate receptors can be classified into a l
151 s in the subunit composition of postsynaptic AMPA-type glutamate receptors can be induced at CNS syna
152 were used to test if positive modulators of AMPA-type glutamate receptors have regionally differenti
155 igate the relationship between the number of AMPA-type glutamate receptors in the PSD and synaptic st
156 investigated whether positive modulators of AMPA-type glutamate receptors influence neurotrophin exp
158 idal cells, TNFalpha drives the insertion of AMPA-type glutamate receptors into synapses, and contrib
159 Here we report that fear conditioning drives AMPA-type glutamate receptors into the synapse of a larg
164 d hippocampal neurons to aggregate NMDA- and AMPA-type glutamate receptors on each other as a way of
165 eurons results in clusters of both NMDA- and AMPA-type glutamate receptors on hippocampal interneuron
166 cally and probably involves up-regulation of AMPA-type glutamate receptors on hypocretin neurons.
167 Spinal axons, which normally cluster only AMPA-type glutamate receptors on other spinal neurons, c
171 changes is the remodeling of the ionotropic AMPA-type glutamate receptors that underlie fast excitat
172 he postsynaptic density, tethering NMDA- and AMPA-type glutamate receptors to signaling proteins and
173 easing the ubiquitination and degradation of AMPA-type glutamate receptors via a mechanism depending
176 ertension alters dendritic spines containing AMPA-type glutamate receptors within NTS, suggesting tha
178 sent study tested if a positive modulator of AMPA-type glutamate receptors would counteract the behav
179 the subunit that limits Ca2+ permeability of AMPA-type glutamate receptors) was markedly and specific
180 se EPSCs were abolished by the antagonist of AMPA-type glutamate receptors, 6-cyano-7-nitro-quinoxali
181 the cell surface expression of NMDA-type and AMPA-type glutamate receptors, along with prominent func
182 eds, wave initiation depends increasingly on AMPA-type glutamate receptors, and an ever increasing fr
183 s onto FSIs, which are mediated primarily by AMPA-type glutamate receptors, glutamate release by chol
184 ), an agent used to block desensitization of AMPA-type glutamate receptors, on heterologously express
185 eversibly modifies the kinetic properties of AMPA-type glutamate receptors, on synaptic responses is
186 e, we show that membrane proteins, including AMPA-type glutamate receptors, rapidly diffuse within th
187 ls with an ampakine, a positive modulator of AMPA-type glutamate receptors, rescues plasticity and re
188 n is of particular importance with regard to AMPA-type glutamate receptors, the multimeric complexes
189 striatum is mediated, in part, by ionotropic AMPA-type glutamate receptors, which are heteromers comp
208 s, we studied the distributions of NMDA- and AMPA-type glutamate receptors; the NMDA receptor-interac
209 tested the role of the postsynaptic NMDA and AMPA type glutamatergic receptors in the lactate-induced
211 nent of the cellular machinery that delivers AMPA-type glutamatergic receptors (AMPARs) into synapses
212 p segments and helices within a region of an AMPA-type iGluR NTD, which has been identified previousl
215 -isomer has been identified as a competitive AMPA-type ionotropic glutamate receptor antagonist, whil
216 Phosphorylation and dephosphorylation of AMPA-type ionotropic glutamate receptors (AMPARs) by kin
219 ly inhibited by activation of either NMDA or AMPA-type ionotropic glutamate receptors in a calcium-de
222 three ionotropic glutamate subfamilies (i.e. AMPA-type, kainate-type, and NMDA-type) assemble as tetr
223 -hydroxy-5-methyl-4-isoxazolepropionic acid (AMPA) type mediate fast excitatory synaptic transmission
224 ent with the role of synaptic trafficking of AMPA-type of glutamate receptors in HSP, Mecp2 KO neuron
225 hdrawal requires activation of NMDA-type and AMPA-type postsynaptic receptors within the abdominal ga
230 tion 5-HT causes the insertion of additional AMPA-type receptors into the postsynaptic membrane of se
231 mulation, IHCs release glutamate to activate AMPA-type receptors on these myelinated type-I neurons,
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