コーパス検索結果 (１語後でソート)
通し番号をクリックするとPubMedの該当ページを表示します
1 tic peptide that prevents internalization of AMPA-type glutamate receptor.
2 hydroxy-5-methyl-4-isoxazole-propionic acid (AMPA)-type glutamate receptor.
3 gh removal and dephosphorylation of synaptic AMPA-type glutamate receptors.
4 al and activate the AIB interneurons through AMPA-type glutamate receptors.
5 control synaptic targeting and insertion of AMPA-type glutamate receptors.
6 y involves activity-dependent trafficking of AMPA-type glutamate receptors.
7 l pentraxin domains mediate association with AMPA-type glutamate receptors.
8 of glutamate receptor type 1 subunits of the AMPA-type glutamate receptors.
9 ell characterized as a negative modulator of AMPA-type glutamate receptors.
10 nase regulates the physiological activity of AMPA-type glutamate receptors.
11 ration of a positive allosteric modulator of AMPA-type glutamate receptors.
12 stitutive, internalization of both NMDA- and AMPA-type glutamate receptors.
13 al activity- and PDZ-dependent regulation of AMPA-type glutamate receptors.
14 ptic activity to postsynaptic endocytosis of AMPA-type glutamate receptors.
15 ths of age, which involves calcium-permeable AMPA-type glutamate receptors.
16 c input from bipolar cells through NMDA- and AMPA-type glutamate receptors.
17 via its regulatory effect on trafficking of AMPA-type glutamate receptors.
18 ayed release, a large quantal size, and fast AMPA-type glutamate receptors.
19 ction in the level of synaptically localized AMPA-type glutamate receptors.
20 NMDA receptors (NMDARs) and Ca2+-impermeable AMPA-type glutamate receptors.
21 -hydroxy-5-methyl-4-isoxazolepropionic acid (AMPA)-type glutamate receptors.
22 o-3-hydroxyl-5-methyl-4-isoxazolepropionate (AMPA)-type glutamate receptors.
23 -hydroxy-5-methyl-4-isoxazolepropionic acid (AMPA)-type glutamate receptors.
24 -hydroxy-5-methylisoxazole-4-propionic acid (AMPA)-type glutamate receptors.
25 -hydroxy-5-methyl-4-isoxazolepropionic acid (AMPA)-type glutamate receptors.
26 se EPSCs were abolished by the antagonist of AMPA-type glutamate receptors, 6-cyano-7-nitro-quinoxali
27 d of synaptic levels of the GluA1 subunit of AMPA-type glutamate receptors after 48 h silencing with
28 the cell surface expression of NMDA-type and AMPA-type glutamate receptors, along with prominent func
29 -hydroxy-5-methyl-4-isoxazolepropionic acid (AMPA)-type glutamate receptors (AMPA-Rs), which mediate
35 NMDA-type glutamate receptor (NMDAR) but not AMPA-type glutamate receptor (AMPAR) mediated currents.
37 synaptic strength in brain are dependent on AMPA-type glutamate receptor (AMPAR) recycling, which is
38 ances have been made in our understanding of AMPA-type glutamate receptor (AMPAR) regulation by trans
40 e that the specific intracellular domains of AMPA-type glutamate receptor (AMPAR) subunits are critic
43 ansmission is mediated primarily through the AMPA-type glutamate receptor (AMPAR); the regulation of
44 excitatory synapses where it associates with AMPA-type glutamate receptors (AMPAR) and enhances synap
45 molecules to synapses and in endocytosis of AMPA-type glutamate receptors (AMPAR) in the dendrites o
47 in, mediates homeostatic synaptic scaling of AMPA type glutamate receptors (AMPARs) via its ability t
48 -hydroxy-5-methyl-4-isoxazolepropionic acid (AMPA)-type glutamate receptors (AMPARs) at Schaffer coll
49 hydroxy-5-methyl-4-isoxazole propionic acid (AMPA)-type glutamate receptors (AMPARs) mediate excitato
50 -hydroxy-5-methyl-4-isoxazolepropionic acid (AMPA)-type glutamate receptors (AMPARs) mediate the majo
51 hydroxy-5-methyl-4-isoxazole propionic acid (AMPA)-type glutamate receptors (AMPARs) to synapses is a
53 ity is the regulated addition and removal of AMPA-type glutamate receptors (AMPARs) at excitatory syn
56 Although the properties and trafficking of AMPA-type glutamate receptors (AMPARs) depend critically
58 ates endocytosis of GluR2 subunit-containing AMPA-type glutamate receptors (AMPARs) in an ATPase-depe
59 ), which activates postsynaptic synthesis of AMPA-type glutamate receptors (AMPARs) in dendrites and
60 /Arg3.1 selectively modulates trafficking of AMPA-type glutamate receptors (AMPARs) in neurons by acc
61 We studied the dynamics of newly synthesized AMPA-type glutamate receptors (AMPARs) induced with lear
64 The synaptic insertion of GluR1-containing AMPA-type glutamate receptors (AMPARs) is critical for s
76 naptic density protein-95 (PSD-95) localizes AMPA-type glutamate receptors (AMPARs) to postsynaptic s
78 xocytic fusion events mediating insertion of AMPA-type glutamate receptors (AMPARs) to the somatodend
80 ynthesis alters endocytosis and recycling of AMPA-type glutamate receptors (AMPARs), implicating PI(3
81 tic strength through changes in postsynaptic AMPA-type glutamate receptors (AMPARs), suggesting the e
83 ransmission in the CNS is mediated mainly by AMPA-type glutamate receptors (AMPARs), whose biophysica
84 ron synapses were dominated by GluA2-lacking AMPA-type glutamate receptors (AMPARs), with little cont
91 hydroxy-5-methyl-4-isoxazole propionic acid (AMPA)-type glutamate receptors and the function of synap
92 -hydroxy-5-methyl-4-isoxazolepropionic acid (AMPA)-type glutamate receptors and the stabilization of
93 wo glycine receptors, one GABA receptor, two AMPA-type glutamate receptors and one purinergic recepto
94 tentiation was expressed postsynaptically by AMPA-type glutamate receptors and required calmodulin-de
95 oning induce similar changes in postsynaptic AMPA-type glutamate receptors and that occluding these c
96 These results suggest an interaction between AMPA-type glutamate receptors and the gap junction prote
97 EAAT2 buffers basal glutamate activation of AMPA-type glutamate receptors and therefore decreases ba
98 -hydroxy-5-methyl-4-isoxazolepropionic acid (AMPA)-type glutamate receptors, and thereby enhance fast
99 eds, wave initiation depends increasingly on AMPA-type glutamate receptors, and an ever increasing fr
101 ve IDRA 21 and other positive modulators of (AMPA)-type glutamate receptors are considered potential
105 Several studies have implicated a change in AMPA-type glutamate receptors as being responsible for t
107 orylation cascades that alter the density of AMPA-type glutamate receptors at excitatory synapses; ho
108 ely increases the level of GluA1 subunits of AMPA-type glutamate receptors at the synapses of the nuc
109 tamate input is necessary for clustering the AMPA-type glutamate receptor but not for clustering the
110 hydroxy-5-methyl-4-isoxazole propionic acid (AMPA)-type glutamate receptors (but not by blockade of N
111 then the present data suggest that forebrain AMPA-type glutamate receptors can be classified into a l
112 s in the subunit composition of postsynaptic AMPA-type glutamate receptors can be induced at CNS syna
113 o-3-hydroxy-5-methyl-4-isoxazole propionate (AMPA)-type glutamate receptors cause the enhanced respon
114 turation by recruitment of calcium-permeable AMPA-type glutamate receptors (CP-AMPARs) after drug wit
115 quires opening of calcium (Ca(2+))-permeable AMPA-type glutamate receptors (CP-AMPARs) and signaling
119 hydroxy-5-methyl-4-isoxazoleproprionic acid (AMPA)-type glutamate receptors during long-term potentia
123 p) has been implicated in the aggregation of AMPA-type glutamate receptors (GluR) at excitatory synap
124 ydroxy-5-methyl-4-isoaxazole propionic acid (AMPA)-type glutamate receptors (GluR1 and GluR2/3) durin
128 s onto FSIs, which are mediated primarily by AMPA-type glutamate receptors, glutamate release by chol
129 alpha-amino-3-hydroxy-5-methyl-4-isoxazole (AMPA)-type glutamate receptor has recently been demonstr
130 -hydroxy-5-methyl-4-isoxazolepropionic acid (AMPA)-type glutamate receptors have distinct roles in co
131 were used to test if positive modulators of AMPA-type glutamate receptors have regionally differenti
132 no 3-hydroxy-5-methyl-4-isoxazolepropionate (AMPA)-type glutamate receptors in rat brain and to test
135 igate the relationship between the number of AMPA-type glutamate receptors in the PSD and synaptic st
136 investigated whether positive modulators of AMPA-type glutamate receptors influence neurotrophin exp
138 idal cells, TNFalpha drives the insertion of AMPA-type glutamate receptors into synapses, and contrib
139 Here we report that fear conditioning drives AMPA-type glutamate receptors into the synapse of a larg
141 -hydroxy-5-methyl-4-isoxazolepropionic acid (AMPA) type glutamate receptors mediate most fast synapti
142 hydroxy-5-methyl-4-isoxazole propionic acid (AMPA)-type glutamate receptors mediate the majority of e
147 display a dramatic reduction in frequency of AMPA-type glutamate receptor-mediated miniature excitato
148 ls exhibit a large and selective decrease in AMPA-type glutamate receptor-mediated synaptic transmiss
149 city that converge on regulation of NMDA and AMPA-type glutamate receptors (NMDAR, AMPAR), including
150 d hippocampal neurons to aggregate NMDA- and AMPA-type glutamate receptors on each other as a way of
151 eurons results in clusters of both NMDA- and AMPA-type glutamate receptors on hippocampal interneuron
152 cally and probably involves up-regulation of AMPA-type glutamate receptors on hypocretin neurons.
153 Spinal axons, which normally cluster only AMPA-type glutamate receptors on other spinal neurons, c
154 ), an agent used to block desensitization of AMPA-type glutamate receptors, on heterologously express
155 eversibly modifies the kinetic properties of AMPA-type glutamate receptors, on synaptic responses is
156 piny neurons as a primary site of persistent AMPA-type glutamate receptor plasticity by two widely us
157 et neurons of ALa in dorsal pallidum possess AMPA-type glutamate receptor profiles resembling those o
158 units for a very different ion channel - the AMPA-type glutamate receptor - prominently regulating ea
159 e, we show that membrane proteins, including AMPA-type glutamate receptors, rapidly diffuse within th
162 ls with an ampakine, a positive modulator of AMPA-type glutamate receptors, rescues plasticity and re
163 ill training induces an increase of synaptic AMPA-type glutamate receptor subunit 1 (GluA1), there is
164 -4-isoxazolepropionic acid receptor (AMPAR) [AMPA-type glutamate receptor subunit 1 (GluR1 subunit)],
166 PICK1 protein interacts in neurons with the AMPA-type glutamate receptor subunit 2 (GluR2) and with
167 AMPA receptor complexes that contain the AMPA-type glutamate receptor subunit 2 (GluR2) are respo
168 h correlates with a significant reduction of AMPA-type glutamate receptor subunit 2 (GluR2) at the sy
169 similar to the decrease in the number of the AMPA-type glutamate receptor subunit 2/3-immunoreactive
170 KAP5 is important for phosphorylation of the AMPA-type glutamate receptor subunit GluA1 on Ser-845 by
171 sociated with enhanced surface levels of the AMPA-type glutamate receptor subunit GluA2, an effect th
172 ctive effect of EphB2 may be mediated by the AMPA-type glutamate receptor subunit GluA2, which can be
173 ever, silenced neurons could not recruit the AMPA-type glutamate receptor subunit GluR1 as efficientl
174 of the presynaptic marker synaptophysin, the AMPA-type glutamate receptor subunit GluR1, and the puta
175 , and PKA form a signalling complex with the AMPA-type glutamate receptor subunit GluR1, which is lin
178 bridization, we show that mRNAs encoding the AMPA-type glutamate receptor subunits (GluRs) 1 and 2 ar
181 changes is the remodeling of the ionotropic AMPA-type glutamate receptors that underlie fast excitat
182 n is of particular importance with regard to AMPA-type glutamate receptors, the multimeric complexes
183 s, we studied the distributions of NMDA- and AMPA-type glutamate receptors; the NMDA receptor-interac
184 he postsynaptic density, tethering NMDA- and AMPA-type glutamate receptors to signaling proteins and
185 rc protein has been demonstrated to regulate AMPA-type glutamate receptor trafficking by recruiting e
187 easing the ubiquitination and degradation of AMPA-type glutamate receptors via a mechanism depending
189 the subunit that limits Ca2+ permeability of AMPA-type glutamate receptors) was markedly and specific
190 -hydroxy-5-methyl-4-isoxazolepropionic acid (AMPA)-type glutamate receptors were studied using equili
192 -hydroxy-5-methylisoxazole-4-propionic acid (AMPA)-type glutamate receptors, which become phosphoryla
193 striatum is mediated, in part, by ionotropic AMPA-type glutamate receptors, which are heteromers comp
194 ertension alters dendritic spines containing AMPA-type glutamate receptors within NTS, suggesting tha
196 sent study tested if a positive modulator of AMPA-type glutamate receptors would counteract the behav
WebLSDに未収録の専門用語（用法）は "新規対訳" から投稿できます。