ライフサイエンスコーパス: AMSA

1 AMSA has the potential for guiding more optimal timing o

2 AMSA is highly associated with pre-hospital ROSC, surviv

3 AMSA predicts the success of electrical defibrillation w

4 AMSA represents a numerical value based on the sum of th

5 AMSA therefore serves to minimize interruptions of preco

6 AMSA was measured prior to each shock and averaged for e

7 AMSA, therefore, has the potential for guiding optimal t

8 AMSA-avg was associated with pre-hospital ROSC (p = 0.00

9 AMSA-IPF will be another great tool to facilitate detect

10 bute to the intercalator resistance of HL-60/AMSA cells.

11 HL-60/AMSA contains a mutant form of topo IIalpha that was tho

12 that expression of topo IIbeta RNA in HL-60/AMSA is only 10% of that in HL-60, and topo IIbeta prote

13 HL-60/AMSA, a human leukemia cell line, is resistant to interc

14 Additionally, AMSA-avg was associated with hospital admission (p < 0.0

15 The anterior middle superior alveolar (AMSA) injection is reported to effectively anesthetize m

16 en ara-C with either doxorubicin, amsacrine (AMSA), or daunorubicin without ATRA, using logistic and

17 An AMSA value of 21 mV x Hz predicted restoration of perfus

18 At an AMSA value of >13.0 mV-Hz, successful defibrillation yie

19 imals, a validation group, confirmed that an AMSA value of 21 mV.Hz predicted restoration of perfusin

20 brillator application, first shock AMSA, and AMSA-avg that could predict pre-hospital ROSC, hospital

21 We found that IMAP and AMSA scores had similar median values (1.75 [interquarti

22 form characteristic amplitude spectral area (AMSA) can predict successful defibrillation and return o

23 ographic predictor, amplitude spectral area (AMSA), for the optimal timing of defibrillation shocks i

24 was defined as the amplitude spectrum area (AMSA).

25 transformed into an amplitude spectrum area (AMSA).

26 hools' American Medical Student Association (AMSA) PharmFree Scorecard and average Institute on Medic

27 ontal surgeries with unilateral or bilateral AMSA injections.

28 pectral-averaging isotope-pattern-filtering (AMSA-IPF) algorithm developed in the computational langu

29 were less likely to report receiving gifts (AMSA score, odds ratio [OR]: 0.37, 95% CI 0.19-0.72; IMA

30 the major target for the antitumour agent m-AMSA (4'-(9-acridinylamino)methanesulphonm-ansidide) in

31 ases in sensitivity to the antitumor agent m-AMSA [4'-(9-acridinylamino)methanesulfon-m-anisidide] we

32 unit) causes resistance to antitumor agent m-AMSA but hypersensitivity to the quinolone oxolinic acid

33 o the DNA intercalating anti-tumour agents m-AMSA and ellipticine, but confer resistance to the non-i

34 acridinylamino)methanesulphon-m-anisidide (m-AMSA) stabilizes the T4 type II topoisomerase at the str

35 -acridinylamino)methanesulfon-m-anisidide (m-AMSA)-resistant bacteriophage T4 topoisomerases have pre

36 leaving subunit) causes resistance to both m-AMSA and oxolinic acid.

37 Inhibition of the purified enzyme by m-AMSA results in formation of a cleavage complex that con

38 ntitumor drugs such as VM-26, doxorubicin, m-AMSA, and mitoxantrone.

39 Finally, m-AMSA induced the production of cleavage complexes involv

40 in the recombinational repair pathway for m-AMSA-induced damage.

41 These agents included m-AMSA (4'-(9-acridinylamino)methanesulfon-m-anisidide), m

42 of cleavage-inducing inhibitors including m-AMSA, VP-16, mitoxantrone, ellipticine, and oxolinic aci

43 oisomerase II poisons such as etoposide or m-AMSA which require micromolar concentrations to elicit c

44 The resulting m-AMSA-dependent repair products do not form in the absenc

45 to be resistant to m-AMSA, indicating that m-AMSA inhibits growth by inducing the cleavage complex ra

46 Second, the m-AMSA sensitivity of the rnh-deletion mutant was shown to

47 Third, m-AMSA stimulated recombination in phage-infected bacteria

48 hown to confer hypersensitivity in vivo to m-AMSA and oxolinic acid.

49 The substitutions alter sensitivity to m-AMSA and to oxolinic acid, sometimes in opposite directi

50 ient mutants were shown to be resistant to m-AMSA, indicating that m-AMSA inhibits growth by inducing

51 ) increased the sensitivity of phage T4 to m-AMSA, strongly suggesting that recombination participate

52 of Rnh protein causes hypersensitivity to m-AMSA.

53 Association of AMSA with defibrillation success was independent of the

54 p of 55 animals yielded a threshold value of AMSA that uniformly predicted successful resuscitation.

55 The negative predictive value of AMSA was 95% and statistically equivalent to that of cor

56 Scores on AMSA and IMAP shared policy dimensions were not closely

57 to interact with marketing representatives (AMSA score, OR 0.33, 95% CI 0.15-0.69; IMAP score, OR 0.

58 nitor/defibrillator application, first shock AMSA, and AMSA-avg that could predict pre-hospital ROSC,

59 First-shock AMSA was also predictive of pre-hospital ROSC, hospital

60 to each shock and averaged for each subject (AMSA-avg).

61 The AMSA injection is a novel anesthetic technique that may

62 The AMSA injection provided promising results for a variety

63 The AMSA-IPF algorithm is applicable to not only compounds c

64 The AMSA-IPF algorithm offers three significant improvements

65 The greater the AMSA value, the greater was the probability of reversal

66 Drawbacks of the AMSA injection included occasionally inadequate buccal h

67 Benefits of the AMSA injection included outstanding palatal hemostatic c

68 is to provide background information on the AMSA injection and demonstrate its use in a variety of m

69 very of a shock, were analyzed utilizing the AMSA algorithm.

70 ture studies are needed to determine whether AMSA computed during resuscitation can identify patients

71 brillation attempt was greatly improved with AMSA (78%) as compared with coronary perfusion pressure