ライフサイエンスコーパス: ANCA-positive

コーパス検索結果 (１語後でソート)

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1 At the time of KTX, 29 patients were ANCA-positive.

2 Speckled ANCA (sANCA) subjects were ANCA positive by ELISA with a speckling over the entire

3 f HNE ANCA-positive CIMDL sera were also PR3 ANCA-positive by at least 1 assay.

4 ]), and antineutrophil cytoplasmic antibody (ANCA)-positive cases were compared with control groups.

5 Fifty-seven percent of HNE ANCA-positive CIMDL sera were also PR3 ANCA-positive by

6 e 3-antineutrophil cytoplasmic antibody (PR3-ANCA)-positive IgG preparations or myeloperoxidase-ANCA

7 gG preparations or myeloperoxidase-ANCA (MPO-ANCA)-positive IgG preparations to activate neutrophils

8 The present study examined the effects of ANCA-positive IgG (ANCA IgG), derived from patients with

9 were perfused in the presence or absence of ANCA-positive IgG over endothelial cells that had been a

10 ositive IgG preparations 0.735 +/- 0.10, PR3-ANCA-positive IgG preparations 0.33 +/- 0.098; P < 0.01)

11 /- 0.35 nmoles; P < 0.001), Ca2+ fluxes (MPO-ANCA-positive IgG preparations 0.735 +/- 0.10, PR3-ANCA-

12 ive IgG preparations 251.98 +/- 26.7 ng, PR3-ANCA-positive IgG preparations 145.19 +/- 19.4 ng; P < 0

13 0.098; P < 0.01), and MPO degranulation (MPO-ANCA-positive IgG preparations 251.98 +/- 26.7 ng, PR3-A

14 ons 9.13 +/- 0.39 nmoles [mean +/- SEM], PR3-ANCA-positive IgG preparations 6.32 +/- 0.35 nmoles; P <

15 greater generation of superoxide anions (MPO-ANCA-positive IgG preparations 9.13 +/- 0.39 nmoles [mea

16 G4 were the predominant isotypes in both MPO-ANCA-positive IgG preparations and PR3-ANCA.

17 In vitro MPO-ANCA-positive IgG preparations are more activating than

18 Activation of PMN by MPO-ANCA-positive IgG preparations compared with PR3-ANCA-po

19 icantly more IgG1 than did PR3-ANCA, and PR3-ANCA-positive IgG preparations contained significantly m

20 -positive IgG preparations compared with PR3-ANCA-positive IgG preparations resulted in greater gener

21 The increased activation seen with MPO-ANCA-positive IgG preparations was not due to increased

22 cell surface or greater IgG3 present in MPO-ANCA-positive IgG preparations.

23 gG preparations are more activating than PR3-ANCA-positive IgG preparations.

24 at incorporate ANCA specificity, such as PR3 ANCA-positive MPA and MPO ANCA-positive MPA, provide a m

25 icity, such as PR3 ANCA-positive MPA and MPO ANCA-positive MPA, provide a more useful tool than the c

26 cificity was predictive of relapse, with PR3 ANCA-positive patients almost twice as likely to relapse

27 Clinical profiles of the ANCA-positive patients with CD were compared with those

28 Nine centers enrolled 197 ANCA-positive patients with either Wegener's granulomato

29 One hundred seven ANCA-positive patients with necrotizing and crescentic g

30 Sera from 61 PR3 ANCA-positive patients with WG or MPA were assayed by ca

31 , in contrast to anti-LAMP-2 antibodies from ANCA-positive patients, these antibodies from ANCA-negat

32 and FACS analysis demonstrate reactivity of ANCA-positive sera and antimyeloperoxidase antibodies wi

33 IgG from NE ANCA-positive sera of patients with CIMDL inhibited the

34 Sixty percent of ANCA-positive sera showed a perinuclear reaction pattern

35 induce antineutrophil cytoplasmic antibody (ANCA)-positive vasculitis (APV) are largely unknown.

36 ed with antineutrophil cytoplasmic antibody (ANCA)-positive vasculitis.

37 e demonstrates that patients with idiopathic ANCA-positive vasculitis may quickly develop a superimpo

38 ickly develop a superimposed drug-associated ANCA-positive vasculitis.

39 azine or propylthiouracil is associated with ANCA-positive vasculitis.

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