ライフサイエンスコーパス: AT-rich region

1 binding sites upstream and downstream of the AT-rich region.

2 separation is not precisely localized in the AT-rich region.

3 eletion of both the Fis binding site and the AT-rich region.

4 e core and then 400 to 600 bp beyond into an AT-rich region.

5 aA box sequences followed by unwinding of an AT-rich region.

6 GC-rich region prevented TBP binding to the AT-rich region.

7 ymines situated at the 5' and 3' ends of the AT-rich region.

8 method, on a test dataset and on relatively AT-rich regions.

9 e of cisplatination at sites upstream of the AT-rich regions.

10 increase in base substitution error rates in AT-rich regions.

11 a Fis binding site, a DnaA binding site, an AT-rich region, an inverted repeat and a 10 bp site betw

12 uct and dinucleotide adduct sites within the AT-rich region and owing to the influence of cisplatinat

13 inverted repeat and a 10 bp site between the AT-rich region and the inverted repeat.

14 ycin binds the minor groove of duplex DNA at AT-rich regions and has been a valuable probe of protein

15 th potential single-stranded character, like AT-rich regions and sequences that can form cruciform st

16 ning 652 base pairs in between contained two AT-rich regions and several regulatory sequences.

17 stone chromatin proteins able to bind DNA in AT-rich regions and to interact with various transcripti

18 narrowed the 1p21.2 breakpoint to a 1990 bp AT-rich region, and junction fragments were amplified by

19 for overall AT content, for distribution of AT-rich regions, and for the abundance of several MAR-re

20 GC bps embedded in AT-rich regions are known to be essential for nogalamyci

21 Flanking sequences, such as the AT-rich region, are known to be important for DNA replic

22 e of the sterile antisense transcripts to an AT-rich region, as is typical for GIARDIA: It is unclear

23 ells, with the most prominent changes in the AT-rich region associated with the dA/dT(23) repeat and

24 e DnaA protein to bring about opening of the AT-rich region at the replication origin.

25 lement (APRE (SIE)) at -171 to -163, and two AT-rich regions at -143 to -138 and -128 to -123.

26 id not detectably footprint to either GC- or AT-rich regions at equivalent doses.

27 slocation occurs within a short, palindromic AT-rich region (ATRR).

28 sponse to the stimulants LPS and LTA and the AT-rich region between nt -2571 and -2338 in the Saa3 pr

29 a localized increase in accessibility at the AT-rich regions bound by homeo-proteins and perhaps at a

30 tion start site, and depend on a TATA box or AT-rich region but not the downstream promoter element (

31 The mutagenesis of the AT-rich regions, but not the AP-1 site, resulted in a lo

32 unit, while pentanucleotides 2 and 4 and the AT-rich region constitute a second, relatively weak, ass

33 Sequences within this AT-rich region determined to be important for TrcR bindi

34 cription activity revealed that the extended AT-rich region does not affect the kinetics of abortive

35 and CspE are U/T stretches, AGGGAGGGA and AU/AT-rich regions, especially AAAUUU, respectively.

36 ze and contain three functional elements: an AT-rich region flanked by binding sites for Cbf1 and CBF

37 enetic assay in yeast, we found that a short AT-rich region (Flex1) within FRA16D increases chromosom

38 ch extension (ext-a1) of the core ORF and an AT-rich region following the core extension (dsORF-a1).

39 statin, the sequence specificity lies in the AT-rich region for hybrids and is similar to that of DNA

40 Similarly, promoters in which the AT-rich region from -17 to -22 is interrupted by several

41 nd can be functionally substituted for by an AT-rich region from the human lamin B2 IR that differs i

42 We identified a novel AT-rich region immediately downstream of the -35 region.

43 with a highly conserved thymine (-14) and an AT-rich region in the middle between the hallmark RpoN-r

44 To further analyze the role of the AT-rich region in TrcR autoregulation, the trcR promoter

45 g proteins that bind to the narrow groove of AT-rich regions in double-stranded DNA.

46 nalysis of Fugu sequences homologous to very AT-rich regions in the human genome may, therefore, be a

47 in vitro and in vivo; (iii) mutation of the AT-rich regions inhibits HMGA1a binding in vitro; and (i

48 t made up of pentanucleotide 2 and 4 and the AT-rich region is initiated by assembly of a hexamer on

49 est that most A. gambiae MITEs are in highly AT-rich regions, many of which are closely associated wi

50 of dnaA to the end of yaaA that includes the AT-rich region melted during the initiation stage of DNA

51 nces within oriC followed by unwinding of an AT-rich region near the left border.

52 A reduction in fidelity in AT-rich regions occurred for pol II despite having an as

53 Mutations in the AT-rich region of gamma also abolished alpha function.

54 o be essential for DNA strand opening at the AT-rich region of the replication origin of the Escheric

55 a forkhead consensus site located within an AT-rich region of the telokin promoter.

56 s in gel mobility shift assays with TrcR, an AT-rich region of the trcR promoter was shown to be esse

57 We and others have previously shown that AT-rich regions of DNA surrounding transcription factor

58 findings, we show that these motifs occur in AT-rich regions of DNA.

59 GA proteins that bind to the minor groove of AT-rich regions of DNA.

60 can inhibit transcription factor binding to AT-rich regions of DNA.

61 ferentiate among bulk DNA and three discrete AT-rich regions of genomic DNA examined by quantitative

62 air of basic repeats to recognize the tandem AT-rich regions of the binding sites.

63 s MvaT and MvaU are thought to bind the same AT-rich regions of the chromosome and function coordinat

64 ements, MvaT and MvaU bind preferentially to AT-rich regions of the chromosome.

65 l patterns seen in A. mellifera that lead to AT-rich regions of the genome.

66 ressing HMGA1a; (ii) HMGA1a protein binds to AT-rich regions of the KL promoter DNA both in vitro and

67 The FRalpha recognition domain mapped to AT rich regions on the promoters.

68 also no evidence of preferential cleavage in AT-rich regions or other target DNA sites implicated in

69 site, as well as by mutations that alter the AT-rich regions or their phasing.

70 tion previously observed for deletion of the AT-rich region results from deletion of both the Fis bin

71 ence of a second repeat bound to an adjacent AT-rich region results in intramolecular cooperativity i

72 es adjacent to cruciform structures abutting AT-rich regions, similar to the CRISPR leader sequence.

73 An AT-rich region that borders the dA/dT repeat was also hi

74 rotein one (AP-1) and RIP60 in vitro, and an AT-rich region that contains a dA/dT(23) dinucleotide re

75 The binding site was localized to an AT-rich region that contains a large number of GANTC sit

76 rom DNase I digestion an approximately 73-bp AT-rich region that includes the entire mntA promoter.

77 onucleotide repeats in two sets, flanking an AT-rich region that may be the site of initial melting.

78 terated initiator protein binding sites), an AT-rich region that melts upon initiator-iteron interact

79 Herein we studied the role of the AT-rich region upstream of -17 in transcription regulati

80 I promoters are characterized by an extended AT-rich region upstream of -17, which is often interrupt

81 the R1-2 clone, but its specificity for the AT-rich region was altered.

82 Eighteen putative DnaA boxes with several AT-rich regions were identified in the dnaA-dnaE interge

83 l proteins that act cooperatively to melt an AT-rich region where the replicative helicase is loaded

84 performance of sequencing and PCR primers in AT-rich regions, where short primers yield poor sequenci

85 hree times higher in GC-rich regions than in AT-rich regions, while the opposite is true for LINE1.

86 site is mapped to a sequence embedded in an AT-rich region with four scattered GC base pairs (bps) (

87 ound that ERVs integrate in late-replicating AT-rich regions with abundant microsatellites, mirror re

88 fied TEF protein can specifically bind to an AT-rich region within the core of the telokin promoter.