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1 hway requiring the cell membrane transporter ATP-binding cassette transporter A1.
2 x and levels of the cholesterol transporters ATP binding cassette transporter A1 (ABCA1) and ABCG1.
3 oER2-mediated signaling on the expression of ATP binding cassette transporter A1 (ABCA1) and choleste
4 chain positively regulates the expression of ATP binding cassette transporter A1 (ABCA1) and of neutr
8 olesterol reservoir and abundantly expresses ATP binding cassette transporter A1 (ABCA1), a key chole
9 f the major cellular cholesterol transporter ATP binding cassette transporter A1 (ABCA1), suggesting
10 ligands for both receptor subtypes activate ATP binding cassette transporter A1 (ABCA1)-mediated cho
11 f serum to decrease ACAT activity depends on ATP binding cassette transporter A1 (ABCA1)-mediated eff
13 LXR agonism upregulates the genes encoding ATP binding cassette transporters A1 (ABCA1) and G1 (ABC
14 erbated by down-modulation and impairment of ATP-binding cassette transporter A1 (ABCA1) activity by
15 Genetic deletion of the sterol transporters ATP-binding cassette transporter A1 (ABCA1) and low-dens
16 tivation resulted in increased levels of the ATP-binding cassette transporter A1 (ABCA1) and stearoyl
17 ty acid homeostasis, including the genes for ATP-binding cassette transporter A1 (ABCA1) and sterol r
18 levels associated with increased enterocyte ATP-binding cassette transporter A1 (Abca1) expression a
19 ell lipid:apoAI ratio due to either elevated ATP-binding cassette transporter A1 (ABCA1) expression a
20 obtained in HEK293 cells transfected with an ATP-binding cassette transporter A1 (ABCA1) expression p
21 -secretase 1 (BACE1) and an up-regulation of ATP-binding cassette transporter A1 (ABCA1) expression,
22 Recent studies have demonstrated that the ATP-binding cassette transporter A1 (ABCA1) facilitates
24 ess lower levels of the cholesterol exporter ATP-binding cassette transporter A1 (ABCA1) in compariso
25 omal triglyceride transfer protein (MTP) and ATP-binding cassette transporter A1 (ABCA1) in these pat
26 port proteins cholesterol 27-hydroxylase and ATP-binding cassette transporter A1 (ABCA1) in THP-1 cel
32 lipoprotein (HDL) particles by the action of ATP-binding cassette transporter A1 (ABCA1) is not defin
39 diated via liver-X receptor alpha (LXRalpha)/ATP-binding cassette transporter A1 (ABCA1) pathway, as
40 of HDL to remove cellular cholesterol by the ATP-binding cassette transporter A1 (ABCA1) pathway.
41 promote cholesterol efflux from cells by the ATP-binding cassette transporter A1 (ABCA1) pathway.
46 ations in the cholesterol efflux transporter ATP-binding cassette transporter A1 (ABCA1) result in im
47 The effect was attributed primarily to the ATP-binding cassette transporter A1 (ABCA1) transcriptio
50 Ang II inhibits macrophage expression of the ATP-binding cassette transporter A1 (ABCA1), a key regul
51 nd very low HDL, resulting from mutations in ATP-binding cassette transporter A1 (ABCA1), an integral
56 e expression of the cholesterol transporter, ATP-binding cassette transporter A1 (ABCA1), was 15- to
57 f cellular cholesterol efflux, including the ATP-binding cassette transporter A1 (ABCA1), which media
58 poA1 Trp72 in MPO-mediated inhibition of the ATP-binding cassette transporter A1 (ABCA1)-dependent ch
59 d-free apoA-I had normal capacity to promote ATP-binding cassette transporter A1 (ABCA1)-dependent ch
60 -containing nascent HDL particles created by ATP-binding cassette transporter A1 (ABCA1)-mediated eff
65 udies of Tangier disease have shown that the ATP-binding cassette transporter A1 (ABCA1)/apolipoprote
66 liver X receptor-responsive genes including ATP-binding cassette transporters A1 (ABCA1) and G1 (ABC
68 ent studies have implicated mutations in the ATP-binding cassette transporter A1, ABCA1, as a cause o
69 he capacity of the mutant protein to promote ATP-binding cassette transporter A1- (ABCA1-) mediated c
73 f the reverse cholesterol transport factors, ATP-binding cassette transporter A1 and high-density lip
75 cardiovascular disease candidate genes (i.e. ATP-binding cassette transporters A1 and C6, peroxisome
77 eveloped mice with efficient deletion of the ATP-binding cassette transporters A1 and G1 (ABCA1 and A
78 ect physical interactions between apoA-I and ATP-binding cassette transporter A1, and can explain Tan
79 ed expression of LXR target genes, including ATP-binding cassette transporter A1, and increased apoli
80 important in HDL metabolism, including human ATP-binding cassette transporter A1, apolipoprotein A-I,
81 pholipid transfer protein) and cell factors (ATP-binding cassette transporter A1, ATP-binding cassett
82 nce of scavenger receptor class B type I and ATP Binding Cassette Transporter A1, but not the ATP Bin
83 e ability of lipid-free apoA-I to facilitate ATP-binding cassette transporter A1 cholesterol transpor
84 roid receptor element-binding protein 1c and ATP binding cassette transporter A1, demonstrating HSL-d
88 lly, miR-223 was found to indirectly promote ATP-binding cassette transporter A1 expression (mRNA and
89 ears that unsaturated fatty acids can reduce ATP-binding cassette transporter A1 gene activity by enh
91 99 with the identification of defects in the ATP-binding cassette transporter A1 gene as the cause of
92 ut mice supports the concept that increasing ATP-binding cassette transporter A1 gene expression may
93 vidence from and studies to suggest that the ATP-binding cassette transporter A1 gene regulates intes
94 aining nascent HDL particles produced by the ATP binding cassette transporter A1 have different sizes
96 dual mechanism that required a reduction in ATP-binding cassette transporter A1-mediated (ABCA1-medi
97 gs suggest that niacin by increasing hepatic ATP-binding cassette transporter A1-mediated apoAI lipid
98 nt stimulation of the liver X receptor alpha-ATP-binding cassette transporter A1-mediated cholesterol
99 amellar phospholipid liposomes and promoting ATP-binding cassette transporter A1-mediated cholesterol
100 plets or shuttled to the plasma membrane for ATP-binding cassette transporter A1-mediated efflux.
101 he central helices of apoA-I, participate in ATP-binding cassette transporter A1-mediated lipid efflu
102 or-activated receptor-gamma/liver X receptor/ATP-binding cassette transporter A1 pathway in macrophag
106 creased scavenger receptor B1, and unchanged ATP-binding cassette transporter A1 protein expression i
107 was mediated through up-regulation of ABCA1 (ATP-binding cassette transporter A1) protein expression.
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