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1 ion by binding to substrate proteins with an ATP-regulated affinity that relies on allosteric couplin
2 aK acts as a molecular chaperone through its ATP-regulated binding and release of polypeptide substra
3 FTR) is a cAMP-dependent protein kinase- and ATP-regulated chloride channel, the activity of which de
4 s a cAMP-dependent protein kinase (PKA)- and ATP-regulated chloride channel, whose gating process inv
5 utside of the IFT complex and demonstrate an ATP-regulated co-immunoprecipitation of heterotrimeric k
6 uman homolog hRAD51, into an operational ADP/ATP-regulated DNA-protein (nucleoprotein) filament is es
7  recognition complex (ORC) is a six-subunit, ATP-regulated, DNA binding protein that is required for
8 ntical to its closest relative, Kir1.3 (Kir1-ATP-regulated inward rectifier K+ [ROMK] family) and dis
9 Glucose metabolism mediates GSIS in part via ATP-regulated K+ (KATP) channels, but multiple lines of
10    Instead, we find robust expression of the ATP-regulated K+ channel at early and late states of emb
11                                              ATP-regulated (K(ATP)) channels are formed by an inward
12 hila homolog of CASK/Lin-2, associates in an ATP-regulated manner with CaMKII to catalyze formation o
13 s between its two seven-membered rings in an ATP-regulated manner.
14 We propose that RNA export complexes have an ATP-regulated mechanism for release from binding sites a
15 MMR that relies on two dynamic and redundant ATP-regulated molecular switches.
16 urement of "charge flux" to characterize the ATP-regulated multiconductance nature of Hsc70, which en
17 ce of a protein docking motif on AGR2 and an ATP-regulated peptide-binding activity for Reptin.
18                                              ATP-regulated potassium (KATP) channel complexes of inwa
19 he MPC is an essential regulator of both the ATP-regulated potassium (KATP) channel-dependent and -in
20             In contrast, rose bengal and the ATP-regulated potassium channel antagonist glyburide inc
21 sitive Na-K-2Cl cotransporter (NKCC2) or the ATP-regulated potassium channel ROMK (KCNJI) have been i
22 te DNA end that MRE11-RAD50 can resect in an ATP-regulated reaction, to produce a 3'-hydroxyl that ca
23 th increased capacities for pluripotency and ATP-regulated release of IL-33.

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