ライフサイエンスコーパス: ATP-sensitive

1 noylaminoethyl methanethiosulfonate) to show ATP-sensitive accessibility of cysteine mutants at the h

2 d to at least three different channel types: ATP-sensitive, Ca(2+)-regulated and voltage-dependent K(

3 on of GIP, GLP-1 and PYY was sensitive to K+(ATP)-sensitive channel modulators tolbutamide and diazox

4 ane depolarisation through the closure of K+(ATP)-sensitive channels.

5 dually released into a smaller (600-800 kDa) ATP-sensitive complex.

6 e that the variant-based channel can form an ATP-sensitive conductance and may contribute to cardiopr

7 We suggest that CNPY3 interacts with the ATP-sensitive conformation of gp96 to promote substrate

8 lowed surface expression and detection of an ATP-sensitive current when coexpressed with Kir6.2.

9 RNA-mediated knockdown of ROMK inhibits the ATP-sensitive, diazoxide-activated component of mitochon

10 d CARD-deleted MDA5 constructs assemble into ATP-sensitive filaments.

11 We demonstrate the ATP-sensitive interaction of the cofilin phosphatase chr

12 ization and vasorelaxation by activating the ATP-sensitive, intermediate conductance and small conduc

13 ports that chloroquine inhibition of cardiac ATP-sensitive inward rectifier K(+) current (I(KATP)) is

14 sing on membrane proteins, we identified the ATP-sensitive inward rectifying potassium channel KIR4.1

15 tion of the bee antiviral immune response by ATP-sensitive inwardly rectifying potassium (KATP) chann

16 < 0.05; 0.03 < P(a) < 0.08), as well as with ATP-sensitive inwardly rectifying potassium channel subu

17 P2X7 receptors (Rs) constitute a subclass of ATP-sensitive ionotropic receptors (P2X1-P2X7).

18 strated that opening adenosine triphosphate (ATP)-sensitive K (KATP) channels or activation of delta-

19 up, which binds to adenosine triphosphatase (ATP)-sensitive K(+) (K(ATP)) channels for insulin secret

20 tric oxide (NO), and adenosine triphosphate (ATP)-sensitive K(+) (K(ATP)) channels in adenosine-induc

21 ATP-sensitive K (K(ATP)) channels are composed of Kir6,

22 dent I(Ca,L) inactivation, combined with the ATP-sensitive K current agonist pinacidil or I(Ca,L) blo

23 Compromised ATP-sensitive K(+) (K(ATP)) channel function renders the

24 ommonly caused by mutations in the beta-cell ATP-sensitive K(+) (K(ATP)) channel genes.

25 The vascular ATP-sensitive K(+) (K(ATP)) channel is targeted by a var

26 Mutations in the pore-forming subunit of the ATP-sensitive K(+) (K(ATP)) channel Kir6.2 cause neonata

27 itus (NDM) can be caused by gain-of-function ATP-sensitive K(+) (K(ATP)) channel mutations.

28 The ATP-sensitive K(+) (K(ATP)) channel opener diazoxide or

29 Mutations in the pancreatic ATP-sensitive K(+) (K(ATP)) channel proteins sulfonylure

30 Increased sarcolemmal ATP-sensitive K(+) (K(ATP)) channel subunit protein expr

31 Comparatively, we examined islets from ATP-sensitive K(+) (K(ATP)) channel-deficient SUR1(-/-)

32 sceptibility is attributable to the vascular ATP-sensitive K(+) (K(ATP)) channel.

33 Pancreatic beta-cell ATP-sensitive K(+) (K(ATP)) channels are composed of Kir

34 ATP-sensitive K(+) (K(ATP)) channels couple cell metabol

35 tor 1 (SUR1) and Kir6.2 subunits, which form ATP-sensitive K(+) (K(ATP)) channels in beta-cells.

36 and the subsequent activation of SUR1/Kir6.2 ATP-sensitive K(+) (K(ATP)) channels inhibit hepatic glu

37 ATP-sensitive K(+) (K(ATP)) channels modulate their acti

38 ATP-sensitive K(+) (K(ATP)) channels, comprised of pore-

39 We evaluated the role of ATP-sensitive K(+) (K(ATP)) channels, somatostatin, and

40 abolic-sensing, cardioprotective sarcolemmal ATP-sensitive K(+) (K(ATP)) channels.

41 ts evoked by NMDA are greatly potentiated by ATP-sensitive K(+) (K-ATP) channel blocking agents in ST

42 transmitter release by activating inhibitory ATP-sensitive K(+) (KATP ) channels, as well as a class

43 ells with RNA interference inhibits SOCE and ATP-sensitive K(+) (KATP) channel activation.

44 lfonylurea receptor 2 (SUR2) subunits of the ATP-sensitive K(+) (KATP) channel as well as two mutatio

45 The opening of sarcolemmal and mitochondrial ATP-sensitive K(+) (KATP) channels in the heart is belie

46 ptide secretion also required the closing of ATP-sensitive K(+) (KATP) channels, as the KATP channel

47 report a novel target of the drug memantine, ATP-sensitive K(+) (KATP) channels, potentially relevant

48 The mitochondrial ATP-sensitive K(+) (mitoK(ATP)) channel plays a central

49 Ba(2+)) nor inhibitors of the mitochondrial ATP-sensitive K(+) channel (5-hydroxydecanoate and glibe

50 cate that this defect lies downstream of the ATP-sensitive K(+) channel (K(ATP) channel) and calcium

51 Mutations in the pancreatic ATP-sensitive K(+) channel (K(ATP) channel) cause perman

52 with inactivating mutations of the beta-cell ATP-sensitive K(+) channel (K(ATP) channel) genes ABCC8

53 he E23K variant in the Kir6.2 subunit of the ATP-sensitive K(+) channel (K(ATP) channel) is associate

54 determine whether closure of the alpha-cell ATP-sensitive K(+) channel (K(ATP) channel) is the mecha

55 eas and most commonly results from recessive ATP-sensitive K(+) channel (K(ATP) channel) mutations.

56 associated with decreased expression of the ATP-sensitive K(+) channel (K(ATP) channel) sulfonylurea

57 Glucose-excited neurons utilize the ATP-sensitive K(+) channel (K(ATP) channel) to sense glu

58 ne (KCNJ11), the pore-forming subunit of the ATP-sensitive K(+) channel (K(ATP) channel), are a commo

59 ne (KCNJ11), the pore-forming subunit of the ATP-sensitive K(+) channel (K(ATP) channel), cause neona

60 )1, the regulatory subunit of the pancreatic ATP-sensitive K(+) channel (K(ATP) channel).

61 The ATP-sensitive K(+) channel (K(ATP)) controls insulin sec

62 Perfusion with the ATP-sensitive K(+) channel (K(ATP)) inhibitor glibenclam

63 Mutations to the ATP-sensitive K(+) channel (KATP channel) that reduce th

64 tal diabetes (ND) caused by mutations in the ATP-sensitive K(+) channel (KATP channel).

65 ial inhibition of insulin secretion with the ATP-sensitive K(+) channel agonist (opener) diazoxide, c

66 Finally, the ATP-sensitive K(+) channel agonist diazoxide (200 microm

67 e Kir6.2 and SUR1 subunits of the pancreatic ATP-sensitive K(+) channel are the most common cause of

68 glucose levels rise, and most use GK and an ATP-sensitive K(+) channel as the ultimate effector of g

69 ility transition pore, and the mitochondrial ATP-sensitive K(+) channel did not change the NADH effec

70 from diagnosis and were diagnosed later than ATP-sensitive K(+) channel mutation carriers (11 vs. 8 w

71 ts of the ruthenium complexes suggested that ATP-sensitive K(+) channel pathways were not involved be

72 perpolarization, indicating activation of an ATP-sensitive K(+) channel via a PI3 kinase-dependent me

73 The Kir6.2 subunit mRNA of the ATP-sensitive K(+) channel was expressed in 42% of GE an

74 ember 6.2), which encode the subunits of the ATP-sensitive K(+) channel, and RNA in situ hybridizatio

75 t encode the Kir6.2 subunit of the beta-cell ATP-sensitive K(+) channel.

76 PKC epsilon in addition to the mitochondrial ATP-sensitive K(+) channel.

77 s are uncertain but may involve cell surface ATP-sensitive K(+) channels (K(ATP) channels) analogous

78 In glucose-sensing neurons, ATP-sensitive K(+) channels (K(ATP) channels) are though

79 ATP-sensitive K(+) channels (K(ATP) channels) couple bet

80 ATP-sensitive K(+) channels (K(ATP) channels) couple cel

81 We also examined the role of hypothalamic ATP-sensitive K(+) channels (K(ATP) channels) in the eff

82 es to hypoglycemia through the modulation of ATP-sensitive K(+) channels (K(ATP) channels) in the ven

83 ATP-sensitive K(+) channels (K(ATP) channels) link gluco

84 cytoplasmic [ATP]/[ADP], causing closure of ATP-sensitive K(+) channels (K(ATP) channels), Ca(2+) en

85 sulfonylurea receptor, the stress-responsive ATP-sensitive K(+) channels (K(ATP) channels), with thei

86 e the cAMP-dependent inhibition of beta-cell ATP-sensitive K(+) channels (K(ATP)) was provided by one

87 an alter the functional expression of cloned ATP-sensitive K(+) channels (K(ATP)).

88 the effect, indicating that H2S acts through ATP-sensitive K(+) channels and nitric oxide synthesis.

89 ilation was insensitive to the inhibitors of ATP-sensitive K(+) channels and voltage-gated K(+) chann

90 Vascular ATP-sensitive K(+) channels are activated by multiple va

91 ATP-sensitive K(+) channels are gated by intracellular A

92 Vascular ATP-sensitive K(+) channels are inhibited by multiple va

93 elected mutant, we examine the regulation of ATP-sensitive K(+) channels via a G(q/11)-coupled recept

94 In addition to closure of the ATP-sensitive K(+) channels with mitochondrial ATP synth

95 tly coupled to the activation of sarcolemmal ATP-sensitive K(+) channels, hastening action potential

96 generated from glucose is assumed to inhibit ATP-sensitive K(+) channels, leading to the depolarizati

97 calcium exchanger, L-type calcium channels, ATP-sensitive K(+) channels, or [Ca(2+)](m) uniporter.

98 mitochondrial proteins such as mitochondrial ATP-sensitive K(+) channels, the mitochondrial permeabil

99 cretion via their ability to open alpha-cell ATP-sensitive K(+) channels.

100 current, a Ca(2+)-activated K(+) current, an ATP-sensitive K(+) current, a plasma membrane calcium-pu

101 ed by oxidative stress activates sarcolemmal ATP-sensitive K(+) currents to form a metabolic sink.

102 aterality determinant in Xenopus laevis: the ATP-sensitive K(+)-channel (K(ATP)).

103 ATP-sensitive K(+)-channels link metabolism and excitabi

104 variable that controls insulin secretion by ATP-sensitive K(+)-dependent and -independent mechanisms

105 ATP-sensitive K+ (K(ATP)) channels are hetero-octamers o

106 ATP-sensitive K+ (K(ATP)) channels are present in the sa

107 ATP-sensitive K+ (K(ATP)) channels link metabolic state

108 racellular Ca2+, phospholipase A2 (PLA2) and ATP-sensitive K+ (KATP) channel activation whereas A2A-m

109 rachidonic acid (AA), are potent sarcolemmal ATP-sensitive K+ (KATP) channel activators.

110 ften caused by inactivating mutations of the ATP-sensitive K+ (KATP) channel in the pancreatic beta c

111 ATP-sensitive K+ (KATP) channel openers are vasodilators

112 Kir6.2 and SUR1 subunits of the plasmalemmal ATP-sensitive K+ (KATP) channel.

113 nti-Kir6.2 antibodies, indicating absence of ATP-sensitive K+ (KATP) channels.

114 the sulfonylurea glibenclamide, implicating ATP-sensitive K+ (KATP) channels; however, tissue ATP wa

115 were examined on membrane potential and the ATP-sensitive K+ channel (K ATP) in INS 832/13 cells.

116 ion on chromosome 6q24, and 14 patients with ATP-sensitive K+ channel (K(ATP) channel) gene mutations

117 The ATP-sensitive K+ channel (KATP channel) couples glucose

118 ntly increased their APF and decreased their ATP-sensitive K+ channel (KATP channel) currents as extr

119 ardioprotective signal and the mitochondrial ATP-sensitive K+ channel (mitoK(ATP)) plays a crucial ro

120 rmacological modulation of the mitochondrial ATP-sensitive K+ channel (mitoKATP) sensitive to diazoxi

121 ts were reversed by a specific mitochondrial ATP-sensitive K+ channel inhibitor, 5-hydroxydecanoate,

122 ectifier subunits Kir 6.1 and Kir 6.2 of the ATP-sensitive K+ channel of the plasma membrane (cellKAT

123 arious types of cells with the mitochondrial ATP-sensitive K+ channel opener, diazoxide, precondition

124 show that tolbutamide, an antagonist of the ATP-sensitive K+ channel, allows these oscillations to t

125 efect either at the level or upstream of the ATP-sensitive K+ channel.

126 ATP-sensitive K+ channels (K(ATP) channels) control elec

127 Glyburide, an inhibitor of ATP-sensitive K+ channels (K(ATP) channels), provided pa

128 via Epac1 and/or Epac2 to inhibit beta-cell ATP-sensitive K+ channels (K(ATP) channels; a hetero-oct

129 ATP-sensitive K+ channels (K(ATP)) contribute to vasomot

130 iding novel insight into the architecture of ATP-sensitive K+ channels (KATP channels) (KIR6.0/SUR)4.

131 Metabolic-sensing ATP-sensitive K+ channels (KATP channels) adjust membran

132 In many cells, ATP-sensitive K+ channels (KATP channels) couple metabol

133 Closure of ATP-sensitive K+ channels (KATP channels) is a key step

134 nty years after the discovery of sarcolemmal ATP-sensitive K+ channels and 12 years after the discove

135 The activation of ATP-sensitive K+ channels by protein kinase A in vascula

136 ATP-sensitive K+ channels composed of the pore-forming p

137 d glucose trigger insulin release by closing ATP-sensitive K+ channels, depolarizing beta cells, and

138 These observations implicate the ANT in ATP-sensitive K+ transport in brain mitochondria.

139 een the degree of coupling and the extent of ATP-sensitive K+-channel activation and illustrates an e

140 protein 1 or mABC1) displayed mitochondrial ATP-sensitive K+-channel activity.

141 ATP-sensitive K+-channels are not involved in the mechan

142 t link ligand binding to the channel gate in ATP-sensitive Kir6.2 channels.

143 The P2X7 receptor is an ATP-sensitive ligand-gated cation channel, expressed pre

144 analysis showed short periods (</=0.5 s) of ATP-sensitive linear motion.

145 They commonly bind actin in an ATP-sensitive manner, exhibit actin-activated ATPase act

146 promoting PP1alpha-MEK1/2 interaction in an ATP-sensitive manner.

147 , like Myo2p, cosediments with F-actin in an ATP-sensitive manner.

148 This suggests that ATP-sensitive metabotropic (P2Y) receptors play a role i

149 viously shown that Ca(2+) directly activates ATP-sensitive microtubule binding by a Chlamydomonas out

150 de of oxidative phosphorylation may activate ATP sensitive mitochondrial potassium (mitoK(ATP)) chann

151 itro-L-arginine methyl ester (L-NAME) or the ATP-sensitive mitochondrial potassium channel (mitoKATP)

152 y chain exhibits a dominant Ca2+-independent ATP-sensitive MT binding activity in vitro that is inhib

153 Here, we identify an endolysosomal ATP-sensitive Na(+) channel (lysoNa(ATP)).

154 ated pressor response, and (2) activation of ATP-sensitive P2X receptors enhances the pressor respons

155 These results demonstrate that the ATP-sensitive P2X(7) receptor regulates fluid secretion

156 Recently, ATP-sensitive P2Y purinergic receptors have emerged as d

157 The mitochondrial ATP sensitive potassium channel (mK(ATP)) is implicated

158 by the putative blocker of the mitochondrial ATP sensitive potassium channel, 5-hydroxydecanoate, bef

159 ich in that dose is a selective inhibitor of ATP sensitive potassium channels (K(ATP)).

160 tion of macrophage polarization by targeting ATP sensitive potassium channels (KATP).

161 ), and the selective adenosine triphosphate (ATP)-sensitive potassium (K(ATP)) channel blocker gliben

162 rming subunit of the adenosine triphosphate (ATP)-sensitive potassium (KATP) channel, cause neonatal

163 ads to activation of adenosine triphosphate (ATP)-sensitive potassium channels.

164 s the majority of LHA MC4R-GFP neurons in an ATP- sensitive potassium channel-dependent manner.

165 Potentiation of ATP-sensitive potassium (K(ATP)) and inhibition of calci

166 signaling in POMC neurons that elevates the ATP-sensitive potassium (K(ATP)) channel activity cell-a

167 resent investigation we examined the role of ATP-sensitive potassium (K(ATP)) channel activity in mod

168 The regulation of ATP-sensitive potassium (K(ATP)) channel activity is com

169 responding to cytoplasmic nucleotide levels, ATP-sensitive potassium (K(ATP)) channel activity provid

170 loss of function mutations of the beta-cell ATP-sensitive potassium (K(ATP)) channel can develop hyp

171 The beta-cell ATP-sensitive potassium (K(ATP)) channel composed of sul

172 The ATP-sensitive potassium (K(ATP)) channel controls insuli

173 The isoform-specific structure of the ATP-sensitive potassium (K(ATP)) channel endows it with

174 Furthermore, leptin indirectly activated an ATP-sensitive potassium (K(ATP)) channel in OX neurons,

175 The pancreatic beta-cell ATP-sensitive potassium (K(ATP)) channel is a multimeric

176 The ATP-sensitive potassium (K(ATP)) channel links cell meta

177 nitric oxide (NO) donor nitroprusside or the ATP-sensitive potassium (K(ATP)) channel opener cromakal

178 Although ATP-sensitive potassium (K(ATP)) channel openers, e.g.,

179 ght involve a NO-cyclic GMP-protein kinase G-ATP-sensitive potassium (K(ATP)) channel pathway.

180 tivating mutations in the genes encoding the ATP-sensitive potassium (K(ATP)) channel subunits Kir6.2

181 irpin RNA (shRNA) inhibited the hypothalamic ATP-sensitive potassium (K(ATP)) channel with glibenclam

182 Kcnj11 (Kir6.2; subunit of ATP-sensitive potassium (K(ATP)) channel) was significan

183 Abcc8 (Sur1; subunit of ATP-sensitive potassium (K(ATP)) channel) was significan

184 The pancreatic ATP-sensitive potassium (K(ATP)) channel, a complex of f

185 ding Kir6.2, the pore-forming subunit of the ATP-sensitive potassium (K(ATP)) channel, are the most c

186 odes Kir6.2, the pore-forming subunit of the ATP-sensitive potassium (K(ATP)) channel, cause permanen

187 The ATP-sensitive potassium (K(ATP)) channel, composed of th

188 r6.2 pore-forming subunit of the sarcolemmal ATP-sensitive potassium (K(ATP)) channel, predisposed to

189 el Kir6.2 is the pore-forming subunit of the ATP-sensitive potassium (K(ATP)) channel, which controls

190 d the mechanism of chloroquine inhibition of ATP-sensitive potassium (K(ATP)) channels (Kir6.2/SUR2A)

191 ATP-sensitive potassium (K(ATP)) channels activate under

192 ATP-sensitive potassium (K(ATP)) channels are critical f

193 ng gated by high-energy nucleotides, cardiac ATP-sensitive potassium (K(ATP)) channels are exquisitel

194 Cardiac ATP-sensitive potassium (K(ATP)) channels are key sensor

195 ATP-sensitive potassium (K(ATP)) channels composed of su

196 ATP-sensitive potassium (K(ATP)) channels comprise four

197 ATP-sensitive potassium (K(ATP)) channels comprise Kir6.

198 ATP-sensitive potassium (K(ATP)) channels conduct potass

199 ATP-sensitive potassium (K(ATP)) channels couple cell me

200 conducted to examine the role of myocardial ATP-sensitive potassium (K(ATP)) channels in exercise-in

201 ulation of EGP by activation of hypothalamic ATP-sensitive potassium (K(ATP)) channels in rodents, wh

202 Here we show that activation of ATP-sensitive potassium (K(ATP)) channels in the medioba

203 The activity of ATP-sensitive potassium (K(ATP)) channels is governed by

204 Ventricular ATP-sensitive potassium (K(ATP)) channels link intracell

205 ATP-sensitive potassium (K(ATP)) channels mediate glucos

206 Inward rectifying, ATP-sensitive potassium (K(ATP)) channels mediated the r

207 ATP-sensitive potassium (K(ATP)) channels regulate insul

208 ATP-sensitive potassium (K(ATP)) channels regulate insul

209 requires high-fidelity metabolic sensing by ATP-sensitive potassium (K(ATP)) channels that adjust me

210 The BTB overexpresses ATP-sensitive potassium (K(ATP)) channels that are barel

211 les with sulfonylurea receptor 1 to form the ATP-sensitive potassium (K(ATP)) channels that regulate

212 mate via H(2)O(2) signaling, which activates ATP-sensitive potassium (K(ATP)) channels to inhibit dop

213 We investigated the participation of ATP-sensitive potassium (K(ATP)) channels, adenosine A1

214 (ROS), coupled to the opening of sarcolemmal ATP-sensitive potassium (K(ATP)) channels, contributes t

215 ATP-sensitive potassium (K(ATP)) channels, so named beca

216 ely 70% of the beta-cells have nonfunctional ATP-sensitive potassium (K(ATP)) channels, whereas the r

217 milar drugs also stimulate hair, implicating ATP-sensitive potassium (K(ATP)) channels.

218 neurons is driven by ATP-mediated closure of ATP-sensitive potassium (K(ATP)) channels.

219 e decrease in SNr firing was not mediated by ATP-sensitive potassium (KATP) channel activity, but if

220 ess, the ATP/ADP ratio increases, leading to ATP-sensitive potassium (KATP) channel closure, which in

221 The beta-cell ATP-sensitive potassium (KATP) channel controls insulin

222 sulphonylurea receptor (SUR1) subunit of the ATP-sensitive potassium (KATP) channel is a member of th

223 Similarly, antihypertensive ATP-sensitive potassium (KATP) channel openers (KCOs) ac

224 ATP-sensitive potassium (KATP) channel openers have emer

225 that Epac exists in a complex with vascular ATP-sensitive potassium (KATP) channel subunits and that

226 mutation, W68R, in the Kir6.2 subunit of the ATP-sensitive potassium (KATP) channel, in a patient wit

227 ggering of insulin secretion mediated by the ATP-sensitive potassium (KATP) channel, was decreased in

228 The ATP-sensitive potassium (KATP) channels are crucial for

229 ATP-sensitive potassium (KATP) channels are heteromultim

230 ATP-sensitive potassium (KATP) channels are widely expre

231 carbamazepine, correct biogenesis defects in ATP-sensitive potassium (KATP) channels composed of sulf

232 ATP-sensitive potassium (KATP) channels comprise four po

233 ATP-sensitive potassium (KATP) channels consisting of su

234 ATP-sensitive potassium (KATP) channels couple cell meta

235 ATP-sensitive potassium (KATP) channels couple the metab

236 In the absence of intracellular nucleotides, ATP-sensitive potassium (KATP) channels exhibit spontane

237 while simultaneously recording currents from ATP-sensitive potassium (KATP) channels in single cells,

238 r 2B (SUR2B) forms the regulatory subunit of ATP-sensitive potassium (KATP) channels in vascular smoo

239 ATP-sensitive potassium (KATP) channels may be involved

240 (NO) synthase, soluble guanylyl cyclase, and ATP-sensitive potassium (KATP) channels nearly abolished

241 ATP-sensitive potassium (KATP) channels play a key role

242 ATP-sensitive potassium (KATP) channels play a prominent

243 sarcolemmal (sarc) and mitochondrial (mito) ATP-sensitive potassium (KATP) channels play crucial rol

244 Because activation of central ATP-sensitive potassium (KATP) channels suppresses EGP i

245 Insulin secretion is under the control of ATP-sensitive potassium (KATP) channels that play key ro

246 ATP-sensitive potassium (KATP) channels were first disco

247 ATP-sensitive potassium (KATP) channels within the hypot

248 nes encoding the Kir6.1 and SUR2 subunits of ATP-sensitive potassium (KATP) channels, respectively.

249 Through their actions on ATP-sensitive potassium (KATP) channels, sulfonylureas b

250 Central signals activate ATP-sensitive potassium (KATP) channels, thereby down-re

251 by leptin mirror those reported recently for ATP-sensitive potassium (KATP) channels, which are criti

252 tabolism of glucose, leading to a closure of ATP-sensitive potassium (KATP) channels.

253 The mitochondrial ATP-sensitive potassium (mitoK(ATP)) channel opener diaz

254 acking the Kir6.2 subunit of the sarcolemmal ATP-sensitive potassium (sK(ATP)) channel after exposure

255 a protein that combines with SUR2 to form an ATP-sensitive potassium channel (K(ATP)) expressed in co

256 The ATP-sensitive potassium channel (K(ATP)) in mouse coloni

257 We have previously shown that an ATP-sensitive potassium channel (K(ATP)) is expressed in

258 ATP-sensitive potassium channel (K(ATP)) openers target

259 odes Kir6.2, the pore-forming subunit of the ATP-sensitive potassium channel (K(ATP)), are the common

260 e perfused with Ringer solution (control), a ATP-sensitive potassium channel (KATP ) inhibitor, an in

261 ABSTRACT: Sarcolemmal ATP-sensitive potassium channel (KATP channel) activatio

262 ells were compared to those of the reference ATP-sensitive potassium channel (KATP channel) openers d

263 ; encoded by ABCC8) and its associated islet ATP-sensitive potassium channel (Kir6.2; encoded by KCNJ

264 ation and preconditioning-like mitochondrial ATP-sensitive potassium channel activation.

265 conditioning with diazoxide, a mitochondrial ATP-sensitive potassium channel agonist, prevented dendr

266 wnstream glucokinase effectors revealed that ATP-sensitive potassium channel and P/Q calcium channel

267 h DZX, supporting a role for a mitochondrial ATP-sensitive potassium channel in the mechanism of card

268 isolation buffer, cardioplegia (CPG)+/-DZX+/-ATP-sensitive potassium channel inhibitor, 5-hydroxydeca

269 preconditioning induced by the mitochondrial ATP-sensitive potassium channel opener BMS-191095.

270 response to stress that is prevented by the ATP-sensitive potassium channel opener, diazoxide (DZX)

271 by covalently modifying (sulfhydrating) the ATP-sensitive potassium channel, as mutating the site of

272 ein kinase G-inhibitor) or glibenclamide (an ATP-sensitive potassium channel-inhibitor) all led to an

273 l, betaIV-spectrin targets ankyrin-B and the ATP-sensitive potassium channel.

274 ATP-sensitive potassium channels (K(ATP) channels) are i

275 Opening of cardiac ATP-sensitive potassium channels (K(ATP) channels) is a

276 ATP-sensitive potassium channels (K(ATP) channels) of ar

277 Sarcolemmal ATP-sensitive potassium channels (K(ATP)) act as metabol

278 IPC may involve activation of ATP-sensitive potassium channels (K(ATP)).

279 ATP-sensitive potassium channels (K(ATP); Kir6.x) are a

280 Sarcolemmal ATP-sensitive potassium channels (KATP channels) in card

281 axin (Syn)-1A interacts with SUR1 to inhibit ATP-sensitive potassium channels (KATP channels).

282 In the vasculature, ATP-sensitive potassium channels (KATP) channels regulat

283 is the prototypical opener of mitochondrial ATP-sensitive potassium channels (mitoK(ATP)) and protec

284 we demonstrated that targeting mitochondrial ATP-sensitive potassium channels (mitoK(ATP)) protects n

285 lucose cotransporter SGLT1, or by closure of ATP-sensitive potassium channels after glucose metabolis

286 gers membrane depolarization both by closing ATP-sensitive potassium channels and because of its upta

287 version to lactate, leading to activation of ATP-sensitive potassium channels and to decreased hepati

288 nhibitor), 5-hydroxydecanoate (mitochondrial ATP-sensitive potassium channels inhibitor), or glibencl

289 tassium channel (Ir), proteins that comprise ATP-sensitive potassium channels regulating hormone secr

290 and unclear and may involve Akt activation, ATP-sensitive potassium channels, and nitric oxide, amon

291 lly identified as an endogenous regulator of ATP-sensitive potassium channels.

292 cium-dependent potassium channels and not by ATP-sensitive potassium channels.

293 parallel with the acquisition of functional ATP-sensitive potassium channels.

294 inhibition of glycolysis, and activation of ATP-sensitive potassium channels.

295 2)), can be antagonized by activators of the ATP-sensitive potassium current (K(ATP)).

296 e composition and signaling of an endogenous ATP-sensitive potassium ion channel (KATP) in HepG2C3A,

297 e composition and signaling of an endogenous ATP-sensitive potassium ion channel.

298 malian SUR genes are associated with K(ATP) (ATP-sensitive potassium) channels, which are involved in

299 s been proposed as a regulator of the 30 pS, ATP-sensitive renal K channel (Kir1.1, also known as ren

300 Myosin's conserved ATP-sensitive tryptophan (AST) is an energy transduction