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1 l, betaIV-spectrin targets ankyrin-B and the ATP-sensitive potassium channel.
2 ll bodies of mNTS neurones via effects on an ATP-sensitive potassium channel.
3 eceptors (SURx) are required subunits of the ATP-sensitive potassium channel.
4 inhibition of glycolysis, and activation of ATP-sensitive potassium channels.
5 lly identified as an endogenous regulator of ATP-sensitive potassium channels.
6 cium-dependent potassium channels and not by ATP-sensitive potassium channels.
7 ific cation channels, chloride channels, and ATP-sensitive potassium channels.
8 family, which associate with Kir6.x to form ATP-sensitive potassium channels.
9 mily that is associated with Kir 6.x to form ATP-sensitive potassium channels.
10 bition of insulin secretion by activation of ATP-sensitive potassium channels.
11 parallel with the acquisition of functional ATP-sensitive potassium channels.
12 ough the activation of adenosine pathways or ATP-sensitive potassium channels.
13 ads to activation of adenosine triphosphate (ATP)-sensitive potassium channels.
14 as act by inhibiting adenosine triphosphate (ATP)-sensitive potassium channels.
15 by the putative blocker of the mitochondrial ATP sensitive potassium channel, 5-hydroxydecanoate, bef
17 ned with an inward rectifier Kir6.2 subunit, ATP-sensitive potassium channel activity is generated.
18 lucose cotransporter SGLT1, or by closure of ATP-sensitive potassium channels after glucose metabolis
19 conditioning with diazoxide, a mitochondrial ATP-sensitive potassium channel agonist, prevented dendr
20 wnstream glucokinase effectors revealed that ATP-sensitive potassium channel and P/Q calcium channel
21 gers membrane depolarization both by closing ATP-sensitive potassium channels and because of its upta
22 version to lactate, leading to activation of ATP-sensitive potassium channels and to decreased hepati
23 and unclear and may involve Akt activation, ATP-sensitive potassium channels, and nitric oxide, amon
24 vation of adenosine receptors and opening of ATP-sensitive potassium channels appear to play a role i
25 by covalently modifying (sulfhydrating) the ATP-sensitive potassium channel, as mutating the site of
28 al-ventricle administration of inhibitors of ATP-sensitive potassium channels, but not of antagonists
30 s that activation of adenosine triphosphate (ATP)-sensitive potassium channels contributes to vascula
32 Activation of either protein kinase C or the ATP-sensitive potassium channel has been shown to induce
33 ide, a selective opener of the mitochondrial ATP-sensitive potassium channel, has been shown to elici
34 tegral component of the pancreatic beta-cell ATP-sensitive potassium channel, IKATP, was investigated
35 h DZX, supporting a role for a mitochondrial ATP-sensitive potassium channel in the mechanism of card
37 ntial clinical consequences of inhibition of ATP-sensitive potassium channels in the heart of diabeti
38 These results suggest that the opening of ATP-sensitive potassium channels in vascular smooth musc
39 le sodium salt of glipizide, an inhibitor of ATP-sensitive potassium channels, in anesthetized and aw
41 hannel inhibitor), glibenclamide (5 mumol/L, ATP-sensitive potassium channel inhibitor), and iberioto
42 isolation buffer, cardioplegia (CPG)+/-DZX+/-ATP-sensitive potassium channel inhibitor, 5-hydroxydeca
43 nhibitor), 5-hydroxydecanoate (mitochondrial ATP-sensitive potassium channels inhibitor), or glibencl
44 ein kinase G-inhibitor) or glibenclamide (an ATP-sensitive potassium channel-inhibitor) all led to an
48 a protein that combines with SUR2 to form an ATP-sensitive potassium channel (K(ATP)) expressed in co
53 odes Kir6.2, the pore-forming subunit of the ATP-sensitive potassium channel (K(ATP)), are the common
56 n beta-cells at a density similar to that of ATP-sensitive potassium channels (K(ATP) channels) and e
57 ites on SUR1 that antagonize the inhibition, ATP-sensitive potassium channels (K(ATP) channels) are d
66 eta-cells demonstrated that leptin activated ATP-sensitive potassium channels (K(ATP)) by increasing
73 methylpropanamides 1, were found to activate ATP sensitive potassium channels (KATP) and represent a
75 e perfused with Ringer solution (control), a ATP-sensitive potassium channel (KATP ) inhibitor, an in
77 lming majority of evidence suggests that the ATP-sensitive potassium channel (KATP channel) is an imp
78 ells were compared to those of the reference ATP-sensitive potassium channel (KATP channel) openers d
79 1 revealed that the F1388 mutation abolished ATP-sensitive potassium channel (KATP) activity in intac
81 g of the lead cardiac selective antiischemic ATP-sensitive potassium channel (KATP) opener (4) are de
84 from the patient of origin, lack functional ATP-sensitive potassium channels (KATP) and also carry a
87 ; encoded by ABCC8) and its associated islet ATP-sensitive potassium channel (Kir6.2; encoded by KCNJ
88 is the prototypical opener of mitochondrial ATP-sensitive potassium channels (mitoK(ATP)) and protec
89 we demonstrated that targeting mitochondrial ATP-sensitive potassium channels (mitoK(ATP)) protects n
93 ve cardioplegia based on the hyperpolarizing ATP-sensitive potassium channel opener pinacidil, the pr
95 response to stress that is prevented by the ATP-sensitive potassium channel opener, diazoxide (DZX)
98 tassium channel (Ir), proteins that comprise ATP-sensitive potassium channels regulating hormone secr
99 th the goal of obtaining an activator of the ATP sensitive potassium channel suitable for use in the
103 ancy; the cells therefore lacked operational ATP-sensitive potassium channels, which results in the f
104 malian SUR genes are associated with K(ATP) (ATP-sensitive potassium) channels, which are involved in
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