ライフサイエンスコーパス: ATP

1 ATP binding leads to formation of an occluded state with

2 ATP citrate-lyase produces acetyl-CoA in the nucleus and

3 ATP injection increased expression of several markers fo

4 ATP is also released from sensory-motor nerves during an

5 ATP promotes octamer polymerization, whereas GTP promote

6 ATP synthases produce ATP by rotary catalysis, powered b

7 ATP, but not NAD(+), causes a conformational shift to a

8 ATP, released from perivascular sympathetic nerves, caus

9 ATP-competitive inhibitors against this complex have bee

10 ATP-independent endocytosis became more significant at 3

11 ATP-independent endocytosis primarily involved retrieval

12 Differential role of pannexin-1/ATP/P2X7 axis in IL-1beta release by human monocytes.

13 t cations, thus, in the presence of Mg(2+) , ATP is inadequate as an activator.

14 LR4 agonist LPS in the absence of additional ATP.

15 5-, 3.17- and 2.12-fold increase in ATP:ADP, ATP:AMP and energy charge after portal venous reperfusio

16 ly, other mitochondrial proteins such as ADP/ATP carrier proteins.

17 e nucleotide translocase (ANT) exchanges ADP/ATP through the mitochondrial inner membrane, and Ant2 i

18 application and receptor deactivation after ATP washout, and makes the receptor pore permeable to NM

19 very and uptake in cells resulted in altered ATP levels, decreased glycolytic flux, Nrf-2 and glutath

20 by Rev in a dose-dependent manner, although ATP-independent helicase activity is not.

21 -kDa protein complex LptB2FG is unique among ATP-binding cassette transporters because it extracts li

22 An ATP analogue that reacts with lysine residues inhibited

23 An ATP-independent form of endocytosis was recruited to acc

24 Furthermore, DDX21 is both an ATP-dependent and ATP-independent helicase, and both ATP

25 have isolated from extracts of HeLa cells an ATP-dependent factor that releases Cdc20 from MCC and id

26 o Rico is closely linked to a mutation in an ATP Binding Cassette subfamily C2 (ABCC2) gene that func

27 olds, resulting in nanomolar potencies in an ATP synthesis inhibition assay.

28 hat open and close clamps around ptDNA in an ATP-fueled reaction.

29 promoting PP1alpha-MEK1/2 interaction in an ATP-sensitive manner.

30 The multidrug resistance protein MRP1 is an ATP-binding cassette (ABC) transporter that confers resi

31 ABCG4 is an ATP-binding cassette transmembrane protein which has bee

32 Key to the chaperone activity is an ATP-induced allosteric regulation of polypeptide substra

33 n eIF3j affinity for the 43S PIC requires an ATP-dependent, but unwinding-independent, activity of eI

34 tubule-attached, dimeric kinesin bound to an ATP analog.

35 Eukaryotes metabolize OP via an ATP-dependent 5-oxoprolinase; most prokaryotes lack homo

36 SlRd2 was an ATP-binding protein that formed homodimers in planta.

37 inhibitory concentration (IC50): 3.8muM] and ATP synthesis (IC50: 0.9muM), and additional findings su

38 TP-independent helicase, and both ATPase and ATP-dependent helicase activities are inhibited by Rev i

39 aking them unavailable for actin binding and ATP hydrolysis.

40 c pattern is optimal to maximize biomass and ATP production; it requires the activity of a branched T

41 expression, spare respiratory capacity, and ATP levels.

42 HKs by targeting the conserved catalytic and ATP-binding (CA) domain.

43 SM high-energy phosphate concentrations and ATP flux rates were normal in HFrEF and HFpEF patients.

44 lism (glucose oxidation, O2 consumption, and ATP production), insulin secretion was almost completely

45 n with the kinetics, voltage-dependence, and ATP-dependence of VDP.

46 thermore, DDX21 is both an ATP-dependent and ATP-independent helicase, and both ATPase and ATP-depend

47 ormancy, maintenance of genome integrity and ATP homeostasis were robustly expressed.

48 homeostasis of glycolytic intermediates and ATP during large shifts in glucose supply or demand.

49 atically phosphorylated by NAD(+) kinase and ATP or (tz) ATP to the corresponding N(tz) ADP(+) .

50 Reduced oxygen levels and ATP consumption rates were simulated to characterize met

51 ed loss in oxygen consumption rate (OCR) and ATP production by mitochondria were ameliorated by NTZ i

52 id content, increased lactate production and ATP levels, reduced expression of peroxisome proliferato

53 bition altered mitochondrial respiration and ATP production, an effect that was abrogated by phosphoc

54 of conserved amino acids that couple RNA and ATP binding to the protein (Motif III).

55 echanistic insights of how sulfonylureas and ATP interact with the KATP channel complex to inhibit ch

56 For both XTT and ATP, which reflect the metabolic activity of the cells,

57 Apical ATP release was attenuated in Calhm1 knockout cultures f

58 Key mitochondrial functions such as ATP production, Ca(2+) uptake and release, and substrate

59 and coupled mitochondrial functions such as ATP synthesis by oxidative phosphorylation, Ca(2+) dynam

60 tain the ability to bind nucleotides such as ATP.

61 e regulated by intracellular pH, in part, at ATP-binding site 1 formed by the nucleotide-binding doma

62 4 T cells, which reduced intracellular ATP, [ATP]i.

63 Because ATP is released extracellularly by nerve and other tissu

64 tumor cell death involves interplay between ATP/AMP efflux pathways and different cell-autonomous ec

65 pacity for Hsp90 and Hsc70 complexes to bind ATP and enhances their ATPase activities in vitro.

66 At room temperature, blocking ATP hydrolysis effectively abolished slow endocytosis an

67 However, combined inhibition of both ATP sources abolished evoked transmission.

68 of the PPIP5Ks elevated [ATP] by 35%; both [ATP] and [5-InsP7] were restored to WT levels by overexp

69 terestingly, neither mutant was activated by ATP, and the Km and Hill coefficient of each mutant assa

70 l changes coupled to substrate activation by ATP to perform C-C bond ligation at a distant Mn center.

71 t with increased synaptic efficacy caused by ATP.

72 ammation utilizing TLR4 ligation followed by ATP or nigericin treatment, inflammasome activation is e

73 Multidrug resistance (MDR) mediated by ATP-binding cassette (ABC) transport proteins remains a

74 dation of metabolites is in part mediated by ATP-binding cassette (ABC) transporters.

75 tion of the bee antiviral immune response by ATP-sensitive inwardly rectifying potassium (KATP) chann

76 The subfamily C ATP-binding cassette (ABCC) transporters mediate multidr

77 mic localization is driven by MinD-catalyzed ATP hydrolysis, stimulated by interactions with MinE's a

78 a novel role of FANCD2 in governing cellular ATP production, and advances our understanding of how de

79 iratory inhibition and reduction of cellular ATP.

80 Blue/green reduced cellular ATP, while red/NIR increased ATP in a biphasic manner.

81 starvation, there is a decline in cellular [ATP]; the unusually low affinity of IP6Ks for ATP compel

82 electron transport chain membrane complexes, ATP synthase, and the mitochondrial contact site and cri

83 transmembrane component of the two-component ATP-binding cassette (ABC) transporter TarGH, which expo

84 rference complex, its Cas10 subunit converts ATP into a cyclic oligoadenylate product, which alloster

85 wo of these, Lys-565 and Lys-663, coordinate ATP in the active site.

86 t mediate p31(comet)-Mad2 binding and couple ATP hydrolysis to local unfolding of Mad2.

87 egrity using MitoTracker Green and cytosolic ATP levels.

88 t at the expense of a reduction in cytosolic ATP pools.

89 nt localization, and resulted in a decreased ATP production, similar to what was observed in non-mono

90 h different K(+) channels, astrocyte-derived ATP differentially modulates the excitability of differe

91 oieties of MCC are disassembled by different ATP-requiring processes.

92 Double ATP injections sustained the activation of phospho-STAT3

93 f phenylephrine (PE; alpha1 -agonist) during ATP or control vasodilatator infusion, before and after

94 Elevated ATP was also paralleled by upregulation of enzymes invol

95 Deletion of the PPIP5Ks elevated [ATP] by 35%; both [ATP] and [5-InsP7] were restored to W

96 cytoplasmic organelles that generate energy (ATP) by oxidative phosphorylation and mediate key cellul

97 itial prerequisite binding of MVAPP enhances ATP binding.

98 acetylation status of Lys-72 may affect ERK1 ATP binding.

99 Similar to exercise, ATP-mediated vasodilatation occurs via activation of inw

100 Ring-forming AAA+ chaperones exert ATP-fueled substrate unfolding by threading through a ce

101 receptor (P2Y2R) activated by extracellular ATP and UTP molecules released following injury/stress.

102 described the accumulation of extracellular ATP /AMP during chemotherapy-induced apoptosis in Jurkat

103 DORN1), have demonstrated that extracellular ATP is a signal involved in plant stress responses, incl

104 er, these results suggest that extracellular ATP signaling directly impacts the JA signaling pathway

105 We examined whether extracellular ATP impacts the stability of JAZ1 in Arabidopsis.

106 cry1, 3-bromo-7-nitroindazole, competes for ATP binding and thereby diminishes FADH degrees formatio

107 onsumption rate) is determined by demand for ATP, with feedback by the energy state ([ATP]/[ADP][Pi ]

108 terface that was thought to be essential for ATP synthesis, and cannot convert gamma-subunit rotation

109 , anaerobic respiration and fermentation for ATP production; (3) the reduced ATP generation results i

110 ans-arginine finger, R158, indispensable for ATP hydrolysis; (iii) the location of this arginine is c

111 TP]; the unusually low affinity of IP6Ks for ATP compels 5-InsP7 levels to fall in parallel (Azevedo,

112 ifferential gene expression was profiled for ATP and +P cultures using suppression subtractive hybrid

113 substrate binding primes the transporter for ATP hydrolysis.

114 d as L1 medium and observed DIP release from ATP into the medium, suggesting that K. mikimotoi cells

115 formation of S-adenosylmethionine (SAM) from ATP and methionine.

116 ergy budget of the chloroplast by generating ATP without net production of NADPH.

117 one produced no significant changes in glial ATP levels.

118 fy the responses of oxidative and glycolytic ATP production to alterations in glycolysis and oxidativ

119 embrane-targeting sequence stimulated higher ATP hydrolysis rates than the full-length protein, indic

120 Hsp90 is a homodimeric ATP-dependent molecular chaperone that remodels its subs

121 In this article, we study how ATP-driven activities and actomyosin cytoskeleton impact

122 will be a useful tool for understanding how ATP hydrolysis is coupled to LPS transport.

123 essing extracellular hydrolases to hydrolyse ATP.

124 ese heads only attach to actin and hydrolyze ATP when they are required.

125 r two AAA+ HslU ring hexamers that hydrolyze ATP to power the unfolding of protein substrates and the

126 , transports two calcium ions per hydrolyzed ATP molecule via an "alternating-access" mechanism.

127 by high expression of the immunosuppressive ATP ecto-nucleotidase CD39.

128 ons in the binding of ATP and thereby impair ATP-induced nucleotide-binding domain dimerization and A

129 isolated from myo2-E1-Sup1 are defective in ATP-dependent contraction in vitro.

130 ith a 2.45-, 3.17- and 2.12-fold increase in ATP:ADP, ATP:AMP and energy charge after portal venous r

131 recently, the dual roles of mitochondria in ATP production (bioenergetics) and apoptosis (cell life/

132 ous molecular chaperone that participates in ATP-dependent protein remodeling in both eukaryotes and

133 Mitochondria are known for their role in ATP production and generation of reactive oxygen species

134 f Hsp70 and ClpB/Hsp104 chaperones, which in ATP-dependent reactions disentangle individual proteins

135 educed cellular ATP, while red/NIR increased ATP in a biphasic manner.

136 Metabolomics analysis reveals increased ATP production and improved energy metabolism in injured

137 Further, we observed that 6-ETI induces ATP depletion and cell death accompanied by S phase arre

138 PGE2 did not inhibit ATP-induced mature IL-1beta production in monocytes.

139 1 inhibits ATP synthase activity from isolated mitochondria and tri

140 , oxygen consumption rates and intracellular ATP concentrations decreased and reactive oxygen species

141 AbetaOs transiently decreased intracellular ATP levels and AMPK activity, as evaluated by its phosph

142 yh) CD4 T cells, which reduced intracellular ATP, [ATP]i.

143 NKA incorporated into the LCP retained its ATP-hydrolysing activity for 7 days, while the solubiliz

144 to overcome the loss of K(ATP) channels in K(ATP) knockout mice.

145 st therefore occur to overcome the loss of K(ATP) channels in K(ATP) knockout mice.

146 t mutant M41L/D67N/K70R/S215Y HIV-2 RT lacks ATP-dependent excision activity, and recombinant virus c

147 ated and stimulated with lipopolysaccharide, ATP, or both.

148 ting precursor and binding to mtHsp70 in low ATP conditions.

149 complex cytochrome c oxidase I/IV and lower ATP levels.

150 odel to achieve input compensation: at lower ATP fraction, the individual hexamers make a shorter cyc

151 sis has been generally recognized as a major ATP-dependent function, which efficiently retrieves more

152 We update theoretical maximum ATP yields by mitochondria and cells catabolizing differ

153 ect of retinoschisin on Na/K-ATPase-mediated ATP hydrolysis and ion transport.

154 25A24 encodes a mitochondrial inner membrane ATP-Mg/Pi carrier.

155 rved in type 1 diabetes alters mitochondrial ATP and IFNgamma production; the latter is correlated wi

156 (FANCD2) functionally impacts mitochondrial ATP production through its interaction with ATP5alpha, w

157 NFalpha that in turn stimulate mitochondrial ATP production in POMC neurons, promoting mitochondrial

158 stress responses, whereas the mitochondrial ATP synthase F0 subunit component is a vasoactive peptid

159 y to the time-dependent pore dilation model, ATP binding opens an NMDG(+)-permeable channel within mi

160 ion is associated with low levels of mucosal ATP, highlighting the importance of mitochondrial functi

161 understanding of the proteasome's multistep ATP-dependent mechanism, its biochemical and structural

162 that a single injection of 6 mul of 150 mum ATP into female rat sciatic nerve quadrupled the number

163 1 enzymatic activity with a nM Kd; has a non-ATP competitive mode of action and a novel putative bind

164 tress responses, ultimately restoring normal ATP production, macromolecule biosynthesis, and growth.

165 ion sites on RBOHD eliminates the ability of ATP to induce stomatal closure.

166 ent of each mutant assayed in the absence of ATP were similar to those of wild-type GC-B assayed in t

167 rupt critical interactions in the binding of ATP and thereby impair ATP-induced nucleotide-binding do

168 aperone function are regulated by binding of ATP/ADP to mtHsp70's nucleotide-binding domain.

169 lucose cotransporter SGLT1, or by closure of ATP-sensitive potassium channels after glucose metabolis

170 Comparison of ATP-bound and nucleotide-free states reveals how reversi

171 effect profile as the high concentrations of ATP required to activate the receptor are usually only f

172 ng with high intracellular concentrations of ATP.

173 Both effects of ATP addition are reversed by phosphorylation of the RLC.

174 Treatment with IVM increases the efficacy of ATP to activate P2X4R, slows both receptor desensitizati

175 xin-1 (Panx1) channels mediate the efflux of ATP and AMP from cancer cells in response to induction o

176 tranded DNA ends, RarA couples the energy of ATP binding and hydrolysis to separating the strands of

177 hanism of condensin depends on the energy of ATP hydrolysis but how this activity specifically promot

178 Using the energy of ATP hydrolysis, ABC transporters catalyze the trans-memb

179 lity index quantifies overall flexibility of ATP supply; and the bioenergetic capacity quantifies the

180 a continuous supply of energy in the form of ATP to support persistent contractile function.

181 chondria efficiently allows the formation of ATP by oxidative phosphorylation.

182 , increases in substrate level generation of ATP and reducing equivalents, and recycling of N and pos

183 s surprising conformational heterogeneity of ATP-bound BiP that distinguishes BiP from its bacterial

184 structure associated with the hydrolysis of ATP and release of inorganic phosphate (Pi) from the nuc

185 and remodeling machines couple hydrolysis of ATP to mechanical unfolding and translocation of protein

186 al motifs, and the binding and hydrolysis of ATP.

187 alternate access mechanism and the impact of ATP, MalE, and maltose on the conformation of the transp

188 ependent IFN-beta induction independently of ATP synthesis.

189 Rather, inhibition of ATP synthesis caused massive spontaneous vesicle exocyto

190 Injection of ATP at 150 mum caused little Wallerian degeneration and

191 sue injury, we hypothesize that injection of ATP into a peripheral nerve might mimic the stimulatory

192 The co-opted PK generates high levels of ATP within the viral replication compartment at the expe

193 monocytes released relatively low levels of ATP, whereas cells stimulated with TLR2 agonists release

194 d with TLR2 agonists released high levels of ATP.

195 microscopy) and biophysical measurements of ATP release indicate that G100V/C103V retards initial fu

196 ytic subunits suggests that the mechanism of ATP generation by rotary ATPases is less strictly conser

197 velop a screening strategy for modulators of ATP-PRT and identify 3-(2-thienyl)-L-alanine (TIH) as an

198 sphorylation are the fundamental pathways of ATP generation in eukaryotes.

199 ataxin3 and p97 associate in the presence of ATP and in the absence of nucleotide, but not in the pre

200 M2B, and NM2C polymerized in the presence of ATP are much too small to explain their substantial diff

201 tion, production of lactate, and presence of ATP.

202 of wild-type GC-B assayed in the presence of ATP.

203 r, recruitment of PK and local production of ATP within the replication compartment allow the virus r

204 DNA-bound RecA protein increases the rate of ATP hydrolysis catalysed by RecN during the DNA pairing

205 As a result, the rate of ATP synthesis, plotted against [ADP], remains low until

206 al stalk is a key determinant of the rate of ATP turnover.

207 We describe here how rates of ATP generation by each pathway can be calculated from si

208 ange rapidly after reperfusion and ratios of ATP/ADP/AMP after reperfusion are significantly correlat

209 The mitochondrial network is a major site of ATP production through the coupled integration of the el

210 e relative roles for the two main sources of ATP production, glycolysis and oxidative phosphorylation

211 ation in the PTP; thus, the only subunits of ATP synthase that could participate in pore formation ar

212 igh-fat diet (HFD) results in suppression of ATP citrate-lyase levels in tissues such as adipose and

213 linked with TAT-Gap19-induced suppression of ATP signaling and activation of the ERK1/2 signaling pat

214 main, thermodynamically driving synthesis of ATP.

215 asite" that relies entirely on the uptake of ATP from the host cell.

216 s provided the first high-resolution view of ATP activity in Enterococcus hirae V1-ATPase.

217 omatin remodeler family, and CSB is the only ATP-dependent chromatin remodeler essential for transcri

218 substrate conditions (various calcium and/or ATP concentrations) promoting particular conformational

219 nd interactions with purified P-gp and other ATP-binding cassette transporters that transport amphipa

220 e insertion of thiol-oligonucleotides into p-ATP monolayers previously oxidized, and the covalent bin

221 uggest a segregation mechanism by which ParA-ATP dimers equilibrate to HDRs shown to be localized nea

222 ditory neurons can be modulated by paracrine ATP signalling, as shown for the cochlear nucleus bushy

223 The Arabidopsis PENETRATION 3 (PEN3) ATP binding cassette (ABC) transporter plays a role in d

224 ABCD1 and its homolog ABCD2 are peroxisomal ATP-binding cassette (ABC) half-transporters of fatty ac

225 replication machinery an access to plentiful ATP, facilitating robust virus replication.

226 P-glycoprotein (P-gp) is a polyspecific ATP-dependent transporter linked to multidrug resistance

227 Indirubins have been identified as potent ATP-competitive protein kinase inhibitors.

228 ATP synthases produce ATP by rotary catalysis, powered by the electrochemical

229 ohydrolase 1 (NTPDase1) degrades the purines ATP and ADP that are key regulators of inflammation and

230 ased mitochondrial fragmentation and reduced ATP and mtDNA copy number in FECD.

231 otential, uncoupled respiration, and reduced ATP levels.

232 te stability and hence significantly reduced ATP sensitivity and a marked increase of channel activit

233 ns, mitochondrial network structure, reduced ATP production, and flight and motor dysfunction.

234 entation for ATP production; (3) the reduced ATP generation results in substantially decreased synthe

235 mage, coupled with hypoxia, leads to reduced ATP production by aerobic respiration, driving cells to

236 In high-frequency regions, ATP responsiveness diminished before hearing onset.

237 source and sink and amplifying wave regulate ATP levels using hydrolysis or secretion, respectively,

238 p53 expression, whereas 1,5-InsP8 regulates ATP levels.

239 ge loading forces can the motor head release ATP at a fast rate, which significantly reduces the velo

240 ng tone of the cerebral vessels by releasing ATP and COX-1 derivatives.

241 of DNA at sites of protein adducts requires ATP hydrolysis at both sites, as does the stimulation of

242 Sarcolemmal ATP-sensitive potassium channels (KATP channels) in card

243 We have investigated the role of the SWI/SNF ATP-dependent nucleosome-remodeling complex in the repai

244 ome protein B (CSB) belongs to the SWI2/SNF2 ATP-dependent chromatin remodeler family, and CSB is the

245 for ATP, with feedback by the energy state ([ATP]/[ADP][Pi ]) regulating the pathway.

246 To address this issue, we studied ATP requirement in three typical forms of endocytosis at

247 ocreatine, a creatine analog that can supply ATP.

248 th receptor desensitization during sustained ATP application and receptor deactivation after ATP wash

249 p97 is a ubiquitin-targeted ATP-dependent segregase that extracts ubiquitylated clie

250 We find that ATP-dependent activities enhance the nanoscale z fluctua

251 We found that ATP supported a similar growth rate and cell yield as L1

252 In vitro analyses reveal that ATP excitation of the preBotC involves P2Y1 receptor-med

253 FAO inhibition abrogated the ATP rescue, diminished survival of the inner matrix deta

254 ated with mutations in the gene encoding the ATP-dependent chromatin remodeler CHD7.

255 CFTR intraburst gating is distinct from the ATP-dependent mechanism that controls channel opening an

256 single-molecule imaging to determine how the ATP-dependent translocase RecBCD travels along DNA occup

257 d by transient or oscillatory changes in the ATP fraction, yet is insensitive to constant changes in

258 elected patients who harbor mutations in the ATP-binding sites of ABCB4.

259 e the dynamic encoding of information in the ATP-induced calcium responses of fibroblast cells, using

260 this region, structural changes occur in the ATP-induced opening of Hsp70 to allow substrate exchange

261 igration is pharmacologically inhibited, the ATP:ADP ratio decreases.

262 Interestingly, the ATP-induced JAZ1 degradation was attenuated in the JA re

263 logically hyperactive and seems to mimic the ATP-bound state of cry1, but the reason for this phenoty

264 t lack of cooperativity in hydrolysis of the ATP in each protomer.

265 esence of glibenclamide, an inhibitor of the ATP-dependent potassium (KATP)-channels, thus suggesting

266 al structures of the founding members of the ATP-dependent RNA ligase family (T4 RNA ligase 1; Rnl1)

267 Among them, the ATP-dependent chromatin remodeling complexes control the

268 face between SUR1 and Kir6.2 adjacent to the ATP site in the propeller form and is disrupted in the q

269 suggest that the pore is associated with the ATP synthase complex and specifically with the ring of c

270 interact with conserved residues within the ATP-binding site.

271 terminus, adjacent to Lys-71, which binds to ATP, suggesting that acetylation status of Lys-72 may af

272 substrates are metabolized and contribute to ATP synthesis in neurons.

273 P, pyrophosphate, and phosphoenolpyruvate to ATP, orthophosphate, and pyruvate and provides diverse f

274 Thus, substrate is released prior to ATP hydrolysis.

275 the JAZ1 stability decreased in response to ATP addition in a proteasome-dependent manner.

276 apacity quantifies the maximum rate of total ATP production.

277 ellular currencies for phosphoryl transfers (ATP), acyl transfers (acetyl-CoA, carbamoyl-P), methyl t

278 was based on the ability of NKA to transform ATP to ADP and free phosphate, the latter reacting with

279 Finally, 1 mm 2,4,6,-trinitrophenyl ATP inhibited all three mutants by as much as 80% but fa

280 l O2 consumption and adenosine triphosphate (ATP) synthesis rates of osteosarcoma cybrid cells were m

281 t in the presence of adenosine triphosphate (ATP) the human RAD51 (HsRAD51) recombinase can form a nu

282 vent coordination of adenosine triphosphate (ATP) to the catalytic site.

283 ABCB4 (MDR3) is an adenosine triphosphate (ATP)-binding cassette (ABC) transporter expressed at the

284 ht on how superfamily 1 and 2 helicases turn ATP hydrolysis into motion along DNA.

285 ndenses nicotinamide mononucleotide and (tz) ATP to yield N(tz) AD(+) , which can be enzymatically ph

286 sphorylated by NAD(+) kinase and ATP or (tz) ATP to the corresponding N(tz) ADP(+) .

287 energetically sustainable and unsustainable ATP concentrations for various energetic demands.

288 etal ions but not organic cofactors, and use ATP to activate substrates through phosphorylation.

289 Together, our results show how various ATP-driven processes differently affect membrane mechani

290 ion is achieved through expending energy via ATP hydrolysis, suggesting that it is coupled to TFIIH's

291 ns down a large electrochemical gradient via ATP synthase located on the folded inner membrane, known

292 membrane at elevated neuronal activity when ATP consumption within nerve terminals increases drastic

293 nveyor belt' model of translocation in which ATP binding allows a Vps4 subunit to join the growing en

294 While ATP binding was sufficient to partially populate the OF

295 While ATP depletion led to cell death, over-acetylated tubulin

296 ce of mitochondrial function associated with ATP production in the pathophysiology of the disease.

297 ent of the Rad50 catalytic head domains with ATP bound at both sites is important for MRN functions i

298 e temperature-grown cells are incubated with ATP [8].

299 hP2X7R channel on sustained stimulation with ATP(4).

300 polysaccharide, followed by stimulation with ATP, led to an activation of caspase 1 and interleukin-1