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1 ar basis for adenosine-induced second-degree AV block.
2 cy stimulation identified GP sites producing AV block.
3 hythm, except for 1 with undetected complete AV block.
4  congenital and childhood nonimmune isolated AV block.
5 ose with abnormalities that increase risk of AV block.
6 epair, 48 (1.1%) underwent PPM placement for AV block.
7  for those with subsequent PPM placement for AV block.
8 holine-induced first-degree and third-degree AV block.
9 injected fibroblasts without the creation of AV block.
10         Acetylcholine induced dose-dependent AV block.
11 tients with atrial fibrillation and advanced AV block.
12 ntricular response during AF without causing AV block.
13 ime, significant sinus bradycardia preceding AV block.
14 icular (AV) block, and 8 congenital complete AV block.
15 ide the homeodomain, and not associated with AV block.
16  used as a candidate gene in 2 children with AV block.
17 ant have been recognized in association with AV block.
18 tricular tachycardia and a high incidence of AV block.
19 ortion of tetralogy of Fallot and idiopathic AV block.
20 ndent hypoglycemia, myocardial ischemia, and AV block.
21 diomyopathy, one of whom had a pacemaker for AV block.
22  characterized by complete atrioventricular (AV) block.
23 , and one had first-degree atrioventricular (AV) block.
24                                              AV block after adenosine was only observed in AT patient
25 ement for atrioventricular conduction block (AV block) after operative repair of perimembranous ventr
26                     Subjects with congenital AV block, all presenting with bradycardia, showed large
27 g conditions of this case, recapitulated 2:1 AV block and arrhythmia.
28                      Only 3 days of complete AV block and autonomic denervation did not affect the pr
29 different myocardial layers in conditions of AV block and His-bundle pacing.
30  12 mongrel dogs, after creation of complete AV block and implantation of a ventricular inhibited pac
31                                    Operative AV block and PPM placement occurred in 1.1% of patients
32 m) of canine left ventricle in conditions of AV block and right ventricular pacing.
33  reentrant tachycardia, the incidence of 2:1 AV block and the response to atropine and a single ventr
34 I AV block frequently progressed to complete AV block and was associated with seizures, death, and pa
35 ar basis for second-degree atrioventricular [AV] block), and the recovery of excitability in rabbit i
36 2 congenital second-degree atrioventricular (AV) block, and 8 congenital complete AV block.
37 eeks of age rapidly progressed into complete AV block as early as 4 weeks of age.
38  which further progressed into second-degree AV block at 4 weeks of age before the development of car
39 lure, left ventricular systolic dysfunction, AV block, atrial or ventricular arrhythmias, and sudden
40 tellate ganglion in dogs with chronic MI and AV block augments sympathetic nerve sprouting and create
41 e presence of first-degree atrioventricular (AV) block, bifascicular block, left bundle branch block,
42          Another 6 dogs with MI and complete AV block but without NGF infusion served as controls (n=
43 rlier reports do not report the incidence of AV block by VSD type.
44 etus with cardiac failure caused by complete AV block (CAVB) may allow delivery of a full-term, stabl
45 athways may account for sporadic examples of AV block, complicating posteroseptal ablation in patient
46                                     Complete AV block created by segmental atrial isolation was achie
47  and ventricular rhythms in 22 (6%)-although AV block decreased (3 hearts, 1%).
48 , from 1980-2009) included 141 children with AV block diagnosed in utero, at birth, or before 15 year
49 al area and marked effects on AV conduction, AV block did not occur.
50                             The first-degree AV block dose prolonged AV and AH intervals by 26% and 2
51                          At the first-degree AV block dose, AVN effective refractory period increased
52                 The incidence of induced 2:1 AV block during AV node reentrant tachycardia is approxi
53                         In patients with 2:1 AV block during AV node reentrant tachycardia, the absen
54 APD90 and ERP were measured before and after AV block during pacing at 120 beats/min.
55 arify the mechanism of 2:1 atrioventricular (AV) block during AV node reentrant tachycardia induced i
56 conduction disturbances, and intraprocedural AV block emerged as predictors of PPM implantation after
57 interval in separate groups of dogs that had AV block for 1 week or 3 days with and without rapid pac
58                             In dogs that had AV block for 1 week, 1 hour of rapid pacing prolonged V-
59                                      Type II AV block frequently progressed to complete AV block and
60                            Patients with 2:1 AV block had a shorter tachycardia cycle length than did
61                                           In AV-block hearts, CRTopt exclusively depended on interven
62              In left bundle-branch block and AV-block hearts, optimal hemodynamic effect of CRT depen
63  2.30; 95% CI, 1.70-3.11), atrioventricular (AV) block (HR, 1.48; 95% CI, 1.11-1.97), and myocardial
64 ands with cardiac anomalies and first-degree AV block, idiopathic AV block, or tetralogy of Fallot.
65 s were uncommon under basal conditions, with AV block in 14 (4%) and junctional rhythms in 4 (1%).
66 achieved without the induction of pathologic AV block in 50 (81%) of 62 patients.
67 onduction, whereas adenosine infusion caused AV block in TG(WT) mice but not TG(N488I) mice with pre-
68 n humans consistently caused bradycardia and AV block in the zebrafish.
69                          The data imply that AV block in utero is accompanied by hypertrophy, which i
70 n have CHB induce complete atrioventricular (AV) block in the human fetal heart perfused by the Lange
71 dies revealed first-degree atrioventricular (AV) block in the transgenic heart at 1 week of age, whic
72 conduction abnormalities, including complete AV block, in the pups.
73 ow and fast pathway, and a Wenckebach type I AV block; in the third ECG, findings compatible with sim
74                     Complications other than AV block included polymorphic ventricular tachycardia 10
75                                   High-grade AV block is a potential risk of lesions placed in the sy
76 ndle-branch block (n=8) or atrioventricular (AV) block (n=6) through atrial (A), right ventricular (R
77 n the cycle length (CL) at which anterograde AV block occurred between group I (305 +/- 63 ms) and gr
78                      Inadvertent high degree AV block occurred in 10 (16%) of 62 patients, with the A
79                               Persistent 2:1 AV block occurred in 13 of 139 patients with AV node ree
80                  Transient atrioventricular (AV) block occurred during eight cryo-applications (1 CA,
81 ccurred in 10 (16%) of 62 patients, with the AV block occurring at the time of the procedure in 6 pat
82 equire a permanent pacemaker for inadvertent AV block or because of AV nodal ablation after a failed
83    Features favoring AT were the presence of AV block or marked shortening of atrial cycle length bef
84 h block (OR 39; P = 0.002), and first-degree AV block (OR 14; P = 0.001) on the baseline ECG are inde
85 malies and first-degree AV block, idiopathic AV block, or tetralogy of Fallot.
86 ffinity for calcium (P < 0.015), and reduced AV block (P = 0.04).
87         The highly penetrant and progressive AV block phenotype seen in human heterozygous missense m
88 ion (AF) without producing atrioventricular (AV) block remains a clinical challenge.
89 ildhood nonimmune isolated atrioventricular (AV) block remains unknown.
90 cts, including progressive atrioventricular (AV) block requiring pacemaker implantation.
91 nd 28+/-8 bpm (first-degree and third-degree AV block, respectively; P<0.05).
92 n parents of children affected by idiopathic AV block revealed a high prevalence of conduction abnorm
93 3; p < 0.01); for patients with first-degree AV block (RR: 1.52; p < 0.01), left anterior hemiblock (
94 eline; and for patients with intraprocedural AV block (RR: 3.49; p < 0.01).
95 s accompanied by ECG abnormalities including AV block, sinus bradycardia, and ventricular dysfunction
96 p developed either second-degree or complete AV block spontaneously.
97  during AT (at doses sufficient to result in AV block) terminated or transiently suppressed focal AT
98                  One group had uninterrupted AV block; the other group underwent a period of rapid le
99                                     Complete AV block was achieved in each of the 10 dogs with 6.5+/-
100 at 10 microg/min were infused until complete AV block was achieved.
101                                     Advanced AV block was also occasionally demonstrated in the mutan
102         The most significant risk factor for AV block was Down syndrome (odds ratio 3.62, 95% confide
103                                       Type I AV block was most often due to digitalis and was reversi
104                                           No AV block was seen in any cohort at 4 weeks.
105                PR prolongation (first degree AV block) was present at 4 weeks, 7 months, and 17 month
106 rest and during exertion, without pathologic AV block, was achieved long term in 45 (73%) of 62 patie
107  dogs with MI and complete atrioventricular (AV) block, we induced cardiac sympathetic nerve sproutin
108 ction, 14% to 30%), atrial fibrillation, and AV block were studied by pressure-volume analysis.
109 al (V-A) conduction, two suffered high-grade AV block when ablation of the systemic venous portion of
110               Associated phenotypes included AV block, which was the primary manifestation of cardiac
111 onal ablation because it can create complete AV block, while in effect permitting the equivalent of H
112  sinus node dysfunction or atrioventricular (AV) block, with need for pacing, is common after the Fon
113  a feasible energy source to create complete AV block within the beating heart without damaging the o

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