ライフサイエンスコーパス: AVP

1 AVP activates arginine vasopressin type 1A (V(1A))/Galph

2 AVP also increased the fluorescence intensity on the sur

3 AVP and OT signaling predominantly occur within a circui

4 AVP exerted the same effects on diestrous and proestrous

5 AVP from the SCN is found in regions important for the r

6 AVP increased extracellular-regulated kinase 1/2 (ERK1/2

7 AVP increases humans' willingness to cooperate.

8 AVP injected into wild-type mice enhanced and reduced, r

9 AVP is activated by two cofactors, the viral DNA and pVI

10 AVP stimulated GLP-1 and PYY release from primary cultur

11 AVP travels through the bloodstream to the kidney, where

12 AVP, initially synthesized as an inactive enzyme, requir

13 AVP, partially activated by being bound to DNA, excises

14 AVP-p demonstrates activity against viruses with the Old

15 AVP-p is unique among self-derived inhibitory peptides i

16 AVP-p may represent a potent, highly specific, novel the

17 AVP/cyclic adenosine monophosphate enhance the phosphory

18 Inhibition of the V(2) AVP receptor represses ENaC activity in Adx mice.

19 By e16.5, AVP cell bodies are formed in the SON and PVN, but are a

20 e potential therapeutic value of a new V1 a -AVP receptor partial agonist with a preferential splanch

21 tested the hypothesis that CREB3L1 activates AVP gene transcription.

22 lfide bond with AVP forming the fully active AVP-pVIc complex bound to DNA.

23 The persistent high urine volume after AVP administration was traced to a reduction in aquapori

24 l connectivity with the ventral pallidum, an AVP receptor-rich region previously associated with AVP-

25 icant positive association between Ang-2 and AVP index, MMP-2, Ang-1, and VEGF in SRA.

26 Fluoxetine exposure also increased 5HT and AVP afferent development to brain areas implicated in ag

27 g immunohistochemistry, examined for 5HT and AVP afferent innervation/development to areas of the bra

28 s aggressive behavior and alters LAH 5HT and AVP development, yet only alterations in AVP afferent de

29 B-Raf and A-kinase anchoring protein 79, and AVP increased this interaction in ADPKD but not NHK cell

30 rences in AVP-ir fiber fractional areas, and AVP-ir cell body numbers, which were mainly observed in

31 n the structural differences between AVP and AVP-pVIc, we present a model that postulates that activa

32 ical modeling to investigate whether CRH and AVP promote distinct patterns of electrical excitability

33 cal modelling to investigate whether CRH and AVP promote distinct patterns of electrical excitability

34 Co-stimulation with CRH and AVP results in complex patterns of excitability includin

35 Co-stimulation with CRH and AVP results in complex patterns of excitability includin

36 rease in excitability in response to CRH and AVP the patterns of electrical excitability and underlyi

37 rease in excitability in response to CRH and AVP the patterns of electrical excitability and underlyi

38 he use of 5-HT-targeted drugs in females and AVP-targeted drugs in males.

39 e via AVP receptor (AVPR)2 in the kidney and AVP receptor 1A in vascular smooth muscle.

40 studying the role and relatedness of OT and AVP in the developing brain.

41 Reviewing the research about OT and AVP in these NDD suggests that altered OT pathways may b

42 1 (Fmr1) gene, resulting in decreased OT and AVP staining cells in some brain regions.

43 We review how the OT and AVP systems have been investigated in Autism Spectrum Di

44 appropriately phased to support rhythms, and AVP receptor signaling is required to impose circuit-lev

45 entry in cells co-transfected with TRPC6 and AVP V1 receptor.

46 Finally, constant light increased VIP and AVP expression, but not VPAC2R.

47 In Ussing chambers, basolaterally applied AVP reduced colonic anion secretion and this effect was

48 homeostasis and behavior is problematic, as AVP is made in multiple nuclei in the hypothalamus (i.e.

49 dependent on circulating ovarian steroids as AVP no longer excited preoptic kisspeptin neurons in ova

50 Once particles are assembled, AVP-mediated cleavage facilitates the release of the mem

51 Their in vitro pharmacological profile at AVP V(2)R, V(1a)R, V(1b)R, and oxytocin receptor was mea

52 s analogue d(CH2)5[D-Tyr(Et2)-Ile4-Eda9]AVP (AVP(an)) have been synthesized using the technetium comp

53 Elevated baseline AVP level was independently predictive of mortality, but

54 There was no interaction of baseline AVP with treatment assignment in terms of survival (P=0.

55 Based on the structural differences between AVP and AVP-pVIc, we present a model that postulates tha

56 [Cl(-)]i is differentially regulated between AVP+ and VIP+ neurons-a low concentration of the loop di

57 versed by bumetanide, and furosemide blocked AVP release, both in vivo and in hypothalamic explants.

58 during the day than during the night in both AVP+ and VIP+ neurons.

59 Aldosterone-independent stimulation by AVP shifts the role of ENaC in the ASDN from protecting

60 that is cleaved at both N- and C-termini by AVP.

61 ive to modulation of systemic and/or central AVP release through PVN inputs to the posterior pituitar

62 t, when subjects make the risky Stag choice, AVP down-regulates the BOLD signal in the left dorsolate

63 tedly, estrogen is found to permit circadian AVP signaling at preoptic kisspeptin neurons rather than

64 the brain and exhibits a prominent circadian AVP rhythm.

65 The 99mTc(NS3)(CN-AVP) and 99mTc(NS3)(CN-AVP(an)) ability of binding to small-cell lung cancer (S

66 he novel vasopressin conjugate 99mTc(NS3)(CN-AVP(an)) is a desirable compound for imaging oncogene re

67 The 99mTc(NS3)(CN-AVP) and 99mTc(NS3)(CN-AVP(an)) ability of binding to sm

68 Consequently, AVP failed to induce the sustained intracellular Ca(2+)

69 In contrast, AVP promotes an increase in single spike frequency, a me

70 In contrast, AVP promotes an increase in single spike frequency, a me

71 (cortisol:ACTH area under curve) during CRF/AVP challenge at 1.5 years and increased adrenomedullary

72 orticotroph excitability and exaggerated CRH/AVP-stimulated ACTH secretion in vitro.

73 on, the stimulatory effect of 1-desamino-8-D-AVP and the defect in AC6(-/-) mice seem to be restricte

74 on of TRPC6 or ROS production also decreased AVP-stimulated membrane currents.

75 ion of adult male rats resulted in decreased AVP mRNA expression and increased methylation of specifi

76 umerous regions in which males have a denser AVP-immunoreactive innervation than females.

77 We describe AVP-immunoreactive (-ir) fibers in midbrain, hindbrain,

78 of its analogue d(CH2)5[D-Tyr(Et2)-Ile4-Eda9]AVP (AVP(an)) have been synthesized using the technetium

79 periments, male participants received either AVP or placebo intranasally and made decisions with fina

80 adjusting for baseline covariates, elevated AVP levels were associated with increased all-cause mort

81 sed the proportion of patients with elevated AVP (P<0.001), but this had no effect on mortality (haza

82 bsence of aldosterone combined with elevated AVP release compromises normal feedback regulation of EN

83 ion of PDE4, and inhibition of PDE1 enhanced AVP-induced ERK activation.

84 nfirm that Caprin-2 over-expression enhances AVP mRNA abundance and poly(A) tail length.

85 smolality and copeptin (surrogate marker for AVP).

86 molality or copeptin (a surrogate marker for AVP).

87 ow a previously unidentified causal role for AVP in social approach behavior in humans, as establishe

88 rnal capsid cement protein and substrate for AVP, is cleaved at two sites, one of which is near the N

89 on of copeptin, an established surrogate for AVP, with parameters of renal function and morphology in

90 However, AVP also causes dephosphorylation of AQP2 at S261.

91 However, AVP-mediated protection against H/R was elicited only vi

92 Remarkably, however, AVP signaling was critically dependent on circulating ov

93 he in females and activation of hypothalamic AVP neurons in males.

94 euroanatomical experiments, we determined if AVP neurons project from the PVN to the RVLM and if argi

95 Importantly, AVP and OT signaling within the SBNN has been shown to d

96 In AVP, however, His-54 Ndelta1 is 7.0 A away from Cys-122

97 In AVP, however, Tyr-84 is more than 11 A away from its pos

98 In AVP-pVIc, the general base His-54 Ndelta1 is 3.9 A away

99 In AVP-pVIc, Tyr-84 forms a cation-pi interaction with His-

100 and AVP development, yet only alterations in AVP afferent development within the LAH correlate with t

101 , for the first time, the role of L-cells in AVP regulated intestinal fluid secretion, potentially li

102 in concentrations correlated with changes in AVP concentrations in controls and patients.

103 lasmalemmal and cytoplamic NK3R densities in AVP-labeled but not in OC-labeled dendrites.

104 nificantly increased nuclear NK3R density in AVP-labeled somata but not in OC-labeled somata.

105 is due in part to age and sex differences in AVP and OT synthesis within the SBNN.

106 nd female mice to examine sex differences in AVP innervation.

107 ions) and sex (10 subregions) differences in AVP-ir fiber fractional areas, and AVP-ir cell body numb

108 However, their subcellular distribution in AVP or OC neurons of the PVN and plasticity following re

109 es nuclear NK3R translocation exclusively in AVP neurons.

110 ditions, NK3Rs were predominantly located in AVP neurons, however sparsely distributed in OC neurons

111 superoxide generating NADPH oxidase (NOX) in AVP-expressing hypothalamic paraventricular nucleus (PVN

112 is more than 11 A away from its position in AVP-pVIc.

113 tigated intracellular chloride regulation in AVP and VIP-expressing SCN neurons and found evidence su

114 relationship between baseline and trends in AVP with outcomes in patients hospitalized for worsening

115 PVN neurons, whereas robust upregulation in AVP neurons accompanied DH and SL rats.

116 ologic states characterized by inappropriate AVP secretion and positive water balance.

117 into rat SONs and PVNs resulted in increased AVP biosynthesis.

118 Tolvaptan treatment increased AVP levels during follow-up, but this incremental increa

119 phase of sepsis is associated with increased AVP levels and suppressed thirst.

120 on of NKCC2 with lipid rafts facilitates its AVP-induced apical translocation and activation at the s

121 concentration in Brattleboro rats that lack AVP.

122 Our results suggest that the BNST/MeA AVP system innervates regions containing major modulator

123 As observed in cryo-electron micrographs, AVP-p treatment causes morphological changes consistent

124 um-induced NDI by potentiating the action of AVP on the CD.

125 t a model that postulates that activation of AVP by pVIc occurs via a 62-amino acid-long activation p

126 There is a conundrum in the activation of AVP in that AVP and pVI are sequence-independent DNA-bin

127 that conundrum by showing that activation of AVP takes place on the one-dimensional contour of DNA.

128 was prevented by pharmacological blockade of AVP receptors.

129 However, a thorough characterization of AVP and OT-immunoreactive (ir) fibers and cell bodies ac

130 on, there is no comprehensive description of AVP distribution in the mouse brain and spinal cord.

131 We also provide a detailed description of AVP-ir innervation in heterogenous regions such as the a

132 ularly robust and widespread distribution of AVP-ir fibers, which, as in other species, originates fr

133 ntrating ability by augmenting the effect of AVP on the kidney and ameliorates lithium-induced NDI by

134 hydration-evoked GABA-mediated excitation of AVP neurons was reversed by bumetanide, and furosemide b

135 adal steroid hormone-dependent expression of AVP in the BNST and MeA and electrolytic lesions to elim

136 In LXRbeta(-/-) mice the expression of AVP was markedly decreased in the magnocellular neurons

137 ell established as an important co-factor of AVP, the role of the N-terminal fragment, pVIn, is curre

138 To understand the function of AVP, it is essential to know the site of origin of vario

139 ed aggression in males, whereas injection of AVP inhibited aggression in females and stimulated aggre

140 An i.p. injection of AVP to LXRbeta(-/-) mice revealed a partial kidney respo

141 istochemistry to corroborate the location of AVP-containing cells and to define the location of AVP-c

142 ntaining cells and to define the location of AVP-containing fibers throughout the mouse central nervo

143 In conclusion, a partial or complete loss of AVP osmoregulation occurs in patients with SIAD.

144 ptin concentration as a surrogate measure of AVP concentration, patients with SIAD could be grouped a

145 t Hes1 may be necessary for the migration of AVP neurons.

146 es1 null mice show continued misplacement of AVP cell bodies in the PVN and SON and additionally exhi

147 Our data also highlight the robust nature of AVP innervation in specific regulatory nuclei, such as t

148 caused by a defect in central production of AVP.

149 the generation, placement and projection of AVP neurons.

150 We thus identify CREB3L1 as a regulator of AVP transcription in the rat hypothalamus.

151 Sliding of AVP-pVIc complexes along DNA illustrates a new biochemic

152 Comparison of the structure of AVP with that of an active form, the AVP-pVIc complex, r

153 P-1) and peptide YY (PYY) may be a target of AVP and contribute to the control of fluid balance.

154 retic bumetanide had differential effects on AVP+ and VIP+ neurons, while blocking the KCCs with VU02

155 males in the near plasmalemmal p47(phox) on AVP dendrites seen in the present study.

156 males in the near plasmalemmal p47(phox) on AVP dendrites seen in the present study.

157 no significant differences in overall CRF or AVP expression at any rostrocaudal level of the PVN.

158 , unlike the static pattern observed for OT, AVP innervation of the forebrain SBNN appears to undergo

159 alian PVN (CCK, CRH, ENK, NTS, SS, VIP, OXT, AVP), we provide the first 3D arrangement map of NPO neu

160 The anterior visual pathway (AVP) conducts visual information from the medulla of the

161 it that we call the anterior visual pathway (AVP).

162 treatment failure, we identified a peptide, AVP-p, derived from the fusion glycoprotein of a nonpath

163 We have identified a GP2-derived peptide, AVP-p, with antiviral activity and no acute cytotoxicity

164 rk in concert with the well-known peripheral AVP actions of controlling homeostasis and stress respon

165 Airway vascular permeability (AVP) index was also assessed.

166 lity, urine sodium concentration, and plasma AVP levels.

167 Plasma [AVP] is significantly elevated in Adx vs. control mice.

168 al intracellular Ca(2+) levels and prevented AVP-induced translocation of aquaporin 2, further sugges

169 The AVP-V1A receptor is the primary AVP receptor in the heart; however, its role in cardiac

170 is mediated by the virally encoded protease AVP.

171 y the virally encoded adenovirus proteinase (AVP) before the virus particle becomes infectious.

172 ly, activation of the adenovirus proteinase (AVP) during maturation and endosome escape following cel

173 The adenovirus proteinase (AVP), the first member of a new class of cysteine protei

174 adenovirus infection, the viral proteinase (AVP) becomes activated to process virion precursor prote

175 d a decrease in cytoplasmic p47(phox) in PVN AVP dendrites.

176 the present study, densities of NK3Rs in PVN AVP- or OC-labeled somatodendritic profiles were measure

177 munoreactive projections; we also quantified AVP-immunoreactive fiber density in gonadectomized and s

178 We therefore quantified AVP- and OT-ir fibers and cell bodies in 22 subregions o

179 Rat AVP promoter deletion constructs revealed that CRE-like

180 3L1 (CREB3L1CA) induce the expression of rat AVP promoter-luciferase reporter constructs, whereas a d

181 for the arginine-vasopressin V(2) receptor (AVP V(2)R) was synthesized using "Click" chemistry.

182 An antagonist of AVPR1B reduced AVP-triggered hormone secretion from murine and human ce

183 l to the B-Raf/MEK/ERK pathway and regulates AVP-induced proliferation of ADPKD cells.

184 educes and testosterone replacement restores AVP expression within the BST.

185 In summary, AVP released by a subset of PVN neurons modulates cardio

186 diphenyleneiodonium significantly suppressed AVP- and angiotensin II-induced whole cell currents and

187 eceptor binding and membrane fusion and that AVP-p may represent a viable therapeutic strategy for ar

188 methylation, we explored the hypothesis that AVP expression in the adult brain is maintained through

189 a conundrum in the activation of AVP in that AVP and pVI are sequence-independent DNA-binding protein

190 s and cryo-electron microscopy indicate that AVP-p induces premature activation of viral fusion prote

191 In conclusion, we propose that AVP activates L-cell AVPR1B, causing GLP-1 and PYY secre

192 a(2+)]i) in multiple cells and revealed that AVP increased [Ca(2+)]i in >80% of diestrous kisspeptin

193 Together, these studies show that AVP exerts a potent and direct stimulatory influence upo

194 By means of Ussing chambers, we show that AVP reduced colonic anion secretion, although this was b

195 slices from kisspeptin-GFP mice showed that AVP dose-dependently increased the firing rate of most k

196 These results suggest that AVP has a pleiotropic role in renal function and may fav

197 Recent evidence suggests that AVP may have additional effects on renal function and fa

198 The AVP bears a fundamental resemblance to the sky-compass p

199 The AVP-V1A receptor is the primary AVP receptor in the hear

200 This is mediated by the AVP receptor 1B, which is highly enriched in colonic L-c

201 ture of AVP with that of an active form, the AVP-pVIc complex, reveals why AVP is inactive.

202 ing of transmembrane proteins, including the AVP-regulated water channel, aquaporin 2.

203 ticle, we have analyzed the formation of the AVP from early larval to adult stages.

204 gh sustained epigenetic modifications of the AVP gene promoter.

205 ship of primary and secondary neurons of the AVP lineages.

206 d by extension of the 3' poly(A) tail of the AVP mRNA, and the up-regulation of the expression of RNA

207 The immature fiber tracts of the AVP, formed by secondary neurons of lineages DALcl1/2 an

208 promotes AQP2 trafficking independent of the AVP-PKA axis.

209 An activated adenovirus proteinase, the AVP-pVIc complex, was shown to slide along viral DNA wit

210 tically stimulated hypothalamus shortens the AVP mRNA poly(A) tail at the same time as reducing trans

211 und to DNA, excises pVIc, which binds to the AVP molecule that cut it out.

212 Direct binding of CREB3L1 to the AVP promoter was shown by chromatin immunoprecipitation

213 methylation of specific CpG sites within the AVP promoter in the BST.

214 inate the SCN, effectively eliminating those AVP-immunoreactive projections; we also quantified AVP-i

215 1) = 1.45 x 10(6) bp(2)/s, until it binds to AVP also on the same DNA molecule.

216 Here we show that Caprin-2 binds to AVP mRNAs, and that lentiviral mediated shRNA knockdown

217 suggesting impaired sensitivity of the CD to AVP in SHR-A3.

218 a larger effect on VIP+ neurons compared to AVP+ neurons.

219 In contrast to AVP, we observed no age or sex differences in OT-ir fibe

220 n undermining coparenting and was related to AVP.

221 d PDEs, also induced a mitogenic response to AVP in NHK cells, similar to the effect of restricting i

222 mote its apical translocation in response to AVP stimulation and low chloride hypotonic stress.

223 ducing ERK1/2 phosphorylation in response to AVP stimulation.

224 with high levels of pS256, despite unchanged AVP and cAMP.

225 n dissecting molecular mechanisms underlying AVP-mediated water reabsorption, evidenced by our identi

226 To better understand AVP-mediated signaling in the heart, we created a transg

227 ent analysis examined baseline and follow-up AVP levels in 3196 EVEREST patients with valid AVP measu

228 nted the lithium-induced increase in urinary AVP excretion and suppressed the lithium-induced increas

229 V1a -AVP receptor expression in rats with cirrhosis and ascit

230 V1a -AVP receptor partial agonism could be a useful pharmacol

231 The effect of the V1a -AVP receptor partial agonist on PP was associated with a

232 P levels in 3196 EVEREST patients with valid AVP measurements.

233 Vasopressin (AVP) is both a neuroendocrine hormone located in magnoce

234 Vasopressin (AVP) signaling via PKA and other kinases activates NKCC2

235 Vasopressin (AVP) stimulates ENaC.

236 ment of the serotonin (5HT) and vasopressin (AVP) neural systems modulating this behavior using puber

237 hool, assayed oxytocin (OT) and vasopressin (AVP), and measured coparenting and child behavior proble

238 a greater increase in basal and vasopressin (AVP)-stimulated cAMP levels and Cl(-) secretion by ADPKD

239 [Arg8]-vasopressin (AVP) activates 3 G-protein-coupled receptors: V1A, V2, a

240 Defining how arginine vasopressin (AVP) acts centrally to regulate homeostasis and behavior

241 Arginine vasopressin (AVP) affects kidney function via vasopressin V2 receptor

242 with the neuropeptide arginine vasopressin (AVP) and clock proteins (PER2 and BMAL1), supporting tha

243 by contributions from arginine vasopressin (AVP) and gastrin-releasing peptide (GRP).

244 urohypophyseal hormone arginine vasopressin (AVP) and in the expression of oxidative stress.

245 eus (PVN) that release arginine vasopressin (AVP) and specifically, that increased SNA evoked by CIH

246 Circulating levels of arginine vasopressin (AVP) are elevated during hypovolemia and during cardiac

247 Oxytocin (OT) and arginine vasopressin (AVP) are two small, related neuropeptide hormones found

248 nt on the neuropeptide arginine vasopressin (AVP) because it was prevented by pharmacological blockad

249 sing hormone (CRH) and arginine vasopressin (AVP) cause a depolarization of corticotrophs, leading to

250 its mammalian homolog arginine vasopressin (AVP) demonstrate several relationships between these neu

251 In A7r5 cells, arginine vasopressin (AVP) evoked a striking Ca(2+) entry response that was si

252 Arginine vasopressin (AVP) has a key role in osmoregulation by facilitating wa

253 e antidiuretic hormone arginine vasopressin (AVP) increases in the hypothalamus, and this is accompan

254 Arginine vasopressin (AVP) is a neurohypophysial hormone regulating hydrominer

255 Arginine vasopressin (AVP) levels are elevated in proportion to heart failure

256 y elevated circulating arginine vasopressin (AVP) levels in SHR-A3 compared with SHR-B2.

257 hich contains axons of arginine vasopressin (AVP) neurons from the hypothalamic paraventricular nucle

258 ncluding VIP, GRP, and arginine vasopressin (AVP) neurons, with each ipRGC innervating specific subdo

259 m numerous stimuli for arginine vasopressin (AVP) production, such as volume depletion, pain, stress,

260 Synthetic arginine vasopressin (AVP) receptor agonists able to induce systemic and mesen

261 Arginine vasopressin (AVP) regulates fluid balance and blood pressure via AVP

262 se in water intake and arginine vasopressin (AVP) secretion to promote fluid expansion and maintain b

263 Arginine vasopressin (AVP) stimulates the release of enteroendocrine L-cell de

264 renal collecting duct, arginine vasopressin (AVP) stimulates the synthesis of cAMP, leading to signal

265 sing hormone (CRH) and arginine vasopressin (AVP) to control the release of adrenocorticotrophic horm

266 sing hormone (CRH) and arginine vasopressin (AVP) to control the release of adrenocorticotrophin horm

267 e antidiuretic hormone arginine vasopressin (AVP) underlies diabetes insipidus, which is characterize

268 inct binding sites for arginine vasopressin (AVP) within its V2 -receptor (V2 R).

269 Here we show that arginine vasopressin (AVP), a neuropeptide that mediates complex mammalian soc

270 d serum sodium, plasma arginine vasopressin (AVP), and plasma copeptin concentrations from 50 patient

271 ption is controlled by arginine vasopressin (AVP), which binds to V2 receptors, resulting in protein

272 bstantially attenuated arginine vasopressin (AVP)-induced Ca(2+) entry in cells co-transfected with T

273 a central modulator of arginine vasopressin (AVP)-induced water transport in the renal collecting duc

274 eness of the kidney to arginine vasopressin (AVP).

275 neuropeptide hormone, arginine vasopressin (AVP).

276 t serotonin (5-HT) and arginine-vasopressin (AVP) act in opposite ways in the hypothalamus to regulat

277 e elevated circulating arginine-vasopressin (AVP) levels, we examined whether AVP can affect the skel

278 us output neuropeptide arginine-vasopressin (AVP) on the activity of preoptic area kisspeptin neurons

279 eleasing factor (CRF), arginine-vasopressin (AVP), histidine decarboxylase (HDC), melanin-concentrati

280 The neuropeptide vasopressin (AVP) has been implicated in the regulation of numerous p

281 The neuropeptides vasopressin (AVP) and oxytocin (OT) have been implicated in the regul

282 eurons expressing either VIP or vasopressin (AVP).

283 f the socially relevant peptide vasopressin (AVP) within the bed nucleus of the stria terminalis (BST

284 99mTc-labeled conjugates of the vasopressin (AVP) peptide and of its analogue d(CH2)5[D-Tyr(Et2)-Ile4

285 lay stress information include vasopressin- (AVP) and oxytocin- (OC) containing neurons of the parave

286 gulates fluid balance and blood pressure via AVP receptor (AVPR)2 in the kidney and AVP receptor 1A i

287 vo environment, heat-disrupted ts-1 virions, AVP-pVIc complexes processed five different precursor pr

288 In vivo, in heat-disrupted immature virus, AVP was also activated by pVI in DNA-dependent reactions

289 In vitro, AVP-pVIc complexes processed a purified virion precursor

290 Whether AVP also affects kidney structure in the general populat

291 asopressin (AVP) levels, we examined whether AVP can affect the skeleton directly as yet another comp

292 We next examined whether AVP signaling in kisspeptin neurons was time and ovarian

293 tive form, the AVP-pVIc complex, reveals why AVP is inactive.

294 eptor-rich region previously associated with AVP-mediated social reward processing in mammals.

295 pVIc then forms a disulfide bond with AVP forming the fully active AVP-pVIc complex bound to D

296 Pretreatment of H9c2 cells with AVP significantly reduced H/R-induced cell death and cas

297 show that CREB3L1 mRNA levels correlate with AVP transcription level in SONs and PVNs following sodiu

298 ly, increases in expression in parallel with AVP expression in supraoptic nuclei (SONs) and paraventi

299 ivity in H9c2 myoblasts after treatment with AVP.

300 ency, either globally or specifically within AVP-expressing neurons, developed central diabetes insip