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1 his may provide new treatment modalities for Acanthamoeba keratitis.
2 reen new anti-infectives in the treatment of Acanthamoeba keratitis.
3 ould test the validity of this treatment for Acanthamoeba keratitis.
4 corneal scrapings from bacterial, viral, or Acanthamoeba keratitis.
5 a) inhibitors may be a therapeutic target in Acanthamoeba keratitis.
6 4 patients underwent diagnostic testing for Acanthamoeba keratitis.
7 al-time PCR assays could be used to diagnose Acanthamoeba keratitis.
8 P-133 produce an additive protection against Acanthamoeba keratitis.
9 ubsequent steps of the pathogenic cascade of Acanthamoeba keratitis.
10 hat mediate latex bead-induced resistance to Acanthamoeba keratitis.
11 astellanii antigens fails to protect against Acanthamoeba keratitis.
12 innate immune apparatus in the resistance to Acanthamoeba keratitis.
13 ils in latex-bead-induced protection against Acanthamoeba keratitis.
14 ge amounts of mannose and is associated with Acanthamoeba keratitis.
15 a, such as herpes simplex virus keratitis or Acanthamoeba keratitis.
16 o chlorhexidine, a drug widely used to treat Acanthamoeba keratitis.
17 ubsequent steps of the pathogenic cascade of Acanthamoeba keratitis.
18 nt an important immunotherapeutic target for Acanthamoeba keratitis.
19 high numbers in the corneas of patients with Acanthamoeba keratitis.
20 topical steroid is used in the management of Acanthamoeba keratitis.
21 e and A. griffini is reported from a case of Acanthamoeba keratitis.
22 08.9 +/- 812.5 cells/mm(2); P < 0.0001), and Acanthamoeba keratitis (1000.2 +/- 1090.3 cells/mm(2); P
31 l microscopy diagnostic criteria to diagnose Acanthamoeba keratitis (AK) using polymerase chain react
32 lls, a key first step in the pathogenesis of Acanthamoeba keratitis (AK), a devastating corneal infec
33 as the causative agent of sight- threatening Acanthamoeba keratitis (AK), serious infections of other
42 (aPA) play key roles in the pathogenesis of Acanthamoeba keratitis by inducing a cytopathic effect o
44 n findings can be useful for differentiating acanthamoeba keratitis from bacterial and fungal keratit
45 immune apparatus are important in preventing Acanthamoeba keratitis from progressing to become an int
47 at the initial phases in the pathogenesis of Acanthamoeba keratitis involve parasite binding and lysi
57 ial keratitis, epithelial regeneration line, Acanthamoeba keratitis, mucus plaque keratopathy, medica
59 , specimens isolated from the faucets at two Acanthamoeba keratitis patients' homes differed from the
61 tis, and 115 patients with laboratory-proven acanthamoeba keratitis seen at Aravind Eye Hospital, Mad
63 he potential of this Rns region for tracking Acanthamoeba keratitis strains in infections and for dis
64 ifferentiating features were more common for acanthamoeba keratitis than for bacterial or fungal kera
65 tex beads induces a remarkable resistance to Acanthamoeba keratitis that is largely, if not entirely,
66 oscopy could be useful in cases of fungal or acanthamoeba keratitis to determine the depth of infecti
70 bacterial or fungal keratitis, patients with acanthamoeba keratitis were more likely to be younger an
71 ral immunization with Ac-CT protects against Acanthamoeba keratitis when administered before corneal
72 s adverse events, particularly bacterial and Acanthamoeba keratitis while using these lenses, althoug
73 with fungal keratitis, and 93 patients with acanthamoeba keratitis who had medical records available
74 dies reporting resolution or modification of Acanthamoeba keratitis without specific antiacanthamoeba
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